ライフサイエンスコーパス: athlete

1 nners, with a median of 13 marathon runs per athlete.

2 er application of the ECG in the care of the athlete.

3 s of this method in the examination of elite athletes.

4 es practicing a non-contact sport and 26 non-athletes.

5 aluation of entrapment neuropathies in elite athletes.

6 ith previously identified mQTLs in non-elite athletes.

7 ignificant albeit still neglected problem in athletes.

8 ce of atrial fibrillation among well-trained athletes.

9 e impaired compared to matched eumenorrhoeic athletes.

10 ac death, particularly in young patients and athletes.

11 essional ASF players and other contact sport athletes.

12 dotypes inducing respiratory symptoms within athletes.

13 al effects associated with age in the alumni athletes.

14 arge cohort of young, white adults including athletes.

15 eart rate, consistent with HCN repression in athletes.

16 e of at least 0.13 m compared to non-amputee athletes.

17 thological sinus node dysfunction in veteran athletes.

18 he proper evaluation of ECG abnormalities in athletes.

19 s and a major cause of sudden death in young athletes.

20 r the Child SCAT3 in a large cohort of young athletes.

21 emporary standards for ECG interpretation in athletes.

22 ated neural activity in former U.S. football athletes.

23 ism and performance of world-class endurance athletes.

24 eable and competent in ECG interpretation in athletes.

25 t of HA has yet to be studied in truly elite athletes.

26 age, which may be more successful in masters athletes.

27 ces for health and performance monitoring of athletes.

28 y in Sport (RED-S) in female elite endurance athletes.

29 me-of-day on physical performance in Olympic athletes.

30 immunological and metabolic effects on these athletes.

31 males, with little available data on female athletes.

32 st active athletes compared with less active athletes.

33 plethysmogram, heart rate, and activities of athletes.

34 etect the misuse of the CRISPR/Cas system by athletes.

35 ading cause of sudden cardiac death in young athletes.

36 cardiomyopathy criteria has been detected in athletes.

37 ssociations in a cohort of endurance trained athletes.

38 power relative to body mass than other rugby athletes (14%; P = 2 x 10(-6)).

39 d motor control were performed in 30 masters athletes (16 male) aged 44-83 years.

40 tio to the history of concussion in 81 youth athletes (18 with a history of concussion, ages 13-18) d

41 46), including 4,720 females (32%) and 2,958 athletes (20%), were evaluated by using a health questio

42 al study was performed in 3281 healthy elite athletes (2039 men and 1242 women) aged 23.1+/-5.7 years

43 t ventricular hypertrophy was present in 144 athletes (27.4%).

44 %) than nonelite swimmers (31%), nonswimming athletes (32%) and controls (24%).

45 Seven hundred twenty Olympic athletes (360 females and 360 age- and sport-matched mal

46 ) compared to controls (67%) and nonswimming athletes (42%).

47 We assessed 152 masters athletes 54.4+/-8.5 years of age (70% male) and 92 contr

48 Most athletes (60%) and controls (63%) had a normal CAC score

49 Of these athletes, 7 had a pattern consistent with previous myoca

50 dy mass for success, compared to other rugby athletes (82%; P = 0.01, OR = 3.34) and controls (84%; P

51 Twenty-eight athletes about to embark on a period of forced detrainin

52 physiological limits in all planes in elite athletes according to static and dynamic cardiovascular

53 For each condition, we quantified athlete aerobic energy expenditure and performed biomech

54 g carbon fiber plates to shoes soles reduces athlete aerobic energy expenditure during running (impro

55 gnificantly larger in a sample of former NFL athletes after adjusting for their size, age, race, and

56 e exists pertaining to metabolic recovery of athletes after endurance races without the utilisation o

57 distance, and star drill time) in youth male athletes age 10-19, in two sessions: fifty-one subjects

58 n 2011 and 2014, 4,925 previously unscreened athletes aged 14 to 35 years were prospectively evaluate

59 ystem included more than 3000 youth football athletes aged 5 to 14 years from 118 teams, providing 31

60 s and 46 saliva samples from 55 male college athletes ages 18-25 years.

