戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  and that these effects are dependent on the aryl hydrocarbon receptor.
2 m cross-talk between the molecular clock and aryl hydrocarbon receptor.
3 ptophan and salicylate) as activators of the aryl hydrocarbon receptor.
4 8-tetrachlorodibenzo-p-dioxin (TCDD) via the aryl hydrocarbon receptor.
5 is pathway through a noncanonical pathway of aryl hydrocarbon receptor.
6 l microbes and innate lymphoid cells via the aryl hydrocarbon receptor.
7                                         AhR; Aryl hydrocarbon receptor.
8 n abundance and the enhanced activity of the aryl hydrocarbon receptor.
9 ammatory properties-partly via activation of aryl hydrocarbon receptors.
10                              A ligand to the aryl hydrocarbon receptor, 2,3,7,8-tetrachlorodibenzo-p-
11 nhibition of transcriptional activity of the aryl hydrocarbon receptor, a key regulator of ILC3 maint
12 cated within a putative binding site for the aryl hydrocarbon receptor, a master regulator of IL-22 p
13  Accordingly, we show that expression of the aryl hydrocarbon receptor, a transcription factor import
14                                TGF-beta1 and aryl hydrocarbon receptor activation enhanced the ERbeta
15                         TGF-beta1 as well as aryl hydrocarbon receptor activation was necessary for t
16 y of Il1rl2(-/-) mice could be rescued by an aryl hydrocarbon receptor agonist, which was sufficient
17 stent with their role in detoxification, the aryl hydrocarbon receptor (Ahr) (r(2) = 0.18, p = 0.045
18 gistically elicit allergic inflammation, and aryl hydrocarbon receptor (AhR) activation and signaling
19                                              Aryl hydrocarbon receptor (AhR) activation by high-affin
20                                              Aryl hydrocarbon receptor (AhR) activation is reported t
21                                              Aryl hydrocarbon receptor (AhR) activation was evaluated
22 udies, we found that ZIKV infection triggers aryl hydrocarbon receptor (AHR) activation.
23 is paper deals with the characterization and aryl hydrocarbon receptor (AhR) agonist activities of a
24 il fractions were tested for the presence of aryl hydrocarbon receptor (AhR) agonist and androgen rec
25  induction of CYP450 activity in response to aryl hydrocarbon receptor (AhR) agonist omeprazole and a
26    Rodent cancer bioassays indicate that the aryl hydrocarbon receptor (AHR) agonist, 2,3,7,8-tetrach
27 an photoproduct and endogenous high-affinity aryl hydrocarbon receptor (AhR) agonist, acts as a nanom
28            Recent data define a role for the aryl hydrocarbon receptor (AHR) and diet-derived AHR lig
29 FAs upregulate IL-22 production by promoting aryl hydrocarbon receptor (AhR) and hypoxia-inducible fa
30 s and controlled IL-22 production through an aryl hydrocarbon receptor (AhR) and IL-23 receptor pathw
31 ro research suggests dioxins may bind to the aryl hydrocarbon receptor (AhR) and induce telomerase ac
32               T stimulated the expression of aryl hydrocarbon receptor (AHR) and its interaction with
33 in LKO mice correlated with the elevation of aryl hydrocarbon receptor (AHR) and mediator 1 (MED1), t
34                                Activation of aryl hydrocarbon receptor (AhR) and Nrf2-mediated oxidat
35                                          The aryl hydrocarbon receptor (AhR) and pregnane X receptor
36 ted the ligand-operated transcription factor aryl hydrocarbon receptor (AhR) and the hepatic enzyme t
37 ia the ligand-activated transcription factor aryl hydrocarbon receptor (AHR) and the suppressor of cy
38 ses a small-molecule ligand that targets the aryl hydrocarbon receptor (AhR) and ultimately induces T
39 sion in pDCs, and the effect was reversed by aryl hydrocarbon receptor (AHR) antagonist.
