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1 acute lung injury have been shown to improve arterial oxygen tension.
2  of inspired oxygen (Fio(2)) and have a high arterial oxygen tension.
3 ged as a potential strategy to rapidly raise arterial oxygen tensions.
4 -expiratory lung volume, but did not improve arterial oxygen tension and caused a reduction in cardia
5 onsistent with the hypothesis that increased arterial oxygen tension and consequently increased CSF P
6 changes occurred in the setting of preserved arterial oxygen tension and myocardial perfusion.
7   Chemoreceptors in the carotid bodies sense arterial oxygen tension and regulate respiration.
8 An inverse relationship was observed between arterial oxygen tension and the magnitude of hyperleukoc
9 iratory volume in 1 second (FEV1), of 20% in arterial oxygen tension, and of 20% in the 6-minute walk
10                        Reduction of maternal arterial oxygen tension by 50% over 30 min resulted in a
11 0.7), PaO2 (35+/-3.1 mm Hg), and alveolar to arterial oxygen tension difference (AaDO2) (26+/-3 mm Hg
12 ped respiratory distress; PaO2, the alveolar-arterial oxygen tension difference (AaPO2) and venous ad
13 f carbon monoxide (DL(CO)), and the alveolar-arterial oxygen tension difference P(A-a)O(2) were measu
14           Most patients maintain an adequate arterial oxygen tension during single-lung ventilation.
15 ignificantly, slowing blood flow velocity at arterial oxygen tension even without additional contribu
16 expected to broaden the permissible range of arterial oxygen tensions for pulmonary/tissue oxygen tra
17 ding on outcomes: need for IMV, evolution of arterial oxygen tension/fraction of inspired oxygen rati
18  associated with improvement in oxygenation (arterial oxygen tension/fraction of inspired oxygen rati
19 eased inspiratory time, and widened alveolar-arterial oxygen tension gradient (all p < or = 0.001); t
20          The postinjury increase in alveolar-arterial oxygen tension gradient (LH, 36.7 +/- 3.5 vs. S
21 d neutrophils, PaO2/FIO2 ratio, and alveolar-arterial oxygen tension gradient in acid-induced lung in
22                  Standard criteria (alveolar-arterial oxygen tension gradient of > 600 torr [> 80 kPa
23  ratio, followed by shunt fraction, alveolar-arterial oxygen tension gradient, FIO2, PaO2, respirator
24        Despite no difference in end-exercise arterial oxygen tension in hypoxia (59 +/- 6 vs. 59 +/-
25 ur after the instillation of perflubron, the arterial oxygen tension increased by 138 percent and the
26 007) and inversely correlated with wake-time arterial oxygen tension (P = 0.0007) and oxygen saturati
27                               A preoperative arterial oxygen tension (PaO(2)) of </= 50 mm Hg alone o
28  essentially proportional to the decrease in arterial oxygen tension (PaO2) below the normoxic level.
29 delines instructing for explicit targets for arterial oxygen tension (PaO2, 55-86 mm Hg) and oxyhemog
30                      In this model, coronary arterial oxygen tension was influenced by the length of
31 canning, and a marked improvement in resting arterial oxygen tension while breathing air to 10.3 kPa