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1 ool-age follow-up (57.8% of 10 563 recruited at birth).
2 date of next pregnancy minus gestational age at birth).
3 typically associated with their sex assigned at birth).
4 istered to infants weighing less than 1000 g at birth.
5  0.9 is associated with asymptomatic disease at birth.
6 ay or may not correspond to the sex assigned at birth.
7  transient early empiric antibiotic exposure at birth.
8 tween their gender identity and sex assigned at birth.
9 tainably improve the quality of data on care at birth.
10 e for these robust levels of circulating AVP at birth.
11 t provide an accurate assessment of ASD risk at birth.
12 nal epithelia are exposed to glucocorticoids at birth.
13 oncentrations of BPA, BPF, and BPS with size at birth.
14 yocytes leads to a metabolic switch as found at birth.
15 es necessary for adaptation to air breathing at birth.
16 op complexity with advancing gestational age at birth.
17 der identity differs from their sex recorded at birth.
18 ssociated with the SMN1 gene and manifesting at birth.
19 l angiogenesis, but they lose the diaphragms at birth.
20 d the lung size phenotype, allowing survival at birth.
21 le intravenous coadministration of CH31 bnAb at birth.
22  and small or large size for gestational age at birth.
23 massive secretory diarrhea that often begins at birth.
24 ely prevented the neuropathy in mice treated at birth.
25 phic representations of the body are present at birth.
26 nt genetic determinant of T cell lymphopenia at birth.
27 ence in high-income countries and prevalence at birth.
28 ferences between anthropometric measurements at birth.
29 a WBKC, were performed at each trimester and at birth.
30 birth and division, regardless of their size at birth.
31  regulatory T-cell populations were measured at birth.
32 n was strongly associated with delivery mode at birth.
33 f nephron number as well as renal hypoplasia at birth.
34 st year of life and sociodemographic factors at birth.
35 appears strongly developmentally constrained at birth.
36 zed in face processing is already functional at birth.
37 re the second decade of life, perhaps mainly at birth.
38 ciently leverage innate immune cells present at birth.
39  the differences in miRNA expression present at birth.
40 to injury during transition to air breathing at birth.
41 tal synchondrosis was completely mineralized at birth.
42 infection by polymerase chain reaction (PCR) at birth.
43 typically associated with their sex assigned at birth.
44 , and (iv) gender minorities assigned female at birth.
45 ted with a low risk for respiratory symptoms at birth.
46           Cord serum cytokines were measured at birth.
47 cy zones exhibit coordinated neural activity at birth.
48 unction remains unknown as Dcc knockouts die at birth.
49 rity of infants with cCMV will have symptoms at birth.
50 , acquiring the infection from their mothers at birth.
51 der identity is different from that recorded at birth.
52 t clear how they adjust to support breathing at birth.
53 diac myocyte and coronary vascular endowment at birth.
54 gM anti-PC, whereas IgM anti-MDA was present at birth.
55 esponding decrease in mechanical ventilation at birth (0.89 [0.81, 0.97]) and following admission (0.
56 n gender, birth year, and number of siblings at birth 1:10 with children born to nontransplanted wome
57 term infants born in Italy who were measured at birth, 1 and 2 wk, and 1, 2, 3, 4, 5, and 6 mo of age
58                       Infants were evaluated at birth, 1 and 3 months of age in terms of anthropometr
59  born in the United States who were measured at birth; 1029 term infants born in Ireland who were mea
60 samples, infants exposed to ACT weighed less at birth: -220.18 g (+/-SE 21.43, P < .001), -140.68 g (
61  +/- 0.3 of the 17 innate interferons tested at birth, 24% showed no T1/3IFN production.