40   In silico pathway evaluation suggested the aryl hydrocarbon receptor (AhR) as one possible target o
41  monocytes underexpressed the IL-1 inhibitor aryl hydrocarbon receptor (AHR) at baseline and accumula
42                                          The aryl hydrocarbon receptor (AHR) belongs to the PAS (PER-
43                 Xenobiotic activation of the aryl hydrocarbon receptor (AHR) by 2,3,7,8-tetrachlorodi
44                           Stimulation of the aryl hydrocarbon receptor (AHR) by xenobiotics is known
45 nes and the whole liver established that the aryl hydrocarbon receptor (AhR) can disrupt G1-phase cel
46                   Structural features of the aryl hydrocarbon receptor (AHR) can underlie species- an
47 aternal exposure to pollutants that bind the aryl hydrocarbon receptor (AhR) correlates with poorer a
48 es link exposure to pollutants that bind the aryl hydrocarbon receptor (AHR) during development with
49 ression in dendritic cells (DCs), as well as aryl hydrocarbon receptor (AhR) expression by CD4(+) T c
50 CN was shown to be inversely correlated with aryl hydrocarbon receptor (AHR) expression in neuroblast
51   Here we show that the transcription factor aryl hydrocarbon receptor (AHR) functions as a biosensor
52 mozygous mutation (c.1861C>T;p.Q621*) in the aryl hydrocarbon receptor (AHR) gene that perfectly co-s
53                                          The aryl hydrocarbon receptor (AhR) has become increasingly
54                                              Aryl hydrocarbon receptor (AhR) has been shown to have p
55                 The evolutionarily conserved aryl hydrocarbon receptor (AhR) has been studied for its
56                                          The aryl hydrocarbon receptor (AhR) has roles in cell prolif
57                  Although Th17 cells and the Aryl hydrocarbon receptor (AhR) have been implicated, th
58 TGF-beta signalling and consequently for the aryl hydrocarbon receptor (AhR) in conversion.
59      We identified a higher level of nuclear aryl hydrocarbon receptor (AhR) in LXA4-treated KSHV-inf
60 s study, we revealed a novel function of the aryl hydrocarbon receptor (AhR) in NASH.
61                 It also explored the role of aryl hydrocarbon receptor (AhR) in NP's effect.
62 ine produced by glioblastoma cells activates aryl hydrocarbon receptor (AHR) in TAMs to modulate thei
63 stream signaling of the canonical pathway of aryl hydrocarbon receptor (AhR) in vitro, thereby induci
64 e, the ligand-activated transcription factor aryl hydrocarbon receptor (AhR) is a candidate target fo
65                                          The aryl hydrocarbon receptor (AhR) is a conserved, environm
66                                          The aryl hydrocarbon receptor (AhR) is a cytoplasmic recepto
67                                          The aryl hydrocarbon receptor (AhR) is a ligand-activated tr
68                                          The aryl hydrocarbon receptor (AhR) is a ligand-activated tr
69                                          The aryl hydrocarbon receptor (AhR) is a ligand-activated tr
70                                              Aryl hydrocarbon receptor (AHR) is a ligand-activated tr
71                                          The aryl hydrocarbon receptor (AhR) is a ligand-activated tr
72                                          The aryl hydrocarbon receptor (AhR) is a ligand-activated tr
73                                          The Aryl hydrocarbon receptor (AHR) is a ligand-activated tr
74                                          The aryl hydrocarbon receptor (AhR) is a ligand-activated tr
75                                          The aryl hydrocarbon receptor (Ahr) is a ligand-activated tr
76                                          The aryl hydrocarbon receptor (AHR) is a ligand-activated tr
77                                          The aryl hydrocarbon receptor (AHR) is a ligand-activated tr
78                                          The aryl hydrocarbon receptor (AhR) is a ligand-dependent tr
79                                          The aryl hydrocarbon receptor (AHR) is a ligand-dependent tr
80                                          The aryl hydrocarbon receptor (AhR) is a nuclear receptor th
81                                          The aryl hydrocarbon receptor (AhR) is a signal-regulated tr
82 cs and phenotypic profiling we show that the aryl hydrocarbon receptor (AhR) is a target of ezutromid
