ライフサイエンスコーパス: asparagine

1 outing even in the presence of glutamine and asparagine.

2 provide non-essential amino acids, including asparagine.

3 ansporter highly selective for glutamine and asparagine.

4 human L-asparaginases have millimolar KM for asparagine.

5 rolysis and compensating for the lack of the asparagine.

6 nonical RhoGAP domains lacking the auxiliary asparagine.

7 the T19A, T116A, and K188M mutants soaked in asparagine.

8 correct positioning of l-glutamine but not l-asparagine.

9 CBG steroid-binding sites is replaced by an asparagine.

10 ach if D21 is mutated into the corresponding asparagine.

11 d residue in the -2 position relative to the asparagine.

12 0.009 for lysine up to 0.061 cm(2)/(V s) for asparagine.

13 oteasomal degradation, a catabolic source of asparagine.

14 Asparagine(11) was found to occupy a pivotal position, a

15 N-glycosylation of asparagine 130 in its extracellular domain (ECD) enhance

16 Mutagenesis analysis in Kir1.1 revealed that asparagine 171 (N171) is the only pore-lining residue re

17 -P-X(140) motif, leucine-66, proline-67, and asparagine-176 may account for the broad substrate speci

18 e and asparagine 1788 to lysine) and Nav1.6 (asparagine 1768 to aspartic acid and leucine 1331 to val

19 both Nav1.1 (arginine 1648 to histidine and asparagine 1788 to lysine) and Nav1.6 (asparagine 1768 t

20 tinal rod photoreceptors, is glycosylated at asparagines-2 and 15 on its N-terminus.

21 P(N) SadP adhesin showed that the amino acid asparagine 285, which is replaced by an aspartate residu

22 cle, we demonstrate that hypersialylation of asparagine 297 (N297) enhances IgG serum persistence.

23 e of a single N-linked glycosylation site at asparagine 297 of the Fc, with deglycosylation resulting

24 a resistant M2 variant encoding a serine to asparagine 31 mutation (S31N) with improved efficacy ove

25 Asparagine 46 is considered to be important for the stru

26 ghest amount of acrylamide was produced from asparagine (5987.5microg/kg) and the lowest from phenyla

27 found across the interaction surface, in the asparagine 90-glycosylation motif and at buried sites.

28 of a carbohydrate to the nitrogen atom of an asparagine, a process referred to as N-linked glycosylat

29 trast, substitution of p5M to a conventional asparagine abolished recognition by the H-2D(b)/Trh4-spe

30 hould aim to identify cultivars that are low asparagine accumulating and are stable across different

31 ffected by location and maintained their low asparagine accumulation trait.

32 sucrose, reducing sugars, free amino acids, asparagine, acrylamide, 3-deoxyglucosone, methylglyoxal,

33 on transport chain (ETC) inhibition depletes asparagine, activating the ISR via the eIF2alpha kinase

34 almitoleic acid were increased while serine, asparagine and arachidonic acid and its derivatives were

35 ict substrate specificity for cleavage after asparagine and aspartic acid residues during in-solution

36 vide S Typhimurium the ability to catabolize asparagine and assimilate nitrogen.

37 The fructose, asparagine and chitosan mixture had more MRPs compared t

38 n a food model system consisting of glucose, asparagine and chitosans.

39 es and their combination including fructose, asparagine and different molecular weight chitosans.

40 e (ASNS) converts aspartate and glutamine to asparagine and glutamate in an ATP-dependent reaction.

41 cids, and effectively transported aspartate, asparagine and glutamate.

42 A constant amount of asparagine and glutamine in palm olein and soy bean oils

43 es, aspartate and glutamate, were mutated to asparagine and glutamine, respectively.

44 levels and lower baseline amino acid levels-asparagine and glutamine-correlate with CAN (lower basel

45 an enzyme that catalyzes the hydrolysis of L-asparagine and isoaspartyl-peptides.

46 ibactin C (6) was converted to N-myristoyl-d-asparagine and its corresponding colibactin by colibacti

47 and partial degradation of acrylamide (AA), asparagine and low molecular weight sugars were evaluate

48 the ability of secoiridoids to interact with asparagine and lysine tuning the formation of dietary ad

49 Acrylamide is produced from free asparagine and reducing sugars during high-temperature c

50 rylamide is formed from the reaction between asparagine and reducing sugars.

51 cysteine, whereas zinc binds at the terminal asparagine and the same critical cysteine.

