1 In violation-of-expectation experiments, we
asked whether 17-mo-old infants (n = 120) would expect a
2 We then
asked whether 17B1.3 prevents signaling by binding to a
3 We also
asked whether a CB1 positive allosteric modulator would
4 displays a relative lack of neurotropism, we
asked whether a chimeric vesicular stomatitis virus (VSV
5 Specifically, we
asked whether a common microcircuit can account for the
6 We therefore
asked whether a distinct transcriptional state in a spec
7 s into the cellular phase of coagulation, we
asked whether a functional vwf gene is present in the At
8 Here, we
asked whether a generalized Bayesian magnitude estimatio
9 To address this question, we
asked whether a missense mutation associated with human
10 Here we
asked whether a primordial function, such as nucleic aci
11 We
asked whether a single transient episode of HSV-1 epithe
12 Here, we
asked whether a south-north expansion wave of L. granate
13 ons with different biological activities, we
asked whether Abeta can assume structurally and function
14 Here we use metabolic modeling to
ask whether acetate and glycerol cross-feeding are espec
15 r protein bearing two familial mutations and
asked whether activation of a phosphatase PTP1B particip
16 xperimentally address this knowledge gap, we
asked whether activation of PZ(Vgat) neurons could atten
17 Here, we
ask whether adaptation to historical conditions can incr
18 enhance the function of a compound, we then
asked whether adding a glucose makes aesculin activate N
19 As proof of concept we
asked whether administering low-level TLR2 ligands in ad
20 infectious disease vaccines, and this study
asked whether Advax (Vaxine Pty Ltd, Adelaide, Australia
21 underlying this cold-induced hyperphagia, we
asked whether agouti-related peptide (AgRP) neurons are
22 Here, we
asked whether alpha-band activity plays a similar role i
23 We
asked whether alphavbeta3-integrin positively regulates
24 of phenotypic and genotypic assortment, and
ask whether altered associations between individuals (as
25 Here, we
asked whether an artificial neural network (with convolu
26 Here, we
asked whether an increase in expression level of the cel
27 We
asked whether analysis of the genomic context of SASs ca
28 different approaches to their treatment, we
ask whether and how an unexpected event occurring at a p
29 Here we
ask whether and how motor control and redirected somatos
30 Here we
ask whether and how the dynamics of finding mates intera
31 ons across the continental United States and
asked whether and how fire-climate relationships vary ge
32 Here, we
ask whether,
and to what extent, landscape simplificatio
33 To test this hypothesis, we
asked whether,
and under what conditions, people are wil
34 Here, we
asked whether any other co-factors associate with TAPBPR
35 o the evolutionary and heterosis mechanisms,
asking whether any effects of genetic mixture occurred i
36 s are sufficient for positional encoding, we
asked whether arbitrary molecules (e.g., green fluoresce
37 A (DORmO) mouse model of type 1 diabetes, we
asked whether autoimmune insulitis was associated with c
38 Here, we
asked whether basic electrostatic interactions are suffi
39 Here we
asked whether BCL6 controlled helper function through do
40 Studies were carried out to
ask whether behavioral differences existed between the t
41 iment with humans with frontal lobe lesions,
asking whether behavioral impairments can be linked to l
42 ange of methods across cognitive science, we
ask whether belief or knowledge is the more basic kind o
43 We
asked whether beta- and gamma-secretases interact to med
44 In this study, we
asked whether beta-cell dedifferentiation can be prevent
45 Here, we
asked whether beta1 integrin, a mechanosensory protein i
46 Here we
asked whether birth order is reflected in the postnatal
47 We
asked whether brain connectomics can predict response to
48 We
asked whether bumblebees specialize over timescales shor
49 Thus, we
asked whether C1q binds to CD33 and if C1q mediates CD33
50 Here, we
asked whether C1q plays a role in experience-dependent s
51 We
asked whether Ca(2+) influx through a single NMDA recept
52 Using this in vivo model, we
asked whether CB-BF populations that express characteris
53 We
asked whether cells have the ability to limit the degree
54 We therefore
asked whether cells in the tumour stroma might explain t
55 Here we
asked whether CellSearch-detected pulmonary venous CTCs
56 Here, we
asked whether changes in chromatin accessibility that oc
57 he pupil response by selective attention, we
asked whether changes in pupil diameter follow internal
58 In this study, we
