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1 g the dural sinuses and the middle meningeal artery.
2 the posterior surface of the right pulmonary artery.
3 r infarction of the left anterior descending artery.
4 omposite graft based on the internal mammary artery.
5 MRI) and confirmed by biopsy of the temporal artery.
6 nteric arteries and its absence in pulmonary arteries.
7 in situ ischemia by cross-clamping the renal arteries.
8 at while acutely sparing nearby the coronary arteries.
9 o 100 W for 4 minutes at sites near coronary arteries.
10 t doses of exercise training on the coronary arteries.
11 rtant role in the remodelling of the carotid arteries.
12 ponses of cerebral and mesenteric resistance arteries.
13 These EC clones engrafted in the pulmonary arteries.
14 re prevented in AKAP5 null arterial myocytes/arteries.
15 f HA from the vessel wall of small pulmonary arteries.
16 flow-induced dilatation in murine mesenteric arteries.
17 isolated aortas, but also in vivo on femoral arteries.
18 ral and popliteal) and uninvolved (brachial) arteries.
19 rough a local niche at the base of the great arteries.
20 the endothelium of mesenteric and pulmonary arteries.
21 itis is a rare inflammatory disease of large arteries.
22 tality, as has been suggested for peripheral arteries.
23 ntly (P < .001) higher than in the pulmonary arteries (0.15 L/min +/- 0.10) and descending aorta and
24 Rasmussen aneurysms (4%), enlarged bronchial arteries (3%), and systemic bronchial collaterals in 1%
25 Bleeding avoidance strategies such as radial artery access and bivalirudin were used infrequently and
26 ium and hemogenic endothelium from the major arteries, an enriched population of prehematopoietic ste
30 dome, presence of hypoplastic or aplastic A1 arteries and hypoplastic or fetal PCoA, perpendicular he
32 f blood with the intimal surface of diseased arteries and propagate and amplify the regional injury.
33 used to reconstruct small, medium, and large arteries and veins in the rat brain and revealed areas o
36 PAAs and their derivatives, the aortic arch artery and its major branches; however, their specific f
37 poxia reduced AMPK activation in the uterine artery and placenta, and AICAR increased AMPK activation
41 n artery with white fat (superior mesenteric artery) and in aorta from both male and female of anothe
42 ntravascular lithotripsy use in the coronary arteries, and future directions for adoption of the tech
43 inical catheters to embolize renal and iliac arteries, and it can achieve rapid hemostasis in acutely
44 easurements from the right atrium, pulmonary artery, and pulmonary capillary wedge pressure were obta
45 tly afflicts young children, causes coronary artery aneurysms and can result in long-term cardiovascu
50 oxia results in vasodilatation of resistance arteries, as well as recruitment of microvessels of the
51 lar disease that included aorta and coronary artery atheroma, cardiac hypertensive disease, myocardia
52 1.32-1.95, P value = 1.9 x 10-6), and large artery atherosclerosis (OR = 2.4, 95% CI, 1.41-4.07, P v
53 incidence was less sensitive as the carotid artery became stiffer in older men and women, while that
54 burst area was more sensitive as the carotid artery became stiffer in older women but not in older me
55 t improve TIMI 3 flow in the infarct-related artery before pPCI or complete ST-segment resolution 1 h
57 dal PCO2 and blood pressure External carotid artery blood flow increased by ~43% during both exercise
58 t stress, with no change in internal carotid artery blood flow Neurovascular coupling (i.e. the relat
59 stress provoked ~16% increases in vertebral artery blood flow, independent of changes in end-tidal P
61 o-localize with IK/SK channels in mesenteric arteries but not in pulmonary arteries, which may explai
62 is to compare HCR and conventional coronary artery bypass graft (CABG) surgery medium-term outcomes.
