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1 es to which LDV belongs, Arteriviridae (aka, arteriviruses).
2  a new virus family which includes the genus arterivirus.
3 aused not by SHFV but by two novel divergent arteriviruses.
4 ree open reading frames not present in other arteriviruses.
5 sitive-sense, single-stranded RNA [ssRNA(+)] arteriviruses.
6 es, and compared it to those of LV and other arteriviruses.
7 are related to ORFs 2 through 4 of the other arteriviruses.
8 three more ORFs on its genome than the other arteriviruses.
9 ree additional ORFs as compared to the other arteriviruses.
10 ection and enables the culture of fastidious arteriviruses.
11 e-wide CRISPR-knockout screens with multiple arteriviruses.
12 mian hemorrhagic fever virus (SHFV; a simian arterivirus), a rare mode of virus entry that is shared
13 ns of evolution differ markedly among simian arteriviruses and among host species.
14 logenetic and geographic range of the simian arteriviruses and define baboons as a natural host for t
15 ng improved live attenuated vaccines against arteriviruses and other viruses encoding similar dual-sp
16 th each other as well as with those of other arteriviruses, and the predicted catalytic residues were
17                                       Simian arteriviruses are a diverse clade of viruses infecting c
18                                       Simian arteriviruses are a diverse group of related viruses tha
19 tive macaque colonies by showing that simian arteriviruses are actively circulating in captive baboon
20                                              Arteriviruses are economically important positive-strand
21                                       Simian arteriviruses are endemic in some African primates and c
22                               Although these arteriviruses are widely distributed among African nonhu
23 dition to SIV, simian pegiviruses and simian arteriviruses are widespread and prevalent among many Af
24  lends credibility to the hypothesis that an arterivirus can manipulate cellular miRNAs to enhance vi
25 ults indicate that multiple divergent simian arteriviruses can cause SHF.
26 ian hemorrhagic fever virus (SHFV), a simian arterivirus, causes asymptomatic infections in African c
27 ur data suggest a need to modify the current arterivirus classification.
28 oss-species infection, and host-directed pan-arterivirus countermeasure development.
29 fection and serves as a molecular barrier to arterivirus cross-species infection.
30 ants provide potential antiviral targets for arterivirus disease control and prevention.
31                             Similar to other arteriviruses, EAV primarily targets cells of the monocy
32 -arterivirus receptor, with implications for arterivirus emergence, cross-species infection, and host
33                                        Other arteriviruses encode one or two active PLP1s.
34 show that FcRn synergizes with another known arterivirus entry factor, CD163, to mediate arterivirus
35  arterivirus entry factor, CD163, to mediate arterivirus entry.
36 ehydrogenase-elevating virus, a benign mouse arterivirus, exacerbates the pathogenicity of IgM anti-p
37  -2/-1 PRF are highly conserved in all known arteriviruses except equine arteritis virus (EAV) and wo
38 s (family Flaviviridae) and two novel simian arteriviruses (family Arteriviridae) in wild African gre
39 onserved mechanism employed in non-EAV/-WPDV arteriviruses for the expression of additional viral pro
40 simarteriviruses and other distantly related arteriviruses for the expression of additional viral pro
41 s and add another layer to the complexity of arterivirus genome expression.
42    Given that at least three distinct simian arteriviruses have caused fatal infections in captive ma
43 netic and geographic diversity of the simian arteriviruses, identify baboons as a natural host of the
44                         SHFV is unique among arteriviruses in having three N-terminal papain-like pro
45                                 Thus, simian arteriviruses in nature may not require major adaptation
46 capsid-forming domain of a distantly related arterivirus indicates a conserved mechanism of nucleocap
47 ody blocked infection and rescued cells from arterivirus-induced death.
48                                              Arteriviruses infect a variety of mammalian hosts, but t
49 n.IMPORTANCE Simarteriviruses are a group of arteriviruses infecting nonhuman primates, and a number
50  that FcRn is required for the entry step of arterivirus infection and serves as a molecular barrier
51                                     However, arterivirus infection in wild nonhuman primates had not
52                  The challenge in control of arterivirus infection reflects our limited knowledge of
53 e is known about deubiquitylation in pig and arterivirus infection.