asked whether CHD7 promotes gene transcription in neural
59 Here we
ask whether children share this preference for fair part
60 We
asked whether chronic early life stress disrupts maturat
61 Therefore, the current study
asked whether CodY also regulates sporulation and CPE pr
62 In this study, we
asked whether combining innate immune activation in the
63 ularly effective opsonin for Francisella, we
asked whether complement-dependent uptake of F. tularens
64 Here we
ask whether computerized CL auditory training can enhanc
65 We
asked whether contaminating phospholipid in myosin prepa
66 d field studies in Ecuador and Madagascar to
ask whether convergence at the behavioral level provides
67 S-expressing B. thetaiotaomicron strains, we
ask whether CPSs can modify the immune responses to spec
68 To do so, we
ask whether crowdsourced trust ratings can effectively d
69 Here we
asked whether CTCF loops are established by a universal
70 Heyes
asks whether cultural learning mechanisms are cognitive
71 given to human subjects, it may be useful to
ask whether cytokine polarization is the appropriate fra
72 Here, we
asked whether de novo gene expression contributes to thi
73 We
asked whether decreased Cyfip1 levels altered the functi
74 We
asked whether development of fundamental electrophysiolo
75 We
asked whether differences between sun-exposed and shaded
76 Here, we
ask whether different codon usage controls gene expressi
77 Here, we
ask whether different ploidy levels > 2x express differe
78 We first
asked whether different combinations of facultative symb
79 Here, we
asked whether different RLCs contribute to this function
80 Finally, we
ask whether differential privacy and k-anonymity are PSO
81 Here we
ask whether disease-relevant modules of genes can be dis
82 Building on these studies, we
ask whether distributions of network variants reflect st
83 ration, and metabolic regulation in vivo, we
asked whether DJ-1 contributes to MG detoxification in v
84 We
ask whether domain interactions and calcium binding affe
85 Here we
asked whether dopaminergic signaling supports a TD learn
86 Here, we
asked whether during tumor suppression p53 might instead
87 Additionally, we
asked whether each phenotype exhibited more rapid change
88 Here, we
asked whether Emx2 mediates this effect by changing loca
89 We
asked whether environmental factors impact SNP309G funct
90 Here we
asked whether epileptic activity induces plastic changes
91 We
asked whether ERalpha expression contributes to the deve
92 in the light-organ transcriptome, led us to
ask whether EsMIF might be the gatekeeper controlling th
93 In this investigation, we
asked whether esophagitis with an eosinophilic component
94 We also
asked whether estimates of reliability were affected by
95 lonization and resulting immunopathology, we
asked whether estrogen use in the standard VVC model mas
96 In this study, we
asked whether ex-T reg Th2 cells develop and contribute
97 In this study, we
asked whether exosomes from uninfected cells could activ
98 Here we
ask whether expanded polyQ is sufficient to cause pathol
99 To test this hypothesis, we
asked whether experimentally elevating levels of the glu
100 ith the bacterium Caulobacter crescentus and
asked whether exposure to a moderate concentration of so
101 Here we
asked whether FcRn is also involved in the induction of
102 ive mutations in Saccharomyces cerevisiae to
ask whether first-step adaptive mutations result in trad
103 Here, we
asked whether folding of a complex RNA can be understood
104 Here, we
asked whether FXGs are a broadly conserved feature of th
105 We have
asked whether glucose metabolism is enhanced by either a
106 Here we
asked whether H3K9me3, through mediating gene repression
107 We
asked whether hAd-PSCs could be converted into Leydig-li
108 Here, we
asked whether HCV-elimination by sofosbuvir-based direct
109 Here, we
asked whether hearing loss-induced behavioral deficits c
110 ion plays a key role in plasticity and so we
asked whether histone acetylation might be dysregulated
111 The ASTIC trial
asked whether HSCT could cure CD.
112 Here, we
asked whether human unexplained repeated pregnancy loss
113 We
asked whether human vision also exhibits a special sensi
114 Here we
ask whether humans are also sensitive to how the other's
115 Here, we
ask whether hybridization can speed adaptive evolution u
116 Here, we
asked whether hyperthermia alters the response to chemot
117 We
asked whether IL-2R subunits could already preassemble a
118 mation transfer as a sign of decoupling, and
asked whether impaired local information processing lead
119 Here we address a further aspect,
asking whether impression formation is mandatory.
