ライフサイエンスコーパス: atrioventricular conduction

1 s, highlighting key regulation processes for atrioventricular conduction.

2 ges and congenital lack of macrophages delay atrioventricular conduction.

3 ncreased left ventricle diameter and delayed atrioventricular conduction.

4 (-15)) with PR interval, a marker of cardiac atrioventricular conduction.

5 ed interatrial conduction without disturbing atrioventricular conduction.

6 ailure, left bundle branch block, and normal atrioventricular conduction.

7 e), while menthol uniquely alters atrial and atrioventricular conduction.

8 us associated with cardiac malformations and atrioventricular conduction abnormalities was mapped to

9 on of NKX2-5 is linked to septal defects and atrioventricular conduction abnormalities, early lethali

10 TAVI is associated with atrioventricular-conduction abnormalities requiring PPM

11 tion during arrhythmias, including accessory atrioventricular conduction activation.

12 onduction system abnormalities with aberrant atrioventricular conduction and an increased rate of arr

13 imaging, to assess Ca(2+) handling, revealed atrioventricular conduction and excitation-contraction w

14 ein kinase (AMPK), most notably, accelerated atrioventricular conduction and impaired AMPK activation

15 T in patients with heart failure with intact atrioventricular conduction and left bundle branch block

16 Galphai2 overexpression suppressed baseline atrioventricular conduction and slowed the heart rate du

17 F incidence to 0% but had adverse effects on atrioventricular conduction and ventricular repolarizati

18 065 patients with sinus-node disease, intact atrioventricular conduction, and a normal QRS interval t

19 he electrocardiographic PR interval reflects atrioventricular conduction, and is associated with cond

20 ophysiology in humans, including heart rate, atrioventricular conduction, and ventricular repolarizat

21 We conclude that while CVMs controlling atrioventricular conduction are distributed with a peak

22 Rapid atrial rates and intrinsic atrioventricular conduction, as well as congenital mitra

23 ough the inhomogeneous and extremely complex atrioventricular conduction axis is much deeper than it

24 e of permanent pacemaker (PPM) placement for atrioventricular conduction block (AV block) after opera

25 Permanent Pacemaker Secondary to High-Grade Atrioventricular Conduction Block After TAVI) is a prosp

26 Atrioventricular conduction block and arrhythmias caused

27 Atrioventricular conduction block is a complication of o

28 bserved change in rhythm including temporary atrioventricular conduction block or tachycardia termina

29 ay contribute to paravalvular regurgitation, atrioventricular conduction block, and mitral or coronar

30 onduction-system disease (sinus bradycardia, atrioventricular conduction block, or atrial arrhythmias

31 ate heart rate variability, sinus pause, and atrioventricular conduction block.

32 itation (Wolff-Parkinson-White syndrome) and atrioventricular conduction block.

33 e that most commonly involves some degree of atrioventricular conduction blockade.

34 dysfunction manifested by atropine-sensitive atrioventricular conduction blocks and bradycardia that

35 owing seizures, SENP2-deficient mice develop atrioventricular conduction blocks and cardiac asystole.

36 All chronic cases demonstrated resumption of atrioventricular conduction, but these required atrial p

37 manifestations of neonatal lupus, comprising atrioventricular conduction defects and cardiomyopathy,

38 nerative failure characterized by persistent atrioventricular conduction defects and contractile dysf

39 he cardiac transcription factor Nkx2-5 cause atrioventricular conduction defects in humans by unknown

40 entricular arrhythmias, atrial fibrillation, atrioventricular conduction defects, and death by 4 mont

41 tify patients at highest risk for developing atrioventricular conduction disease requiring permanent

42 cations for HBP were sinus node dysfunction, atrioventricular conduction disease, and cardiac resynch

43 ands and family members was characterized by atrioventricular conduction disturbances (61% and 44%, r

44 high incidence of atrial extra systoles and atrioventricular conduction disturbances in Holt-Oram sy

45 omposite of new-onset atrial fibrillation or atrioventricular conduction disturbances requiring the i

46 of the homeodomain, were not associated with atrioventricular conduction disturbances, and were not f

47 d for patients with underlying sinus node or atrioventricular conduction disturbances, for patients w

48 Patients with DM develop atrioventricular conduction disturbances, the principal

49 5 in the myocardium leads to prolongation of atrioventricular conduction, due in part to activation o

50 1) diverse atrial rhythms and mechanisms of atrioventricular conduction during 2 degrees AVB; 2) jun

51 ate, only a few genes for familial sinus and atrioventricular conduction dysfunction are known, and t

52 RATIONALE: Familial sinus node and atrioventricular conduction dysfunction is a rare disord

53 ular response since many drugs that decrease atrioventricular conduction have negative inotropic effe

54 praventricular tachycardia mechanism was 1:1 atrioventricular conduction in 22 patients and supravent

55 Transplantation of EECTs in vivo restored atrioventricular conduction in a rat model of complete h

56 of the carboxyl zinc-finger of Gata6 alters atrioventricular conduction in postnatal life as assesse

57 tionally selective effects on heart rate and atrioventricular conduction in the rat.

58 adycardia (n=1), and atrial flutter with 1:1 atrioventricular conduction (n=1); none required treatme

59 one made during pacing to measure changes in atrioventricular conduction (P-R interval) independent o

60 ysiological testing demonstrated alternative atrioventricular conduction pathways consistent with WPW

61 l [CI] = 1.3 to 5.1; p trend <0.001), slower atrioventricular conduction (PR interval +7.2 ms, 95% CI

62 electrocardiographic measures of heart rate, atrioventricular conduction (PR interval), ventricular r

63 entricular rate (p < 0.001) and reduced both atrioventricular conduction (PR segment-p = 0.02; PR int

64 f new pacemaker features designed to promote atrioventricular conduction, preserve ventricular conduc

65 2 +/- 1.7 RF applications) without affecting atrioventricular conduction properties.

66 Maximal changes in atrioventricular conduction resulted from more rostral s

67 stem for ablating CMs that reside within the atrioventricular conduction system (AVCS).

68 hat subtle changes in TBX3 expression affect atrioventricular conduction system function.

69 To explore whether and to what extent the atrioventricular conduction system is affected by Tbx3 d

70 as maintained in other tissues including the atrioventricular conduction system, lungs, and liver.

71 d synergize to activate transcription in the atrioventricular conduction system.

72 he inner AV canal muscle layer at e.d. 13.5, atrioventricular conduction through the canal was abolis

73 motropic state of the heart (P-R interval or atrioventricular conduction time) in response to selecti

74 and aortic bodies caused an increase in the atrioventricular conduction time.

75 We demonstrate that a ring of atrioventricular conduction tissue develops at 40 hours

76 notch1b are necessary for the development of atrioventricular conduction tissue.

77 Overall, the ratio of the change in atrioventricular conduction to the change in heart rate

78 34) with a dual-chamber device and preserved atrioventricular conduction underwent CMR in 2 asynchron

79 electrophysiology study-derived measures of atrioventricular conduction were the most significant pr

80 lrhodopsin-2-expressing macrophages improves atrioventricular conduction, whereas conditional deletio

81 He is remembered for his insight into atrioventricular conduction, which is as valid today as

82 l-chamber asynchronous) to compare intrinsic atrioventricular conduction with forced RV pacing.

83 Delivery of 130 Gy caused disturbance of atrioventricular conduction with transition into complet