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1 s no effect on the fall in FEV1 during early asthmatic response.
2 13 are thought to be central to the allergic asthmatic response.
3 a key role in the activation of the allergic asthmatic response.
4 ory T cells into the BAL during the allergic asthmatic response.
5 cumulative component of the allergen-induced asthmatic response.
6 diator that is released in airways during an asthmatic response.
7  returned to baseline levels during the late asthmatic response.
8 e mechanism by which PGE(2) blocks the early asthmatic response.
9 nt effect on the allergen-induced dual-phase asthmatic response.
10 gnificantly reduce either the early- or late-asthmatic response.
11  after allergen challenge for early and late asthmatic responses.
12 sceptibility of children to allergen-induced asthmatic responses.
13  measures of allergen-induced early and late asthmatic responses.
14  clinically attractive therapy for allergic, asthmatic responses.
15 ibutes to the development of severe allergic asthmatic responses.
16 nt for diminishing allergic and IgE-mediated asthmatic responses.
17 significantly attenuated both late and early asthmatic responses after allergen provocation in patien
18 lia and who also had biphasic early and late asthmatic responses after laboratory-based allergen prov
19 he role of eosinophils in mediating the late asthmatic response and causing airway hyper-responsivene
20  to increase during the immediate Ag-induced asthmatic response and continued to increase over 2-fold
21 s study introduce a novel participant in the asthmatic response and indicate that factor Xa functions
22 erged as a major contributor to allergic and asthmatic responses, and as such it represents an attrac
23 lammation in the lungs, inhibit allergic and asthmatic responses, and contribute to tolerance to inha
24                                     Enhanced asthmatic response as a result of ovalbumin sensitizatio
25           The primary end point was the late asthmatic response as quantified by the change in the ar
26           The primary end point was the late asthmatic response, as measured 3 to 7 hours after the a
27  whether selective inhibition of COX-2 makes asthmatic responses better or worse.
28  attenuated the fall in FEV1 during the late asthmatic response (both doses led to an 8.7% fall) vers
29  Inhaled prostaglandin (PG) E2 might inhibit asthmatic responses, but the mechanisms involved remain
30 t rich in IL-4, these CD8(+) T cells mediate asthmatic responses, but the mechanisms regulating the c
31 fter 28 days, SB010 attenuated the mean late asthmatic response by 34%, as compared with the baseline
32                       Inhibition of the late asthmatic response by SB010 was associated with attenuat
33      We hypothesized that a pre-existing Th2/asthmatic response can promote Th2 responses to newly en
34                              Mice with a low asthmatic response colonized with microbiota from Pglyrp
35 tically significant attenuation of the early asthmatic response (EAR) by GSK2190915; treatment differ
36 y of cat-allergic mild asthmatics with early asthmatic response (EAR) during cat allergen exposure in
37 ist (LABA) vilanterol (VI) on early and late asthmatic responses (EAR/LAR) and airway hyper-responsiv
38 ies in preventing cat allergen-induced early asthmatic responses (EARs) in cat-allergic patients with
39                 The enhanced severity of the asthmatic response from disruption of the Nrf2 pathway w
40 ponse, whereas nitrosylation during the late asthmatic response generates SNO, safe reservoirs for re
41                                         Late asthmatic response (>=15% reduction in FEV1 within 24 h
42 specific chemokine receptors during allergic asthmatic responses has been relatively undefined.
43 combinant PAF-AH (rPAF-AH) on the dual phase asthmatic response in atopic subjects with mild asthma.
44 used to determine effects of factor X on the asthmatic response in mice.
45 and placebo, for 7 days, on allergen-induced asthmatic responses in 18 non-smoking and 17 smoking ato
46 ach allergen (CRA), an allergen that induces asthmatic responses in both humans and mice, to further
47           Lipoxin A4 (LXA4) signaling blocks asthmatic responses in human and experimental model syst
48                                     Allergic asthmatic responses in the airway are associated with ai
49 e feces and oropharynx correlated with lower asthmatic responses in the lungs.
50                                  In allergic asthmatic responses increased lung homing of HPCs may be
51 btained at 4 h (n = 7), the time of the late asthmatic response (LAR) (n = 5), or 24 h (n = 4) after
52            Primary efficacy outcome was late asthmatic response (LAR) at Week 13, defined as area und
53 stically significant attenuation of the late asthmatic response (LAR) by GSK2190915; the treatment di
54                                     The late asthmatic response (LAR) was suppressed by FP in smokers
55           To identify predictors of the late asthmatic response (LAR), we reviewed data from 60 asthm
56 rgen would seem to dictate the nature of the asthmatic response, little is known regarding how tolera
57 t of monoclonal antibody to IL-5 on the late asthmatic response or on airway hyper-responsiveness to
58  results show that Pglyrp1 enhances allergic asthmatic responses primarily through its effect on the
59  dose of allergen needed to provoke an early asthmatic response, reduced the mean maximal fall in FEV
60 malayi can result in development of a severe asthmatic response termed tropical pulmonary eosinophili
61 demonstrated a positive skin test and a dual asthmatic response to allergen inhalation challenge.
62   Th2 and mast cells are participants in the asthmatic response to allergens, and both cell types pro
63    Clinical experiments demonstrate that the asthmatic response to an aeroallergen can be enhanced by
64                                     However, asthmatic response to corticosteroids also varies widely
65 HR1 have pharmacogenetic effects influencing asthmatic response to corticosteroids, provide a rationa
66 ediatric asthma morbidity and may modify the asthmatic response to indoor PM.
67 clinical and significant effect on the early asthmatic response to mite, cat, and birch and grass pol
68 treatment greatly reduces the severity of an asthmatic response to several types of bronchoconstricto
69 is sufficient to mediate key features of the asthmatic responses to IL-13 in murine models.
70 tenuated the early (0-2 h) and late (4-10 h) asthmatic responses to inhaled allergen compared with pl
71                                              Asthmatic responses were found to involve sequential ind
72 reactive oxygen species during the immediate asthmatic response, whereas nitrosylation during the lat
73                                    The early asthmatic response with SB010 was attenuated by 11% as m
74 heep effectively antagonized antigen-induced asthmatic responses, with 70-75% blockade of the early r