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1 lial protein C receptor (EPCR) in hemophilic arthropathy.
2 l) and suffers from hemarthroses and chronic arthropathy.
3 ead to chronic disabilities with haemophilic arthropathy.
4 th effusion, meniscal tear, and degenerative arthropathy.
5 and knees, and may be associated with severe arthropathy.
6 tis, differentiating it from acute Charcot's arthropathy.
7 uromuscular disease, skeletal dysplasias and arthropathy.
8 s bleeding, with joint bleeds progressing to arthropathy.
9 thritis (RA) is the most common inflammatory arthropathy.
10  in musculoskeletal conditions, such as knee arthropathy.
11 mortality and morbidity of chronic crippling arthropathy.
12 bows, which can result in the development of arthropathy.
13 or as seen in hemophilia in general leads to arthropathy.
14 and prevention of foot ulcers and neurogenic arthropathy.
15 or anular hyperintense zone (HIZ), and facet arthropathy.
16 ritical to the development of B27-associated arthropathy.
17 ubrication is an early event in inflammatory arthropathy.
18  as it had been proposed to do in hemophilic arthropathy.
19 nfected individuals who presented with acute arthropathy.
20 d the relationship of chopsticks use to hand arthropathy.
21 al skin rash, and a characteristic deforming arthropathy.
22  and are associated with severe degenerative arthropathy.
23 y susceptible mouse can mimic B27-associated arthropathy.
24 J, and BALB/cJ beta2m(0) mice developed this arthropathy.
25 mino-transferase values, and hemochromatotic arthropathy.
26 tive arthritis, Reiter's syndrome, and other arthropathies.
27 nt enzymes and inhibitors in the destructive arthropathies.
28  patients with acromegaly, results in severe arthropathies.
29 ing of cartilage-subchondral interactions in arthropathies.
30 imulated renewed interest in crystal-induced arthropathies.
31 dvances in the management of crystal-induced arthropathies.
32 ial fluid samples from patients with various arthropathies.
33 argets for management of the crystal-induced arthropathies.
34 om patients with OA or other noninflammatory arthropathies.
35 s not associated with cancer or inflammatory arthropathy?
36 ncluding 14 patients with spinal neuropathic arthropathy (12 radiographic, seven CT, and six MR studi
37                             Of the 20, 5 had arthropathy, 5 had gastrointestinal symptoms, 4 had a ri
38 axis use and its impact on key indicators of arthropathy across the life-span among participants with
39 iency protects against developing hemophilic arthropathy, administration of a single dose of EPCR-blo
40 anged from -0.2% (95% CI, -1.0% to 0.6%) for arthropathies among males to 23.9% (95% CI, 22.7%-25.0%)
41 s can reliably differentiate between chronic arthropathies and inflammatory conditions with discrete
42 II activity may prove beneficial in limiting arthropathies and other degenerative bone diseases.
43 ails in patients with more severe peripheral arthropathy and axial involvement, and alternative treat
44 crystal deposition diseases (including gouty arthropathy and calcium pyrophosphate deposition disease
45  as a pathogenic factor in both degenerative arthropathy and chondrocalcinosis in aging.
46 ion might be linked to development of severe arthropathy and chronic infection.
47 ate data collected in participants with knee arthropathy and controls.
48 ted with human aging-associated degenerative arthropathy and directly stimulate chondrocalcinosis, ma
49 s not associated with cancer or inflammatory arthropathy and do not apply to the management of acute
50 lomerulonephritis, liver fibrosis, deforming arthropathy and increased anti-DNA antibodies.
51  with EPCR in the pathogenesis of hemophilic arthropathy and its treatment with recombinant FVIIa (rF
52 s of TRPV4 function is associated with joint arthropathy and osteoarthritis.
53 rol bleeding and complications, particularly arthropathy and physical disability.
54 autosomal-dominant human motor neuropathies, arthropathies, and skeletal malformations of varying sev
55 an be affected by many types of inflammatory arthropathies, and the most common autoimmune diseases w
56 and chronic red cell aplasia, fetal hydrops, arthropathy, and other disorders.
57 ents, herpes zoster or simplex, arthritis or arthropathy, and pneumonia.
