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1 able in risk stratification of patients with arterial occlusive disease.
2 scularization for lower extremity peripheral arterial occlusive disease.
3 9 years; range, 54-80 years) with peripheral arterial occlusive disease.
4 tic arteries of patients suspected of having arterial occlusive disease.
5 and economical assessment of lower extremity arterial occlusive disease.
6 d on the order of testing or the severity of arterial occlusive disease.
7 ively assess the severity of lower extremity arterial occlusive disease.
8 for the treatment of atherosclerotic carotid arterial occlusive disease.
9 tion of diagnosis of chronic lower-extremity arterial occlusive disease.
10 mechanism that protects against ischemia in arterial occlusive disease.
11 5% CI, 1.24-3.33]; P = .002), and peripheral arterial occlusive disease (adjusted HR, 2.15 [95% CI, 1
13 alities may contribute to the development of arterial occlusive disease and associated clinical event
14 patients treated with dasatinib, peripheral arterial occlusive disease and other arterial disorders
15 s in patients suspected of having peripheral arterial occlusive disease, and diagnostic performance w
17 tion procedure (aortoiliac and infrainguinal arterial occlusive disease) at VQI-participating centers
18 ity due to worsening pre-existing peripheral arterial occlusive disease in a patient who had received
20 induced fatty liver, atrial thrombus, severe arterial occlusive disease of lower extremities, pleuro-
21 ng abdominal aortic aneurysm (33 percent) or arterial occlusive disease of the legs (67 percent).
23 Intermittent claudication due to peripheral arterial occlusive disease (PAOD) is a common cause of p
25 ls in 60 randomized patients with peripheral arterial occlusive disease referred for 64-section multi
28 mocysteine, which are frequently elevated in arterial occlusive disease, we hypothesized that folic a