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1 and that these effects are dependent on the aryl hydrocarbon receptor.
2 m cross-talk between the molecular clock and aryl hydrocarbon receptor.
3 ptophan and salicylate) as activators of the aryl hydrocarbon receptor.
4 8-tetrachlorodibenzo-p-dioxin (TCDD) via the aryl hydrocarbon receptor.
5 is pathway through a noncanonical pathway of aryl hydrocarbon receptor.
6 l microbes and innate lymphoid cells via the aryl hydrocarbon receptor.
7 AhR; Aryl hydrocarbon receptor.
8 n abundance and the enhanced activity of the aryl hydrocarbon receptor.
9 ammatory properties-partly via activation of aryl hydrocarbon receptors.
11 nhibition of transcriptional activity of the aryl hydrocarbon receptor, a key regulator of ILC3 maint
12 cated within a putative binding site for the aryl hydrocarbon receptor, a master regulator of IL-22 p
13 Accordingly, we show that expression of the aryl hydrocarbon receptor, a transcription factor import
16 y of Il1rl2(-/-) mice could be rescued by an aryl hydrocarbon receptor agonist, which was sufficient
17 stent with their role in detoxification, the aryl hydrocarbon receptor (Ahr) (r(2) = 0.18, p = 0.045
18 gistically elicit allergic inflammation, and aryl hydrocarbon receptor (AhR) activation and signaling
23 is paper deals with the characterization and aryl hydrocarbon receptor (AhR) agonist activities of a
24 il fractions were tested for the presence of aryl hydrocarbon receptor (AhR) agonist and androgen rec
25 induction of CYP450 activity in response to aryl hydrocarbon receptor (AhR) agonist omeprazole and a
26 Rodent cancer bioassays indicate that the aryl hydrocarbon receptor (AHR) agonist, 2,3,7,8-tetrach
27 an photoproduct and endogenous high-affinity aryl hydrocarbon receptor (AhR) agonist, acts as a nanom
29 FAs upregulate IL-22 production by promoting aryl hydrocarbon receptor (AhR) and hypoxia-inducible fa
30 s and controlled IL-22 production through an aryl hydrocarbon receptor (AhR) and IL-23 receptor pathw
31 ro research suggests dioxins may bind to the aryl hydrocarbon receptor (AhR) and induce telomerase ac
33 in LKO mice correlated with the elevation of aryl hydrocarbon receptor (AHR) and mediator 1 (MED1), t
36 ted the ligand-operated transcription factor aryl hydrocarbon receptor (AhR) and the hepatic enzyme t
37 ia the ligand-activated transcription factor aryl hydrocarbon receptor (AHR) and the suppressor of cy
38 ses a small-molecule ligand that targets the aryl hydrocarbon receptor (AhR) and ultimately induces T
40 In silico pathway evaluation suggested the aryl hydrocarbon receptor (AhR) as one possible target o
41 monocytes underexpressed the IL-1 inhibitor aryl hydrocarbon receptor (AHR) at baseline and accumula
45 nes and the whole liver established that the aryl hydrocarbon receptor (AhR) can disrupt G1-phase cel
47 aternal exposure to pollutants that bind the aryl hydrocarbon receptor (AhR) correlates with poorer a
48 es link exposure to pollutants that bind the aryl hydrocarbon receptor (AHR) during development with
49 ression in dendritic cells (DCs), as well as aryl hydrocarbon receptor (AhR) expression by CD4(+) T c
50 CN was shown to be inversely correlated with aryl hydrocarbon receptor (AHR) expression in neuroblast
51 Here we show that the transcription factor aryl hydrocarbon receptor (AHR) functions as a biosensor
52 mozygous mutation (c.1861C>T;p.Q621*) in the aryl hydrocarbon receptor (AHR) gene that perfectly co-s
62 ine produced by glioblastoma cells activates aryl hydrocarbon receptor (AHR) in TAMs to modulate thei
63 stream signaling of the canonical pathway of aryl hydrocarbon receptor (AhR) in vitro, thereby induci
64 e, the ligand-activated transcription factor aryl hydrocarbon receptor (AhR) is a candidate target fo
82 cs and phenotypic profiling we show that the aryl hydrocarbon receptor (AhR) is a target of ezutromid
95 y, we show that an environmental sensor, the aryl hydrocarbon receptor (AhR) is highly induced upon B
98 l exposure to contaminants that activate the aryl hydrocarbon receptor (AHR) lead to suppression of i
100 ,2-b]carbazole (FICZ), a naturally-occurring aryl hydrocarbon receptor (AhR) ligand, allowed its biol
102 icrobiota-derived metabolites, especially in aryl hydrocarbon receptor (AhR) ligands, bile acids and
