ライフサイエンスコーパス: atrioventricular

1 block, sinus arrest, 2 degrees and 3 degrees atrioventricular (A-V) block and supraventricular escape

2 optical mapping studies indicated high-grade atrioventricular (A-V) block in ZO-1cKO comparing to CTL

3 e at different increments of heart rate, and atrioventricular and interventricular delay.

4 The cardiac sodium channel SCN5A regulates atrioventricular and ventricular conduction.

5 mogenic foci can originate in areas near the atrioventricular annuli, we hypothesized that focal annu

6 the developing heart, including cells in the atrioventricular (AV) and outflow tract (OFT) cushions.

7 th chronic left bundle-branch block (n=8) or atrioventricular (AV) block (n=6) through atrial (A), ri

8 n atrial fibrillation (AF) without producing atrioventricular (AV) block remains a clinical challenge

9 genital heart defects, including progressive atrioventricular (AV) block requiring pacemaker implanta

10 Tamoxifen-induced loss of ZO-1 led to atrioventricular (AV) block without changes in heart rat

11 In pigs with complete atrioventricular (AV) block, transcription factor T-box

12 s often accompanied by atrial tachycardia or atrioventricular (AV) block.

13 ) and pressure gradients (nablaP) across the atrioventricular (AV) canal.

14 Atrioventricular (AV) conduction disturbances requiring

15 of SSS in humans, including bradycardia and atrioventricular (AV) dysfunction (heart block).

16 cells (EPDCs) have demonstrated that at the atrioventricular (AV) junction EPDCs contribute to the m

17 The atrioventricular (AV) junction plays a critical role in

18 e novo ventricular rate faster than the slow atrioventricular (AV) junctional escape rhythm observed

19 ntributing to the longitudinal motion of the atrioventricular (AV) plane.

20 cases), ictal bradycardia (25 cases), ictal atrioventricular (AV)-conduction block (11 cases), posti

21 f ablation (82% versus 97%; P=0.04), risk of atrioventricular block (14 versus 0%; P=0.004), and need

22 (five [1%] vs one [<0.5%]), and first-degree atrioventricular block (17 [5%] vs seven [2%]).

23 al dominant sinus node dysfunction (SND) and atrioventricular block (AVB) and to characterize the mut

24 Torsades de pointes (TdP) +/-2 degrees atrioventricular block (AVB) are not always attributed t

25 ably expressed and may cause cardiomyopathy, atrioventricular block (AVB), or atrial arrhythmias (AAs

26 mines the risk for torsade de pointes during atrioventricular block (AVB).

27 icular Pacing in Heart Failure Patients with Atrioventricular Block (BLOCK HF) trial randomized patie

28 Complete atrioventricular block (CAVB) is a life-threatening arrh

29 te to cardiomyopathy, the impact of complete atrioventricular block (cAVB) on heart failure (HF) deve

30 Delayed high-grade atrioventricular block (DH-AVB) has not been systematica

31 ting fetal LQTS arrhythmias: TdP+/-2 degrees atrioventricular block (group 1, n=7), isolated 2 degree

32 lar block (group 1, n=7), isolated 2 degrees atrioventricular block (group 2, n=4), and sinus bradyca

33 High-grade atrioventricular block (H-AVB) is a well-described in-ho

34 acing was permanent atrial fibrillation with atrioventricular block (n=22, 67%).

35 s to investigate the association of parental atrioventricular block (PR interval, >=0.2 s), complete

36 icular Pacing in Heart Failure Patients With Atrioventricular Block [BLOCK HF]; NCT00267098).

37 ratio, 95% CI, 1.32-2.07) for manifesting an atrioventricular block and a 1.62-fold odds (95% CI, 1.0

38 istics on adenosine dose required to produce atrioventricular block and duration of effect were also

39 n, nonsustained ventricular tachycardia, and atrioventricular block and inversely correlated with the

40 al right ventricular pacing in patients with atrioventricular block and left ventricular systolic dys

41 with biventricular pacing for patients with atrioventricular block and LV systolic dysfunction.