52 amide and HMF in baked biscuits, nor between asparagine and the sum of glucose and fructose concentra

53 rate through key interactions with conserved asparagine and tyrosine residues within the binding pock

54 insically disordered, enriched in glutamines/asparagines and depleted in hydrophobic residues.

55 isoleucine metaclusters, along with serine, asparagine, and the previously studied phenylalanine, ar

56 We found that the amino acids glutamine, asparagine, arginine, aspartate, and serine activate TOR

57 hionine sulfoximine abolished the ability of asparagine, arginine, aspartate, or serine, but not that

58 Here we describe a role for asparagine as an amino acid exchange factor: intracellul

59 Gold nanoparticles synthesized using L-Asparagine as reducing and stabilization agent were empl

60 y the Maillard reaction of glucose (GL) with asparagine (AS).

61 The M6 asparagine Asn-905 stood out as being essential for the

62 was able to simultaneously separate Gln and asparagine (Asn) deamidation products even for those pep

63 ography, and gel filtration experiments with asparagine, aspartate, and valine as PKM2 ligands, we ex

64 ed significant accumulation of riboflavin, L-asparagine, aspartate, glycerol, nicotinamide, and 3-hyd

65 MS analysis of alanine, alpha-ketoglutarate, asparagine, aspartic acid, cystathionine, total cysteine

66 evels and higher threonine, serine, proline, asparagine, aspartic acid, phenylalanine, tyrosine, and

67 hain dicarboxylacylcarnitine metabolites and asparagine/aspartic acid could serve as biomarkers assoc

68 roxyisovalerylcarnitine/malonylcarnitine and asparagine/aspartic acid were associated with worse clin

69 trations of total amino acids, aspartic acid/asparagine (Asx), glutamic acid/glutamine and alanine ar

70 contrast, only those animals homozygous for asparagine at codon 146 (NN animals) succumbed to oral c

71 s concluded that only animals homozygous for asparagine at codon 146 succumb to scrapie under natural

72 l distribution of the animals homozygous for asparagine at codon 146 was significantly shorter than t

73 ycosylated in their Fc domain at a conserved asparagine at position 297.

74 for permissive CD4 alleles, which encode an asparagine at position 39 of the receptor.

75 The ubiquitous mutation from serine (WT) to asparagine at residue 31 (S31N) in the influenza A M2 ch

76 bispecificity can be attributed to a unique asparagine at the cofactor binding loop.

77 The identification of asparagine availability as a critical limiting factor fo

78 ic and chemical evidence to demonstrate that asparagine availability plays a critical role in efficie

79 2-oxoglutarate dependent oxygenase aspartate/asparagine-beta-hydroxylase (AspH) catalyses the hydroxy

80 Human aspartate/asparagine-beta-hydroxylase (AspH) is a 2-oxoglutarate (

81 absence of glutamine, which is necessary for asparagine biosynthesis.

82 ragine synthetase (ASNS), which provides for asparagine biosynthesis.

83 sion of alphaENaC lacking these glycosylated asparagines blunted this effect.

84 Initially, N-CDs were prepared from L-asparagine by pyrolysis and characterized by different s

85 Collectively, these results indicate that asparagine catabolism contributes to S Typhimurium virul

86 the translational fidelity of glutamine and asparagine codons.

87 but substitutions of serine by aspartate or asparagine completely abolished substrate transport.

88 tipping point in the ratio below which free asparagine concentration could affect acrylamide formati

89 The association of asparagine concentration in Canadian bread wheat with cu

90 The need to produce wheat with low asparagine concentration is of great importance as a mea

91 The asparagine concentration ranged from 168.9 to 1050 ug/g

92 scuits with glucose, also having the highest asparagine concentration.

93 e synthetase knockdown and altering of media asparagine concentrations, we show that intracellular as

94 rylamide is formed directly from the maltose-asparagine conjugate.

95 al barrier to VACV replication due to a high asparagine content of viral proteins and a rapid demand

96 o be responsible for feruloylserotonin and l-asparagine content variation across populations, respect

97 Rather, conserved asparagines contributed to suppression of the substrate

98 his terpene synthase revealed an active site asparagine critical for water capture and specificity du

99 of methionine oxidation and higher levels of asparagine deamdiation were observed.

100 that modifies a eukaryotic target through an asparagine deamidase activity, which in turn elicits hos

101 , which is relatively poorly understood, and asparagine deamidation, which has been more thoroughly s

102 Insufficient expression of ASNS leads to asparagine deficiency, which facilitates ATF4-independen

103 vel beta-sandwich adhesion domain and unique asparagine-dependent super-helical stalk.

