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1 and angiopoietin signalling (NO-Tie-mediated arteriogenesis).
2 odeling of pre-existing collateral arteries (arteriogenesis).
3 l for initiating the inflammatory process in arteriogenesis.
4  the GK model includes both angiogenesis and arteriogenesis.
5 ontribute to native collateral formation and arteriogenesis.
6 ndicate VEGF and VEGFR-1 are determinants of arteriogenesis.
7  epicardial beta-catenin pathway in coronary arteriogenesis.
8 in endothelial regeneration and compensatory arteriogenesis.
9 ntial for embryonic vascular development and arteriogenesis.
10  collateral arterial formation and branching arteriogenesis.
11  not sufficient, condition for nerve-induced arteriogenesis.
12 ndothelial-monocyte interactions involved in arteriogenesis.
13 ortance of vasculogenesis, angiogenesis, and arteriogenesis.
14 s that both resolve inflammation and promote arteriogenesis.
15 understood, much remains to be learned about arteriogenesis.
16 as associated with impaired angiogenesis and arteriogenesis.
17 ltistep inflammatory process responsible for arteriogenesis.
18  by VEGF withdrawal in the face of unimpeded arteriogenesis.
19 shaping physiological and ischemia-triggered arteriogenesis.
20 nlarges the arterial lumen, a process called arteriogenesis.
21 d adult angiogenesis, lymphangiogenesis, and arteriogenesis.
22 ndothelial growth factor A (VEGF) to enhance arteriogenesis.
23 actor A (VEGF-A) expression and induction of arteriogenesis.
24 d its role in regulation of angiogenesis and arteriogenesis.
25 hat myoglobin overexpression does not affect arteriogenesis.
26 genesis but impaired developmental and adult arteriogenesis.
27 ative contributions of various cell types to arteriogenesis.
28 erturbs nerve-vessel alignment and abolishes arteriogenesis.
29  signaling pathway can effectively stimulate arteriogenesis.
30            The extent and the time course of arteriogenesis after femoral artery ligation was evaluat
31                               The process of arteriogenesis after occlusion of a major artery is poor
32 of ADMA on angioadaptation (angiogenesis and arteriogenesis) after hindlimb ischemia, as assessed by
33 quely demonstrate that functional collateral arteriogenesis alone is not necessarily sufficient for a
34                                              Arteriogenesis also requires endothelial expression of N
35          Mice lacking Nogo-B exhibit reduced arteriogenesis and angiogenesis that are linked to a dec
36 play differential roles in ischemia-mediated arteriogenesis and angiogenesis, partly because of their
37 into the critical role of Notch signaling in arteriogenesis and angiogenesis.
38 ne for beta1 integrin (Itgb1) inhibited both arteriogenesis and angiogenesis.
39 dothelial ERK signaling is critical for both arteriogenesis and arterial-venous patterning and that R
40 ese results can serve as a genomic model for arteriogenesis and as a database for developing new ther
41                                              Arteriogenesis and collateral formation are complex proc
42 s a key regulator of adult and developmental arteriogenesis and collateral formation.
43 findings suggest a novel role for PECAM-1 in arteriogenesis and collateral remodeling.
44 ized that PECAM-1 plays an important role in arteriogenesis and collateral remodeling.
45 S knockout mice [eNOS (-/-)] have defects in arteriogenesis and functional blood flow reserve after m
46 ascular lumen formation as well as defective arteriogenesis and heart malformation.
47 th intrarenal angiogenesis and peri-stenosis arteriogenesis and increased the expression of angiogeni
48              Bmx regulates ischemia-mediated arteriogenesis and lymphangiogenesis, but its role in tu
49            Serial changes in lower extremity arteriogenesis and muscle perfusion were evaluated after
50 ive synthetic phenotype during physiological arteriogenesis and pathological conditions such as ather
51 essing vessels, transport vessels undergoing arteriogenesis and peritumor vessels influenced by tumor
52  are central to both developmental and adult arteriogenesis and provide a model for future studies of
53 tification of regional changes in CT-defined arteriogenesis and quantification of 201Tl perfusion.
54 n increase in the biomarkers of angiogenesis/arteriogenesis and reduction in the markers of thrombosi
55 g permits serial, regional quantification of arteriogenesis and resting tissue perfusion after limb i
56 tify the endocardium as a site of endogenous arteriogenesis and source of endothelial cells to promot
57          Evidence that sensory nerves direct arteriogenesis and that the membrane-spanning Notch sign
58 ; collectively, these pathways contribute to arteriogenesis and the recovery of blood perfusion.
59 ized by day 7, mimicking relative changes in arteriogenesis and tissue perfusion.
60 pre-existing vessels to collateral arteries (arteriogenesis) and angiogenic capillary growth.
