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1 pared with those presenting with hemorrhagic arteriovenous malformation.
2 emorrhage after embolization of a cerebellar arteriovenous malformation.
3 s with pituitary tumor, medulloblastoma, and arteriovenous malformation.
4 uch as the cardiofaciocutaneous syndrome and arteriovenous malformation.
5  stroke in patients with an unruptured brain arteriovenous malformation.
6 nt feeding artery, which was consistent with arteriovenous malformation.
7 mab to treat the macular oedema in a case of arteriovenous malformation.
8 ted] as assessed by development of pulmonary arteriovenous malformation.
9 can trigger cardiovascular diseases, such as arteriovenous malformations.
10 ication of nascent blood vessels and develop arteriovenous malformations.
11 in 18 patients with head and neck tumors and arteriovenous malformations.
12 d this finding is not always associated with arteriovenous malformations.
13 ary venous collateral channels and pulmonary arteriovenous malformations.
14 der characterized by nosebleeds, anemia, and arteriovenous malformations.
15 aracterized by extensive telangiectasias and arteriovenous malformations.
16 utations are frequently observed in sporadic arteriovenous malformations.
17  developing aktDelta/Delta DA uncovers early arteriovenous malformations.
18 ctasia, a devastating disorder that leads to arteriovenous malformations.
19 ding from mucocutaneous telangiectasias, and arteriovenous malformations.
20  et al. describing a mouse model of cerebral arteriovenous malformations.
21 G12D or G12V) are sufficient to induce brain arteriovenous malformations.
22 adult patients with severe KRAS G12C-related arteriovenous malformations.
23 ular assist devices (LVADs) and is caused by arteriovenous malformations.
24 well as the presence of multiple and massive arteriovenous malformations.
25 on congenital heart disease and intracranial arteriovenous malformations.
26 , congestive heart failure due to a cerebral arteriovenous malformation (1), tracheal occlusion incur
27  4905 patients), meningiomas (1490 [30.4%]), arteriovenous malformations (1089 [22.2%]), trigeminal n
28                                Treatment for arteriovenous malformations aims to prevent and resolve
29 ovascular accident caused by a right pontine arteriovenous malformation and destruction of the right
30 n is subclassified into fast-flow, including arteriovenous malformation and portwine stain, and slow-
31             The patients were diagnosed with arteriovenous malformations and bilateral retinoblastoma
32                                  Hemorrhagic arteriovenous malformations and cavernous malformations
33 both lungs is essential to prevent pulmonary arteriovenous malformations and cyanosis.
34    Mice mutant for Notch1 and Notch3 develop arteriovenous malformations and display hallmarks of the
35                                           In arteriovenous malformations and dural arteriovenous fist
36  risk of intracranial bleeding, such as pial arteriovenous malformations and dural fistulas.
37 r EC-specific deletion of Otulin resulted in arteriovenous malformations and embryonic lethality.
38 and unexposed children and young adults with arteriovenous malformations and in those exposed to feru
39 f some other conditions, such as parenchymal arteriovenous malformations and intracerebral hemorrhage
40 y related Fox transcription factors, exhibit arteriovenous malformations and lack of induction of art
41 cular malformations (VMs) including visceral arteriovenous malformations and mucosal telangiectasia.
42 ed by the age-dependent development of focal arteriovenous malformations and telangiectases.
43 raphy or to estimate the risk of bleeding in arteriovenous malformations and therefore to evaluate th
44 ic deletion of Alk1 in mice and found severe arteriovenous malformations and vascular leakage.
45 ous malformation, arteriovenous fistula, and arteriovenous malformation) and vascular tumors allows f
46 tive tool for the treatment of brain tumors, arteriovenous malformation, and functional conditions.
47          Defects in the hyaloid vasculature, arteriovenous malformations, and coarctation of the aort
48 terized by multisystemic vascular dysplasia, arteriovenous malformations, and focal dilatation of pos
49 chronic obstructive pulmonary disease, brain arteriovenous malformations, and select cancers.