62  patients randomized (median gestational age at birth, 26.3 [interquartile range {IQR}, 24.9-27.6] we
63  for assessment according to gestational age at birth (30 + 1 to 41 + 6 wk) and postnatal age at meas
64 a cohort of n = 64 preterm infants (mean age at birth = 32.0 weeks), we tested whether cortical alter
65 in n = 292 healthy newborn infants (mean age at birth = 39.9 weeks) with regional patterns of gene ex
66 eight, length/height, and head circumference at birth, 5 mo, 12 mo, and 5 y were included in a path m
67 er cohort of healthy adults who received BCG at birth, 53% of mycobacteria-reactive-activated CD8 T c
68      Glyburide-exposed neonates were heavier at birth (58.20 g, 95% confidence interval [CI] 10.10-10
69                          Arm A received bOPV at birth, 6 and 10 weeks, bOPV+IPV at week 14 and IPV at
70 cenarios explored the effect of BCG delivery at birth, 6 weeks, 6 months, or 9-12 months, on tubercul
71 was 42 years (IQR 34-50); 27% (n=280) female at birth; 73% (n=763) white race.
72  as high as children who were "nondeficient" at birth (9.3 vs. 4.7; P = 0.002).
73                                              At birth, a higher proportion of activated regulatory T
74 rnal condition is associated with sex ratios at birth, a result inconsistent with the Trivers-Willard
75 lations of baseline characteristics, acuity (at birth, across hospitalization), major morbidities, an
76 ol group were very large for gestational age at birth (adjusted relative risk, 1.30; 95% CI, 1.02 to
77 is in the last 12 months were city residence at birth [adjusted odds ratio (95% confidence interval)
78 enitor driven phase (embryonic day 14.5) and at birth-after neurons from all six cortical layers are
79              In 2 infants with confirmed HIV at birth, all tests were negative at week 6, possibly be
80 deletion of all LAR-RPTPs in the hippocampus at birth also did not alter synaptic connectivity as mea
81 ave previously reported that BCG vaccination at birth alters in vitro cytokine responses to heterolog
82 vg/kg birth weight), resulting in ~3.0% hFIX at birth and 0.6-6.8% over 19-51 mo.
83                            A 100-g higher FM at birth and 100-g/month higher FM accretion from 0 to 3
84  and during pregnancy and 1) gestational age at birth and 2) gestational age- and sex-specific birth
85  ( n = 3; 3.57 x 10(13) vg/kg) expressed <1% at birth and 9.4-27.9% up to 42 mo.
86 arts of South America, often become infected at birth and account for up to 95% of newly reported chr
87 curves are lacking that allow for assessment at birth and across the first 6 mo of life.
88 ers at ~16 week gestation and their children at birth and ages 3, 8, and 12 years.
89  concentrations of PFOS and PFOA in children at birth and at 3, 6, 12, and 24 months of age.
90 s at obstetric health facilities in Tanzania at birth and at postpartum weeks 1, 2, 3, and 6 in HIV-e
91 igate associations of air pollution exposure at birth and at the time of later biosampling with IgE s
92 d on broad measures, such as life expectancy at birth and child and infant mortality, and have shown
93 e infections in participants assigned female at birth and cis-heterosexual males.
94 protocol basis, adjusted for gestational age at birth and country, with multiple imputation used to a
95                 DNAmAge and age acceleration at birth and early childhood were not associated with mi
96 rmal positioning of retinal progenitor cells at birth and ectopic presence of photoreceptors and seco
97 s who were at least 34 weeks gestational age at birth and enrolled within 48 h of birth, born to wome
98                                   Higher FFM at birth and FFM accretion in infancy were associated wi
99                                    Higher FM at birth and FM accretion from 0 to 3 months were associ
100 rons of both regions have similar properties at birth and follow the same developmental profile, with
101               PROBIT enrolled 17 046 infants at birth and followed them up at 6.5 years (n = 13 889).
102 ls and spiral ganglion neurons, which begins at birth and follows a base to apex developmental gradie
103 h care, higher IQ was seen with greater size at birth and greater weight gain in infancy.
104 the effects of low supply on life expectancy at birth and high mortality across ages, even as recentl
105           Universal hepatitis B immunisation at birth and in infancy is the key strategy for global e
106 oss the entire width of the muscle cell both at birth and in mature muscle.
107  EC gene expression data from boy-girl twins at birth and in non-twin adults to detect sex difference
108  associated with changes in placental weight at birth and in placental-to-birth weight ratio (PFR).