61  in ischemic muscle to enhance angiogenesis, arteriogenesis, and perfusion recovery in experimental P
62 ay in macrophage polarization, angiogenesis, arteriogenesis, and perfusion recovery.
63 type ischemic muscle decreased angiogenesis, arteriogenesis, and perfusion, whereas transfer of wild-
64 -93-25(-/-) PAD mice increased angiogenesis, arteriogenesis, and the extent of perfusion, which corre
65 macrophage at the center of ischemia-induced arteriogenesis, and they establish a novel role for Myos
66 ying the mechanisms underlying angiogenesis, arteriogenesis, and vascular disorders, such as HHT and
67 mia, and includes 3 processes: angiogenesis, arteriogenesis, and vasculogenesis.
68 cluding angiogenesis, vascular permeability, arteriogenesis, and vasospasm.
69  in preserving blood flow, thereby promoting arteriogenesis, angiogenesis, and mural cell recruitment
70 mpaired limb perfusion recovery with reduced arteriogenesis, angiogenesis, and recruitment of inflamm
71       This result may occur through impaired arteriogenesis, angiogenesis, or mobilization of stem an
72 ium and bone marrow plays a critical role in arteriogenesis/angiogenesis in vivo and suggests that Bm
73 o study the role of Bmx in ischemia-mediated arteriogenesis/angiogenesis.
74 rge into large conduit arteries in ischemia (arteriogenesis) are major determinants of the severity o
75 n additional phenotype of defective coronary arteriogenesis associated with RXRalpha deficiency and i
76 percholesterolemia results in delayed native arteriogenesis because of reduced early monocyte/macroph
77 be used to assess endogenous and therapeutic arteriogenesis before recovery of tissue perfusion.
78 diabetes-related impairment of angiogenesis, arteriogenesis, blood flow, and functional recovery in a
79 1, which are 2 stretch-induced regulators of arteriogenesis, both in vitro and in vivo.
80    In vivo, DACH1 was expressed during early arteriogenesis but was down in mature arteries.
81 size and promoted vascular proliferation and arteriogenesis, but did not surpass single therapies wit
82 that nerve-derived VEGF may be important for arteriogenesis, but its role in vivo remains unclear.
83  Myeloid cells are important contributors to arteriogenesis, but their key molecular triggers and cel
84                Such selective enhancement of arteriogenesis by therapeutically administered PlGF-1 de
85 r (VEGF) expression gene on angiogenesis and arteriogenesis by using cardiovascular magnetic resonanc
86 mice were correlated with ischemia-initiated arteriogenesis, capillary formation, and vessel maturati
87 3-25(-/-)) showed decreased angiogenesis and arteriogenesis correlating with increased M1-like macrop
88  during vasculogenesis and ischemia-mediated arteriogenesis could be temporally assessed by noninvasi
89 strategies that target both angiogenesis and arteriogenesis could yield the most efficacious treatmen
90 flow recovery after femoral artery ligation (arteriogenesis) dependent on the attenuation of leukocyt
91                          In a mouse model of arteriogenesis (femoral artery ligation), we found that
92 ptor signaling, which limits pial collateral arteriogenesis following cerebrovascular occlusion.
93  approaches for therapeutic angiogenesis and arteriogenesis for PAD.
94 he first evidence that VEGF is necessary for arteriogenesis from a primitive capillary plexus in vivo
95  miR-17 approximately 92 to ischemia-induced arteriogenesis has not been investigated in an in vivo m
96 eft DM and arise only after-and dependent on-arteriogenesis, implicating arteries as drivers of gut l
97 erial branches and collaterals) and impaired arteriogenesis in adult mice.
98   Synectin modulates developmental and adult arteriogenesis in an endothelial cell-autonomous fashion
99 ligand-independent manner, thereby promoting arteriogenesis in diseased tissues.
100 rted enhanced cerebrovascular remodeling and arteriogenesis in experimental type 2 diabetes.
101                                 The delay in arteriogenesis in HCE mice correlated with delayed tissu
102 ovide evidence for an inductive function for arteriogenesis in heart development distinct from its ro
103 ice displayed improved vascular survival and arteriogenesis in ischemic hind limbs, leading to the ac
104 ted arterial growth in adult mice, restoring arteriogenesis in mice lacking synectin and in atheroscl
105 paired angiogenesis in the setting of normal arteriogenesis in response to femoral artery ligation, a
106 d bone marrow-derived cells is essential for arteriogenesis in response to hindlimb ischemia in mice.
107 or induction of therapeutic angiogenesis and arteriogenesis in the chronically ischemic heart.
108  physiologically significant angiogenesis or arteriogenesis in the chronically ischemic porcine heart
109                            Here we show that arteriogenesis in the DM begins during gut rotation and
110 roles for NRP1 in postnatal angiogenesis and arteriogenesis in the heart and retina, pathological neo
111 results yield further evidence for a role of arteriogenesis in the induction of impulse-conducting Pu
112 n endothelial cells, is required for FMD and arteriogenesis in the ischemic hindlimb.
