戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ion of attacks and nature of the most recent attack).
2 and drag polar diagrams at various angles of attack.
3 d activation of TNLs in response to pathogen attack.
4 hways are induced to ultimately fend off the attack.
5 recent ischemic stroke or transient ischemic attack.
6 e activity of T cells and prevent autoimmune attack.
7 males and fpC1 in females, control dimorphic attack.
8  to environmental inputs, including pathogen attack.
9 ody to reactivate T cells function for tumor attack.
10  successfully an organism can defend against attack.
11 ractions in the case of a syn-facial hydride attack.
12 nabling mechanosensitive-triggered predatory attack.
13 ants, and participants with a previous heart attack.
14  the feasibility of the initial nucleophilic attack.
15 an attack, and follow-up EDSS 6 months after attack.
16 cells invade only the stroma during an acute attack.
17  NMDAR-dependent synaptic potentiation after attack.
18 ous sources of immunity to mitigate parasite attack.
19 al cleavage sites en masse from ribonuclease attack.
20 s inhibitor that "kills" PLA2 upon enzymatic attack.
21 om the predator once it has committed to its attack.
22  faster and/or stronger response to pathogen attack.
23 ommon ATG16L1 variant from allogeneic T-cell attack.
24 es that protect them against imminent immune attack.
25 routinely be interpreted as part of a single attack.
26 forts to sensitize tumors to immune-mediated attack.
27 e bias to influence the site of nucleophilic attack.
28  after ischemic stroke or transient ischemic attack.
29 enicity but also enhance macrophage-mediated attack.
30 ignal and coordinates a discrete action: the attack.
31 t no subjects had side effect such as asthma attack.
32 lar disease epidemiology would prevent heart attacks.
33  and increases chances of surviving predator attacks.
34 ience against both local targeted and global attacks.
35 cryptographic keys are at risk of extraction attacks.
36   Influenza infection is a trigger of asthma attacks.
37  the host environment and counter the immune attacks.
38 eacon participants against re-identification attacks.
39 at all showed a benefit in preventing future attacks.
40 he privacy of participants against inference attacks.
41 ited during intermale aggression and promote attacks.
42 tepped down without any recurrence of asthma attacks.
43 ying bodily damage caused by failed predator attacks.
44 for controlling asthma and preventing asthma attacks.
45 forming "corpse barriers" that block further attacks.
46 e prophylaxis of hereditary angioedema (HAE) attacks.
47  re-identification (or membership inference) attacks.
48 eptual model for the progression of migraine attacks.
49  community (Estonia), hardly any models were attacked.
50 ment (0.33-0.61 vs 1.66) and moderate/severe attacks (0.31-0.48 vs 1.33, all P <= .001).
51 nute occlusions mimicking transient ischemic attacks (14/19 vs 6/18 with vehicle, relative risk = 2.2
52 pulation included 6 patients during an acute attack, 18 patients in the chronic phase, and 1 patient
53 e feeding, in predation, and in defence when attacked [4].
54 98 (88%) patients experienced a total of 483 attacks, 77% of which required treatment at a health car
55 t mRNA processing and transcription, thereby attacking a cancer vulnerability, transcriptional addict
56               lPAG neurons are selective for attack action and exhibit short-latency, time-locked spi
57  to 6, previous stroke or transient ischemic attack, age >=75, and no previous coronary artery diseas
58              The risk of transient ischaemic attack also seems to be increased in people with migrain
59  with advanced sycamore budburst, parasitoid attack and (marginally) D. platanoidis emergence.
60  exhibit increased resistance to host immune attack and are proposed to contribute to the latency of
61 as defined as the difference in EDSS between attack and follow-up.
62 -host interactions exploitable for antiviral attack and holds promise for further drug development.
63 r disease and the predominant cause of heart attack and ischemic stroke.
64 olling the face selectivity of both chlorine attack and lactone closure.
65 lives of such labile ligands due to nuclease attack and limited cellular uptake due to their structur
66 rrespective of age, with transient ischaemic attack and minor ischaemic stroke occurring between Marc
67  cohort of patients with transient ischaemic attack and minor stroke on intensive medical management.
68 nts with recent stroke or transient ischemic attack and no known coronary heart disease.
69 ns resulting in microbial growth, phagocytic attack and secretion of host antimicrobial factors.
70 n acute cardiovascular events, such as heart attack and stroke.
71 erosclerosis is the process underlying heart attack and stroke.
72 clinical symptoms such as transient ischemic attack and stroke.
73 t common cause of vascular death after heart attack and stroke.
74 cluding both the chance of the fly surviving attack and the relative fecundity/fertility of the survi
75 as not due to the ICL labeling or complement attack and was observed after injection of both syngenei
76 ses (i) significant decrease in the power of attacks and (ii) substantial increase in the number of q
77                                    The acute attacks and chronic stromal deposits mainly involve the
78 s act as sentinel cells that sense microbial attacks and initiate a protective immune response and pr
79  factors (previous therapy for prevention of attacks and nature of the most recent attack).