109 associated with shorter blood cell telomeres at birth and individuals with type 2 diabetes have short
110  being in the highest socioeconomic quintile at birth and infections (OR, 1.39; 95% CI, 1.09-1.79) in
111 A; mechanisms by which an altered microbiota at birth and later on in childhood may influence disease
112  identified sex differences that are present at birth and maintained throughout life, and those that
113 n born to women with syphilis were recruited at birth and matched with 333 syphilis-uninfected childr
114 al age, allowing term infants to be assessed at birth and preterm infants to be monitored until they
115 2 ASD, 475 neurotypical), who were recruited at birth and prospectively followed at the Boston Medica
116  but quickly drops below prepregnancy levels at birth and remains suppressed during the postpartum pe
117 cell division as a discrete map between size at birth and size at division with stochastic fluctuatio
118 ed endocardial fibroelastosis, 1 degrees CHB at birth and skin rash.
119 d tracking we show that persisters are small at birth and slowly replicating.
120 ysed the association between life expectancy at birth and socioeconomic status at the subcity-unit le
121 exposed mothers have increased heart weights at birth and susceptibility to hypertension during adult
122 describe the network of cord serum cytokines at birth and test whether they are associated with subse
123 ed the inverse correlation between cell size at birth and the duration of the G(1) phase.
124 r, the mitochondrial reticulum is not formed at birth and the mechanisms driving network development
125 ess may contribute to increased heart weight at birth and the programming of susceptibility to hypert
126 association between cord serum interleukin-7 at birth and the trajectories of children's anxiety-depr
127             We aimed to study how FM and FFM at birth and their accretion during infancy were associa
128       We analysed associations of FM and FFM at birth and their accretion in infancy with height, wai
129 children, we estimated individual FM and FFM at birth and their accretion over 0-3 and 3-6 months of
130 seases (IBD) in persons with critical events at birth and within 1 year of age.
131 eventy-five (21.8%) had respiratory symptoms at birth, and 34 (10.0%) required neonatal lung resectio
132 e as a 1 - ratio of prevalence to prevalence at birth, and expected number of patients worldwide base
133 ticipants (aged >18 years, assigned male sex at birth, and identifying as a gender different from mal
134 fine predictors of fetal infection, symptoms at birth, and long-term sequelae.
135 fine predictors of fetal infection, symptoms at birth, and long-term sequelae; 31 covariates were tes
136 e, education, marital status, neonate weight at birth, and neonate sex] 1.8, 95% CI 1.3-2.6) than tho
137 d obtained from an adult donor cow, starting at birth, and repeated every other week until six weeks
138          All mammals must suckle and swallow at birth, and subsequently chew and swallow solid foods,
139  in infants that do not display microcephaly at birth, and the full impact of these more subtle neuro
140 t developed food allergy had decreased nTreg at birth, and the labour-associated decrease in nTreg at
141   We report that oxygen tension, which rises at birth, and the von Hippel-Lindau (VHL)-hypoxia-induci
142  colonized vaginally with serotype-Ia or III at birth, and their healthy infants were eligible as mat
143 d in airway Club cells and endothelial cells at birth, and then AT2 cells at one year of age.
144 ce, hindlimb muscles possess fewer myofibers at birth, and those myofibers are reduced in size and ha
145            Controlling for sex, parental age at birth, and, for half siblings, history of major depre
146 rm infants born in Ireland who were measured at birth; and 149 term infants born in the United States
147 oeconomic status, birth weight, maternal age at birth, anisometropia, astigmatism, spherical equivale
148                              Gestational age at birth appeared to be a confounding factor as TBSS-obs
149 ll known that white matter injuries observed at birth are associated with adverse neurodevelopmental
150 emale and higher family socioeconomic status at birth are strong and consistent predictors of lower p
151 s highlight regional cortical microstructure at birth as a potential sensitive biomarker in predictin
152 he male population, prevalence of hemophilia at birth as a proportion of cases to live male births by
153 hers before conception and from the children at birth as well as at 1 and 2 y after birth.
154 is associated with a younger gestational age at birth, Asian ethnicity, and aggressive posterior ROP.
155 deaths and in life years and life expectancy at birth, attributable to changes in PM2.5.
156 that were differentially associated with sex at birth, birth outcomes, and fetal neurodevelopment.
