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1 ube, and communicate during formation of the atrioventricular canal.
2 al cells derived from the endocardium at the atrioventricular canal.
3 at populated the pulmonary veins, atria, and atrioventricular canal.
4 ssion of versican and bmp4 to the developing atrioventricular canal.
5 gulating the morphogenesis of the RV and the atrioventricular canal.
6 rdium of the developing heart, including the atrioventricular canal.
7 al cells in the outflow tract but not in the atrioventricular canal.
8 ntricle and outflow tract in addition to the atrioventricular canal.
9 s resolve to VC identity within the expanded atrioventricular canal.
10 , may regulate the expression of RhoU at the atrioventricular canal.
11                                We identified atrioventricular canal 1 (avc1), a mouse mutation that c
12          Mutant ybx1 hearts have an expanded atrioventricular canal, abnormal sino-atrial valves and
13      Loss of RhoU function recapitulated the atrioventricular canal and cardiac looping defects obser
14 ions like ventricular septal defects, common atrioventricular canal and double outlet right ventricle
15 is required for the proper remodeling of the atrioventricular canal and for cardiac looping, and that
16 icantly enlarged endocardial cushions in the atrioventricular canal and in the outflow tract.
17 essed in the outflow tract, inner curvature, atrioventricular canal and inflow tract, corresponding t
18 first expressed in the epicardium around the atrioventricular canal and later becomes localized mainl
19 ing pathways resulted in failure to form the atrioventricular canal and loop the linear heart tube.
20 ndocardial-mesenchymal transformation in the atrioventricular canal and outflow tract regions.
21  from E9.5 to E11.5 in endocardium along the atrioventricular canal and outflow tract.
22 e outflow tract, the interatrial groove, the atrioventricular canal and right and left ventricles, as
23 l defects are observed in development of the atrioventricular canal and septation of the outflow trac
24 n the myocardial cells of the outflow tract, atrioventricular canal, and future right ventricle.
25 zed by a thin ventricular myocardium, common atrioventricular canal, and the tetralogy of Fallot malf
26 l-to-mesenchymal transdifferentiation in the atrioventricular canal, and thus play a key role in form
27 highest intensity in the right ventricle and atrioventricular canal, as well as in the sinus venosus.
28 ed to repress chamber differentiation in the atrioventricular canal at 9.5 dpc.
29  marking experiments in ovo suggest that the atrioventricular canal, atria and conotruncus are added
30 chick heart, in the myocardium overlying the atrioventricular canal (AV) and outflow tract (OFT) cush
31 s more cranial in the pSHF contribute to the atrioventricular canal (AVC) and atria, whereas those mo
32 ital malformations involving the non-chamber atrioventricular canal (AVC) and inner curvature (IC) re
33 in the proximal outflow tract (pOFT) but not atrioventricular canal (AVC) cushions.
34 th an approximately 1,000-fold enrichment of atrioventricular canal (AVC) defects that disrupt the ju
35   Alk3 CKO embryos also displayed defects in atrioventricular canal (AVC) endocardial cushion formati
36 lls (EMT) at the outflow tract (OFT) but not atrioventricular canal (AVC) endocardial cushions.
37  embryogenesis, the formation of the cardiac atrioventricular canal (AVC) facilitates the transition
38                                   First, the atrioventricular canal (AVC) is closed by the mechanical
39                     Proper patterning of the atrioventricular canal (AVC) is essential for delay of e
40 d that cellularized cushions of the superior atrioventricular canal (AVC) morph into valve leaflets v
41 lium-mesenchymal-transformation (EMT) in the atrioventricular canal (AVC) region; the number of mesen
42 ise from the endocardial cushions within the atrioventricular canal (AVC) through dynamic interaction
43 e, we studied the formation of the zebrafish atrioventricular canal (AVC) where cardiac valves develo
44 cardiographic variables of unbalanced common atrioventricular canal (CAVC) that could aid in appropri
45                     These embryos had common atrioventricular canal (CAVC), double outlet right ventr
46 tation frozen ventricle (frv) causes ectopic atrioventricular canal characteristics in the ventricula
47              Snai1 overexpression studies in atrioventricular canal collagen I gel explants indicate
48 common human congenital heart abnormalities, atrioventricular canal defect (AVCD).
49 h homologue dachsous1b resulted in a cardiac atrioventricular canal defect that could be rescued by w
50                                              Atrioventricular canal defects (AVCDs) are most commonly
51  Wnt signaling, which has been implicated in atrioventricular canal development (Verhoeven et al., 20
52 rhgef7b/Pak kinase pathway in order to guide atrioventricular canal development and cardiac looping.
53 a role of frv in the regional restriction of atrioventricular canal differentiation.
54 nd discovered that RhoU was expressed at the atrioventricular canal during the time when it forms.
55             As Wnt signaling is critical for atrioventricular canal formation, bone health, and pulmo
56 rs of death include the presence of complete atrioventricular canal (hazard ratio 4.76, 95% CI 1.59 t
57 r, conduction is markedly slowed through the atrioventricular canal in the e.d. 10.5 heart, forming t
58 ocardial cells at the ventricular end of the atrioventricular canal, intensifies and extends from E9.
59 , contributing exclusively to left ventricle/atrioventricular canal (LV/AVC) or atrial myocytes.
60        In the mutant, the expressions of the atrioventricular canal marker genes, such as tbx2b, hyal
61 dant function in the endocardium to regulate atrioventricular canal morphogenesis and outflow tract f
62  = 433), pulmonary atresia (n = 121), common atrioventricular canal (n = 17), absent pulmonary valve
63 efect (n = 11), tetralogy of Fallot (n = 3), atrioventricular canal (n = 3), and others (n = 8).
64 irectly represses Nmyc1 in outflow tract and atrioventricular canal of the developing heart, resultin
65  lacZ expression in mesenchymal cells of the atrioventricular canal of Tie2-Cre;CAG-CAT-Z double-tran
66 rst intron, was expressed exclusively in the atrioventricular canal region of the heart.
67 lly localized to the dorsal venous valve and atrioventricular canal regions.
68 uence, the cardiac chambers twist around the atrioventricular canal resulting in torsion of the heart
69                                       In the atrioventricular canal, Tbx5 haploinsufficiency caused a
70  is normally restricted to outflow tract and atrioventricular canal, throughout the heart.
71            Endothelial cells emerge from the atrioventricular canal to form coronary blood vessels in
72  endocardial endothelial cells that line the atrioventricular canal undergo an EndMT to form the endo
73 d population of endocardial cells within the atrioventricular canal undergoes an endothelial-to-mesen
74 involves twisting of the chambers around the atrioventricular canal, which requires correct tissue pa