戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 e molecular determinants of human congenital asplenia.
2 ut PS-exposed autophagic vesicles because of asplenia.
3 Q) in a child with both severe 46,XY-DSD and asplenia.
4 7.1 years and 64% had functional or anatomic asplenia.
5 ciency causes organ growth defects including asplenia.
6 eptibility to infection caused by functional asplenia.
7 bone marrow failure, and isolated congenital asplenia.
8 t underlie spleen development and congenital asplenia, a condition linked to increased risk of overwh
9 ents with severe sepsis or septic shock with asplenia and 52 without asplenia were included.
10 s, we searched for patients with and without asplenia and community-acquired severe sepsis/septic sho
11 ctive lateralization including dextrocardia, asplenia and intestinal malrotation, suggesting that BCO
12                               A patient with asplenia and multiple red blood cell transfusions acquir
13 ng Diamond-Blackfan anemia (DBA), congenital asplenia and T cell leukemia.
14 or C3 deficiencies, 1 of isolated congenital asplenia, and 2 of Bruton disease (X-linked agammaglobul
15 pressive therapy, immunoglobulin deficiency, asplenia, and/or other rare conditions.
16 ion of hyposplenism and proper management of asplenia are warranted to prevent overwhelming post-sple
17 ression, our study further demonstrated that asplenia, fever, and reduced O(2) saturation, along with
18                          Although functional asplenia from infarctions may be a major contributor to
19 tion including moderately immunocompromised (asplenia, hematologic malignancies, solid organ malignan
20 nocompromise included AIDS, aplastic anemia, asplenia, hematological cancer, chemotherapy, neutropeni
21  with immunocompromising conditions (IC) and asplenia/hyposplenia (AH).
22                          Isolated congenital asplenia (ICA) is characterized by the absence of a sple
23 esis and the etiology of isolated congenital asplenia (ICA), a life-threatening human condition, are
24                                              Asplenia imparts susceptibility to life-threatening seps
25 spleen development, and Tlx1 deletion causes asplenia in mice.
26 n proposed in the pathogenesis of congenital asplenia in patients carrying mutations of the gene-enco
27                                              Asplenia is predominantly due to splenectomy for either
28 posplenism in older mice, recapitulating the asplenia of an HO-1-deficient patient.
29                                       Hence, asplenia or hyposplenism increases susceptibility to sev
30                                   Functional asplenia or hyposplenism may predispose patients to spon
31 % of OPSI patients vs 5% of patients without asplenia (P = .038).
32 quently in OPSI patients (42% vs 12% without asplenia; P < .001) and more frequently manifested as bl
33 ow overall (42% vs 8% among patients without asplenia; P < .001).
34 iary and organ laterality defects as well as asplenia, paralleling symptoms of the human condition ri
35 g chronic mucocutaneous candidiasis, AI, and asplenia, respectively, in 49 of 121 (40%) and 10 of 121
36 rees C), reduced oxygen saturation (<95) and asplenia significantly differed when compared to those o
37 itulating the clinical symptoms of the human asplenia syndrome.
38                                              Asplenia (the congenital or acquired absence of the sple
39                                              Asplenia was present in 76 patients (54%).
40         On multivariable Poisson regression, asplenia was the only predictive variable independently
41 or septic shock with asplenia and 52 without asplenia were included.
42 iary cholangitis, hyposplenism or functional asplenia with severe bacterial infection, type 1 diabete
43 by a perinatal lethal skeletal dysplasia and asplenia, with severe malformation or absence of specifi