105 udies have shown that the toxicant-activated aryl hydrocarbon receptor (AHR) may disrupt fat metaboli
107 d on a few biological pathways, for example, aryl hydrocarbon receptor (AhR) or estrogen receptor (ER
108 diates its effects via the activation of the aryl hydrocarbon receptor (AhR) pathway and the subseque
110 e expression data revealed activation of the aryl hydrocarbon receptor (AhR) pathway in laquinimod-tr
111 athway was significantly upregulated and the aryl hydrocarbon receptor (AhR) pathway was significantl
112 acco smoke contains numerous agonists of the aryl hydrocarbon receptor (AhR) pathway, and activation
123 eceptor repressor (AhRR) is known to repress aryl hydrocarbon receptor (AhR) signaling, but very litt
127 tions, we show that MSI2 directly attenuates aryl hydrocarbon receptor (AHR) signalling through post-
128 xia inducible factor-1alpha (HIF1-alpha) and aryl hydrocarbon receptor (AHR) supports the differentia
129 ammatory M-MO, upregulates the expression of aryl hydrocarbon receptor (AhR) target genes, and stimul
130 e a pattern of regulation in the host by the aryl hydrocarbon receptor (AhR) that is critically depen
131 we used lentivirus-mediated knockdown of the aryl hydrocarbon receptor (AHR) to demonstrate that 1023
132 th the ligand-activated transcription factor aryl hydrocarbon receptor (AHR) to drive the generation
133 orodibenzo-p-dioxin (TCDD) interact with the aryl hydrocarbon receptor (Ahr) to induce osteoclastic b
134 the therapeutic potential of activating the aryl hydrocarbon receptor (AHR) to limit ECM accumulatio
135 t loss, liberated PCBs act as ligands of the aryl hydrocarbon receptor (AhR) to negatively influence
136 y, StemRegenin 1 (SR1), an antagonist of the aryl hydrocarbon receptor (AhR) transcription factor kno
137 rrepresentation of cognate sequences for the aryl hydrocarbon receptor (AhR) transcription factor, wh
138 promote NK cell IL-10, and activation of the aryl hydrocarbon receptor (AHR) was also required for ma
140 ealthspan in worms and flies depend upon the aryl hydrocarbon receptor (AHR), a conserved detector of
141 cities require binding and activation of the aryl hydrocarbon receptor (AhR), a ligand activated tran
144 the modulation of the immune response by the aryl hydrocarbon receptor (AhR), a ligand-activated tran
147 or Gb3 synthesis, and it also identified the aryl hydrocarbon receptor (AHR), a ligand-activated tran
149 lecular weight PAHs are known ligands of the aryl hydrocarbon receptor (AhR), a nuclear receptor that
150 ly polar structure, kynurenine activates the aryl hydrocarbon receptor (AHR), a PER, ARNT, SIM (PAS)
153 -derived metabolites that signal through the aryl hydrocarbon receptor (AHR), a transcription factor
156 y, bioassays indicative of activation of the aryl hydrocarbon receptor (AhR), activation of the pregn
158 DO2 in TNBC cells was sufficient to activate aryl hydrocarbon receptor (AhR), an endogenous kynurenin
160 amely, the pregnane X receptor (PXR) and the aryl hydrocarbon receptor (AhR), and itsinteractions wit
162 nes represent known ligands of the mammalian aryl hydrocarbon receptor (AHR), and UPEC infection of A
165 cript levels of five gene targets, including aryl hydrocarbon receptor (Ahr), interleukin-1 beta (Il1
166 to compare relative risks of activating the aryl hydrocarbon receptor (AhR), nuclear factor erythroi
167 ntrols, we found that coal tar activated the aryl hydrocarbon receptor (AHR), resulting in induction
168 and the anti-lipogenic transcription factor aryl hydrocarbon receptor (Ahr), the latter of which we
170 is the ligand-activated transcription factor aryl hydrocarbon receptor (AhR), which binds TB virulenc
171 -induced Jag1 expression was mediated by the aryl hydrocarbon receptor (AhR), which bound to and acti
173 primarily depend on its ability to activate aryl hydrocarbon receptor (AhR), which is a ligand-depen
174 their toxic action through activation of the aryl hydrocarbon receptor (AhR), which is believed to re
175 te, are agonists of the transcription factor aryl hydrocarbon receptor (AhR), which is widely express
176 heir barrier functions through activation of aryl hydrocarbon receptor (AhR)- nuclear factor erythroi
177 sed in vitro and in vivo studies to quantify aryl hydrocarbon receptor (AhR)-dependent effects of PCB
178 uction of anti-inflammatory cytokines via an aryl hydrocarbon receptor (AhR)-dependent mechanism.