157 d that crAss-like phages are rarely detected at birth but are increasingly prevalent in the infant mi
158 expected Mendelian ratio, are grossly normal at birth but have shorter lifespans than those lacking o
159 Skeletal muscle energy requirements increase at birth but little is known regarding the development o
160  auditory system in many mammals is immature at birth but precisely organized in adults.
161 jority of infants with cCMV are asymptomatic at birth, but 10%-15% will develop hearing loss.
162 ty of infants with cCMVi have normal hearing at birth, but are at risk of developing late-onset SNHL.
163  human neonates is usually devoid of viruses at birth, but quickly becomes colonized, which-in some c
164 inguishable from their wild-type littermates at birth, but they rapidly worsened and died by postnata
165 hed control group, HEU children were smaller at birth, but this difference decreased with time and is
166  GSH levels, we disrupted its synthesis both at birth by breeding a Gclc loxP mouse with a thy1-cre m
167        We calculated average life expectancy at birth by sex and subcity unit with life tables using
168                        Advanced maternal age at birth can have pronounced consequences for offspring
169 rs had significantly reduced islet cell mass at birth, caused by decreased endocrine progenitor produ
170 clear whether rapid or gradual lung aeration at birth causes less lung injury.Objectives: To examine
171 nclusions: Rapidly aerating the preterm lung at birth creates heterogeneous volume states, producing
172 BS colonized 7%, 21%, and 23% of the infants at birth, D21, and D60, respectively, of which 10%, 11%,
173 We conclude that infusion of CD4bs bnAb CH31 at birth does not interfere with de novo antibody respon
174 t that the location of the VWFA is earmarked at birth due to its connectivity with the language netwo
175 posure corresponding to concentrations found at birth (E2-P4 C(0)) and over 7 days old (E2-P4 C(7)).
176   We calculated DNAmAge and age acceleration at birth, early childhood, and midchildhood based on the
177               Cerebral cortical architecture at birth encodes regionally differential dendritic arbor
178 nment from the significant exotropia present at birth, ending with approximately 10 degrees of exotro
179 ncluded sex and maternal education, assessed at birth, family income at 23 years, and smoking status
180 xtent of CIN during prenatal development and at birth, following IVF treatment, is well understood.
181     Placentas and lung tissue were collected at birth for morphometric and Western blot measurements
182                            DBS are collected at birth for the child.
183                     We enrolled 647 newborns at birth from a malaria-holoendemic region of Tanzania.
184        We saw an increase in life expectancy at birth from east to west in Panama City and from north
185 ovember 7, 2012, MLPT infants were recruited at birth from the neonatal unit and postnatal wards of t
186 ealth and Human Development and Saving Lives at Birth Grand Challenges.
187                           MID mice generated at birth had lower muscle and circulating IGF-I, decreas
188  in close proximity to a greener environment at birth has a protective effect on the development of a
189                Previously, CTG repeat length at birth has been correlated to patient age at symptom o
190                              Telomere length at birth has been related to life expectancy.
191 regulatory T-cell (Treg) number and function at birth have been linked with subsequent allergic disea
192 rance is normal, 1-month-old lambs with IUGR at birth have higher rates of hindlimb glucose uptake, w
193 e used GBD 2016 estimates of life expectancy at birth, healthy life expectancy, all-cause and cause-s
194  low birth weight, small for gestational age at birth; height, height-for-age, weight, weight-for-age
195 gth exists in yeast, mice, plants and humans at birth; however, major effect loci underlying such pol
196     Daily administration of kinetin starting at birth improves sensory-motor coordination and prevent
197 dicated a higher degree of immune activation at birth in children who were subsequently diagnosed wit
198                     BCG has been recommended at birth in countries with a high tuberculosis burden fo
199 atial differences in average life expectancy at birth in Latin American cities, with the largest P90-
200 gression was used to examine gestational age at birth in relation to all-cause and cause-specific mor
201 32 years.Methods: Participants were enrolled at birth in the Tucson Children's Respiratory Study and