83                                          The Aryl hydrocarbon Receptor (AhR) is a transcription facto
84                                          The aryl hydrocarbon receptor (AhR) is a transcription facto
85                                              Aryl hydrocarbon receptor (AhR) is a transcription facto
86                       Here, we show that the aryl hydrocarbon receptor (AhR) is activated in cells in
87                                          The Aryl hydrocarbon receptor (AHR) is an environment-sensin
88                                          The aryl hydrocarbon receptor (AhR) is an environment-sensin
89                                          The aryl hydrocarbon receptor (AHR) is an important regulato
90                                          The aryl hydrocarbon receptor (AhR) is an intracellular rece
91                                          The aryl hydrocarbon receptor (AHR) is critically involved i
92                                              Aryl hydrocarbon receptor (Ahr) is crucial for the maint
93                                          The aryl hydrocarbon receptor (AHR) is emerging as a pleiotr
94                                          The aryl hydrocarbon receptor (AHR) is expressed by immune c
95 y, we show that an environmental sensor, the aryl hydrocarbon receptor (AhR) is highly induced upon B
96                                          The aryl hydrocarbon receptor (AhR) is involved in the regul
97                    We have demonstrated that aryl hydrocarbon receptor (AhR) is overexpressed in lung
98 l exposure to contaminants that activate the aryl hydrocarbon receptor (AHR) lead to suppression of i
99                             The prototypical aryl hydrocarbon receptor (AHR) ligand, 2,3,7,8-Tetrachl
100 ,2-b]carbazole (FICZ), a naturally-occurring aryl hydrocarbon receptor (AhR) ligand, allowed its biol
101                                              Aryl hydrocarbon receptor (AhR) ligands are important fo
102 icrobiota-derived metabolites, especially in aryl hydrocarbon receptor (AhR) ligands, bile acids and
103 lize tryptophan into metabolites that act as aryl hydrocarbon receptor (AHR) ligands.
104 els are decreased in mice fed a diet free of aryl hydrocarbon receptor (AhR) ligands.
105 udies have shown that the toxicant-activated aryl hydrocarbon receptor (AHR) may disrupt fat metaboli
106                                          The aryl hydrocarbon receptor (AHR) mediates the toxic effec
107 d on a few biological pathways, for example, aryl hydrocarbon receptor (AhR) or estrogen receptor (ER
108 diates its effects via the activation of the aryl hydrocarbon receptor (AhR) pathway and the subseque
109                            We identified the aryl hydrocarbon receptor (AHR) pathway as a potent tumo
110 e expression data revealed activation of the aryl hydrocarbon receptor (AhR) pathway in laquinimod-tr
111 athway was significantly upregulated and the aryl hydrocarbon receptor (AhR) pathway was significantl
112 acco smoke contains numerous agonists of the aryl hydrocarbon receptor (AhR) pathway, and activation
113 sistance is linked to down regulation of the aryl hydrocarbon receptor (AHR) pathway.
114                                          The aryl hydrocarbon receptor (AHR) plays an important physi
115              The endogenous ligand-activated aryl hydrocarbon receptor (AHR) plays an important role
116                                          The aryl hydrocarbon receptor (AhR) plays an important role
117                                          The aryl hydrocarbon receptor (AHR) plays crucial roles in i
118                            Activation of the aryl hydrocarbon receptor (AHR) promoted mo-DC different
119                                          The aryl hydrocarbon receptor (AHR) recognizes xenobiotics a
120                                          The aryl hydrocarbon receptor (AhR) represents an environmen
121                          While repression of aryl hydrocarbon receptor (AHR) signaling has been shown
122  TGF-beta signaling pathway is influenced by aryl hydrocarbon receptor (AHR) signaling pathways.
123 eceptor repressor (AhRR) is known to repress aryl hydrocarbon receptor (AhR) signaling, but very litt
124 of IL-23, IL-22 production is independent of aryl hydrocarbon receptor (AhR) signaling.
125 d negatively correlates with inducibility of aryl hydrocarbon receptor (AHR) signaling.