58 stosis, crystalline arthropathy, neuropathic arthropathy, and tendinopathy.
59 sis; multicentric osteolysis, nodulosis, and arthropathy; and Winchester syndromes, skeletal dysplasi
60                          The crystal-induced arthropathies are characterized by self-limiting episode
61  comorbidities in patients with inflammatory arthropathy are important.
62 is, including poor general health, diabetes, arthropathies, arrhythmias, impotence, and skin pigmenta
63 e participant, who had substantial, advanced arthropathy at baseline, administered factor for bleedin
64                                Patients with arthropathy, B. cepacia infection, or younger age derive
65 members had multiple shoulder, hip, and knee arthropathies, beginning in the pre-teen years and conti
66 atients with already existing advanced joint arthropathy benefit from tertiary prophylaxis with signi
67 e acute arthritis and occasionally a chronic arthropathy, both in children and adults.
68 ay that has been associated with neuropathic arthropathy but has not been assessed in knee osteoarthr
69 ints is closely associated with OA and other arthropathies, but the precise role of HA in arthritis p
70 inflammation seen in primary osteoarthritis, arthropathy caused by excess GH affects all joint tissue
71        Rheumatoid arthritis is a destructive arthropathy characterized by chronic synovial inflammati
72                       The crystal deposition arthropathies comprise a host of disorders that may occu
73 f-function mutations result in camptodactyly-arthropathy-coxa vara-pericarditis (CACP) syndrome, whic
74 n autosomal recessive disorder camptodactyly-arthropathy-coxa vara-pericarditis syndrome (CACP).
75 e autosomal recessive disorder camptodactyly-arthropathy-coxa vara-pericarditis syndrome (CACP; MIM 2
76 omal recessive, human disorder camptodactyly-arthropathy-coxa vara-pericarditis syndrome.
77 athy and vascular disease (DI), with Charcot arthropathy (DA), and without complications (D), and in
78 ebilitating, crippling arthritis, hemophilic arthropathy, develops.
79    Tendon diseases are among the most common arthropathy disorders; thus knowledge of tendon gene reg
80  lupus erythematosus, and a group of chronic arthropathies, expression of CD44-dependent primary adhe
81 ontributors to the development of hemophilic arthropathy following hemarthrosis.
82 Not surprisingly, osteoarthritis and crystal arthropathy frequently coexist.
83 ks may help differentiate spinal neuropathic arthropathy from infection.
84 en bleeding occurs in the joints, hemophilic arthropathy (HA) may develop, resulting in hemarthroses
85 tion in the knee joint tissues of hemophilic arthropathy (HA) patients using quantitative susceptibil
86 nto joints in hemophilia leads to hemophilic arthropathy (HA), a debilitating joint disease.
87 te inflammation and tissue damage in crystal arthropathies have been further clarified.
88 tervertebral discs, bulging, and facet joint arthropathy have been documented in almost 90% of asympt
89 vival of less than 50% without B. cepacia or arthropathy have improved survival.
90 ticarial rash, aseptic meningitis, deforming arthropathy, hearing loss, and mental retardation.
91 a, a congenital bleeding disorder, can cause arthropathy, impaired mobility, pain, and life-threateni
92 oad suggests the negative role of hemophilic arthropathy in bone density loss.
93                The development of hemophilic arthropathy in EPCR-overexpressing FVIII-/- mice did not
94           Progression of preexisting chronic arthropathy in one participant was the only serious adve
95                                        Joint arthropathy in primary prophylaxis develops over many ye
96                    Effective ways to prevent arthropathy in severe hemophilia are unknown.
97 se acute arthritis, and occasionally chronic arthropathy, in infected adults.
98 any of the first descriptions of crystalline arthropathies, including gout, calcium pyrophosphate dep
99 olderia cepacia, and cystic fibrosis-related arthropathy increased the post-transplantation hazard of
100 s; unspecified/mixed CTD; other inflammatory arthropathy), increased ASCVD rates were found in nearly
101 or effusion, meniscal tear, and degenerative arthropathy, independent of one another.