104 aragine depletion, and administration of the asparagine depletion enzyme l-asparaginase is an importa

105 Also, asparagine depletion via the ASNS inhibitor amino sulfox

106 lastic leukemia (ALL) cells are sensitive to asparagine depletion, and administration of the asparagi

107 nts its transcriptional expression following asparagine depletion.

108 esponse to ATF4-independent apoptosis during asparagine depletion.

109 property to allow the required therapeutic l-asparagine depletion.

110 asparaginase activity levels and sufficient asparagine depletion.

111 uently given for months to obtain continuous asparagine depletion.

112 hese studies reveal an axis of adaptation to asparagine deprivation and present a rationale for clini

113 In addition, we previously showed that asparagine deprivation such as that mediated by l-aspara

114 ed that tau cleavage after amino acid 368 by asparagine endopeptidase (AEP) is upregulated in Alzheim

115 delta-secretase, also known as asparagine endopeptidase (AEP) or legumain, is a lysosom

116 Asparagine endopeptidase (AEP), also called legumain, is

117 Delta-secretase, a lysosomal asparagine endopeptidase (AEP), simultaneously cleaves b

118 We mimicked these events in situ by asparagine endopeptidase degradation of alpha-syn fibril

119 are sensitive to proteases (cathepsin B and asparagine endopeptidase) that are over-expressed by res

120 truncations, resulting from CtsB, CtsL, and asparagine endopeptidase.

121 that the substrate 2-ABA itself supplies the asparagine-equivalent amino function that assists in cat

122 ere we report beta5i-selective inhibition by asparagine-ethylenediamine (AsnEDA)-based compounds and

123 We recently reported asparagine ethylenediamines (AsnEDAs) as immunoproteasom

124 an amino acid exchange factor: intracellular asparagine exchanges with extracellular amino acids.

125 ant with His(273) and His(274) exchanged for asparagines exhibits a much less pronounced pH dependenc

126 nella utilizes the Amadori compound fructose-asparagine (F-Asn) as a nutrient through the successive

127 terica fra locus, which encodes the fructose-asparagine (F-Asn) utilization pathway, are highly atten

128 Salmonella-specific nutrient source fructose-asparagine (F-Asn), to the probiotic bacterium Escherich

129 racteristic arginine finger and an auxiliary asparagine for catalysis.

130 te lymphoblastic leukemia, acts by depleting asparagine from the blood.

131 Asparagine further proved crucial in glutamine-deprived

132 C and are tolerant of spectating tryptophan, asparagine, glutamine, and threonine residues.

133 mination of six amino acids namely (alanine, asparagine, glutamine, proline, serine and valine) for S

134 One of the mutations, N1000G (asparagine glycine at residue 1000), significantly impro

135 While both antibodies share a canonical asparagine-glycine (NG) motif in a structural loop, this

136 formation of acrylamide directly from sugar-asparagine glycoconjugates.

137 ed with AGY codons (denoted S"), glycine and asparagine, (GS"N).

138 rge primarily from a single AA substitution (asparagine-->threonine) via a single nucleotide mutation

139 h the same pUS9 arginine residues mutated to asparagine (HSV-1pUS9KBDM) and then restored them being

140 droxylase domain (PHD) enzymes and HIF-alpha asparagine hydroxylase factor inhibiting HIF (FIH).

141 The asparagine hydroxylase, factor inhibiting HIF (FIH), con

142 Here, we identify asparagine hydroxylation as a novel posttranslational mo

143 Studies investigating whether asparagine hydroxylation is a general regulatory oxygen-

144 trates, can be modulated by oxygen-dependent asparagine hydroxylation, suggesting that Cezanne is reg

145 inhibiting HIF1 (FIH1)- and oxygen-dependent asparagine hydroxylation.

146 glutamine-derived nitrogen and aspartate to asparagine) impaired EC sprouting even in the presence o

147 site that, together with the presence of an asparagine, imply the use of the distal arginine as a ca

148 There was much greater free asparagine in potatoes grown at the Doncaster site compa

149 s (OMWP) were added to glucose and lysine or asparagine in silica model systems to mimic water activi

150 Our structures reveal bound asparagine in the active site that has unambiguously not

151 thionine residue and deamidation of the same asparagine in the corresponding acidic fractions generat

152 Substitution of the aspartate or the asparagine in the DxN motif abolished the activity of Lt

153 vates Rho GTPases by deamidating a conserved asparagine in the GTPase switch-I region.