179 r findings uncover an activin-A-induced IRF4-aryl hydrocarbon receptor (AhR)-dependent transcriptiona
182 roduction in murine T cells through inducing aryl hydrocarbon receptor (AhR)-retinoic acid receptor-r
183 this induction was abrogated by CH223191, an aryl hydrocarbon receptor (AhR)-specific antagonist.
197 ls, IAA activated an inflammatory nongenomic aryl hydrocarbon receptor (AhR)/p38MAPK/NF-kappaB pathwa
198 ently been shown to be endogenous ligands of aryl hydrocarbon receptor (AhR; a unique cellular chemic
199 forms together with the transcription factor aryl-hydrocarbon receptor (AhR), compared to unprimed co
200 pothesis that coplanar PCBs act at adipocyte aryl hydrocarbon receptors (AhRs) to promote adipose inf
201 netic and functional similarities in species aryl hydrocarbon receptors (AHRs), which mediate DLC sen
203 ould parallel that in other clients like the aryl hydrocarbon receptor and HIF1alpha, which also inte
204 hobicity in two bioassays, indicative of the aryl hydrocarbon receptor and oxidative stress response
206 ghly expressed CD90 ( approximately 63%) and aryl hydrocarbon receptor and produced IL-17 ( approxima
207 ed in this process through activation of the aryl hydrocarbon receptor and subsequent mitochondrial r
208 te the ligand-activated transcription factor aryl hydrocarbon receptor and, consequently, antiinflamm
209 leotide-polymorphisms and upon activation of aryl-hydrocarbon-receptor and oxygen-mediated pathways.
210 particulate matter caused activation of the aryl hydrocarbon receptor, and phosphorylation of histon
212 on the ligand-activated transcription factor aryl hydrocarbon receptor, and the third on how docosano
213 associated with increased transcription from aryl hydrocarbon receptor- and oxidative stress-regulate
214 drocarbon receptor signaling pathway, as the aryl hydrocarbon receptor antagonist GNF-351 modified ap
215 ility and safety of StemRegenin-1 (SR-1), an aryl hydrocarbon receptor antagonist that expands CD34+
216 ve and tolerant populations, we identify the aryl hydrocarbon receptor-based signaling pathway as a s
220 nduces the expression of CYP2E1, CYP1A2, and aryl hydrocarbon receptor, but not of CYP3A4, hepatocyte
221 ound mutations further demonstrated that the aryl hydrocarbon receptor, but not RORgammat, was requir
222 ted with increased levels of MUC2, LYZ1, and aryl hydrocarbon receptor, but reduced levels of SLC2A5.