61 089 participants, comprising 530 elite rugby athletes and 559 non-athletes, DNA was collected and gen

62 havioural impairment in retired professional athletes and a point of comparison for future neuroimagi

63 specific populations such as collision sport athletes and certain military personnel are of particula

64 nd nonelite swimmers compared to nonswimming athletes and controls.

65 rhinitis in swimming compared to nonswimming athletes and controls.

66 elite swimmers (22%) compared to nonswimming athletes and controls.

67 e, affecting in equal measure women and men, athletes and couch potatoes, spinach eaters and fast foo

68 e of coronary atherosclerosis in middle-aged athletes and describe strategies for the clinical manage

69 ial growth in the populations of competitive athletes and highly active people (CAHAP).

70 in the training-induced bradycardia in human athletes and investigate the role of microRNAs (miRs) in

71 roblem for both recreational and competitive athletes and is the most common chronic condition among

72 n concussion sustained by professional/elite athletes and long-term cognitive impairment.

73 of 23 of the world's most successful memory athletes and matched controls with fMRI during both task

74 juries are mild, such as those that occur in athletes and military personnel exposed to repetitive he

75 rmTBI resembles the alterations observed in athletes and military personnel exposed to rmTBI, these

76 nces were also recorded between recreational athletes and non-athletes (p = 0.044).

77 ant differences between males and females or athletes and non-athletes in distress levels, which stat

78 national consensus for ECG interpretation in athletes and provides expert opinion-based recommendatio

79 nd real-time competition assistance for both athletes and referees.

80 e-off between maximum speed and endurance in athletes and some animals that has been explained by cos

81 ed differences in LD size and number between athletes and T2DM.

82 tern of exercise which is analogous to human athletes and the cardiovascular risks in both species ar

83 wards upper airway disorders in the swimming athletes and to ensure adequate management.

84 t discriminate elite athletes from non-elite athletes and to identify those associated with endurance

85 However, endurance trained athletes and type 2 diabetes mellitus (T2DM) patients st

86 bow pain can cause disability, especially in athletes, and is a common clinical complaint for both th

87 sed incidence of bradyarrhythmias in veteran athletes, and it will be important to understand the und

88 problem that affects millions of civilians, athletes, and military personnel yearly.

89 Athletes are a vulnerable population for methicillin-res

90 Male athletes are more likely to have a CAC score >300 Agatst

91 Evidence suggests masters athletes are more successful at motor unit remodelling,

92 the particular injuries to which these young athletes are prone.

93 signal and white matter changes in young NFL athletes are related to later onset of neuropsychiatric

94 History has taught us that athletes are subjected to unmonitored trials with experi

95 We tested 15 athletes as they ran at 3.5 m/s in four footwear conditi

96 isms involved in development of EIB in elite athletes as well as essential aspects to ultimately prop

97 HCN remodeling likely occurs in human athletes, as well as in rodent models.

98 heart disease in longshoremen and in college athletes at Harvard University and University of Pennsyl

99 e common (94%) in selected elite rugby union athletes (back three and centre players) who are most re

100 interval, 1.01-1.15; P=0.016); 15 (14%) male athletes but none of the controls revealed late gadolini

101 rdiac mass and volumes are often elevated in athletes, but it is not known whether moderate physical

102 or their metabolites in urine samples of the athletes by GC/MS(n) or LC/MS(n).

103 e is a form of burnout, defined in endurance athletes by unexplained performance drop associated with

104 For instance, using NeuroFeedback (NF), athletes can learn to self-regulate specific brain rhyth

105 sociated with specific risk factors, such as athletes, children, men who have sex with men, prisoners

106 Compared to matched eumenorrhoeic athletes, chronic OCP use impaired the sweating onset th

107 One in two female athletes chronically take a combined, monophasic oral co

108 Serum samples of 16 athletes collected 24 h before, immediately after, as we

109 solution metabolomics profiling in 490 elite athletes, common variant metabolic quantitative trait lo

110 adults and left-footedness was increased in athletes compared to the general population.