126             These metabolites are ligands of aryl hydrocarbon receptor (AHR) signaling.
127 tions, we show that MSI2 directly attenuates aryl hydrocarbon receptor (AHR) signalling through post-
128 xia inducible factor-1alpha (HIF1-alpha) and aryl hydrocarbon receptor (AHR) supports the differentia
129 ammatory M-MO, upregulates the expression of aryl hydrocarbon receptor (AhR) target genes, and stimul
130 e a pattern of regulation in the host by the aryl hydrocarbon receptor (AhR) that is critically depen
131 we used lentivirus-mediated knockdown of the aryl hydrocarbon receptor (AHR) to demonstrate that 1023
132 th the ligand-activated transcription factor aryl hydrocarbon receptor (AHR) to drive the generation
133 orodibenzo-p-dioxin (TCDD) interact with the aryl hydrocarbon receptor (Ahr) to induce osteoclastic b
134  the therapeutic potential of activating the aryl hydrocarbon receptor (AHR) to limit ECM accumulatio
135 t loss, liberated PCBs act as ligands of the aryl hydrocarbon receptor (AhR) to negatively influence
136 y, StemRegenin 1 (SR1), an antagonist of the aryl hydrocarbon receptor (AhR) transcription factor kno
137 rrepresentation of cognate sequences for the aryl hydrocarbon receptor (AhR) transcription factor, wh
138 promote NK cell IL-10, and activation of the aryl hydrocarbon receptor (AHR) was also required for ma
139         These metabolites are ligands of the aryl hydrocarbon receptor (AhR)(6), and AhR-mediated sig
140 ealthspan in worms and flies depend upon the aryl hydrocarbon receptor (AHR), a conserved detector of
141 cities require binding and activation of the aryl hydrocarbon receptor (AhR), a ligand activated tran
142            TCDD is a potent activator of the aryl hydrocarbon receptor (AHR), a ligand activated tran
143                                          The aryl hydrocarbon receptor (AhR), a ligand-activated memb
144 the modulation of the immune response by the aryl hydrocarbon receptor (AhR), a ligand-activated tran
145                                          The aryl hydrocarbon receptor (AhR), a ligand-activated tran
146                                          The aryl hydrocarbon receptor (AhR), a ligand-activated tran
147 or Gb3 synthesis, and it also identified the aryl hydrocarbon receptor (AHR), a ligand-activated tran
148                           Interestingly, the aryl hydrocarbon receptor (AhR), a ligand-dependent tran
149 lecular weight PAHs are known ligands of the aryl hydrocarbon receptor (AhR), a nuclear receptor that
150 ly polar structure, kynurenine activates the aryl hydrocarbon receptor (AHR), a PER, ARNT, SIM (PAS)
151                                          The aryl hydrocarbon receptor (AhR), a regulator of xenobiot
152                        Here we show that the aryl hydrocarbon receptor (AhR), a transcription factor
153 -derived metabolites that signal through the aryl hydrocarbon receptor (AHR), a transcription factor
154                                          The aryl hydrocarbon receptor (AhR), a transcription factor
155                          We propose that the aryl hydrocarbon receptor (AhR), a unique chemical senso
156 y, bioassays indicative of activation of the aryl hydrocarbon receptor (AhR), activation of the pregn
157                                          The aryl hydrocarbon receptor (AHR), also known as the dioxi
158 DO2 in TNBC cells was sufficient to activate aryl hydrocarbon receptor (AhR), an endogenous kynurenin
159                                              Aryl hydrocarbon receptor (AhR), an important regulator
160 amely, the pregnane X receptor (PXR) and the aryl hydrocarbon receptor (AhR), and itsinteractions wit
161 tors constitutive androstane receptor (CAR), aryl hydrocarbon receptor (AhR), and Nrf2.
162 nes represent known ligands of the mammalian aryl hydrocarbon receptor (AHR), and UPEC infection of A
163          Dioxin exposure, acting through the aryl hydrocarbon receptor (AHR), blocked eyelid closure
164                                          The aryl hydrocarbon receptor (AhR), for many years almost e
165 cript levels of five gene targets, including aryl hydrocarbon receptor (Ahr), interleukin-1 beta (Il1
166  to compare relative risks of activating the aryl hydrocarbon receptor (AhR), nuclear factor erythroi
167 ntrols, we found that coal tar activated the aryl hydrocarbon receptor (AHR), resulting in induction
168  and the anti-lipogenic transcription factor aryl hydrocarbon receptor (Ahr), the latter of which we
169              Because IS is an agonist of the aryl hydrocarbon receptor (AHR), we first examined the r
170 is the ligand-activated transcription factor aryl hydrocarbon receptor (AhR), which binds TB virulenc
171 -induced Jag1 expression was mediated by the aryl hydrocarbon receptor (AhR), which bound to and acti
172                                          The aryl hydrocarbon receptor (AHR), which has been central
173  primarily depend on its ability to activate aryl hydrocarbon receptor (AhR), which is a ligand-depen
174 their toxic action through activation of the aryl hydrocarbon receptor (AhR), which is believed to re
175 te, are agonists of the transcription factor aryl hydrocarbon receptor (AhR), which is widely express
176 heir barrier functions through activation of aryl hydrocarbon receptor (AhR)- nuclear factor erythroi
177 sed in vitro and in vivo studies to quantify aryl hydrocarbon receptor (AhR)-dependent effects of PCB
178 uction of anti-inflammatory cytokines via an aryl hydrocarbon receptor (AhR)-dependent mechanism.
