戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 sed for studies of neointima hyperplasia and arterial stenosis.
2  for TAG functional assessment of a coronary arterial stenosis.
3 no benefit in suppressing the development of arterial stenosis.
4  CCR2 as a viable therapeutic target for NF1 arterial stenosis.
5 ial atherosclerosis increases with degree of arterial stenosis.
6 terial definition, venous contamination, and arterial stenosis.
7 nd progression of neointimal hyperplasia and arterial stenosis.
8 e to warfarin for patients with intracranial arterial stenosis.
9 n assessing the clinical importance of renal arterial stenosis.
10 ve a predisposition for premature and severe arterial stenosis.
11  assessed for location and extent of carotid arterial stenosis.
12 + cells did not correlate with the degree of arterial stenosis.
13 t contributes to the development of critical arterial stenosis.
14 nformation in vivo, we used a mouse model of arterial stenosis.
15 k patients with atherosclerotic intracranial arterial stenosis.
16 timal proliferation in a murine model of NF1 arterial stenosis.
17 < .001), and for imaging of an area after an arterial stenosis (164 vs 7; P < .001).
18 Nf1 in myeloid cells amplified the degree of arterial stenosis after arterial injury.
19                In patients with intracranial arterial stenosis, aggressive medical management was sup
20  diarrhea, sphincter of Oddi dysfunction and arterial stenosis; all responded to directed treatments.
21 mmunities relates to the correlation between arterial stenosis and acute ischaemic events, including
22 hologies associated with thrombosis, such as arterial stenosis and myeloproliferative neoplasms (MPNs
23  mild, transient graft damage due to gradual arterial stenosis and the development of arterial collat
24  for months to years; LMP causes progressive arterial stenosis and thrombosis and is composed of uniq
25 iovascular abnormalities, including pulmonic arterial stenosis and ventricular septal defects accompa
26  on individual "vulnerable plaque," coronary arterial stenosis, and inducible myocardial ischemia to
27 FMD is a systemic arteriopathy presenting as arterial stenosis, aneurysm, and dissection in virtually
28 al and could lead to inaccuracies in carotid arterial stenosis assessment.
29 l MR DSA improves the delineation of carotid arterial stenosis by virtually eliminating saturation ef
30                                  Progressive arterial stenosis (cardiac allograft vasculopathy (CAV))
31 ozygous Nf1 (Nf1(+/-)) mice develop a marked arterial stenosis characterized by proliferating smooth
32 f renal transplant arteries and detection of arterial stenosis comparable with those at contrast-enha
33  grade and hemodynamic significance of renal arterial stenosis, diagnostic quality, and presence of a
34  VEC MR imaging at baseline, single coronary arterial stenosis, dipyridamole stress, and reactive hyp
35 PAD) is characterized by varying severity of arterial stenosis, exercise induced claudication, malper
36 tential novel therapeutic approach to reduce arterial stenosis following revascularization in CAD and
37 which is based on the concept that occlusive arterial stenosis generally provokes ischaemic events.
38                                 Intracranial arterial stenosis (ICAS) is an important cause of stroke
39 y, reduces in-stent neointimal formation and arterial stenosis in hypercholesterolemic rabbits.
40 specificity (92%) for the detection of renal arterial stenosis, including all four distal stenoses en
41                                              Arterial stenosis is a nonneoplastic manifestation of NF
42                 Atherosclerotic intracranial arterial stenosis is an important cause of stroke that i
43                 Atherosclerotic intracranial arterial stenosis is an important cause of stroke.
44       As compared with kidneys without renal arterial stenosis, kidneys with renal arterial stenosis
45 hed definitions; FCA was defined as cerebral arterial stenosis not attributed to specific diagnoses s
46                         With MR angiography, arterial stenosis, occlusion, or tortuosity was identifi
47 ial plaque and an estimated internal carotid arterial stenosis of 50%-100%.
48 multiphoton microscopy revealed that in vivo arterial stenosis of a damaged carotid artery markedly i
49 to 79% sensitive and 83% to 99% specific for arterial stenosis of at least 50%.
50 a non-lacunar brain infarct without proximal arterial stenosis or cardioembolic sources, with a clear
51 flow reserve and (a) assessments of coronary arterial stenosis severity by quantitative coronary angi
52  renal arterial stenosis, kidneys with renal arterial stenosis showed 50% (0.14/0.28) EF reduction (P
53                          Persistent proximal arterial stenosis was more frequent in focal cerebral ar
54 ry groups, an approximately 50% reduction in arterial stenosis was observed with targeted NP treatmen
55      Antithrombotic therapy for intracranial arterial stenosis was recently evaluated in the Warfarin
56 0.44 to 3.57+/-0.65 and 3.45+/-0.58 mm2, and arterial stenosis was reduced from 58+/-11% to 37+/-8% a
57 nner in which clinically significant carotid arterial stenosis was reported varied widely.
58 icity of DSC CT for detecting 50% or greater arterial stenosis were calculated by using a bivariate s
59                Seven pigs with induced renal arterial stenosis were studied.
60 ading to graft loss, whereas two episodes of arterial stenosis were successfully treated with percuta
61 sal status is a risk factor for intracranial arterial stenosis when compared with premenopausal statu
62 R 1.42, 95% CI 0.96 to 2.10) and ipsilateral arterial stenosis with 50%-99% narrowing (HR 1.54, 95% C