42 For patients with atrioventricular block and systolic dysfunction, biventr

43 dP), and in an adult woman with QTc >500 ms, atrioventricular block and TdP.

44 out biventricular pacing in HF patients with atrioventricular block because they are typically exclud

45 d spontaneously all patients had evidence of atrioventricular block before etrasimod exposure.

46 ed against dofetilide-induced TdP in chronic atrioventricular block dogs.

47 nduced sinus pauses, asystole, and transient atrioventricular block in both groups showing a strong v

48 h of acquired and often reversible causes of atrioventricular block in childhood.

49 tion because of a presumed risk of prolonged atrioventricular block in denervated hearts.

50 Adenosine induces atrioventricular block in healthy pediatric and young ad

51 There was no permanent atrioventricular block in patients who underwent cryoabl

52 five (1%) versus one (<1%), and first-degree atrioventricular block in three (1%) versus six (1%).

53 High-degree atrioventricular block is the most common presentation o

54 e or asystole; (2) high-grade Mobitz type II atrioventricular block or complete heart block; (3) vent

55 ntly predicted the adenosine dose to produce atrioventricular block or duration of effect.

56 e (n=35), 31.4% had newly diagnosed advanced atrioventricular block or severe bradycardia before TAVR

57 (AF)/atrial tachycardia (AT) in 28, advanced atrioventricular block or severe bradycardia in 24, nons

58 ars) with cardioinhibitory syncope, advanced atrioventricular block or sinus arrest, and no structura

59 monitoring showed no increased incidence of atrioventricular block or sinus pause with ozanimod.

60 ugh evaluation is required to determine when atrioventricular block requires treatment.

61 -onset left bundle-branch block and advanced atrioventricular block requiring permanent pacemaker imp

62 nts had a transient, asymptomatic, low-grade atrioventricular block that resolved spontaneously all p

63 The median longest atrioventricular block was 1.9 seconds (interquartile ra

64 Out of 7 irradiated animals, complete atrioventricular block was achieved in 6 animals of all

65 n following a dose-escalation protocol until atrioventricular block was achieved.

66 resence of severe metabolic imbalance, while atrioventricular block was largely an independent primar

67 Atrioventricular block was observed in 77 patients (96%;

68 Complete atrioventricular block was present in 11 patients; 3 pat

69 bradycardia or second-degree or third-degree atrioventricular block was reported.

70 ons at other anatomic sites, and no cases of atrioventricular block were encountered.

71 lly, PR interval prolongation and high-grade atrioventricular block were exclusively associated with

72 No late complications including atrioventricular block were noted.

73 al fibrillation and second- and third-degree atrioventricular block were observed, respectively, in 5

74 ymptom onset in Killip class I to II without atrioventricular block were randomized 1:1 to IV metopro

75 nd no second-degree or third-degree cases of atrioventricular block were reported.

76 diagnosis of LC in patients with high-degree atrioventricular block will facilitate the identificatio

77 ied exhibited novel fetal rhythms, including atrioventricular block with 3:1 conduction ratio, QRS al

78 ained ventricular arrhythmia, and high-grade atrioventricular block within 30 days including index ev

79 icular Pacing in Heart Failure Patients With Atrioventricular Block) trial demonstrated that biventri

80 n, nonsustained ventricular tachycardia, and atrioventricular block), which carries increased risk fo

81 ion, (2) early-onset atrial fibrillation and atrioventricular block, and (3) left ventricular noncomp

82 on; the composite of pacemaker implantation, atrioventricular block, and sinoatrial dysfunction: 0.94

83 al duration prolongation, occasionally a 2:1 atrioventricular block, and slowing of conduction veloci

84 ores an adequate heart rate in patients with atrioventricular block, but high percentages of right ve