63 [95% CI, 0.59-0.79]; P<0.0001) and coronary artery bypass grafting (hazard ratio, 0.61 [95% CI, 0.45
64 95% CI, 1.33-2.58) and lowest after coronary artery bypass grafting + mitral valve surgery (1.38; 95%
65 raft Patency in Patients Undergoing Coronary Artery Bypass Grafting Surgery) investigated whether tic
66 s who underwent elective colectomy, coronary artery bypass grafting, abdominal aortic aneurysm repair
67 formance to predict adjudicated non-coronary artery bypass grafting-related GUSTO (Global Use of Stra
68 dy: TAXUS Drug-Eluting Stent Versus Coronary Artery Bypass Surgery for the Treatment of Narrowed Arte
71 cardial infarction hospitalization; coronary artery bypass surgery; heart valve repair/replacement; p
72 sis identified donor age >40 years, coronary artery bypass, and no aspirin after LT as independent ri
73 defined by a combined metric of the coronary artery calcification score and 2-dimensional vascular ul
74 ladaptations, including accelerated coronary artery calcification, exercise-induced cardiac biomarker
76 otid and femoral plaque volume; and coronary artery calcium score (CACS) at baseline and 2.8 years la
78 Herein, we review the use of the coronary artery calcium score as a decision aid in individuals wi
79 ctive of cardiovascular risk score, coronary artery calcium score, or coronary artery area stenosis.
80 se 2b trial compared progression of coronary artery calcium volume score and other measurements of ca
81 primary end point was change in log coronary artery calcium volume score from baseline to week 52.
83 lusion (PO) of the cervical internal carotid artery (cICA) can be caused by distal ICA occlusion.
84 as middle cerebral artery occlusion, femoral artery clipping, and complete or incomplete cortical ves
86 ssure, heart rate, and simultaneous brachial artery diameter and blood velocity were recorded at rest
88 vessel-forming cells infused into the renal artery did not penetrate the renal vascular network to g
89 dministration of PRRT via the proper hepatic artery did not reproduce the increase in hepatic tumor u
90 in the MIS-C group (4%) manifested coronary artery dilatation (z score = 3.15) in the acute phase, s
92 dence interval [CI], 1.60 to 2.41), coronary artery disease (10.2%, vs. 5.2% among those without dise
93 The most common cause of SCD was coronary artery disease (40%), followed by sudden arrhythmic deat
97 rculation of patients with unstable coronary artery disease (CAD), and their recruitment to inflamed
98 trate one or more associations with coronary artery disease (CAD), atrial fibrillation, or reduced le
101 cal read and no known macrovascular coronary artery disease (n=783), MPR remained independently assoc
102 4.22 [95% CI, 1.71-10.4], P=0.002), coronary artery disease (odds ratio, 0.35 [95% CI, 0.16-0.79], P=
103 interval (CI), 1.2-3.4, P = 0.009), coronary artery disease (OR, 1.9; 95% CI, 1.1-3.7; P = 0.04), and
111 atients: 14 095 (43.8%) with stable coronary artery disease and 18 046 (56.1%) with acute coronary sy
112 cation over time in patients with peripheral artery disease and the association of changes in symptom
117 CI, 1.92-4.85] P=2.30x10(-6)) for peripheral artery disease in the inverse variance-weighted analysis
118 schaemia resulting from obstructive coronary artery disease is a major cause of morbidity and mortali
123 gene expression associations to key coronary artery disease processes and clinical phenotypes in the
124 hypertension to slow and stabilize coronary artery disease progression and improve clinical outcome.
125 h de novo 3-vessel and/or left main coronary artery disease randomized to treatment with PCI or CABG
131 of paclitaxel DCBs for treatment of coronary artery disease was not associated with increased mortali
132 ent elevation, and absence of known coronary artery disease were independent predictors of unstable l
133 ocardial infarction and multivessel coronary artery disease were randomly assigned to one of the foll
135 D (Examining Use of Ticagrelor in Peripheral Artery Disease) was a randomized clinical trial that ass
136 l (Examining Use of Ticagrelor in Peripheral Artery Disease), we examined the changes in Rutherford c
138 75 years ranged from 17% to 78% for coronary artery disease, 13% to 76% for breast cancer, and 11% to
139 LI) is the most advanced stage of peripheral artery disease, associated with significant risk of limb
141 atrial fibrillation (AF), stroke, peripheral artery disease, cancer, liver-, rheumatic-, and chronic
143 shown that in patients with chronic coronary artery disease, ischemic episodes lead to a global patte
144 associated with the genetic risk of coronary artery disease, lower intelligence, lower socioeconomic
145 s secondary prevention, concomitant coronary artery disease, particularly with prior myocardial infar
147 In revascularisation of left main coronary artery disease, PCI was associated with an inferior clin
149 ating the effects of prediabetes in coronary artery disease, stroke and chronic kidney disease, compl
150 ents with angina and nonobstructive coronary artery disease, those with coronary microvascular dysfun
151 duces the incidence and severity of coronary artery disease, whereas supplementation with nitrate can
152 rdiovascular disease-in particular, coronary artery disease-and its contribution to disease pathogene
153 urther establishing a role for this coronary artery disease-associated gene in fundamental SMC proces
154 tial remoteness was associated with coronary artery disease-related SCD (odds ratio, 1.44 [95% CI, 1.