202 F and BPS was inversely associated with size at birth in this cohort.
203  to development of asthma, and link deficits at birth in tptef/te with HRCT-assessed structural airwa
204                              Life expectancy at birth increased by 5.4 (95% UI 3.7-7.2) years, with h
205 ive ventilation support during stabilisation at birth increased significantly (1.26 [1.15, 1.38]) wit
206                                    Hyperoxia at birth increases the severity of influenza A virus inf
207 e their early exposure to oxygen (hyperoxia) at birth increases the severity of respiratory viral inf
208                                              At birth, infants who remained healthy had significantly
209                      The proportion of nTreg at birth is a major determinant of the proportion presen
210                      Establishing prevalence at birth is a milestone toward assessing years of life l
211 mitted to a PICU, increasing gestational age at birth is associated with a substantial reduction in t
212 o 2.6).Conclusions: Higher exposure to NO(2) at birth is associated with persistently low levels of C
213                              Gestational age at birth is declining, probably because more deliveries
214 ohort, ZIKV RT-PCR positivity in the neonate at birth is strongly associated with microcephaly.
215                 Placenta, normally discarded at birth, is a potentially rich source of DNA methylatio
216  of gestation) receiving respiratory support at birth, it may be reasonable to begin with 21% to 30%
217                 A higher proportion of nTreg at birth, larger birth size and male sex was each associ
218               A single dose of hybrid vector at birth led to life-long restoration of bile compositio
219 rine CD11b-expressing mononuclear phagocytes at birth led to severe acute hyperoxia-induced lung inju
220                                 Maternal age at birth, maternal level of education, household income,
221  were diagnosed with HIV; 10 (66.7%) of them at birth (median HIV-RNA 4570 copies/mL).
222 th (PTB) and small for gestational age (SGA) at birth mediate the association between maternal educat
223                                              At birth, microbes rapidly colonize our epithelial surfa
224 t noninvasive measures of cortical structure at birth mirror areal differences in cortical gene expre
225 events that cause severe respiratory failure at birth, most extremely preterm infants now survive, bu
226 l compartment was measured by flow cytometry at birth (n = 463), 6 (n = 600) and 12 (n = 675) months.
227                      Offspring were examined at birth (n = 613) and at 3 months (n = 602) and 18 mont
228 ome, including hydrops, respiratory distress at birth, need for supplemental oxygen, neonatal ventila
229                                              At birth, neonatal triceps and subscapular skinfold thic
230  number of past pregnancies of young mothers at birth of the index child.
231 hese issues, we herein leverage the presence at birth of two types of locomotor-like movements, spont
232 f CD4 binding site (CD4bs) bnAb administered at birth on de novo antibody responses elicited by concu
233 ine the effect of gradual and rapid aeration at birth on: 1) the spatiotemporal volume conditions of
234 asurements and Main Results: NO(2) exposures at birth or age 6 were available for 777 children with o
235 NO(2) exposure at participants' home address at birth or age 6 with CC16 levels from age 6 to 32.Meas
236  than those delivering by ERCS to breastfeed at birth or hospital discharge (63.6%, n = 14,906 versus
237 increased adverse maternal or fetal outcomes at birth or in the first year of life.
238                          After Igf1 deletion at birth or in young adult mice, evaluations of muscle p
239  fetal congenital anomalies, either apparent at birth or later in life.
240 ion, resulted in lethal respiratory distress at birth or shortly after.
241  antibodies were related to PM(2.5) exposure at birth (OR = 3.33 [95% CI = 1.40-7.94] and OR = 4.98 [
242 ace-specific cortical circuit already active at birth, or does its specialization develop slowly as a
243 d a significantly smaller head circumference at birth (P = 0.004), with a distinctive pattern of catc
244 he ninth and first decile of life expectancy at birth (P90-P10 gap) across subcity units in cities.
245                                   Prevalence at birth (per 100 000 males) is 24.6 cases for all sever
246 ctive gestational age (GA) was defined as GA at birth plus postnatal age at the time of sample collec
247                Adjusting for gestational age at birth, postmenstrual age at scan, maternal age, socio
248                                              At birth (postnatal day 0, PND0), four males and four fe
249                            Feeding mice S100 at birth prevented the expansion of Enterobacteriaceae,
250    The umbilical artery lumen closes rapidly at birth, preventing neonatal blood loss, whereas the um
251                                100% coverage at birth reduced tuberculosis deaths by 16.5% (0.7-41.9)
252 arios in which 92% BCG coverage was achieved at birth reduced tuberculosis deaths in the global birth
253  while number, size and quality of offspring at birth remained unaffected.