111 of 206 former National Football League (NFL) athletes compared with 759 male subjects from the DHS-2

112 cular morbidity and mortality, was higher in athletes compared with controls, and was higher in the m

113 controls, and was higher in the most active athletes compared with less active athletes.

114 ons are slightly larger in young competitive athletes compared with sedentary controls, but rarely >4

115 Similarity with memory athlete connectivity patterns predicted memory improveme

116 Male athletes demonstrated predominantly calcific plaques (72

117 able conditions should be explored in former athletes demonstrating CTE-linked clinical phenotypes or

118 diet impairs performance in elite endurance athletes despite a significant improvement in peak aerob

119 the increased longevity typical of endurance athletes despite the presence of more coronary atheroscl

120 prising 530 elite rugby athletes and 559 non-athletes, DNA was collected and genotyped for the FTO rs

121 h (SCD) is the leading cause of mortality in athletes during sport.

122 gy recommendations for ECG interpretation in athletes, ECG standards have evolved quickly over the la

123 urgeons who had previously served as Olympic athletes, elite musicians, or expert military personnel.

124 Endurance athletes exceed the usual recommendations for exercise b

125 Masters athletes exhibit age-related declines in neuromuscular f

126 sport has a relevant impact, with endurance athletes exhibiting the greatest degree of RV and LV dim

127 hool Reporting Information Online injury and athlete-exposure data, and used generalized estimating e

128 During an estimated 2,697,089 athlete-exposures, 190 EHIs were reported.

129 un metagenomic analysis in a cohort of elite athletes, finding that every gene in a major pathway met

130 lectrocardiographic (ECG) screening of young athletes for cardiac disease.

131 Studies including professional/elite athletes from any sport were considered.

132 candidate biomarkers discriminate concussed athletes from controls and are associated with duration

133 h control groups and discriminated concussed athletes from controls with areas under the curve of 0.6

134 the network organization that distinguishes athletes from controls.

135 s quantified the discrimination of concussed athletes from controls.

136 nguishes normal physiological adaptations in athletes from distinctly abnormal findings suggestive of

137 fluenced metabolites that discriminate elite athletes from non-elite athletes and to identify those a

138 tly improved the discrimination of concussed athletes from noncontact controls compared to symptom se

139 tted (group 3), 4 to 5 sessions; and Masters athletes (group 4), 6 to 7 sessions plus regular competi

140 Male athletes had a higher prevalence of atherosclerotic plaq

141 Overall, 1,072 (21.8%) athletes had an abnormal ECG on the basis of 2010 ESC re

142 ignificantly higher proportion of former NFL athletes had an aorta of >40 mm (29.6% versus 8.6%; P<0.

143 Accordingly, these athletes had greater peak power relative to body mass th

144 Athletes had participated in endurance exercise for an a

145 a population-based control group, former NFL athletes had significantly larger ascending aortic diame

146 to have been potentially identifiable if the athletes had undergone preparticipation screening.

147 Equine athletes have a pattern of exercise which is analogous t

148 sk of future adverse events, elite endurance athletes have an increased life expectancy compared with

149 In the longer term, some endurance athletes have an increased prevalence of coronary artery

150 embranes in leg muscles of endurance-trained athletes have an increased ratio of surface per mitochon

151 A large proportion of endurance athletes have raised levels of cardiac biomarkers (tropo

152 rtension, or coronary heart disease, or even athletes, heart rate lowering consistently increases cen

153 s are thought to be able to identify at-risk athletes; however, the efficacy of these programs remain

154 d a significant positive correlation between athletes' improvement in CVSA abilities and the increase

155 CI, 1.54-13.68; P=0.006) and in competitive athletes in comparison with nonathletes (odds ratio, 16.

156 etween males and females or athletes and non-athletes in distress levels, which statistically signifi

157 'techno doping') and inclusion of Paralympic athletes in Olympic events are matters of ongoing debate

158 tle objective cognitive impairment in alumni athletes in the context of high subjective complaints an

159 ated performance benefits in elite endurance athletes, in part due to reduced exercise economy.