50 estigate the proper management of unruptured arteriovenous malformations, and the key factors in endo
51                                        Brain arteriovenous malformations are currently attracting inc
52       A Randomised trial of Unruptured Brain Arteriovenous malformations (ARUBA) aims to compare the
53    In A Randomized trial of Unruptured Brain Arteriovenous malformations (ARUBA), randomisation was h
54 especially in the venous drainage areas, and arteriovenous malformations as determined by increased c
55 el density was comparable for hemangioma and arteriovenous malformation (AVM) but significantly great
56 n about whether to treat an unruptured brain arteriovenous malformation (AVM) depends on a comparison
57                                   Congenital arteriovenous malformation (AVM) in the pelvic area is u
58                                              Arteriovenous malformation (AVM) is a fast-flow, congeni
59                                    A uterine arteriovenous malformation (AVM) is a rare cause of uter
60                                              Arteriovenous malformation (AVM) is an abnormal connecti
61                                 Extracranial arteriovenous malformation (AVM) is most commonly caused
62 entation of intracranial hemorrhage in brain arteriovenous malformation (AVM) patients.
63                                         This arteriovenous malformation (AVM), which shunts nearly al
64  better outcome prediction for patients with arteriovenous malformation (AVM)-related intracerebral h
65 age (ICH) is a serious complication of brain arteriovenous malformation (AVM).
66 n endothelial cells (ECs) cause extracranial arteriovenous malformation (AVM).
67                                     As brain arteriovenous malformations (AVM) and intracranial aneur
68                                        Liver arteriovenous malformations (AVM) in hereditary hemorrha
69 reotactic radiosurgery planning for cerebral arteriovenous malformations (AVM) is complicated by the
70 n may result in angiogenesis, and ultimately arteriovenous malformations (AVM), through transforming
71 epistaxis, mucocutaneous telangiectases, and arteriovenous malformations (AVM).
72 proportion of strokes are caused by cerebral arteriovenous malformations (AVM).
73 , exhibit retarded retinal vessel growth and arteriovenous malformations (AVM).
74  ventricular aneurysms (n = 3/24), pulmonary arteriovenous malformations (AVMs) (n = 5/24), and proxi
75                                        Brain arteriovenous malformations (AVMs) are abnormal connecti
76                                        Brain arteriovenous malformations (AVMs) are abnormal tangles
77                                        Brain arteriovenous malformations (AVMs) are an important caus
78                                              Arteriovenous malformations (AVMs) are characteristic of
79                                 Extracranial arteriovenous malformations (AVMs) are characterized by
80                                     Cerebral arteriovenous malformations (AVMs) are common vascular m
81                                        Brain arteriovenous malformations (AVMs) are currently being t
82                                              Arteriovenous malformations (AVMs) are fragile direct co
83                            Congenital pelvic arteriovenous malformations (AVMs) are high-flow vascula
84                        Mechanisms underlying arteriovenous malformations (AVMs) are poorly understood
85                                 Extracranial arteriovenous malformations (AVMs) are rare but dangerou
86                                     Cerebral arteriovenous malformations (AVMs) are the most common v
87                                              Arteriovenous malformations (AVMs) are tortuous vessels
88                                       Pelvic arteriovenous malformations (AVMs) are uncommon lesions
89                                              Arteriovenous malformations (AVMs) are vascular anomalie
90                                       Spinal arteriovenous malformations (AVMs) can lead to developme
91                                              Arteriovenous malformations (AVMs) have an inherent capa
92                                   Background Arteriovenous malformations (AVMs) in eloquent brain reg
93 cell cycle regulation and the development of arteriovenous malformations (AVMs) in HHT is not known.
94 cterized by mucocutaneous telangiectases and arteriovenous malformations (AVMs) in internal organs.
95       This disease is characterized by small arteriovenous malformations (AVMs) in mucocutaneous area
96                                              Arteriovenous malformations (AVMs) in organs, such as th
97                                              Arteriovenous malformations (AVMs) in the lungs, liver a
98 age (ICH) for patients with unruptured brain arteriovenous malformations (AVMs) in the natural course
99 n about the frequency and clinical course of arteriovenous malformations (AVMs) of the brain in adult
100      The natural history of paediatric brain arteriovenous malformations (AVMs) remains uncertain, pa
101 enetic bleeding disorder leading to systemic arteriovenous malformations (AVMs), is caused by loss-of
102              Because mutations in ALK1 cause arteriovenous malformations (AVMs), our findings suggest
103 onnections between arteries and veins called arteriovenous malformations (AVMs), which can hemorrhage
104 g capillary beds is the primary pathology of arteriovenous malformations (AVMs).
105 inherited vascular disorder characterized by arteriovenous malformations (AVMs).
106 after stereotactic radiosurgery for cerebral arteriovenous malformations (AVMs).
107 characterized by excessive angiogenesis with arteriovenous malformations (AVMs).
108 t connections between arteries and veins, or arteriovenous malformations (AVMs).
109 es to life-threatening visceral and cerebral arteriovenous malformations (AVMs).