85 , cardiogenic shock, ventricular arrhythmia, atrioventricular block, cardiac arrest, or death of a ca

86 In FHS (n=371 cases with first-degree atrioventricular block, complete bundle branch block, or

87 h 3:1 conduction ratio, QRS alternans in 2:1 atrioventricular block, long-cycle length TdP, and slow

88 ck (BLOCK HF) trial randomized patients with atrioventricular block, New York Heart Association sympt

89 The BLOCK HF trial randomized patients with atrioventricular block, NYHA symptom class I to III hear

90 including bradycardic events, sinus pauses, atrioventricular block, premature ventricular contractio

91 tion are ventricular arrhythmias or complete atrioventricular block, presenting clinically as syncope

92 isease, PR interval prolongation, high-grade atrioventricular block, significant left ventricular dys

93 signature LQTS rhythms: functional 2 degrees atrioventricular block, T-wave alternans, and torsade de

94 dditional patients, during adenosine-induced atrioventricular block, the minimum CF significantly inc

95 bradycardia due to sinus node dysfunction or atrioventricular block.

96 ave increased risk of procedural failure and atrioventricular block.

97 use, macrophage ablation induces progressive atrioventricular block.

98 S with atypical features, including neonatal atrioventricular block.

99 ted VT reinduction with anticipated complete atrioventricular block.

100 ERT2) mice show episodes of sinus pauses and atrioventricular block.

101 dia/fibrillation or bradyarrhythmias such as atrioventricular block.

102 5 s mean heart rate, longest RR, pauses, and atrioventricular block.

103 ilateral chronic conjunctivitis and complete atrioventricular block.

104 lete left bundle branch block or high-degree atrioventricular block.

105 (new left bundle branch block or high-degree atrioventricular block; area under the receiver operatin

106 Atrioventricular blocked dogs were immunosuppressed, ins

107 is of electrocardiographic bundle branch and atrioventricular blocks is not fully understood.

108 s tachycardia and bradycardia, asystole, and atrioventricular blocks) are observed in patients follow

109 cle exit of TBX3+ myocytes in the developing atrioventricular bundle during the period of atrioventri

110 ening and prolonged QRS duration, as well as atrioventricular bundle hypoplasia after birth in hetero

111 ide-gated channel, subtype 4 staining in the atrioventricular bundle, but has no significant effect o

112 in the proximal outflow tract (pOFT) but not atrioventricular canal (AVC) cushions.

113 Proper patterning of the atrioventricular canal (AVC) is essential for delay of e

114 Loss of RhoU function recapitulated the atrioventricular canal and cardiac looping defects obser

115 ing pathways resulted in failure to form the atrioventricular canal and loop the linear heart tube.

116 h homologue dachsous1b resulted in a cardiac atrioventricular canal defect that could be rescued by w

117 Wnt signaling, which has been implicated in atrioventricular canal development (Verhoeven et al., 20

118 rhgef7b/Pak kinase pathway in order to guide atrioventricular canal development and cardiac looping.

119 nd discovered that RhoU was expressed at the atrioventricular canal during the time when it forms.

120 In the mutant, the expressions of the atrioventricular canal marker genes, such as tbx2b, hyal

121 endocardial endothelial cells that line the atrioventricular canal undergo an EndMT to form the endo

122 al cells derived from the endocardium at the atrioventricular canal.

123 , may regulate the expression of RhoU at the atrioventricular canal.

124 s coordinate cell junction formation between atrioventricular cardiomyocytes to promote cell adhesive

125 entricular rate (p < 0.001) and reduced both atrioventricular conduction (PR segment-p = 0.02; PR int

126 on of NKX2-5 is linked to septal defects and atrioventricular conduction abnormalities, early lethali

127 onduction system abnormalities with aberrant atrioventricular conduction and an increased rate of arr

128 F incidence to 0% but had adverse effects on atrioventricular conduction and ventricular repolarizati

129 ate heart rate variability, sinus pause, and atrioventricular conduction block.