160 nuing concern about effects of concussion in athletes, including risk of the neurodegenerative diseas

161 nantly calcific morphology of the plaques in athletes indicates potentially different pathophysiologi

162 However, extensive training in competitive athletes is associated with an increased atrial fibrilla

163 imple reaction time (VRT) is faster in elite athletes is contentious.

164 on long-term health and wellbeing in former athletes is limited.

165 ted, the arrhythmogenic substrate for VTs in athletes is unknown.

166 lates to aortic enlargement in older, former athletes is unknown.

167 tarium of diagnostic tests used routinely in athletes, its accuracy in the detection of entrapment ne

168 n (EIB) without asthma (EIBwA ) occurring in athletes led to speculate about different endotypes indu

169 ning world class octogenarian female masters athletes (MA, 80.9 +/- 6.6 years).

170 Masters athletes maintain high levels of activity into older age

171 n of glucose (polymers) only to help trained athletes maximize endurance performance during prolonged

172 enuous exercise session.Eight male endurance athletes (mean +/- SEM age: 29 +/- 2 y; peak oxygen cons

173 and improve patients' medical progression or athletes' monitoring capabilities that are already beyon

174 tional potential for patients as well as for athletes, musicians and other groups, depends largely on

175 r interest as it is common practice to train athletes, musicians or perform rehabilitation exercises

176 olving elite (n = 101) and nonelite swimming athletes (n = 107), nonswimming athletes (n = 38) and se

177 ite swimming athletes (n = 107), nonswimming athletes (n = 38) and sex- and age-matched controls (n =

178 ball players (n = 84), and non-contact-sport athletes (n = 50).

179 In a subgroup of rugby athletes (n = 77), we assessed muscle power during a cou

180 Alumni athletes (N=33, aged 34-71 years) and an age-matched sam

181 athological change (CTE-NC) were identified (athletes, n = 10; long-term survivors of moderate or sev

182 e for age or luminal stenosis >/=50% in male athletes (odds ratio, 1.08; 95% confidence interval, 1.0

183 re larger in youth athletes than high school athletes (odds ratio, 6.23; 95% CI, 1.02-37.98).

184 ass index, BMI: 23.4 +/- 1.7 kg/m(2)) female athletes, of which 25 belong to the weight loss and rega

185 We conclude that athletes on a LCHF diet do not compensate for reduced gl

186 igher rates of glucose synthesis compared to athletes on a mixed diet.

187 h we compared to a group of normally trained athletes on behavioral tasks performed during fMRI scann

188 risk or to detect the presence of disease in athletes or military recruits.

189 corded between recreational athletes and non-athletes (p = 0.044).

190 al heat illness (EHI) among U.S. high school athletes participating in preseason sports activities, i

191 ing musculoskeletal system makes the growing athlete particularly vulnerable to specific types of inj

192 bow pain is a frequent presenting symptom in athletes, particularly athletes who throw.

193 Endurance athlete performance is greatly dependent on sufficient p

194 s, but rarely with skeletal muscle and elite athlete phenotypes.

195 ng to CRF remains unclear, especially in non-athlete population.

196 Compared to T2DM, trained athletes possess higher levels of perilipin 5 (PLIN5), a

197 ll players and a control group comprising 27 athletes practicing a non-contact sport and 26 non-athle

198 ristic differs according to sex, with female athletes progressing to a slower firing pattern that was

199 ting results from a trial of trained javelin athletes, providing new estimates for key performance pa

200 ove their distance-running performance, many athletes race with carbon fiber plates embedded in their

201 The major roles of an athlete's daily diet are to supply the substrates needed

202 ach for interpreting the complex features of athlete's heart in women.

