ライフサイエンスコーパス: atrial contraction

1 ationship, quantifying work performed during atrial contraction).

2 ocated on atrial axial tubules that regulate atrial contraction.

3 cardiac phases: midsystole, middiastole, and atrial contraction.

4 ufficient to compensate for an impairment of atrial contraction.

5 eak velocity of pulmonary venous flow during atrial contraction.

6 cle when the foramen ovale was closed during atrial contraction.

7 lectrical activity resulting in asynchronous atrial contraction.

8 ation of the myocardial stretch generated by atrial contraction.

9 consistent with its positive impact on left atrial contraction.

10 tcome was the mean number of daily premature atrial contractions.

11 k A wave velocity minus velocity at onset of atrial contraction]).

12 stole, and the shortest were obtained during atrial contraction (40% variation per cardiac cycle).

13 The mean depth was greater during atrial contraction (6.3 mm +/- 2.1) than in midsystole (

14 points of the waveform: point 1, peak of the atrial contraction (a wave); point 2, the left atrial pr

15 rdinated atrial contractions, rather than no atrial contraction, achieved lower overall absolute simu

16 e Mlc2a gene resulted in severely diminished atrial contraction and consequent embryonic lethality at

17 ide important insight into the regulation of atrial contraction and describe potential therapeutic ta

18 ons and flow, and myh6 morphants, which lack atrial contraction and exhibit reduced flow.

19 FD exhibited a higher incidence of premature atrial contractions and increased risk of atrial fibrill

20 , right ventricular pacing without preceding atrial contraction, and dual-chamber pacing at AV delays

21 les--in particular, reverse flow velocity at atrial contraction; and 5) increasing LA pressure result

22 s to understand how the processes regulating atrial contraction are remodelled during ageing and prov

23 offee was associated with 58 daily premature atrial contractions as compared with 53 daily events on

24 ection within the venous waveform signifying atrial contraction] at or below baseline; DV no A).

25 ation, atrial late potentials, and premature atrial contraction burden at 24 months and no atrial fib

26 ary veins and transmitral flow duration with atrial contraction correlated with LV filling pressure i

27 of E (initial peak velocity), A (velocity at atrial contraction), deceleration time (time from E velo

28 We then use a premature atrial contraction detection algorithm to have more accu

29 filling rates of the early filling phase and atrial contraction [E/A ratio]).

30 ract, and LV free wall, as well as premature atrial contractions, from the left atrial appendage at a

31 The difference between flow duration with atrial contraction in the pulmonary veins and transmitra

32 terdepolarizations and more severe premature atrial contractions in atrial trabeculae of patients wit

33 ak pulmonary venous reverse flow velocity at atrial contraction increased significantly, without bein

34 naling complex that may enhance the speed of atrial contraction independently of phospholamban regula

35 ned analysis of both flow velocity curves at atrial contraction is a reliable, feasible predictor of

36 These data suggest that organized atrial contraction is needed to maintain normal endocard

37 ection fraction (P=0.03), lower LA strain at atrial contraction (LASac; P<0.001), higher LAV (P<0.003

38 ) weak-atriumm58 mutant (wea) with inhibited atrial contraction leading to a highly undeveloped ventr

39 se in peak velocity flow in late diastole by atrial contraction (MV A Peak) indicating poorer left at

40 During atrial contraction, MVG was similarly low (P<0.01) in bo

41 Augmented right atrial contraction optimizes RV performance, whereas ver

42 e lengths, left atrial dimensions, premature atrial contraction (PAC) frequency, and atrial vulnerabi

43 al isolation was able to eliminate premature atrial contractions (PACs) and AF in six of 21 patients

44 nt SVT is initiated by spontaneous premature atrial contractions (PACs) and is terminated by spontane

45 Premature atrial contractions (PACs) are independent predictors of

46 Premature atrial contractions (PACs) play a critical role in AF pa

47 ed as the presence of either >/=30 premature atrial contractions (PACs)/hour daily or any runs of >/=

48 P < .001) and LA left atrium strain rate at atrial contraction peak ( SRA LA strain rate at atrial c

49 at atrial contraction peak LA strain rate at atrial contraction peak (beta coefficient -0.0028, P = .

50 0.0019, P = .016) and SRA LA strain rate at atrial contraction peak (beta coefficient = -0.0022, P =

51 ial contraction peak ( SRA LA strain rate at atrial contraction peak ) (-1.50 +/- 0.62 vs -1.78 +/- 0

52 0.0016, P = .027), and SRA LA strain rate at atrial contraction peak LA strain rate at atrial contrac

53 ), early diastolic (SRe), and late diastolic atrial contraction phases (SRa) were analyzed by dedicat

54 ed a linear relation with LA pressure before atrial contraction (r = 0.80, p < 0.005), confirming the

55 ng rate [E wave] and late diastolic filling [atrial contraction] rate [A wave]) time points by using

56 ling atrial activity in AF as unco-ordinated atrial contractions, rather than no atrial contraction,

57 eaks 1 and 2) reflected rapid LV filling and atrial contraction, respectively.

58 ced late I(Na) and correlated with premature atrial contraction severity.

59 ial peak atrial longitudinal strain and peak atrial contraction strain showed a similar, although non

60 bal peak atrial longitudinal strain and peak atrial contraction strain significantly decreased after

61 result in significantly more daily premature atrial contractions than the avoidance of caffeine.

62 ing time indicated augmented contribution of atrial contraction toward LV filling (P<.05).

63 Peak left atrial contraction velocities (measured from the transmi

64 ximum LA volumes (VOLmax) and volumes before atrial contraction (VOLbac) were measured; LAPEF was cal

65 e time-velocity integral of each flow during atrial contraction was measured.

66 manometer catheter, and LA dP/dt(max) during atrial contraction was obtained.