1 anti-GD1b ganglioside antibody in autoimmune ataxic neuropathies.

2 ole of human anti-GD1b antibodies in sensory ataxic neuropathies.

3 y sensory neuropathy (HSN, 6.1%), 38 sensory ataxic neuropathy (2.5%), 72 hereditary neuropathy with

4 clonal antibodies from patients with chronic ataxic neuropathies and Miller Fisher syndrome were stud

5                                   Autoimmune ataxic neuropathies are a subset of the sensory ataxic n

6 oglobulin G (IgG) antibodies, and in chronic ataxic neuropathies associated with persistently elevate

7 Huttenlocher syndrome to adult-onset sensory ataxic neuropathy dysarthria and ophthalmoparesis.

8 -dependent, predominantly sensory, sometimes ataxic neuropathy, frequently accompanied by upper limb

9         The major known causes of autoimmune ataxic neuropathies include sensory variants of the Guil

10    The major site of pathology in autoimmune ataxic neuropathies is the dorsal root ganglion, but dor

11 reviously under the acronym CANOMAD: chronic ataxic neuropathy, ophthalmoplegia, IgM paraprotein, col

12                             CANOMAD (chronic ataxic neuropathy, ophthalmoplegia, immunoglobulin M [Ig

13 should be considered in all cases of sensory ataxic neuropathy, particularly, but not only, if cerebe

14          The combination of CPEO and sensory ataxic neuropathy (SANO, incomplete SANDO) was found in

15 xic neuropathies are a subset of the sensory ataxic neuropathies which are characterized by ataxia as

16 c neuropathy syndrome termed chronic sensory ataxic neuropathy with anti-disialosyl IgM antibodies is

17                                  The sensory ataxic neuropathy with dysarthria and ophthalmoparesis (

18                   The combination of sensory ataxic neuropathy with ophthalmoparesis (SANO) was obser