223 Effects of developmental activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-
225 ndogenous ligand of the transcription factor aryl hydrocarbon receptor, could change the expression o
227 that IL-23-mediated restoration of IL-22 is aryl hydrocarbon receptor dependent, whereas IL-17 requi
228 s close homolog Cyp1a1 was upregulated in an aryl hydrocarbon receptor-dependent manner, hence indica
231 , which, along with the environmental sensor aryl hydrocarbon receptor, forms a multipartite transcri
232 ted to xenobiotic metabolism (pregnane X and aryl hydrocarbon receptors), hormone-mediated modes of a
233 derivatives of tryptophan that activated the aryl-hydrocarbon receptor in CD4(+) T cells, allowing Th
234 ly increase cellular viability, activate the aryl hydrocarbon receptor, increase double-strand DNA br
239 lic mutations in the photoreceptor-expressed aryl hydrocarbon receptor interacting protein-like 1 (AI
241 of P351Delta12 hAIPL1 and the mouse isoform, aryl hydrocarbon receptor interacting protein-like 1 (mA
244 , a genome-wide CRISPR screen identified the aryl hydrocarbon receptor-interacting protein (AIP), a c
248 of PDE6 relies on the chaperone activity of aryl hydrocarbon receptor-interacting protein-like 1 (AI
249 ations in either the enzyme itself or AIPL1 (aryl hydrocarbon receptor-interacting protein-like 1), l
250 erogenic molecule (ligand for the endogenous aryl-hydrocarbon receptor; ITE) were injected i.p. four
251 ith other PDE4 inhibitors, an agonist of the aryl hydrocarbon receptor, Janus kinase inhibitors, and
252 on of the aryl hydrocarbon receptor, and the aryl hydrocarbon receptor ligand restores FLG expression
253 inarof, a bacteria-derived polyphenol, is an aryl hydrocarbon receptor ligand that attenuated inflamm
254 ct 6-formylindolo[3,2-b]carbazole (FICZ), an aryl hydrocarbon receptor ligand, has been found to be a
256 llowed the ranking of wetland sites based on aryl hydrocarbon receptor-mediated end points; EROD acti
258 ve model was applied to predict variation of aryl hydrocarbon receptor-mediated toxic potencies among
259 We postulate a hypothesis where the AhR (aryl hydrocarbon receptor) mediates aberrant cell growth
261 eted the HIF-alpha dimerization partner, the aryl hydrocarbon receptor nuclear translocator (ARNT) in
264 hypoxia-inducible factor complex (HIF-alpha.aryl hydrocarbon receptor nuclear translocator (ARNT)) r
266 and NPAS3 must each heterodimerize with the aryl hydrocarbon receptor nuclear translocator (ARNT), t
267 and a constitutively expressed beta subunit, aryl hydrocarbon receptor nuclear translocator (ARNT).
268 With hypoxia, the stabilized HIF-alpha and aryl hydrocarbon receptor nuclear translocator (ARNT, al
269 d the transport of two transcription factors-aryl hydrocarbon receptor nuclear translocator and sine
270 he circadian clock transcriptional activator aryl hydrocarbon receptor nuclear translocator-like (Bma
271 f circadian clock transcriptional activators aryl hydrocarbon receptor nuclear translocator-like (Bma
274 rcadian reporter construct [brain and muscle aryl hydrocarbon receptor nuclear translocator-like:luci
275 ptional factor BMAL1 (brain and muscle ARNT [aryl hydrocarbon receptor nuclear translocator]-like pro
276 pted to manipulate the expressions of FLRL2, aryl-hydrocarbon receptor nuclear translocator-like (Arn
277 etabolic enzyme activity, likely through the aryl hydrocarbon receptor pathway, and generation of rea
278 hanced by IL-21 expression through the c-Maf/aryl hydrocarbon receptor pathway, independent of APCs.
283 okers, including an intragenic region of the aryl hydrocarbon receptor repressor gene (AHRR; cg055759
284 prominent example is hypomethylation of the aryl hydrocarbon-receptor repressor (AHRR) locus, which
286 ng heaviness and also DNA methylation at the aryl-hydrocarbon receptor repressor (AHRR) locus, but we
287 questionnaires as well as DNA methylation in aryl-hydrocarbon receptor repressor (AHRR), a sentinel e
288 (LIMR), enhanced the interaction of LIMR and aryl-hydrocarbon receptor repressor (AHRR), and promoted
290 e to PPIs was partially mediated through the aryl hydrocarbon receptor signaling pathway, as the aryl
291 of immunology, presenting information on the aryl hydrocarbon receptor signaling pathway, the immunom
294 h were associated with gene dysregulation in aryl hydrocarbon receptor, stress-response, and thyroid
295 transcription 3 and the cell cycle regulator aryl hydrocarbon receptor, the data suggest a disturbed
296 ferentiation, indole acts via the xenobiotic aryl hydrocarbon receptor to increase expression of the
297 IDO1 interacted non-enzymatically with the aryl hydrocarbon receptor to inhibit activation of NOTCH
298 ese regulatory factors, we identify AHR, the aryl hydrocarbon-receptor which controls a healthy immun
299 totype, is independent of suppression of the aryl hydrocarbon receptor, which targets cells with more
300 l for Th9, via suppressing the expression of aryl hydrocarbon receptor, without an increase in IL-10.