130 e that most commonly involves some degree of atrioventricular conduction blockade.

131 owing seizures, SENP2-deficient mice develop atrioventricular conduction blocks and cardiac asystole.

132 entricular arrhythmias, atrial fibrillation, atrioventricular conduction defects, and death by 4 mont

133 tify patients at highest risk for developing atrioventricular conduction disease requiring permanent

134 cations for HBP were sinus node dysfunction, atrioventricular conduction disease, and cardiac resynch

135 ands and family members was characterized by atrioventricular conduction disturbances (61% and 44%, r

136 ate, only a few genes for familial sinus and atrioventricular conduction dysfunction are known, and t

137 RATIONALE: Familial sinus node and atrioventricular conduction dysfunction is a rare disord

138 Transplantation of EECTs in vivo restored atrioventricular conduction in a rat model of complete h

139 of the carboxyl zinc-finger of Gata6 alters atrioventricular conduction in postnatal life as assesse

140 Maximal changes in atrioventricular conduction resulted from more rostral s

141 hat subtle changes in TBX3 expression affect atrioventricular conduction system function.

142 To explore whether and to what extent the atrioventricular conduction system is affected by Tbx3 d

143 as maintained in other tissues including the atrioventricular conduction system, lungs, and liver.

144 d synergize to activate transcription in the atrioventricular conduction system.

145 Overall, the ratio of the change in atrioventricular conduction to the change in heart rate

146 Delivery of 130 Gy caused disturbance of atrioventricular conduction with transition into complet

147 he electrocardiographic PR interval reflects atrioventricular conduction, and is associated with cond

148 All chronic cases demonstrated resumption of atrioventricular conduction, but these required atrial p

149 lrhodopsin-2-expressing macrophages improves atrioventricular conduction, whereas conditional deletio

150 ges and congenital lack of macrophages delay atrioventricular conduction.

151 s, highlighting key regulation processes for atrioventricular conduction.

152 Impaired atrioventricular coupling and lower LA compliance correl

153 However, atrioventricular coupling was impaired and the curviline

154 and diastolic LA chamber stiffness, impaired atrioventricular coupling, and decreased left ventricula

155 lar precursors give rise to the endocardium, atrioventricular cushions and coronary vascular endothel

156 sal and ejection flow would occur at optimal atrioventricular delay (AVD), contributing to its hemody

157 During variation of atrioventricular delay while LV pacing, and ventriculo-v

158 n was compared with the AAI mode at multiple atrioventricular delays (AVD).

159 Three of the 4 tachycardias had atrioventricular dissociation ruling out extranodal acce

160 chycardia was <70 ms in 3, 1 had spontaneous atrioventricular dissociation, and in 1 the atria were d

161 We find that initially atrioventricular endocardial cells migrate collectively

162 ield, pharyngeal endoderm, outflow tract and atrioventricular endocardial cushions and post-migratory

163 curred at right atrium (N=105, 48%) and left atrioventricular groove (N=67, 31%), followed by Bachman

164 s challenging due to the high risk for fatal atrioventricular groove disruption and significant parav

165 he right atrium, Bachmann's bundle, the left atrioventricular groove, and the pulmonary vein area was

166 ventricular function and compression of the atrioventricular groove, which worsened during stress an

167 nt steps for first, second, and third-degree atrioventricular heart block in pediatric patients.

168 ith CTS (p < 0.0001 for atrial fibrillation, atrioventricular heart block, and pacemaker implantation

169 pitated inappropriate depolarizations in the atrioventricular (His)-bundle associated with lethal ven

170 showed major mechanical dyssynchrony at left atrioventricular, interventricular, and left intraventri

171 ither sham irradiation or irradiation of the atrioventricular junction (55, 50, 40, and 25 Gy).