154 d hydrogen-bonding network to reposition the asparagine in the McrB signature motif for optimal catal

155 A conserved asparagine in the oxyanion hole, Asn-169, is found to be

156 Glycosylation of this asparagine in zebra finch CBG does not influence its ste

157 Furthermore, we identified two conserved asparagines in MTN1 and MTN2 from Arabidopsis that confe

158 In summary, glycosylated asparagines in the palm and knuckle domains of alphaENaC

159 red by replacing four of five glutamines for asparagines in various combinations via site-directed mu

160 or more conservative residues, glutamine or asparagine, in the GERAMT-binding site.

161 coma cells combined with depletion of plasma asparagine inhibited tumor growth in vivo.

162 -asparaginases to catalyze the hydrolysis of asparagine into aspartic acid and ammonia has been recen

163 Collectively, our results indicate that asparagine is an important regulator of cancer cell amin

164 sm and nucleotide synthesis, suggesting that asparagine is involved in coordinating protein and nucle

165 However, the auxiliary asparagine is missing in the RhoGAP domain of Myo9b (Myo

166 Asparagine is present in plums at relatively high concen

167 tes 96 and 107 are conservatively mutated to asparagine is strongly impaired in dimer formation but m

168 ginase, an antileukemic enzyme that depletes asparagine, is a common clinical problem.

169 examine the metacluster formation of serine, asparagine, isoleucine, and tryptophan.

170 Several carbon sources (L-Asparagine, L-Aspartic Acid, L- Glutamic Acid, m- Erythr

171 mmediately adsorbed by the char, including l-asparagine, l-glutamine, and l-arginine.

172 ed by a group of amino acids that includes l-asparagine, l-glutamine, l-threonine, l-arginine, l-glyc

173 r levels of L-acetylcarnitine, creatinine, L-asparagine, L-glutamine, linoleic acid, pyruvic acid, pa

174 ric parallel beta-helix containing a 10-step asparagine ladder characteristic of alginate-converting

175 The continuous hydrogen-bond network (asparagine ladder) formed among the asparagine residues

176 a single-domain architecture with an intact asparagine ladder, a three-domain architecture with the

177 s of NL docking, cover-neck bundle (CNB) and asparagine latch (N-latch) formation, during force gener

178 from increased branched chain amino acid and asparagine levels and altered expression of key enzymes

179 y, we show that maintenance of intracellular asparagine levels is critical for cancer cell growth.

180 e concentrations, we show that intracellular asparagine levels regulate uptake of amino acids, especi

181 idase) that is significantly associated with asparagine levels, with an effect that is independent of

182 Asparagine limitation in melanoma and pancreatic cancer

183 es the transfer of fucose from GDP-fucose to asparagine-linked GlcNAc of the N-glycan core in the med

184 of post-translational modification, notably asparagine-linked glycans.

185 2 most prevalent types of type I CDG, ALG6 (asparagine-linked glycosylation protein 6)-deficiency CD

186 GlcNAc(2)Fuc(0-1) were confirmed as the main asparagine-linked oligosaccharides on the surface of TBE

187 Asparagine-linked protein N-glycosylation, the most comp

188 ee domains of life in terms of structure and asparagine location in the sequon of the protein.

189 N61 on binding Loop D, suggesting these two asparagines may interact.

190 novel link between endothelial glutamine and asparagine metabolism in vessel sprouting.

191 serine, tryptophan, phenylalanine, tyrosine, asparagine, methionine, and lysine.

192 namine Zn(II)(NA) and manganese complex with asparagine Mn(II)(Asp)(2).

193 he mutant containing an aspartic acid (D) to asparagine (N) substitution at position 405 (D405N) muta

194 A missense mutation, replacing asparagine (N) with lysine (K), at position 46 in the Gl

195 In the secretory pathway, misfolded asparagine (N)-linked glycoproteins are selectively sort

196 The asparagine (N)-linked Man9GlcNAc2 is required for glycop

197 Previously, antibodies specific to a gluco-asparagine (N-Glc) glycopeptide, CSF114(N-Glc), were ide

198 icate that the composition of the five CD16a asparagine(N)-linked carbohydrates (glycans) impacts aff

199 The composition of asparagine(N)297-linked glycans can modulate the binding

200 We found that a highly conserved asparagine (N220) in the first transmembrane segment is

201 en bond between a glutamic acid (E90) and an asparagine (N258) residue suffices to keep the gate of C

202 ynonymous amino acid substitution, replacing asparagine (N40) with aspartate (D40), and has been link

203 on following ETC inhibition neither depletes asparagine nor activates the ISR, reflecting an altered

204 The substitution of Ser-254 with either an asparagine or a glutamine increases the l-asparagine spe

205 n conversion involved the gain or loss of an asparagine or glutamine residue.