120 Here we
asked whether,
in the context of motor learning where er
121 In this genetic study, we
ask whether inactivating mutations in further genes of t
122 We
asked whether increased serotonin levels in newly identi
123 Here, we
asked whether increasing attention to (i.e., the precisi
124 cross a gradient of agricultural land use to
ask whether indicators of nutritional physiology includi
125 Finally, we
asked whether individual variation in wheel use within a
126 We
ask whether individuals with more general education and
127 and mechanisms of multisensory processing we
ask whether inputs from two senses (auditory and visual)
128 Here we
asked whether interaction of gD with a cognate entry rec
129 rate cohorts from the general population, we
asked whether intraparietal sulcus functional connectivi
130 ables, or "interoceptive illusions." Here we
ask whether it is possible to produce an illusory percep
131 Presently, we
asked whether it is possible for multiple types of categ
132 ed genes is higher in wake than in sleep, we
asked whether it is specifically wake associated with sy
133 sented with severe upper limb impairment and
asked whether it was possible to differentiate those wit
134 In the present study, we
asked whether it was possible to immediately change comp
135 is automatic has previously been studied by
asking whether it can occur implicitly (outside awarenes
136 glucopyranoside, suppresses inflammation, we
asked whether its anti-inflammatory activity is associat
137 Here, we
asked whether keratinocyte activity is also required for
138 Here we
asked whether lactate has any paracrine role via activat
139 As avian eyes differ from mammals, we
asked whether local mechanisms also underlie FD myopia i
140 erience-dependent development, leading us to
ask whether loss of Shank3 might impair homeostatic plas
141 The current study
asks whether loss of Arc/Arg3.1 can affect brain circuit
142 Here we
asked whether LRRK2 modifies inflammatory signaling and
143 We
asked whether MarA expression also has a stochastic comp
144 We
asked whether mechanical coupling of alpha(M)beta(2) int
145 Here, we
asked whether mesoaccumbal GABA (gamma-aminobutyric acid
146 cation of epidermal metabolite profiling, we
asked whether metabolites extracted from the skin surfac
147 mplitude of afterhyperpolarization led us to
ask whether METH alters the activity of Ca(2+)-activated
148 We
asked whether mice that lack HDAC6 and have potent suppr
149 We
asked whether mnemonic prediction errors promote hippoca
150 e we used multimodal neuroimaging methods to
ask whether modulating reward-related signaling in the A
151 Next, we
asked whether monkeys would generalize the middle concep
152 Thus, we
asked whether MRs regulate CA2 neuron properties and CA2
153 L1 association with ankyrin-G; therefore, we
asked whether NAP promotes the dissociation of ankyrin-G
154 an alpha-neurexins, however, which led us to
ask whether neurexophilins modulate the function of alph
155 These puzzling results prompted us to
ask whether neuroligin-3 mutant phenotypes may be reshap
156 Here we
asked whether neurons in adult mice, born during the fin
157 Here we
asked whether noisy spiking and/or crude information pro
158 We
asked whether NPC quality control is compromised in agin
159 Here we
asked whether Nurr1 is causing LID.
160 Using the early Drosophila larval brain, we
asked whether nutrient-dependent growth of neural stem c
161 to pure autotrophy and heterotrophy), and to
ask whether observed patterns are consistent with a trai
162 contact forces when feeling a surface and we
ask whether,
on a given trial, discrimination performanc
163 Here, we
ask whether opiate exposure alters differentiation and d
164 Here we
ask whether PACAP is also involved in producing reinstat
165 We first
asked whether pairs of young children (M = 74 months) wo
166 Additionally, we
asked whether parameter distributions from each of the c
167 Tremblay and colleagues
asked whether patients receiving either routine anticoag
168 Specifically, we
ask whether patterns of social contact (group membership
169 Using a genetic ablation model, we
asked whether pericyte loss alone is sufficient for capi
170 molecule (NCAM) activates FGF receptors, we
asked whether peripherally administered NCAM peptide mim
171 We
asked whether pharmacological stimulation of endogenous
172 We also test Cheverud's conjecture by
asking whether phenotypic patterns provide an acceptable
173 is pathway is activated in human cancers, we
asked whether phosphorylated Dicer1 contributed to tumor
174 Here, we focus on nitrogen and
ask whether plant community nitrogen uptake rate is dete
175 his short-term effect of hurricanes, we then
asked whether populations and species that more frequent
176 We next
asked whether potentiation of the endogenous anti-inflam
177 Finally, we
asked whether predictions would be improved by taking in
178 Here, we