179 r findings uncover an activin-A-induced IRF4-aryl hydrocarbon receptor (AhR)-dependent transcriptiona
180                         All extracts induced aryl hydrocarbon receptor (Ahr)-regulated mRNAs, malform
181                                              Aryl hydrocarbon receptor (AhR)-related CYP1A4 mRNA leve
182 roduction in murine T cells through inducing aryl hydrocarbon receptor (AhR)-retinoic acid receptor-r
183 this induction was abrogated by CH223191, an aryl hydrocarbon receptor (AhR)-specific antagonist.
184 narof are mediated through activation of the aryl hydrocarbon receptor (AhR).
185 its hepatotoxicity through activation of the aryl hydrocarbon receptor (AhR).
186 mediated primarily through activation of the aryl hydrocarbon receptor (AHR).
187 insults and express the transcription factor aryl hydrocarbon receptor (AhR).
188 ynurenine pathway, producing ligands for the aryl hydrocarbon receptor (AHR).
189 r adverse outcomes through activation of the aryl hydrocarbon receptor (AhR).
190 e il22 promoter and its interaction with the aryl hydrocarbon receptor (AhR).
191 12 and FA function as natural antagonists of aryl hydrocarbon receptor (AhR).
192 ar translocation of the transcription factor aryl hydrocarbon receptor (AHR).
193 ion and activity of the transcription factor aryl hydrocarbon receptor (AhR).
194 s in vitro and in vivo, which is mediated by aryl hydrocarbon receptor (AhR).
195 onmental contaminant, is a potent ligand for aryl hydrocarbon receptor (AhR).
196 kin homeostasis is largely controlled by the aryl hydrocarbon receptor (AhR).
197 ls, IAA activated an inflammatory nongenomic aryl hydrocarbon receptor (AhR)/p38MAPK/NF-kappaB pathwa
198 ently been shown to be endogenous ligands of aryl hydrocarbon receptor (AhR; a unique cellular chemic
199 forms together with the transcription factor aryl-hydrocarbon receptor (AhR), compared to unprimed co
200 pothesis that coplanar PCBs act at adipocyte aryl hydrocarbon receptors (AhRs) to promote adipose inf
201 netic and functional similarities in species aryl hydrocarbon receptors (AHRs), which mediate DLC sen
202                         Mechanistically, the aryl hydrocarbon receptor, along with STAT3 and c-Maf, a
203 ould parallel that in other clients like the aryl hydrocarbon receptor and HIF1alpha, which also inte
204 hobicity in two bioassays, indicative of the aryl hydrocarbon receptor and oxidative stress response
205 es known to be specifically regulated by the aryl hydrocarbon receptor and oxidative stress.
206 ghly expressed CD90 ( approximately 63%) and aryl hydrocarbon receptor and produced IL-17 ( approxima
207 ed in this process through activation of the aryl hydrocarbon receptor and subsequent mitochondrial r
208 te the ligand-activated transcription factor aryl hydrocarbon receptor and, consequently, antiinflamm
209 leotide-polymorphisms and upon activation of aryl-hydrocarbon-receptor and oxygen-mediated pathways.
210  particulate matter caused activation of the aryl hydrocarbon receptor, and phosphorylation of histon
211        SIRT1 also promotes activation of the aryl hydrocarbon receptor, and the aryl hydrocarbon rece
212 on the ligand-activated transcription factor aryl hydrocarbon receptor, and the third on how docosano
213 associated with increased transcription from aryl hydrocarbon receptor- and oxidative stress-regulate
214 drocarbon receptor signaling pathway, as the aryl hydrocarbon receptor antagonist GNF-351 modified ap
215 ility and safety of StemRegenin-1 (SR-1), an aryl hydrocarbon receptor antagonist that expands CD34+
216 ve and tolerant populations, we identify the aryl hydrocarbon receptor-based signaling pathway as a s
217          Four CpGs border sequences carrying aryl hydrocarbon receptor binding sites and enhancer-spe
218                                              Aryl hydrocarbon receptor blockade prevented differentia
219                 Both depend on RORgammat and aryl hydrocarbon receptor, but NKp46(+)ILC3s also requir
220 nduces the expression of CYP2E1, CYP1A2, and aryl hydrocarbon receptor, but not of CYP3A4, hepatocyte
221 ound mutations further demonstrated that the aryl hydrocarbon receptor, but not RORgammat, was requir
222 ted with increased levels of MUC2, LYZ1, and aryl hydrocarbon receptor, but reduced levels of SLC2A5.