166 rotective marker, in particular for coronary artery disease; however, HDL particle concentration (HDL
167 Over the past decade, spontaneous coronary artery dissection (SCAD) has emerged as an important cau
170 analyzed all CT angiograms of the pulmonary arteries done in patients with suspected pulmonary embol
171 hanisms underlying the formation of coronary arteries during development and during cardiac neovascul
173 underwent myomectomy) and 127 to the uterine-artery embolization group (of whom 98 underwent emboliza
176 how that an 18 h exposure of human pulmonary artery endothelial cells to the different nanoparticles
177 ex (CTOI) and iii) PBV relative to pulmonary artery enhancement (PBV/PAenh); PBV/PAenh was also compa
178 icles accumulate at disease-prone regions of arteries exposed to disturbed flow patterns, and promote
179 ilution led to a marked increase in brachial artery flow-mediated dilatation in humans The increase i
180 was induced by ligating the left circumflex artery followed by the left anterior descending coronary
181 AR (n = 195) or PAD (n = 190) of the encased artery for LAPC between January 1, 2003 and April 30, 20
184 e increased responses to vasoconstrictors in arteries from Jak2V617F mice resulting from a disturbed
194 show that vascular injury in rodent carotid arteries induces YY1 expression along with reduced expre
196 P < .001), carotid injuries versus vertebral artery injuries (49 of 420 [11.7%] vs 35 of 667 [5.2%];
197 ase (CAD), and their recruitment to inflamed arteries is implicated in events leading to mortality fo
201 e interval [CI] = 44-113%) in risk for large artery ischemic stroke, a 57% (95% CI = 29-91%) increase
202 arly detection of occluded potential culprit arteries leads to interventions that improve outcomes re
204 went left anterior descending (LAD) coronary artery ligation to mimic vulnerable atherosclerotic plaq
205 permanent left anterior descending coronary artery ligation which, 8-10 weeks later, led to systolic
206 [CI]: 1.1, 2.8; P = .02) and middle cerebral artery location (OR, 1.9; 95% CI: 1.2, 3.0; P = .008).