110 l hypervascularization and the appearance of arteriovenous malformations (AVMs).
111 acterized by local telangiectases and larger arteriovenous malformations (AVMs); but how ENG function
112  of Notch signaling is associated with brain arteriovenous malformation (bAVM), a disease that lacks
113         Here, we used a mouse model of brain arteriovenous malformation (bAVM)-mediated by endothelia
114                                        Brain arteriovenous malformations (BAVMs) are a rare but poten
115                                        Brain arteriovenous malformations (bAVMs) are an important cau
116                                        Brain arteriovenous malformations (BAVMs) are congenital vascu
117                                        Brain arteriovenous malformations (BAVMs) can cause devastatin
118 r morphology (i.e. compact/diffuse) of brain arteriovenous malformations (bAVMs) correlates with the
119 nterventional treatment for unruptured brain arteriovenous malformations (bAVMs) is uncertain because
120           The pathogenesis of sporadic brain arteriovenous malformations (BAVMs) remains unknown, but
121 cally associated with capillary malformation-arteriovenous malformation, but sporadic reports of lymp
122 s achieved satisfactory obliteration of deep arteriovenous malformations, but with increased actuaria
123 rds: Pulmonary, Soft Tissues/Skin, Vascular, Arteriovenous Malformation (C) RSNA, 2023.
124               Congenital heart diseases with arteriovenous malformations carry a high risk of mortali
125 antly increased CBF in the presumed nidus of arteriovenous malformation causing haemorrhage (n = 1),
126       The wide scope of pathology, including arteriovenous malformations, cavernous malformations and
127 giopathy (CPA) is a rare subtype of cerebral arteriovenous malformation, characterized by unique angi
128                       Capillary malformation-arteriovenous malformation (CM-AVM) is a blood and lymph
129                       Capillary malformation-arteriovenous malformation (CM-AVM) is an autosomal domi
130 ant vascular disorder capillary malformation-arteriovenous malformation (CM-AVM).
131 RASA1 mutations cause capillary malformation-arteriovenous malformation (CM-AVM); whether it also fun
132 rhage, including age at initial diagnosis of arteriovenous malformation, co-existing extranidal aneur
133                                         Some arteriovenous malformations considered inoperable in the
134            Tissue samples from patients with arteriovenous malformations displayed strong endothelial
135  including aneurysmal formation and rupture, arteriovenous malformations, dural arteriovenous fistula
136       This review focuses on new concepts in arteriovenous malformation etiology, classification, tre
137 n in the endothelium is sufficient for brain arteriovenous malformations, even in the setting of unin
138  or stroke in patients with unruptured brain arteriovenous malformations followed up for 33 months.
139            Ang-2 therefore may contribute to arteriovenous malformation formation and subsequent blee
140 ss of flow-migration coupling as a driver of arteriovenous malformation formation in hereditary hemor
141 enous Alk1 deletion was sufficient to induce arteriovenous malformation formation in the postnatal re
142 n the international, multicentre Genetics of Arteriovenous Malformation (GEN-AVM) consortium.
143                                    Pulmonary arteriovenous malformation has been traditionally diagno
144           Finally, many reports of recurrent arteriovenous malformations have coincided with new theo
145 data regarding factors associated with brain arteriovenous malformation hemorrhage and different trea
146 al and genetic factors associated with brain arteriovenous malformation hemorrhage, as well as studie
147 s, dieulafoy lesion in 1 (1.5%) patient, and arteriovenous malformation in 4 (6%) patients.
148 lformation: the most common and severe brain arteriovenous malformation in neonates.
149 reatment options in patients with unruptured arteriovenous malformation in the future.
150 ges but with concerns about the formation of arteriovenous malformation in the lung excluded from hep
151 inking increased PI3K signaling to fast-flow arteriovenous malformations in hereditary hemorrhagic te
152 c tracheal connections that resemble certain arteriovenous malformations in humans.
153               Inhibition of MDM2 reduced the arteriovenous malformations in the mouse model.
154 emorrhagic telangiectasia, especially occult arteriovenous malformations in visceral organs that may
155  Finally, we demonstrate that KRAS-dependent arteriovenous malformations in zebrafish are refractory
156 ing effective multidisciplinary treatment of arteriovenous malformations, including those previously
157                              The presence of arteriovenous malformations is often described when endo
158  This resulted in defective angiogenesis and arteriovenous malformations, leading to embryonic lethal
159 moptysis caused by thoracic vascular injury, arteriovenous malformation, leaking thoracic aneurysm wi
160  underlie hemorrhagic events associated with arteriovenous malformation lesions.