172 evelopmental processes that occur within the atrioventricular junction (AVJ) of the heart: conduction

173 5 Gy were delivered during expiration to the atrioventricular junction (n=5) and left ventricular myo

174 as tracked by pacemaker interrogation in the atrioventricular junction group, time-course magnetic re

175 radiation for catheter-free ablation of the atrioventricular junction in intact pigs.

176 CNTfs were surgically sewn across the right atrioventricular junction in rodents, and acute (n=3) an

177 Structures (atrioventricular junction or left ventricular myocardium

178 Gy were applied in forced-breath-hold to the atrioventricular junction, left atrial pulmonary vein ju

179 uption of cardiac impulse propagation at the atrioventricular junction.

180 , CNTf maintain conduction for 1 month after atrioventricular nodal ablation in the absence of inflam

181 In 5 patients, atrioventricular nodal ablation was performed with direc

182 ing standard HBP procedures or combined with atrioventricular nodal ablation.

183 tes was 10.0 (1.3) mm in patients undergoing atrioventricular nodal ablation.

184 antation and if it was effective in blocking atrioventricular nodal conduction in these patients.

185 is followed by an increase in heart rate and atrioventricular nodal conduction properties and might b

186 in dormant AP conduction times shorter than atrioventricular nodal conduction times before adenosine

187 Of these, 50% recovered atrioventricular nodal conduction within 1 month; 2 pati

188 n the ECG reflects atrial depolarization and atrioventricular nodal delay which can be partially diff

189 The recovery of atrioventricular nodal function after CIHB is high and f

190 ad been either SP ablation (no residual dual atrioventricular nodal physiology) or SP modulation (res

191 Atrioventricular nodal radiofrequency ablation (AVNA) wi

192 evidence that atypical fast-slow and typical atrioventricular nodal re-entrant tachycardia (AVNRT) do

193 ause of its low prevalence, data on atypical atrioventricular nodal reentrant tachycardia (AVNRT) are

194 Atrioventricular nodal reentrant tachycardia (AVNRT) is

195 ting pathway (SP) is treatment of choice for atrioventricular nodal reentrant tachycardia (AVNRT).

196 y was performed on patients with CHD who had atrioventricular nodal reentrant tachycardia and were tr

197 Atrioventricular nodal reentrant tachycardia can complic

198 SR), differentiates NF reentrant tachycardia/atrioventricular nodal reentrant tachycardia from perman

199 The relationship of atrioventricular nodal reentrant tachycardia to congenit

200 Nineteen patients with 20 SVTs (atypical atrioventricular nodal reentrant tachycardia without [n=

201 illation/atrial flutter, atrial tachycardia, atrioventricular nodal reentrant tachycardia, monomorphi

202 receding atrial reset was observed in 98% of atrioventricular nodal reentries during 4+/-1.1 cycles;

203 We analyzed 51 atypical atrioventricular nodal reentry (45% posterior type) and

204 a effectively distinguishes between atypical atrioventricular nodal reentry and atrioventricular reen

205 septal accessory pathways (AP) and atypical atrioventricular nodal reentry can be challenging.

206 n = 25) or focal atrial tachycardia (n = 8), atrioventricular nodal reentry tachycardia (n = 13), ree

207 ar reentry tachycardia (n=104, 90 patients), atrioventricular nodal reentry tachycardia (n=33, 29 pat

208 ia mechanisms were seen in 3 of the 4 cases (atrioventricular nodal reentry tachycardia [2] and atrio

209 coexisted with other tachycardia mechanisms (atrioventricular nodal reentry tachycardia and atriovent

210 ventricular premature beats, atrial flutter, atrioventricular nodal reentry, and atrial tachycardia,

211 eentry tachycardia (n=33, 29 patients), twin atrioventricular nodal tachycardia (n=3, 2 patients), ma

212 emonstrate the feasibility of high frequency atrioventricular-nodal stimulation (AVNS) to reduce the