203 graphic and morphological features of female athlete's heart, with special attention to differences r

204 ipid (LD size and number) in relation to the athlete's paradox.

205 nts of lipids in their muscle; the so-called athlete's paradox.

206 f LDs with PLIN5 cannot causally explain the athlete's paradox.

207 e insulin sensitivity and contributes to the athlete's paradox.

208 5 abundance cannot be causally linked to the athlete's paradox.

209 n fatalities have raised questions regarding athlete safety.

210 or all testing modalities from 21 adolescent athletes (seven concussive and fourteen healthy) in thre

211 s responsible for the cardiovascular care of athletes should be knowledgeable and competent in ECG in

212 compared with sedentary males, and only male athletes showed a CAC >/=300 Agatston units (11.3%) and

213 ereas world class octogenarian female master athletes showed attenuated indices of denervation and gr

214 injustice of unfair advantage for dishonest athletes, significant potential health risks are associa

215 be to accept that there is a delay from when athletes start experimenting with novel agents to the ti

216 ommendations for ECG interpretation in young athletes state that ATWI beyond lead V1 warrants further

217 mellitus, and lipid profile, the former NFL athletes still had significantly larger ascending aortas

218 As more elite athletes suffer from stress, targeting the microbiota of

219 and more often only calcified plaques among athletes, suggesting a more benign composition of athero

220 lysis of world record performances by master athletes suggests an essentially linear decline with age

221 per 100,000 athlete-years, with 43.8% of the athletes surviving until they were discharged from the h

222 of performance enhancing drugs in sports as athletes take diuretics to dilute the concentration of d

223 n 24 hours after injury were larger in youth athletes than high school athletes (odds ratio, 6.23; 95

224 pectively; p < 0.0001) and more common among athletes than in nonathletes (3.5% vs. 2.0%, respectivel

225 heart rate was significantly lower in human athletes than in nonathletes, and in all subjects, the r

226 ubstrate for fast VT in high-level endurance athletes that can be successfully treated by ablation.

227 Compared with Masters athletes, the exercise group demonstrated lower absolute

228 c disorders are associated with SCD in young athletes, the majority of which can be identified or sug

229 In the athlete, there is a reduced threshold for imaging to cla

230 different than in the 1950s, but for current athletes, this study provides information on the risk of

231 e sports medicine physician is to return the athlete to competition-balanced against the need to prev

232 ion tensor imaging (DTI) data for 28 contact athletes to compare WM changes.

233 Attribution of candidate symptoms in retired athletes to CTE is complicated by the presence of multip

234 However, genotyping of athletes to determine their dementia risk remains a dist

235 This provides a window of opportunity for athletes to exploit with relative immunity.

236 rnational sporting events will expose female athletes to hot environments differing in their thermal

237 arge international events will expose female athletes to hot environments, and studies evaluating aer

238 Focusing on daily nutrition is important for athletes to perform and adapt optimally to exercise trai

239 nce of continuously challenging patients and athletes to promote neural plasticity, skilled performan

240 for indirect effects caused through enabling athletes to push beyond normal physiological limits with

241 SA) abilities, which are essential for these athletes to reach high performances.

242 the differences between pediatric and adult athletes to recognize the particular injuries to which t

243 sensitivity, albeit not in endurance-trained athletes (Trained).

244 Among the competitive athletes, two deaths were attributed to hypertrophic car

245 s are not suitable for a practical use since athletes typically go through strenuous activities durin

246 Thirty-eight Scandinavian female elite athletes underwent a day-long exercise test.

247 v(max) was 3.8-10.7% faster when athletes used J-shaped versus C-shaped RSP models (p < 0

248 mer London 2012 Olympic and Paralympic Games Athletes' Village that was repurposed on active design p

249 le); between-sport comparison (contact sport athletes vs non-contact sports ones); external compariso

250 n-contact sports ones); external comparison (athletes vs samples of the general population or populat

251 Metabolic recovery of the athletes was attained within 48 h post-marathon, most li

252 al injury and inflammation in the airways of athletes was demonstrated.