161 tracranial stenosis, intracranial aneurysms, arteriovenous malformations, malignant gliomas, and meni
162       Two recent studies in unruptured brain arteriovenous malformation management - ARUBA (a multice
163 dy, 18 patients with tumors (n = 14), facial arteriovenous malformations (n = 3), and vertebral arter
164                                              Arteriovenous malformations of the brain (AMB) can cause
165                                              Arteriovenous malformations of the brain are congenital
166   Final histology showed that 2 patients had arteriovenous malformations: one had a benign hemangioma
167 ow in the setting of a previously unruptured arteriovenous malformation or aneurysm.
168                                        Known arteriovenous malformations or aneurysms are considered
169                                No congenital arteriovenous malformations or congenital aneurysms demo
170 , unruptured intracranial aneurysm, cerebral arteriovenous malformation, or cerebrospinal fluid shunt
171        Future studies are needed to identify arteriovenous malformation patients at the greatest risk
172 tural course, and treatment outcome in adult arteriovenous malformation patients.
173 resent a promising therapeutic treatment for arteriovenous malformation patients.
174 pulmonary angiography in detecting pulmonary arteriovenous malformation (PAVM) in children with cavop
175            We report on a case of pancreatic arteriovenous malformation (PAVM) that obliterated short
176 d includes arrhythmias, edema, and pulmonary arteriovenous malformations (PAVM) among others.
177            Within the past decade, pulmonary arteriovenous malformations (PAVMs) have evolved from ra
178 ss is a recognized complication of pulmonary arteriovenous malformations (PAVMs) that allow systemic
179  standard of care for treatment of pulmonary arteriovenous malformations (PAVMs).
180 clusion Smoking is associated with pulmonary arteriovenous malformation persistence after embolizatio
181                                        Renal arteriovenous malformation (RAVM) is a rare disease.
182                       The recently published arteriovenous malformation-related intracerebral haemorr
183 f preventive eradication of unruptured brain arteriovenous malformations remains uncertain.
184  as how active KRAS signaling contributes to arteriovenous malformations, remains unknown.
185                                     Cerebral arteriovenous malformations represent the most common fo
186  These mice die by mid-gestation with severe arteriovenous malformations resulting from fusion of maj
187 cyclines to decrease the rate of spontaneous arteriovenous malformation rupture.
188 areful angiographic assessment of individual arteriovenous malformations should be performed before e
189 atients had rapid reductions in symptoms and arteriovenous malformation size.
190                         Specific methods for arteriovenous malformations, such as micro-computed tomo
191 A1-related disorders (capillary malformation-arteriovenous malformation syndrome).
192 ability, progressive cyanosis from pulmonary arteriovenous malformations, systemic to hepatic venoven
193  angiodysplastic lesions (telangiectases and arteriovenous malformations) that affect many organs.
194 st the hypothesis that, for unruptured brain arteriovenous malformations, there is no difference betw
195  a lipomatous mass, an epidermoid naevus and arteriovenous malformation tissue, all of which were sam
196 ny age, and had Gamma Knife radiosurgery for arteriovenous malformation, trigeminal neuralgia, or ben
197 levant for understanding the causes of human arteriovenous malformations, tumor angiogenesis, and dia
198 ing to a diagnosis of capillary malformation/arteriovenous malformation type 1.
199 hese conditions include renal hemangiomas or arteriovenous malformations, ureteropelvic junction obst
200 t common and most severe of congenital brain arteriovenous malformations, we performed an integrated
201 ients (>/=18 years) with an unruptured brain arteriovenous malformation were enrolled into this trial
202                     Patients with unruptured arteriovenous malformations were found to be more suscep
203 ), the hallmark vascular lesion is termed an arteriovenous malformation, which involves the direct co
204 ellular and molecular perturbations in brain arteriovenous malformations, which are a leading cause o
205 ariety of pathological conditions, including arteriovenous malformations, which are prone to bleed, c
206  stroke in patients with an unruptured brain arteriovenous malformation who are allocated to either m
207 n 21 subjects with epilepsy, brain tumor, or arteriovenous malformation who had undergone IAP and MEG
208  adults, matched for age and sex, with brain arteriovenous malformations who received at least one do
209 18 years) diagnosed with an unruptured brain arteriovenous malformation, who had never undergone inte
210 icentre randomized trial of unruptured brain arteriovenous malformations) will be of major importance
211 ts of the angioarchitecture of the principal arteriovenous malformations with risk of intracranial bl

 
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