213 Thirty-minute exposure to MEHP increased the atrioventricular node (147 versus 107 ms) and ventricula

214 The atrioventricular node (AVN) coordinates the timing of at

215 y subepicardially via a thin needle into the atrioventricular node (AVN) region of adult rats to crea

216 of interconnected structures, including the atrioventricular node (AVN), the central connection poin

217 th chronic atrial fibrillation who underwent atrioventricular node ablation and pacemaker implant dem

218 Patients with cAVB were identified by an atrioventricular node ablation or diagnosis of third-deg

219 G telemetry devices underwent radiofrequency atrioventricular node ablation to produce AVB.

220 s, beam positions, and particle numbers) for atrioventricular node ablation was conducted.

221 Atrioventricular node ablation with pacemaker insertion

222 gation, we tested the feasibility of in vivo atrioventricular node ablation, in Langendorff-perfused

223 ial fibrillation should be considered before atrioventricular node ablation.

224 entricular response rates and have undergone atrioventricular node ablation.

225 ys (AP) are uncommon connections between the atrioventricular node and the fascicles or ventricles.

226 mitral) AT that can be overcome by injecting atrioventricular node blockers and signal averaging, res

227 lopment and postnatal function of the murine atrioventricular node by promoting cell-cycle exit of sp

228 tly higher expressed in the right atrium and atrioventricular node compared with left ventricle (P=5.

229 ablation of the right inferior extension of atrioventricular node in 3 cases and by observing a VA i

230 hmias (bradyarrhythmias, atrial tachycardia, atrioventricular node reentrant tachycardia) are signifi

231 Current maneuvers for differentiation of atrioventricular node reentry tachycardia (AVNRT) and at

232 The atrioventricular node size was unaffected.

233 atrioventricular bundle during the period of atrioventricular node specification, which results in fe

234 d channel, subtype 4 staining in the compact atrioventricular node with some retention of hyperpolari

235 ate electrical conduction through the distal atrioventricular node, where conducting cells densely in

236 t half maximum of 10 mm was delivered to the atrioventricular node.

237 the remaining 7 for pathway proximity to the atrioventricular node.

238 ding: Zone I-sinoatrial node region; Zone II-atrioventricular node/His region; and Zone III-bundle br

239 All dogs had atrioventricular-node ablation and ventricular pacemaker

240 via an accessory pathway (n = 4) or via twin atrioventricular nodes (n = 4), ventricular tachycardia

241 Transcriptomic analysis of atrioventricular nodes isolated by laser capture microdi

242 Notably, Tbx3 (+/-) atrioventricular nodes showed increased expression of wo

243 The APs were not atrioventricular pathways because the septal VA interval

244 cating tachycardia [ORT] using a decremental atrioventricular [permanent form of junctional reciproca

245 including distal conduction disease, AF, and atrioventricular pre-excitation.

246 RR interval was not available) or inducible atrioventricular re-entrant tachycardia.

247 entricular nodal reentry tachycardia [2] and atrioventricular reentrant tachycardia [1]).

248 ay antegrade refractory period (P<0.001) and atrioventricular reentrant tachycardia initiating atrial

249 Atrioventricular reentrant tachycardia is common in chil

250 atypical atrioventricular nodal reentry and atrioventricular reentrant tachycardia mediated by septa

251 this phenomena was observed in 6 (8%) of the atrioventricular reentrant tachycardia mediated by septa

252 rioventricular nodal reentry tachycardia and atrioventricular reentrant tachycardia).

253 and 13 had frequent symptomatic episodes of atrioventricular reentrant tachycardia.

254 ricular node reentry tachycardia (AVNRT) and atrioventricular reentry tachycardia (AVRT) lack sensiti

255 Arrhythmia mechanisms included atrioventricular reentry tachycardia (n=104, 90 patients

256 pigenetic data, we identified Tbx3-dependent atrioventricular regulatory DNA elements (REs) on a geno

257 We also show that Glis1 knockdown causes atrioventricular regurgitation in developing hearts in z

258 In adult mammalian hearts, atrioventricular rings (AVRs) surround the atrial orific

259 n shown to result in abnormal development of atrioventricular septa.

