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1 the ischemically damaged small intestine and ascending colon.
2 ut first in the stomach, small intestine and ascending colon.
3 ein expression was significantly enhanced in ascending colon.
4 of the most proximal inflection point in the ascending colon.
5 lly, overlying the position of the cecum and ascending colon.
6 wel, and most commonly affects the cecum and ascending colon.
7 ps were created at the posterior wall of the ascending colon.
8  Right-sided colon was defined as caecum and ascending colon.
9 -41.5 months; P <.001), primary tumor in the ascending colon (17.9 months; 95% CI, 0-37.5 months; P <
10 patic flexure in 4 patients (36%), cecum and ascending colon (4 pts, 36%), rectosigmoid (2 pts, 18%)
11 on for metachronous peritoneal recurrence of ascending colon adenocarcinoma.
12 ion (positive criteria); (c) air or stool in ascending colon and (d) cecal air or stool (negative cri
13 erry extract by microbiota obtained from the ascending colon and descending colon compartments of a d
14  galectins were significantly reduced in the ascending colon and duodenum in ART-treated SIV-infected
15 be divided into three major clusterings: (1) ascending colon and transverse colon, (2) descending col
16 h, antrum, corpus, duodenum, terminal ileum, ascending colon, and descending colon.
17 lon, two in the transverse colon, one in the ascending colon, and one in the cecum.
18 c biopsy specimens from the duodenum, ileum, ascending colon, and rectum.
19                              The jejunum and ascending colon are the most common sites of involvement
20                                To test this, ascending colon biopsy tissues from 35 children with ASD
21 ells are normally present in human cecum and ascending colon, but are rarely found in descending colo
22 tly correlated with SIVgag DNA levels in the ascending colon during necropsy.
23 mucosal biopsies from the terminal ileum and ascending colon during surgery and post-operative colono
24                  Overall colonic transit and ascending colon emptying (AC t((1/2))) were significantl
25 as a significant overall treatment effect on ascending colon emptying half-time (P = .015) and overal
26 ificantly faster overall colonic transit and ascending colon emptying with the 4-mg dose.
27 y short inspection duration and insufficient ascending colon examination.
28 omparison of gene expression profiles in the ascending colon from a subset of patients with ASD, chro
29 nd paired biopsies of the terminal ileum and ascending colon from healthy individuals (male=3, female
30            Areas of incidental uptake in the ascending colon had the greatest chance (42 %) of being
31 s defined as any tumor arising in the cecum, ascending colon, hepatic flexure, or transverse colon.
32 sigmoid colon, or rectum vs appendix, cecum, ascending colon, hepatic flexure, or transverse colon.
33 sigmoid colon, or rectum vs appendix, cecum, ascending colon, hepatic flexure, or transverse colon.
34 thelial cell type in both terminal ileum and ascending colon (hereon as 'LND') with high expression o
35 ing) colon but not of the right (proximal or ascending) colon (HR = 1.03, 95% CI: 0.89, 1.18), while
36 ormal colorectal anatomical locations (i.e., ascending colon (n = 182), transverse colon (n = 249), d
37  subsites of the colorectum: cecum (n = 63), ascending colon (n = 44), transverse colon (n = 32), des
38 e were cecum (n = 65), sigmoid (n = 60), and ascending colon (n = 50).
39 e of cytological dysplasia, primarily in the ascending colon (n = 6/42, 14%), Transverse colon (n = 2
40 GCPII in the 400 mug supplement group in the ascending colon, no treatment differences were observed
41 could be detected in the small intestine and ascending colon of the normoxia group.
42                               We describe an ascending colon perforation after implantation of a peri
43 signal intensity (r = 0.88, P = .033 for the ascending colon; r = 0.82, P = .006 for the descending c
44 g sensitivities (SE) and specificities (SP) (ascending colon: SE: 1.10%, SP: 91.02; transverse colon:
45 ntent (SBWC), colonic volumes, and T1 of the ascending colon (T1AC) as measures of colonic water.
46 ering all subjects, the percentage change in ascending colon volume rose significantly after CRF.
47 ructose malabsorption, as shown by increased ascending colon volumes.
48            Mean thickness of the wall of the ascending colon was significantly greater in patients wi
49                   The right colon (cecum and ascending colon) was the most common location for polyps
50                              The jejunum and ascending colon were the most common sites of gastrointe
51 ative lesions from the terminal ileum to the ascending colon with a non-specific histo-pathologic fin