ライフサイエンスコーパス: artery dilation

1 e, but had no significant effect on brachial artery dilation.

2 significant effect of treatment on brachial artery dilation.

3 d 1.3, respectively; P<0.05), as was femoral artery dilation.

4 ted N-methyl-D-aspartate (NMDA)-induced pial artery dilation.

5 ischemic impairment of NOC/oFQ-mediated pial artery dilation.

6 ction between opioids and NO in hypoxic pial artery dilation.

7 g conduit artery constriction and resistance artery dilation.

8 opioids, in turn, contribute to hypoxic pial artery dilation.

9 sites in their contributions to hypoxic pial artery dilation.

10 2 channels contribute to opioid-induced pial artery dilation.

11 , partially restored attenuated NOC/oFQ pial artery dilation 1 h after I+R (9+/-1 and 18+/-1 vs. 3+/-

12 rtan therapy improved flow-mediated brachial artery dilation (1.4+/-0.9% to 3.2+/-0.8%, P=0.03) but h

13 Carotid artery dilation (2.5 atm) and complete endothelial denud

14 ine decreased impairment of hypotensive pial artery dilation after fluid percussion brain injury in f

15 ral channels and access for branch pulmonary artery dilation, all of which results in pulmonary arter

16 including ventricular dysfunction, coronary artery dilation and aneurysms, arrhythmia, and conductio

17 Mac-1 to a novel form of mechanical carotid artery dilation and complete endothelial denudation.

18 s, with the potential of leading to coronary artery dilation and coronary artery aneurysms in 25% of

19 Endothelial-dependent coronary artery dilation and increased blood flow in healthy subj

20 diagnosis; their associations with coronary artery dilation and inflammatory markers have not been w

21 c abnormalities are associated with coronary artery dilation and laboratory evidence of inflammation

22 , MR, and aortic root dilation with coronary artery dilation and laboratory inflammatory markers.

23 e effect of H/I on Katp and Kca induced pial artery dilation and the roles of tPA and ERK during/afte

24 coronary cardiac abnormalities with coronary artery dilation and with laboratory inflammatory markers

25 othelial NO release), flow-mediated brachial artery dilation (as an index of vascular NO bioactivity)

26 diagnosis predicted greater odds of coronary artery dilation at 1 and 5 weeks after diagnosis (5-week

27 These data show that NOC/oFQ elicits pial artery dilation, at least in part, via cAMP, K(ATP), and

28 FPI blunted PGE2 pial artery dilation, but U 0126 and SP 600125 (10(-6) M) (ER

29 These data show that pial artery dilation by Kca channel activation is not mediate

30 We have shown that artery dilation by lithocholate (LC) and related cholane

31 203580 did not prevent impairment of PG pial artery dilation by NOC/oFQ.

32 Since recent studies show that pial artery dilation during a 20 or 40 min hypoxic exposure w

33 These data suggest that diminished pial artery dilation during longer hypoxic exposure results f

34 tivation and cAMP contribute to hypoxic pial artery dilation in a stimulus duration-dependent manner.

35 ) channel activation in NOC/oFQ-induced pial artery dilation in newborn pigs equipped with a closed c

36 annel activation in hypotension induced pial artery dilation in newborn pigs equipped with a closed c

37 a+2 channels and cAMP in opioid-induced pial artery dilation in newborn pigs equipped with closed cra

38 nd cAMP-dependent mechanisms to hypoxic pial artery dilation in piglets equipped with a closed crania

39 However, tPA potentiates impairment of pial artery dilation in response to hypotension after hypoxia

40 (Kca) activation contribute to hypoxic pial artery dilation in the piglet, responses to the NO relea

41 Hypotension induced pial artery dilation is prostaglandin-dependent in the newbor

42 (ca)) K channels and cAMP contribute to pial artery dilation observed during a 10-min exposure to hyp

43 In the lung, early pulmonary artery dilation observed in untreated MFS mice was delay

44 eatment difference in flow-mediated brachial artery dilation of 1.7% or larger as statistically signi

45 During hypotension, pial artery dilation (PAD) was impaired more in the male than

46 of subjects and was associated with coronary artery dilation, seen in 29% (p = 0.004).

47 ating system blunted mastoparan induced pial artery dilation similar to FPI (10+/-1 and 17+/-1 vs. 2+

48 Mastoparan (10(-8), 10(-6) M) elicited pial artery dilation that was blunted by FPI and partially re

49 ize the role of vasopressin in impaired pial artery dilation to activators of the ATP sensitive K (K(

50 prostaglandins contributes to impaired pial artery dilation to the newly described opioid, nocicepti

51 hannel-dependent mechanisms in impaired pial artery dilation to the newly described opioid, nocicepti

52 FQ), which contributes to impairment of pial artery dilation to the prostaglandins (PG) PGE2 and PGI2

53 Vasopressin so administered attenuated pial artery dilation to these K(+) channel activators under c

54 of N-methyl-D-aspartate (NMDA)-induced pial artery dilation to vasoconstriction.

55 lin (10(-10), 10(-8), 10(-6) M)-induced pial artery dilation was also inhibited within 1 h of FPI, bu

56 NMDA induced pial artery dilation was attenuated by I+R or H+I+R; but such

57 Cromakalim and NS1619 induced pial artery dilation was attenuated following FPI and MEAVP p

58 Cromakalim and NS1619 induced pial artery dilation was attenuated following FPI, while indo

59 at 1 month after surgery, and early brachial artery dilation was defined as the change in postoperati

60 Nitrite-induced radial artery dilation was enhanced by administration of acetaz

61 ies in piglets show that opioid-induced pial artery dilation was impaired following fluid percussion

62 Flow-mediated brachial artery dilation was impaired in the diabetics compared w

63 sion and fluid percussion brain injury, pial artery dilation was impaired more in males.

64 Flow-mediated brachial artery dilation was measured by ultrasound.

65 Coronary artery dilation was present in 13 of the 174 (7.5%) pati

66 eucine enkephalin and dynorphin-induced pial artery dilation were similarly altered by FPI and partia