260 and management of right dominant unbalanced atrioventricular septal defect (AVSD) remains challengin

261 duals and their affected offspring shared an atrioventricular septal defect or a common atrium along

262 thalamic hamartoma and microcephaly), heart (atrioventricular septal defect), skeleton (postaxial pol

263 of severe CHDs including Ebstein's anomaly, atrioventricular septal defect, and others.

264 Atrioventricular septal defects (AVSD) are a severe cong

265 Atrioventricular septal defects (AVSDs) are a common sev

266 Nonsyndromic atrioventricular septal defects (AVSDs) are an important

267 Also detected by micro-CT were atrioventricular septal defects (n=22), tricuspid hypopl

268 Conotruncal defects and atrioventricular septal defects are over-represented in

269 mmon CHD observed, whereas outflow tract and atrioventricular septal defects were the most prevalent

270 th a pleitropic syndrome of progressive RCM, atrioventricular septal defects, and a high prevalence o

271 ng with diverse cardiac anomalies, including atrioventricular septal defects, Ebstein malformation of

272 enchymal protrusion, and partially penetrant atrioventricular septal defects, including ostium primum

273 both Hoxb1 and its paralog Hoxa1 results in atrioventricular septal defects.

274 d heart field (SHF) are required for OFT and atrioventricular septation and OFT alignment.

275 cardium with reduced cell proliferation, and atrioventricular septation defects similar to Gata4;Tbx5

276 ocyte proliferation, outflow tract (OFT) and atrioventricular septation, and OFT alignment.

277 nts that are essential for outflow tract and atrioventricular septation.

278 The atrioventricular septum was mapped via EAM, and His bund

279 t 25 years of age for patients with a common atrioventricular, single tricuspid, single mitral, and 2

280 We identified an atrioventricular-specific enhancer and a pan-cardiac enh

281 accessibility profiles (ATAC sequencing) of atrioventricular tissue and other epigenetic data, we id

282 ive small GTPases, RhoA and Rac1, coordinate atrioventricular valve (AV) differentiation and morphoge

283 1 is necessary for proper development of the atrioventricular valve (AV).

284 monary bypass time, operation prior to 1991, atrioventricular valve (AVV) replacement at the time of

285 a low incidence of semilunar valve defects, atrioventricular valve defects and double outlet right v

286 Atrioventricular valve failure (moderate or greater regu

287 This study determined the incidence of atrioventricular valve failure and its clinical impact o

288 The cumulative incidence of atrioventricular valve failure at 25 years of age for pa

289 Atrioventricular valve failure occurs frequently in pati

290 versely affects transplant-free survival and atrioventricular valve function.

291 ediating intracellular kinase activation for atrioventricular valve morphogenesis using well defined

292 quent impact on transplant-free survival and atrioventricular valve regurgitation (AVVR) as well as t

293 irculatory failure, ventricular dysfunction, atrioventricular valve regurgitation, arrhythmia, protei

294 rtension, pulmonary regurgitation, pulmonary atrioventricular valve regurgitation, pulmonary and syst

295 own of the ortholog in zebrafish resulted in atrioventricular valve regurgitation.

296 had a single mitral valve, 130 had a common atrioventricular valve, and 97 had a single tricuspid va

297 Using the zebrafish atrioventricular valve, we focus on the valve interstiti

298 rings (AVRs) surround the atrial orifices of atrioventricular valves and are hotbed of ectopic activi

299 ular, single tricuspid, single mitral, and 2 atrioventricular valves was 56% (95% confidence interval

300 Six hundred eighty-six patients had 2 atrioventricular valves, 286 had a single mitral valve,