113 ort term, nicotine promotes angiogenesis and arteriogenesis in the setting of ischemia.
114 at IL-12 disruption rescued angiogenesis and arteriogenesis in type 2 diabetic mice.
115 nockout on VEGF signaling, angiogenesis, and arteriogenesis in vitro and in vivo.
116            Quantitative tools for evaluating arteriogenesis in vivo are not readily available, and th
117 neurons and/or Schwann cells is required for arteriogenesis in vivo.
118 ently, ischemia-initiated collateral growth (arteriogenesis) in the upper limb and capillary formatio
119                             Angiogenesis and arteriogenesis induced by VEGF genes improved regional m
120                                     Coronary arteriogenesis is a central step in cardiogenesis, requi
121                                              Arteriogenesis is the process of formation of arterial c
122 rmation of arterial vasculature, here termed arteriogenesis, is a central process in embryonic vascul
123  a novel candidate that negatively regulates arteriogenesis, meriting additional studies to unravel t
124 vidence of tortuous arterioles indicative of arteriogenesis (n=6-8 per group).
125 IGN AND Angiogenesis (capillary density) and arteriogenesis (number of collaterals and intratree anos
126                                  Substantial arteriogenesis occurred in the primary collaterals suppl
127  showing that endocardial flowers develop by arteriogenesis of Cx40(-) cells and by outgrowth of pre-
128                               While adaptive arteriogenesis of the pial vessels and angiogenesis at t
129 atistical differences in perfusion recovery, arteriogenesis, or angiogenesis 28 days after FAL.
130                                              Arteriogenesis, or the lumenal expansion of pre-existing
131 h factor-B (VEGF-B), which promotes coronary arteriogenesis, physiological cardiac hypertrophy, and i
132 romotes epicardial activation and myocardial arteriogenesis post MI.
133 ved cells (EPDCs) production, and myocardial arteriogenesis post MI.
134 icantly improved limb perfusion recovery and arteriogenesis, preserved vasculogenic paracrine signali
135                This process, referred to as "arteriogenesis," relies on local recruitment of leukocyt
136                         Mechanisms directing arteriogenesis remain unclear.
137 ever, these vessels failed to undergo proper arteriogenesis, resulting in poor perfusion.
138 ow us to uncouple nerve-vessel alignment and arteriogenesis, revealing that nerve-derived Cxcl12 stim
139 te the functional and clinical importance of arteriogenesis, the biology of the process is poorly und
140 Though alterations in shear stress stimulate arteriogenesis, the migration of monocytes into the peri
141 tress (FSS) is a key initiating stimulus for arteriogenesis, the outward remodeling of collateral art
142                                              Arteriogenesis, the shear stress-driven remodeling of co
143 py on circulating biomarkers of angiogenesis/arteriogenesis, thrombosis, inflammation, and leukocyte
144 rs and promote pathogen/debris clearance and arteriogenesis/tissue regeneration, respectively.
145 age polarization to promote angiogenesis and arteriogenesis to revascularize ischemic muscle in exper
146  to determine whether nicotine could enhance arteriogenesis, to compare the magnitude of this effect
147 e relative contributions of angiogenesis and arteriogenesis toward the overall reperfusion response a
148  plays an essential role in angiogenesis and arteriogenesis, two processes critical to restoration of
149            Plasma biomarkers of angiogenesis/arteriogenesis (vascular endothelial growth factor-A, fi
150                                    Increased arteriogenesis was demonstrated by observations of faste
151 us VEGF in ischemia-induced angiogenesis and arteriogenesis was measured by capillary density and mic
152 he specific role of beta-catenin in coronary arteriogenesis, we have generated conditional beta-caten
153                    To address its effects on arteriogenesis, we investigated arterial growth in hyper
154       Increased collateral artery diameters (arteriogenesis) were observed in adductor muscles of GSO
155  myogenic marker expression, blood flow, and arteriogenesis when transplanted into the ischemic hindl
156 n the vascular compartment: MC1568 increased arteriogenesis whereas MS275 inhibited it.
157 mined the impairment to be defective induced arteriogenesis, whereas developmental vasculogenesis was
158  be useful in studies of stroke and cerebral arteriogenesis, which require the accurate assessment of
159 nsory nerves or Schwann cells prevent proper arteriogenesis, while those that disorganize the nerves
160    BMC injection had a substantial effect on arteriogenesis, with normalization of total arterial are
161 or (PlGF) stimulated robust angiogenesis and arteriogenesis without significant side effects, whereas
162 inates macrophage-mediated inflammation with arteriogenesis, wound healing, and blood flow control.

 
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