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1 ng assisted ventilation presented without an arterial pulse.
4 to characterize the relationships among the arterial pulse (AP) and the cardiac-related and 10-Hz rh
5 (SND) relative to the systolic phase of the arterial pulse (AP) and thus pulse-synchronous barorecep
6 e levels above which coherence of SND to the arterial pulse at the frequency of the heart beat became
9 ctive report of the throbbing rhythm and the arterial pulse in human subjects of either sex with thro
10 Textile MEG was demonstrated to convert the arterial pulse into electrical signals with a low detect
11 he magnitude and statistical significance of arterial pulse-modulated activity of single neurones and
12 ed aortic flow monitoring devices, one using arterial pulse power (LiDCOplus) and the other esophagea
13 the normotensive participants, elevation of arterial pulse pressure (a surrogate of arterial stiffne
14 dy was to examine the progression of central arterial pulse pressure (cPP) in women and the degree to
17 ation (PPV), which quantifies the changes in arterial pulse pressure during mechanical ventilation, i
18 cular outflow through the bypass widened the arterial pulse pressure from 41 to 115 mm Hg at similar
19 et al. found that an analytical method using arterial pulse pressure recording (pressure recording an
23 ubstantially from LV pacing (18+/-4% rise in arterial pulse pressure, which correlates with cardiac o
24 and randomly assigned subjects with resting arterial pulse pressures >60 mm Hg and systolic pressure
26 hat reconstructs an accurate estimate of the arterial pulse signal independent of sensing location fr
27 del which maps the features of the estimated arterial pulse signal to systolic and diastolic BP readi
29 f her irregular heart action by using radial arterial pulse tracings and experimental atrial and vent
32 Secondary outcomes included decreases in arterial pulse wave velocity and carotid artery echodens