102 esting as cutaneous vasculitis, inflammatory arthropathy, intermittent polyclonal lymphoproliferation
103 is and are often associated with destructive arthropathies involving cartilage degeneration.
104           Distal interphalangeal (DIP) joint arthropathy is characteristic of both psoriatic arthriti
105 f events from joint bleeding to synovitis to arthropathy is well documented, the component or compone
106 = 0.55), Modic changes (kappa = 0.59), facet arthropathy (kappa = 0.54), and posterior HIZ (kappa = 0
107 = 0.74), Modic changes (kappa = 0.64), facet arthropathy (kappa = 0.69), and posterior HIZ (kappa = 0
108 te and chronic parvoviral B19 infections and arthropathy may provide insights into virus-host interac
109 rding rubella as a possible cause of chronic arthropathy, more negative evidence accumulated with two
110 (2- to 2.3-fold), but was not affected by an arthropathy mutation (1.1-fold).
111 c entrapment (n = 19), neoplasm (n = 9), and arthropathy (n = 11).
112 diopathic skeletal hyperostosis, crystalline arthropathy, neuropathic arthropathy, and tendinopathy.
113                                   Hemophilic arthropathy occurs in all patients with severe and moder
114      An unusual destructive and hypertrophic arthropathy of the atlantoaxial joint in calcium pyropho
115 images in patients with diabetic neuropathic arthropathy of the foot were examined by two reviewers i
116 e of any increased risk of new onset chronic arthropathies or neurologic conditions in women receivin
117 f Philadelphia were examined for evidence of arthropathy or arthritis.
118         Transplanted adults with B. cepacia, arthropathy, or a 5-year predicted survival of greater t
119 elated systemic pathology (e.g., early onset arthropathy, premature aging, ovulation, late onset of p
120 sorders) and 9 disease outcomes (gout, gouty arthropathy, pyogenic arthritis, essential hypertension,
121                                 Degenerative arthropathy resulting from recurrent hemarthrosis remain
122      Patients with the systemic inflammatory arthropathy, rheumatoid arthritis (RA), were evaluated a
123 arthritis (RR 4.9 [2.7-7.6]) or inflammatory arthropathy (RR 1.4 [1.2-1.7]), operation under general
124                                        Joint arthropathy secondary to recurrent hemarthroses remains
125 nti-IL-17 antibody prevented the destructive arthropathy seen in vaccinated and challenged IFN-gamma(
126 CHIKV disease, usually a self-limiting viral arthropathy, share multiple inflammatory processes.
127  study suggest that treatment of acromegalic arthropathy should involve inhibition of ectopic chondro
128                                          The arthropathy showed many features characteristic of human
129                          These include viral arthropathies such as O'nyong-nyong, chikungunya, and de
130 ssociated with inflammatory and degenerative arthropathies such as rheumatoid arthritis.
131 were less likely to be seen for nontraumatic arthropathies than nonpostpartum women (4.5% vs 7.2%, P=
132 rthritis is a heterogeneous group of chronic arthropathies that are influenced by complex genetic and
133 described as an inflammatory crystal-induced arthropathy that afflicts peripheral joints.
134  to end-stage joint degeneration (hemophilic arthropathy), the major morbidity of hemophilia.
135          Among the 124 subjects with chronic arthropathy, the onset occurred between 1 week and 6 wee
136  joint has been referred to as an "analgesic arthropathy." This article discusses common treatments o
137 ) were classified as having undifferentiated arthropathy (UA).
138 collateral ligament injury, and degenerative arthropathy was recorded.
139 eumatoid arthritis [RA], 20 OA, and 27 other arthropathies) was evaluated.
140 osteoarthritis (OA) or other noninflammatory arthropathies were used as controls.
141 findings for diagnosis of spinal neuropathic arthropathy were vacuum disk on radiographs and CT image
142 me assessment as an indicator of early joint arthropathy when followed by ultrasound or magnetic reso
143                    Gout is a form of crystal arthropathy where monosodium urate (MSU) crystals deposi
144 y represent a familial type of pyrophosphate arthropathy with a phenotype that includes peripheral an
145 d rubella vaccine in the US and some chronic arthropathy with an onset between 1 week and 6 weeks aft

 
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