254 tively associated with medical complications at birth, respectively.
255 of affected sibling, low level of Hemoglobin at birth, respiratory distress syndrome, low Hemoglobin
256 wever, after controlling for gestational age at birth, ROI analyses still showed positive correlation
257               The WHO recommendation for BCG at birth should be maintained and emphasised.
258                                              At birth, SMC contraction drives inner layer buckling an
259                                The sex ratio at birth (SRB) may be patterned by maternal condition an
260                                The sex ratio at birth (SRB; ratio of male to female live births) imba
261 icroorganisms that rapidly populate the body at birth, subject to a complex interplay that is depende
262    Since kicking and ground-stepping coexist at birth, switching between the two behaviors may depend
263 se that a topographic scaffolding is present at birth that both directs and constrains experience-dri
264                                     Starting at birth, the immune system of newborns and children enc
265  of gestation) receiving respiratory support at birth, the initial use of 21% oxygen is reasonable.
266                                              At birth, the lungs rapidly transition from a pathogen-f
267 e SAN junction did not cause a malformed SAN at birth, the mutant mice manifested sinus node dysfunct
268                                              At birth, the Xpert-PoC and Xpert-DBS were 100% sensitiv
269 uctures and occlusion comparable to controls at birth, they all acquired typical LF morphology with a
270 ers had much higher use of a health facility at birth, this did not appear to convey a comparable sur
271                Offspring were cross-fostered at birth to a dam on the same (CC,OO) or alternate diet
272                               The transition at birth to diets less enriched in PUFA is especially ch
273 f pathogen-specific antibodies were measured at birth to identify correlates of infant susceptibility
274 rn before 32 weeks' gestation and randomized at birth to receive early high-dose recombinant human er
275 ambs (125 +/- 1 d gestation) were randomized at birth to receive: 1) tidal ventilation without an int
276 ROP), 4099 infants weighing less than 1251 g at birth underwent sequential ophthalmic examinations, b
277                The mean gestational age (GA) at birth was 32 weeks and 3 days (32w3d) (95% CI, +/- 3
278      The median duration of maternal therapy at birth was 55 days (IQR 33-77).
279                      Younger gestational age at birth was found to be an independent predictor of per
280                              Gestational age at birth was inversely associated with mortality from in
281  and the labour-associated decrease in nTreg at birth was more evident among infants with subsequent
282 SV-A IC80 titer >239 or RSV-B IC80 titer >60 at birth was significantly associated with being a healt
283                       Median gestational age at birth was similar in the 2 groups (24.5 and 24.7 week
284 d proportion of activated regulatory T cells at birth was specific to the sensitized-tolerant infants
285 rtional [PM] or disproportional [DM]) or SGA at birth were evaluated with anthropometric measurements
286 rtional (PM) or disproportional (DM)] or SGA at birth were evaluated with anthropometric measurements
287                          Placentas collected at birth were investigated using stereology to establish
288                           Body size measures at birth were lower in infants exposed to repeat prenata
289 or mortality associated with gestational age at birth were: 66.14 (95% CI 63.09-69.34) for extremely
290 ision circles (TRECs) and T cell lymphopenia at birth, who carried heterozygous loss-of-function FOXN
291 of future behavior with brain microstructure at birth will reveal structural basis of behavioral emer
292 des an E3 ubiquitin ligase, led to lethality at birth with a striking reduction of lung size to a sin
293             Clinical presentation was mostly at birth with hypotonia and breathing and feeding diffic
294 is infection remains endemic and vaccination at birth with M. bovis bacille Calmette-Guerin (BCG) is
295  (birthweight above 4,000 g or 90th centile) at birth with positive likelihood ratios (LRs) of 8.74 (
296                      Mice lacking NMNAT2 die at birth with severe axon defects in both the peripheral
297 red with controls, HEU children were smaller at birth, with an adjusted difference in mean WAZ and LA
298 others (1.85 (1.38-2.48)); area of residence at birth, with children born in a small town or in the c
299      Reducing delays and increasing coverage at birth would substantially reduce global paediatric tu
300 d infants had generally similar Apgar scores at birth, yet the ACT-treated infants received greater m

 
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