223   Effects of developmental activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-
224       Activation of the transcription factor aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-
225 ndogenous ligand of the transcription factor aryl hydrocarbon receptor, could change the expression o
226 he group 3 innate lymphoid cell (ILC3) in an aryl hydrocarbon receptor dependent manner.
227  that IL-23-mediated restoration of IL-22 is aryl hydrocarbon receptor dependent, whereas IL-17 requi
228 s close homolog Cyp1a1 was upregulated in an aryl hydrocarbon receptor-dependent manner, hence indica
229 ugh reactive oxygen species production in an aryl hydrocarbon receptor-dependent mechanism.
230                The transcription factor AHR (aryl hydrocarbon receptor) drives the expression of gene
231 , which, along with the environmental sensor aryl hydrocarbon receptor, forms a multipartite transcri
232 ted to xenobiotic metabolism (pregnane X and aryl hydrocarbon receptors), hormone-mediated modes of a
233 derivatives of tryptophan that activated the aryl-hydrocarbon receptor in CD4(+) T cells, allowing Th
234 ly increase cellular viability, activate the aryl hydrocarbon receptor, increase double-strand DNA br
235              The pattern of PER-, BMAL-, and aryl hydrocarbon receptor-induced P450 gene expression a
236 hen PP cells were treated with CH-223191, an aryl hydrocarbon receptor inhibitor.
237          The molecular mechanisms leading to aryl hydrocarbon receptor interacting protein (AIP) muta
238                Here, we have identified AIP (aryl hydrocarbon receptor interacting protein) as a new
239 lic mutations in the photoreceptor-expressed aryl hydrocarbon receptor interacting protein-like 1 (AI
240            Mutant P351Delta12 human isoform, aryl hydrocarbon receptor interacting protein-like 1 (hA
241 of P351Delta12 hAIPL1 and the mouse isoform, aryl hydrocarbon receptor interacting protein-like 1 (mA
242                                   Defects in aryl hydrocarbon receptor interacting protein-like1 (AIP
243                                              Aryl hydrocarbon receptor-interacting protein (AIP) muta
244 , a genome-wide CRISPR screen identified the aryl hydrocarbon receptor-interacting protein (AIP), a c
245                                              Aryl hydrocarbon receptor-interacting protein-like 1 (AI
246                                              Aryl hydrocarbon receptor-interacting protein-like 1 (AI
247                                              Aryl hydrocarbon receptor-interacting protein-like 1 (AI
248  of PDE6 relies on the chaperone activity of aryl hydrocarbon receptor-interacting protein-like 1 (AI
249 ations in either the enzyme itself or AIPL1 (aryl hydrocarbon receptor-interacting protein-like 1), l
250 erogenic molecule (ligand for the endogenous aryl-hydrocarbon receptor; ITE) were injected i.p. four
251 ith other PDE4 inhibitors, an agonist of the aryl hydrocarbon receptor, Janus kinase inhibitors, and
252 on of the aryl hydrocarbon receptor, and the aryl hydrocarbon receptor ligand restores FLG expression
253 inarof, a bacteria-derived polyphenol, is an aryl hydrocarbon receptor ligand that attenuated inflamm
254 ct 6-formylindolo[3,2-b]carbazole (FICZ), an aryl hydrocarbon receptor ligand, has been found to be a
255                                    Activated aryl hydrocarbon receptor may interact with latent Epste
256 llowed the ranking of wetland sites based on aryl hydrocarbon receptor-mediated end points; EROD acti
257 lonies based on the efficacy of eliciting an aryl hydrocarbon receptor-mediated response.