ask whether preparation is engaged during mid-reach modi
179 We
ask whether primates are sensitive to global changes tha
180 Here, we
asked whether pro-inflammatory microglial/macrophage act
181 We
asked whether proactive and reactive mothers differed in
182 We then
asked whether prolonged PSAP/IntA experience would never
183 Here, we
asked whether PrP undergoes liquid-liquid phase separati
184 the formation of P. aeruginosa biofilms, we
asked whether pyruvate likewise contributes to the maint
185 In this paper, we
ask whether rapidly displacing coal electricity generati
186 We
ask whether rate forms a distinct perceptual metric, or
187 We first
asked whether reassembled communities differ from undist
188 ncer-a phenomenon called "Minority MOMP." We
asked whether reflux-induced esophageal carcinogenesis o
189 n of global protein synthesis, leading us to
ask whether resistance was due to the direct antiviral a
190 A separate item
asked whether respondents used a condom at last sexual i
191 lored factors that govern such patterns, and
asked whether reward specialists made more foraging bout
192 We next
asked whether ribosomes lacking SNORA24-guided pseudouri
193 man A3H and RNA binding-defective mutants to
ask whether RNA may be a factor in the functional intera
194 We
asked whether ROM1 could modify the phenotype of a PRPH2
195 In this Review, we
ask whether sensory system abnormalities are specific to
196 ter Yellowstone (Wyoming, United States) and
asked whether short-interval (<30 years) stand-replacing
197 Here we
ask whether similar cognitive processes are at play in p
198 Using a spiking network model, we
asked whether simplified replay could arise from three b
199 isite for sleep-like states, prompting us to
ask whether sleep is present in Cnidaria.
200 With this in mind, we
ask whether some rhizosphere microbial communities might
201 In the present article, we
asked whether Sox17 may act downstream of Wnt/beta-caten
202 natomical foundations of this hypothesis, we
asked whether space-time interactions can be observed in
203 oups of putative target genes (regulons) and
asked whether specific regulons are enriched for genes a
204 community in Boston, Massachusetts, who were
asked whether strabismus was present.
205 We
ask whether such 1D responses are consistent with the sy
206 Here, we
ask whether such directional post-mating (that is, crypt
207 To
ask whether such variability indeed existed, we assessed
208 Here, we
asked whether such abstract knowledge immediately guides
209 Here, we
asked whether such modulation of visual responses might
210 In Experiment 3, we
asked whether symmetrical views also convey an advantage
211 Here, we
asked whether tagging and stimulating cells along the do
212 Here, we
asked whether targeting of ribosome biogenesis can be us
213 nce than clinical research, it is natural to
ask whether the dynamic patterns of neural circuit activ
214 We
ask whether the effects of anthropogenic habitat convers
215 Here we
ask whether the excitability of the motor pathways to th
216 splicing modifies different channels, and to
ask whether the functional consequences of this splicing
217 es developed from small-scale experiments to
ask whether the functional diversity within a range of w
218 We show that an important first step is to
ask whether the functional unit of pathogenesis is a bac
219 llowed up on the previous proposals [1,2] to
ask whether the image features that increase seizure lik
220 Here we
ask whether the loss of various dopaminergic neuron popu
221 Here we
ask whether the precision of stereopsis varies with prof
222 murine cardiomyocytes soon after fusion and
ask whether the process is stochastic or involves conser
223 Here, we
ask whether the putative role of the hippocampal-entorhi
224 We
ask whether the resulting "empirically optimal similarit
225 In this study, we first
ask whether the same genes underlie polymorphic mimicry
226 Here we used high-density EEG recordings to
ask whether the underlying neural sources are common as
227 We
asked whether the >80% sequence differences throughout t
228 Here, we
asked whether the anterior temporal lobe (ATL) serves as
229 Hence, we
asked whether the CD8(+) Treg cells could directly targe
230 Here, we
asked whether the central electrosensory system actually
231 minals of the MF is target specific, we here
asked whether the corelease of glutamate and GABA was al
232 Finally, we
asked whether the defining molecular signature of J-RGCs
233 at NKCC1 is critical for ion homeostasis, we
asked whether the disruption to myelinated axons in slc1
234 We therefore
asked whether the DNA-binding signature of ERalpha diffe
235 s genomes enter the nucleus as naked DNA, we
asked whether the HIRA chaperone complex affects herpesv
236 chanisms associated with this phenomenon, we
asked whether the intrinsic membrane properties in hippo
237 ith PLA gene expression information and were
asked whether the lesions should be biopsied.