54 g body of evidence that suggests that simian arterivirus infections are common in Old World monkeys o
55                  The 3'(-)NCR RNA of another arterivirus, lactate dehydrogenase-elevating virus C (LD
56 l frameshifting (PRF) is a mechanism used by arteriviruses like porcine reproductive and respiratory
57 ther, these features suggest that the simian arteriviruses may be "preemergent" zoonotic pathogens.
58 ts that one of these virus types, the simian arteriviruses, may have the potential to jump between di
59 n/UMPylation activities were reported for an arterivirus NiRAN-RdRp nsp and suggested to generate a t
60  that the conformation of the 1B domain from arterivirus nsp10 undergoes a dynamic transition.
61 we report the first dimeric structure of the arterivirus nsp11 from PRRSV at 2.75-A resolution.
62 we report the first crystal structure of the arterivirus nsp11 protein from PRRSV, which exhibits a u
63  mechanisms associated with NEMO cleavage by arterivirus nsp4 and describes a novel substrate recogni
64 e response for viral survival.IMPORTANCE The arterivirus nsp4-encoded 3C-like protease (3CL(pro)) pla
65                                        Other arterivirus PLP1s cleave only in cis at a single downstr
66 FV PLP1alpha catalytic Cys63 is unique among arterivirus PLP1s in being adjacent to an Ala instead of
67 stead of the canonical Tyr observed in other arterivirus PLP1s.
68                                          The arterivirus porcine reproductive and respiratory syndrom
69                                   Like other arteriviruses, porcine reproductive and respiratory synd
70                                        Among arteriviruses, porcine reproductive and respiratory synd
71                              Unusually, this arterivirus PRF signal lacks an obvious stimulatory RNA
72                          During replication, arteriviruses produce a 3' coterminal, nested set of sub
73 irst structure of full-length nsp10 from the arterivirus PRRSV at 3.0- angstrom resolution.
74 rt, we demonstrate that the N protein of the arterivirus PRRSV participates in viral RNA replication
75 stal structure of full-length nsp10 from the arterivirus PRRSV, which has multiple domains: an N-term
76                                          The arteriviruses recently identified in wild monkeys have h
77 r, this study identifies FcRn as a novel pan-arterivirus receptor, with implications for arterivirus
78 cation of two novel, highly divergent simian arteriviruses related to SHFV, Mikumi yellow baboon viru
79  may help elucidate the mechanism underlying arterivirus replication and may represent great potentia
80                 This enzyme is essential for arterivirus replication by cleaving a site within the vi
81         Our studies provide new insight into arterivirus replication mechanisms, which may have impli
82 nsp11 endoribonuclease plays a vital role in arterivirus replication, but its precise roles and mecha
83 ndoU superfamily and plays a crucial role in arterivirus replication.
84 ndrome virus (PRRSV) is a recently described arterivirus responsible for disease in swine worldwide.
85 f the cellular proteins are conserved in all arterivirus RNAs and that these cell proteins may be uti
86 he negative-strand RNA [3'(-)NCR RNA] of the arterivirus simian hemorrhagic fever virus (SHFV) is 209
87   Nine TRSs were previously reported for the arterivirus Simian hemorrhagic fever virus (SHFV).
88 ated by heterotypic nsp1betas of all non-EAV arteriviruses tested.
89  Simian hemorrhagic fever virus (SHFV) is an arterivirus that causes severe disease in captive macaqu
90 ed RNA viruses, comprising coronaviruses and arteriviruses, that employ a unique strategy of disconti
91 der Nidovirales, including coronaviruses and arteriviruses, use a unique discontinuous transcription
92 ome virus (PRRSV), and apparently most other arteriviruses, use an additional PRF mechanism to access
93    Using a panel of cell lines and divergent arteriviruses, we demonstrate that FcRn is required for
94 s known of the association of PRRSV or other arteriviruses with gonadal tissues.