258 ve model was applied to predict variation of aryl hydrocarbon receptor-mediated toxic potencies among
259     We postulate a hypothesis where the AhR (aryl hydrocarbon receptor) mediates aberrant cell growth
260       AHR and its heterodimerization partner aryl hydrocarbon receptor nuclear translocator (ARNT) be
261 eted the HIF-alpha dimerization partner, the aryl hydrocarbon receptor nuclear translocator (ARNT) in
262        We demonstrate that the expression of aryl hydrocarbon receptor nuclear translocator (ARNT) is
263                        Our data suggest that aryl hydrocarbon receptor nuclear translocator (ARNT) pl
264  hypoxia-inducible factor complex (HIF-alpha.aryl hydrocarbon receptor nuclear translocator (ARNT)) r
265                            The expression of aryl hydrocarbon receptor nuclear translocator (ARNT), a
266  and NPAS3 must each heterodimerize with the aryl hydrocarbon receptor nuclear translocator (ARNT), t
267 and a constitutively expressed beta subunit, aryl hydrocarbon receptor nuclear translocator (ARNT).
268   With hypoxia, the stabilized HIF-alpha and aryl hydrocarbon receptor nuclear translocator (ARNT, al
269 d the transport of two transcription factors-aryl hydrocarbon receptor nuclear translocator and sine
270 he circadian clock transcriptional activator aryl hydrocarbon receptor nuclear translocator-like (Bma
271 f circadian clock transcriptional activators aryl hydrocarbon receptor nuclear translocator-like (Bma
272                 The circadian rhythm-related aryl hydrocarbon receptor nuclear translocator-like 2 (A
273                         The brain and muscle aryl hydrocarbon receptor nuclear translocator-like prot
274 rcadian reporter construct [brain and muscle aryl hydrocarbon receptor nuclear translocator-like:luci
275 ptional factor BMAL1 (brain and muscle ARNT [aryl hydrocarbon receptor nuclear translocator]-like pro
276 pted to manipulate the expressions of FLRL2, aryl-hydrocarbon receptor nuclear translocator-like (Arn
277 etabolic enzyme activity, likely through the aryl hydrocarbon receptor pathway, and generation of rea
278 hanced by IL-21 expression through the c-Maf/aryl hydrocarbon receptor pathway, independent of APCs.
279                  This effect depended on the aryl hydrocarbon receptor pathway.
280 n Hepa-1 cells but not for expression of the aryl hydrocarbon receptor protein.
281                 Transgenic overexpression of aryl hydrocarbon receptor repressor (AhRR) and AhR-media
282                                          The aryl hydrocarbon receptor repressor (AhRR) is known to r
283 okers, including an intragenic region of the aryl hydrocarbon receptor repressor gene (AHRR; cg055759
284  prominent example is hypomethylation of the aryl hydrocarbon-receptor repressor (AHRR) locus, which
285                    Four CpG sites within the aryl-hydrocarbon receptor repressor (AHRR) associate wit
286 ng heaviness and also DNA methylation at the aryl-hydrocarbon receptor repressor (AHRR) locus, but we
287 questionnaires as well as DNA methylation in aryl-hydrocarbon receptor repressor (AHRR), a sentinel e
288 (LIMR), enhanced the interaction of LIMR and aryl-hydrocarbon receptor repressor (AHRR), and promoted
289                         We further found the aryl hydrocarbon receptor signaling pathway plays an imp
290 e to PPIs was partially mediated through the aryl hydrocarbon receptor signaling pathway, as the aryl
291 of immunology, presenting information on the aryl hydrocarbon receptor signaling pathway, the immunom
292  antigen-specific TH22 cells is dependent on aryl hydrocarbon receptor signaling.
293  sporadic differentiation through xenobiotic aryl hydrocarbon receptor signaling.
294 h were associated with gene dysregulation in aryl hydrocarbon receptor, stress-response, and thyroid
295 transcription 3 and the cell cycle regulator aryl hydrocarbon receptor, the data suggest a disturbed
296 ferentiation, indole acts via the xenobiotic aryl hydrocarbon receptor to increase expression of the
297   IDO1 interacted non-enzymatically with the aryl hydrocarbon receptor to inhibit activation of NOTCH
298 ese regulatory factors, we identify AHR, the aryl hydrocarbon-receptor which controls a healthy immun
299 totype, is independent of suppression of the aryl hydrocarbon receptor, which targets cells with more
300 l for Th9, via suppressing the expression of aryl hydrocarbon receptor, without an increase in IL-10.

 
Page Top