206 Ts carry a nonprotonatable amide amino acid, asparagine or glutamine, respectively, at the central hi

207 The presence of asparagine or threonine in over 99% of all human seasona

208 lycocalyx, replacement of these glycosylated asparagines, or removal of N-glycans.

209 midotransferase that converts aspartate into asparagine, produced the strongest inhibitory effect on

210 any prion-forming proteins contain glutamine/asparagine (Q/N) rich domains, and there are conflicting

211 Most yeast prion proteins contain glutamine/asparagine (Q/N)-rich prion domains that drive prion act

212 Asparagine quickly decreased; contrary, the concentratio

213 Asparagine reacted with fructose to form a Schiff base b

214 Through its exchange factor role, asparagine regulates mTORC1 activity and protein synthes

215 In addition, we show that asparagine regulation of serine uptake influences serine

216 to develop a mechanistic model, based on the asparagine-related pathway, for acrylamide and HMF forma

217 absence of hydrogen bonding with a conserved asparagine residue at position 132.

218 n 1 reveals that a glycan emanating from the asparagine residue at position 25 (Asn-25) is located wi

219 T2 N722S mutation) which targets a conserved asparagine residue in the second PDZ domain of Mint2 tha

220 echanism, involving the outward motion of an asparagine residue in transmembrane helix 3, might be a

221 ent of Ca(2+) Remarkably, replacing a single asparagine residue in ZnT10 (Asp-43) with threonine (ZnT

222 vated EcPlt toxin modifies a proximal lysine/asparagine residue instead.

223 A conserved CTE asparagine residue is required for ubiquitylation and de

224 r study on one peptide (PepB2) pinpointed an asparagine residue necessary for CPP activity.

225 id-linked oligosaccharide (LLO) donor to the asparagine residue of a nascent polypeptide chain is cat

226 elop a bidentate interaction with a critical asparagine residue resulted in the incorporation of a py

227 asaccharide with a beta-glucose linked to an asparagine residue which is not located in the typical s

228 y introducing multiple negatively charged or asparagine residues at the edges of CDR3, whereas other

229 Peptides with deamidation sites at asparagine residues but lacking a typical asparagine-X-s

230 We show that the serine and asparagine residues in the G5 motif likely play a role i

231 Mass spectrometry analysis identified two asparagine residues in the helicase 2i domain of RIG-I t

232 attachment of an oligosaccharide to selected asparagine residues in the sequence N-X-S/T (X not equal

233 network (asparagine ladder) formed among the asparagine residues on the concave surfaces of neighbori

234 tein by converting particular N-glycosylated asparagine residues to aspartic acid.

235 typically involved mutating the glycosylated asparagine residues to structurally similar glutamines o

236 modifications, including N-glycosylation of asparagine residues.

237 tact when Asp506 and Asp571 are mutated into asparagine residues.

238 Two glycosylated asparagines, respectively their N-glycans localized in t

239 anslational reprogramming in the survival of asparagine-restricted cancer cells.

240 ation of MAPK inhibitors in combination with asparagine restriction approaches.

241 nsitizing melanoma and pancreatic tumours to asparagine restriction, reflected in inhibition of their

242 e clustered lysine residues with alanines or asparagines results in recombinant PrP amyloid fibrils w

243 que in size and composition: glutamine free, asparagine rich, and the smallest defined to date.

244 An N-terminal glutamine/asparagine-rich nucleation domain is required for nuclea

245 Although prion activity of glutamine/asparagine-rich proteins is predominantly determined by

246 the leaves for aspartate or in the roots for asparagine, serine and glycine.