238 Additionally, we
asked whether the maximum hydraulic conductance in the s
239 Here, we
asked whether the parietal cortex integrates acoustic fe
240 Here, we
asked whether the position of the solar disk per se is r
241 We
asked whether the presence of kin might act on energy al
242 We
asked whether the proteome of secreted cytokines and sig
243 this within-mutation disease variability, we
asked whether the PRPH2 binding partner rod OS membrane
244 To address this issue, we
asked whether the rate and magnitude of perceptual learn
245 In this issue of the JCI, Lu and colleagues
asked whether the results of neural stem cell transplant
246 We
asked whether the same brain regions and neural codes su
247 We
asked whether the same framework could explain the contr
248 Yet we have never
asked whether the same is true for animals.
249 Specifically, we
asked whether the SC contributes to enhancing sensitivit
250 Here, we
asked whether the size of IPS is affected by manipulatio
251 ow the intestinal microbiome develops, so we
asked whether the transfer of an antibiotic-perturbed mi
252 Here, we
asked whether the variance introduced by selecting diffe
253 We
asked whether the visual system uses statistical regular
254 le of fibril structure and hydrophobicity by
asking whether the V18, A21, V40, and A42 side chains wh
255 We tested this hypothesis by
asking whether the visual word form area (VWFA), an expe
256 ms and cardiac function have recovered often
ask whether their medications can be stopped.
257 in alloantigen-induced T cell activation and
asked whether their absence might impact in vivo allores
258 ophic, and heterotrophic nanoflagellates, to
ask whether there are broad patterns in the prevalence o
259 In the current study, we have
asked whether there are subpopulations of neurons, clust
260 The patient
asked whether there is any role for definitive local the
261 tive significance of amylose are unknown, we
asked whether there is natural genetic variation in amyl
262 We
ask whether these differences can be partly explained by
263 Here, we
ask whether these documented latitudinal patterns can be
264 nections between alpha-Syn and tau led us to
ask whether these proteins might be regulated through a
265 To
ask whether these sensilla might receive pheromonal inpu
266 Here, we
asked whether these behavioral deficits could be reverse
267 Here we
asked whether these proneural genes also regulate lamina
268 Here, we
asked whether these SOC components and Homer1 are presen
269 The present study
asks whether these contradictory conclusions result from
270 emporally distinct modes of inactivation and
ask whether they can be distinguished pharmacologically.
271 ironments arise naturally it is important to
ask whether they can instead be turned to our advantage.
272 Here, we have
asked whether they also support vascular integrity when
273 Patients were
asked whether they felt like they had a choice to receiv
274 , early-stage breast cancer, physicians were
asked whether they intended to administer adjuvant CT be
275 associated with aberrant decidualization, we
asked whether they share molecular features with sPE.
276 Participants were
asked whether they would be willing to provide additiona
277 We investigated this by
asking whether they also encode the value of spatial goa
278 immunofluorescence microscopy approaches to
ask whether this mechanism is shared with the closely re
279 , using whole genomes of 92 individuals, and
ask whether this variation can be explained by heterogen
280 We
asked whether this could be attributed to aberrant expre
281 epeatedly cultured at one medical center and
asked whether this lineage might constitute an emerging
282 repeatedly cultured at 1 medical center and
asked whether this lineage might constitute an emerging
283 Here, we
asked whether this mechanism generalizes to OFF-selectiv
284 sa first touches an object, and we therefore
asked whether this parameter could provide the rat with
285 We
asked whether this pathway had an in vivo role in mice.
286 Here, we
asked whether this recovery simply reflects changes in s
287 Recently, a number of authors have
asked whether this type of motor learning problem might
288 infections during the first year of life and
asked whether this was modified by prenatal and early-li
289 l activity during spatial working memory and
asked whether time cells depend on mEC inputs.
290 We
asked whether top-scoring PPARgamma activators identifie
291 Here, we
asked whether topographic representations of the body ar
292 Moreover, we
ask whether TRPV4-mediated Ca(++)-influx evokes mast cel
293 In this study, we
asked whether tumor clone-initiating cells (TcICs) were
294 We
asked whether V1 inputs to different V2 stripes arise fr
295 ells by association with perlecan, led us to
ask whether vascular smooth muscle cells express an IL-2
296 sequencing in mouse collecting duct cells to
ask whether vasopressin signaling selectively increases
297 Here, we
asked whether virally mediated neurotrophin3 (NT3) overe
298 We
asked whether viruses could instead use such activated o
299 Here we
ask whether we can correct abnormal functional connectiv
300 anges in species across multiple regions, we
ask whether we can discriminate an urban signature of ph