253 Fourteen Masters athletes were added for comparison.

254 and fat intake among female participants and athletes were discovered.

255 High school and collegiate athletes were enrolled between 2015 and 2018.

256 Former NFL athletes were twice as likely to have an aorta >40 mm af

257 Coronary plaques are more abundant in athletes, whereas their stable nature could mitigate the

258 he U.S. National Registry of Sudden Death in Athletes (which uses news media, Internet searches, Lexi

259 form of overtraining in a group of endurance athletes, which we compared to a group of normally train

260 We collected helmet data for the athletes, which we correlated with DTI data.

261 cise training on coronary atherosclerosis in athletes who are middle-aged and older and aims to contr

262 ured as coronary artery calcification, among athletes who are middle-aged and older.

263 Data of athletes who made it to the finals (N = 144, 72 female)

264 rative disorders have been reported in elite athletes who participated in contact sports.

265 presenting symptom in athletes, particularly athletes who throw.

266 overuse injuries characteristic to pediatric athletes will be discussed.

267 ue in the long jump, which provided the best athlete with BKA a performance advantage of at least 0.1

268 terpretation of Child SCAT3 scores for young athletes with a concussion in athletic and clinical sett

269 stent deficits in psychomotor ability in the athletes with a history of concussion that may have impl

270 Athletes with a lifelong exercise volume >2000 MET-min/w

271 postinjury relative to baseline in concussed athletes with a loss of consciousness or amnesia.

272 Most lifelong masters endurance athletes with a low atherosclerotic risk profile have no

273 alence of coronary artery disease in masters athletes with a low atherosclerotic risk profile.

274 uth, high school, and college-level football athletes with a mean (SD) of 5.48 (3.06) symptoms.

275 t that due to different movement strategies, athletes with and without BKA should likely compete in s

276 Studies in middle-age and older (masters) athletes with atherosclerotic risk factors for coronary

277 the overall vertical force from both legs of athletes with BKA compared to non-amputees.

278 m intrinsically lower sprinting abilities of athletes with BKA or from more complex adaptions in spri

279 We show that athletes with BKA utilize a different, more effective ta

280 We found slower maximum sprinting speeds in athletes with BKA, but did not find a difference in the

281 be strategies for the clinical management of athletes with coronary atherosclerosis to guide physicia

282 in clinical decision making and treatment of athletes with elevated coronary artery calcification sco

283 The majority of athletes with no or spotty LGE pattern had ventricular a

284 Further studies are needed in athletes with overt and pathological hypertrophy to esta

285 motion perception and sports performance in athletes with real visual impairment.

286 persistent glial cell activity in brains of athletes with sports-related traumatic brain injury.

287 cific prosthetic (RSP) configuration used by athletes with transtibial amputations (TTAs) likely affe

288 Athletes with unilateral TTAs are prescribed C- or J-sha

289 Ten athletes with unilateral TTAs ran at 3 m/s to v(max) on

290 t not stiffness or height, affects v(max) in athletes with unilateral TTAs.

291 ccurs to a similar extent in male and female athletes, with discharge rates progressively slowing in

292 significantly affects cardiac remodeling in athletes, with females presenting a different electrical

293 most common chronic condition among Olympic athletes, with obvious implications for their competing

294 identifying the metabolic recovery trend of athletes within 48 h after a marathon.

295 ernal comparison (concussed vs non-concussed athletes within the same sample); between-sport comparis

296 hieved jump distances similar to world-class athletes without amputations, using a carbon fibre runni

297 Our findings suggest that high school athletes would benefit from enactment of the 2009 NATA-I

298 ompetitive sports was 0.76 cases per 100,000 athlete-years, with 43.8% of the athletes surviving unti

299 ompetitive sports was 0.76 cases per 100,000 athlete-years.

300 ess and endurance performance in Chinese non-athlete young females.