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1 ed with dyspnea, voice changes, and imminent asphyxiation.
2 g a reticular network that leads to death by asphyxiation.
3 ce are rendered highly susceptible to lethal asphyxiation.
4 inflammatory cells and prevention of lethal asphyxiation.
5 muscular transmission, resulting in death by asphyxiation.
6 even the upper respiratory tract leading to asphyxiation.
7 lated stereotyped escape caused by perceived asphyxiation.
8 ases from donors who died following ligature asphyxiation.
9 ases from donors who died following ligature asphyxiation.
10 from DCD donors who died following ligature asphyxiation.
11 sion, causing flaccid paralysis and death by asphyxiation.
12 nto contiguous alveoli and leads to death by asphyxiation.
13 ent that even mild anesthesia often leads to asphyxiation [4], cognitive cardiovascular control has n
14 ng (AAPC, 2.7; 95% CI, 1.0-4.4), hanging and asphyxiation (AAPC, 2.4; 95% CI, 0.2-4.6), and other mea
16 y self-poisoning alone, attempted hanging or asphyxiation (adjusted OR 2.70 [1.53-4.78], p=0.001) and
17 neys from donors who died following ligature asphyxiation and those who received organs from donors d
18 dicates that larval death is attributable to asphyxiation brought on by fluid-congested tracheal tube
19 inases (MMPs) is necessary to prevent lethal asphyxiation, but mechanistic insight into this essentia
26 carring in the lung that ultimately leads to asphyxiation; however, the cascade of events that promot
33 on of inspired oxygen, mechanism of death by asphyxiation or drowning, history of cigarette use (>=20
36 eases from donors who die following ligature asphyxiation suffer an additional warm ischemic insult,
40 eview of research on stress responses during asphyxiation, we estimate 10 (1.9-21.7) min of moderate