1                 Of the 16, only one type III atelectatic alveolus recruited at the first inflection p

2 ventilated aerated regions without expanding atelectatic areas.

3 severe respiratory distress, with cystic and atelectatic changes in the lungs complicated by tracheom

4        X-ray films revealed inflammatory and atelectatic changes with mediastinal shift to the right.

5 ere allowed to passively collapse and remain atelectatic for 1 hr.

6 jury-of aerated lung regions; recruitment of atelectatic lung may protect these regions by attenuatin

7         Cyclical tidal volume recruitment of atelectatic lung regions causes acute lung injury, as do

8 liquid ventilation to enhance recruitment of atelectatic lung regions may be an important means by wh

9 respiratory rates with the intent to recruit atelectatic lung, reduce peak inflating pressures and li

10                        Reexpansion of normal atelectatic lungs with total liquid ventilation was asso

11 tilation in normal and surfactant-deficient, atelectatic lungs.

12 tions were grouped based upon the fractional atelectatic mass in expiration (< 20%, 20-40%, and >= 40

13 al recruitment/derecruitment with fractional atelectatic mass in expiration greater than or equal to

14 ume were negatively correlated in fractional atelectatic mass in expiration greater than or equal to

15 s (r = 0.56 and 0.40), but not in fractional atelectatic mass in expiration less than 20% group (r =

16 olume in the dependent region and fractional atelectatic mass in expiration negatively correlated wit

17 examination of mutants revealed immature and atelectatic regions in the lung, severely reduced capill

18       We assessed the regional proteomics of atelectatic versus normally-aerated lung tissue to test