208 location (at proximal/distal middle cerebral artery (MCA), within/beyond diffusion-weighted imaging (
209 rebral blood velocity in the middle cerebral artery (MCAv) was obtained by transcranial Doppler sonog
210 dial infarction with nonobstructive coronary arteries (MINOCA) occurs in 6% to 15% of myocardial infa
212 found vascular remodeling in which pulmonary arteries narrow because of medial thickening and occlusi
214 markers highlight a decline in capillary and artery numbers, but not of perivascular cells in pancrea
215 rization type (occlusive into the ophthalmic artery [OA] vs. nonocclusive; P < 0.001) were included i
216 e present for the first time central retinal artery occlusion (CRAO) and central retinal vein occlusi
218 g guidelines for assessment of the pulmonary artery occlusion pressure (a frequent surrogate of left
219 uidelines do not accurately assess pulmonary artery occlusion pressure in ventilated critically ill p
221 Imaging guidelines, the predicted pulmonary artery occlusion pressure was indeterminate in 48 of the
222 rdiographic predictors of a normal pulmonary artery occlusion pressure were a lateral e'-wave greater
226 ansplant gavage 3 days after middle cerebral artery occlusion using young donor biome (2-3 months) or
228 urring even in the absence of acute coronary artery occlusion, and contributes to high rates of postc
229 rovascular settings, such as middle cerebral artery occlusion, femoral artery clipping, and complete
236 lated with desmosine (p<0.001), and coronary artery (p=0.002) and thoracic aortic (p<0.001) calcifica
237 external carotid or posterior communicating artery; P < 0.001), and catheterization type (occlusive
238 mic monitoring with an implantable pulmonary artery (PA) sensor is approved for patients with New Yor
239 abundantly expressed in the large pulmonary arteries (PAs) of healthy lung tissues from humans and r
242 ate that the dendritic nature of the hepatic artery, portal vein and hepatic vein can be predicted, t
244 t 6 months after PEA, who had mean pulmonary artery pressure >=25 mm Hg or pulmonary vascular resista
245 t advances have clarified the mean pulmonary artery pressure (mPAP) range that is above normal and is
247 neurysm (n = 1/24), left internal mammillary artery pseudoaneurysm (n = 1/24), left ventricular aneur
248 small and large retinal arteries, sclerotic arteries, regions of vascular nonperfusion, cotton-wool
250 s in surgical technique, including bronchial artery revascularization, for incorporation into the ove
252 participants from the CARDIA study (Coronary Artery Risk Development in Young Adults), a community-ba
253 and discontinuity of small and large retinal arteries, sclerotic arteries, regions of vascular nonper
255 Use of Strategies to Open Occluded Coronary Arteries) severe/life-threatening/moderate and TIMI (Thr
256 In this ex vivo imaging study of coronary artery specimens, the non-invasive imaging radiotracer,
258 ability of coronary CTA to rule out coronary artery stenosis (>=50% stenosis) in the entire populatio
260 ce the stroke impact of asymptomatic carotid artery stenosis has proved difficult over the last decad
263 increased risk of any ischemic stroke, large artery stroke, and small vessel stroke in all univariabl
266 nfection is associated with higher pulmonary artery systolic pressure (PASP) and prevalent echocardio
267 ements in central venous pressure, pulmonary artery systolic pressure, RV/left ventricular ratio, and
268 yndrome (HLHS) or transposition of the great arteries (TGA), diagnoses with lowest fetal cerebral sub
272 creased rate of graft failure due to hepatic artery thrombosis <=14 days from initial LT was observed
274 ed from ischaemic heart disease and coronary artery thrombosis, and one patient assigned fluoxetine h
276 parameters that were associated with basilar artery tip aneurysms (BTA) in a location-specific manner
277 ular eccentricity index (EI), main pulmonary artery-to-aorta (PA/AO) diameter ratio, and pulmonary ar
279 ither sham or TBE targeting the left gastric artery using an occlusion balloon microcatheter to admin
281 reactivity studies were conducted in uterine arteries (UtA) isolated from pregnant mice exposed to hy
282 on (>1 y) have been associated with coronary artery vasculopathy (CAV) in pediatric heart transplant
283 sel grade, and vessel type (internal carotid artery, vertebral artery) with BCVI-associated stroke.
284 05), catheterization route (internal carotid artery vs. external carotid or posterior communicating a
286 Thrombosis of small and mid-sized pulmonary arteries was found in various degrees in all 11 patients
287 AD, a second ITA grafted to a less important artery was associated with higher risk of operative mort
289 Macroscopic arteriosclerosis of the renal artery was independently associated with kidney discard
290 nography and arterial pressure in the radial artery was obtained by tonometry, in the supine and sitt
291 sistance >240 dyn-sec/cm(-5) , and pulmonary artery wedge pressure <=15 mm Hg without another cause o
292 chaemic lesion, swelling and hyperattenuated arteries were associated with 7-day death (OR (95% CI):
295 s C, for up to 6 days, the structures of the arteries were significantly disrupted, especially the tu
297 in mesenteric arteries but not in pulmonary arteries, which may explain TRPV4(EC) -IK/SK channel cou
298 Parenchymal segments supplied by segmental arteries with PTE showed a prevalent consolidation patte
299 ssisted stress relaxation was observed in an artery with white fat (superior mesenteric artery) and i