247 to interference by the amide function of the asparagine side chain with Na(+)-coordinating residues i

248 s coupled to the nitrogen atom (N-linked) of asparagine side chains or to the oxygen atom (O-linked)

249 Significantly, two highly conserved asparagine side chains, each one located between two try

250 Like glutamine, asparagine signals to mTORC1 through Arf1 in the absence

251 While ECs can take up asparagine, silencing asparagine synthetase (ASNS, which

252 berrant glycan modification on this specific asparagine site of E-cadherin was demonstrated to affect

253 ctin isoform is N-glycosylated at a specific asparagine site that is required for interactions with s

254 an asparagine or a glutamine increases the l-asparagine specificity but only when combined with the E

255 into the molecular basis for the increased l-asparagine specificity.

256 aluation of immunosensors fabricated using L-Asparagine stabilized gold nanoparticles and citrate sta

257 d gold nanoparticles and (3) directly onto L-Asparagine stabilized gold nanoparticles modified electr

258 Here we show that a single serine-to-asparagine substitution [Ser(139)-->Asn(139) (S139N)] in

259 Here we demonstrate that the asparagine substitution of the aspartate associated with

260 combination of TCA cycle replenishment plus asparagine supplementation restored the metabolic aberra

261 be fully rescued from glutamine depletion by asparagine supplementation.

262 interestingly, the preference is because the asparagine supply for efficient viral protein synthesis

263 Furthermore, we show that sufficient asparagine supply is required for efficient VACV protein

264 Our study highlights that the asparagine supply, the regulation of which has been evol

265 as genetic and chemical manipulation of the asparagine supply, we provide evidence demonstrating tha

266 , showing enhanced energy production to fuel asparagine synthesis and amino acid uptake.

267 anistically, glutamine provided nitrogen for asparagine synthesis to sustain cellular homeostasis.

268 Asparagine synthetase (ASNS) converts aspartate and glut

269 er Genomic Atlas, and demonstrated that high asparagine synthetase (ASNS) expression correlated with

270 Silencing of asparagine synthetase (ASNS), an amidotransferase that c

271 ion of ATF4 and the expression of its target asparagine synthetase (ASNS), sensitizing melanoma and p

272 he amino acid response induces expression of asparagine synthetase (ASNS), which provides for asparag

273 While ECs can take up asparagine, silencing asparagine synthetase (ASNS, which converts glutamine-de

274 NS gene have been clinically associated with asparagine synthetase deficiency (ASD).

275 Through asparagine synthetase knockdown and altering of media as

276 In WMS glucose/asparagine systems, examination of three different conce

277 ubstrate binding and catalysis, including an asparagine that is proposed to activate the GSH cofactor

278 tic acid in position 163 was substituted for asparagine (the most common in prion susceptible species

279 otein replenishment ranged from 0.73 g/d for asparagine to 3.61 g/d for proline.

280 cacy of L-asparaginases is micromolar KM for asparagine to allow for complete depletion of this amino

281 ic properties and analyzed the conversion of asparagine to aspartate using NMR.

282 The use of asparaginase to convert asparagine to aspartic acid may provide a means to reduc

283 acid route, i.e., reducing sugars react with asparagine to form the Schiff base before decarboxylatio

284 of CD28-based CAR-T cells and changing this asparagine to phenylalanine (CD28-YMFM) promoted durable

285 f the relationship between the ratio of free asparagine to reducing sugars and the levels of acrylami

286 synthesis of aminoacylated glutamine and/or asparagine tRNAs, involving the glutamine amidotransfera

287 ne, methionine, cycloleucine, aspartic acid, asparagine, tyrosine, and glutamic acid).

288 modulation of key metabolites like proline, asparagine, valine and several flavonoids.

289 Asparagine was < 0.5 muM in 96% and 67% of the PEGaspara

290 r residues revealed that the amide moiety of asparagine was crucial for GlyR activation.

291 Erwinia asparaginase 3 times per week, and l-asparagine was measured to monitor asparaginase efficacy

292 ls and animal fat using a constant amount of asparagine was measured.

293 0.95 and 0.94 respectively (p<0.05), whereas asparagine, was poorly correlated (p>0.05).

294 In dry glyoxal/asparagine waxy maize starch (WMS) systems, 9 out of 10

295 olar ratios of total fructose and glucose to asparagine were investigated.

296 ) increasing and most amino acids (including asparagine which is the precursor of acrylamide formed d

297 vidence demonstrating that the production of asparagine, which exclusively requires glutamine for bio

298 Heating asparagine with various cooking oils and animal fat at 1

299 DNA sequences encoding N-glycosylation sites asparagine-X-serine/threonine (N-gly sites) within the V

300 at asparagine residues but lacking a typical asparagine-X-serine/threonine sequons (N-X-S/T, X is any