80 tivity greatly influences the probability of attacks and that flying rapidly is effective for escapin
81  organ in the face of immune and therapeutic attacks and various other stresses.
82  with steroids and plasma exchange for acute attacks and with immunosuppressants (in particular, B ce
83 es (such as coronary heart disease and heart attack) and many beta-adrenoceptor acting drugs are list
84 EDSS) score within 30 days after onset of an attack, and follow-up EDSS 6 months after attack.
85 increased risk of cerebral infarction, heart attack, and reduced cardiac ejection fraction.
86 terations that facilitate escape from immune attack, and sensitivity to PD-1 blockade therapy.
87 ic, 2014 Ebola outbreak, 2001 anthrax letter attacks, and 1984 Rajneeshee Salmonella attacks, the thr
88 ty in research quantifying episodic migraine attacks, and identifying attack onset, a sound theoretic
89 mptomatic in 2 patients; 2 reported sporadic attacks; and 1 reported recurrent attacks (>4 attacks/ye
90 shed by thrusting the flukes while adjusting attack angle with their flippers [1].
91                             Because migraine attacks are hardly predictable and thereby the premonito
92 ulating microparticles during P. vivax acute attacks are indirectly associated with severity.
93 ly that attitudinal changes due to terrorist attacks are more likely to be voiced if norms erode.
94 l ligands facilitates the water nucleophilic attack, as in the case of the highly efficient cobalt po
95 bacterial, fungicidal, virucidal properties, attacking bacteria and destroying the cell membranes.
96 are here chemically engineered to create and attack bulge-loop regions upon hybridization to target R
97 c neuritis and are associated with recurrent attacks, but MOG-IgG optic neuritis has a better visual
98 ketimine, which is subjected to nucleophilic attack by acetoacetate to form the new C(gamma)-C(delta)
99 n individual's ability to avoid or escape an attack by an ambush predator.
100 ce to many environmental stresses, including attack by arthropod herbivores whose feeding activity is
101 plug, and SNF itself, such as radiolysis and attack by constituents of hydrothermal brines, coupled w
102 fer a variety of systems that defend against attack by genetically distinct phages; phages can neutra
103 nts of physical force and to resist external attack by herbivores and pathogens but can in many cases
104          The ability of an insect to survive attack by natural enemies can be modulated by the presen
105 E2 exposing the restrained thioester bond to attack by substrate nucleophile.
106 into new areas, acquire nutrients, and avoid attack by the immune system.
107 ytic mechanism, invoking direct nucleophilic attack by the substrate alpha-amino group on the sn-2 es
108 BPP results revealed class-specific proteins attacked by mHAMs or mHAAs.
109 eavable groups on the backbone, which can be attacked by stimuli in ambient conditions, causing undes
110 ation, deposition of aerosols and particles, attacks by microbes and arthropods.
111  T6SS apparatus only after detecting initial attacks by other bacterial prey cells; this targeted att
112 rived danger signals released in response to attacks by pathogens or herbivores and activate immune r
113 oration of coronary blood flow after a heart attack can cause reperfusion injury potentially leading
114 2 people per 100 000, severe immune-mediated attacks can quickly lead to blindness and paralysis if u
115                               With recurring attacks, central oval stromal opacities accumulated.
116                 Data including demographics, attack characteristics and response to treatments were r
117 nt, painful, and potentially lifethreatening attacks characterized by swelling of subcutaneous or sub
118 nfection had a stroke or transient ischaemic attack, compared with five (6%) of 96 patients with Zika
119                      The complement membrane attack complex (MAC) is classically known as a cytolytic
120 mplement protein C1q, to induce the membrane attack complex and reactive super-oxygen species for pro
121 mplement system involving C1q-C3-C4-membrane attack complex and ROS regulates exosome-mediated, ethan
122                   Historically, the membrane attack complex, composed of complement components C5b-9,
123 uced ethanol-induced C3a/b, C4, and membrane attack complex/C5b9 formations; ROS production; and ulti
124 ant, carrying a S205F mutation in a membrane-attack-complex/perforin (MACPF)-domain protein, harbour
125 to the peptide with adoption of an 'in-line' attack conformation for catalysis.
126 st binding precedes rate-limiting chlorenium attack, controlling the face selectivity of both chlorin
127          Arthropod predators and parasitoids attack crop pests, providing a valuable ecosystem servic
128 se results along with in situ humpback whale attack data to model how predator speed and engulfment t
129  Patients can experience acute neurovisceral attacks, debilitating chronic symptoms, and long-term co
130 xacerbated by a litany of successful privacy attacks demonstrating that traditional statistical discl
131 ction produced by Spiroplasma following wasp attack depended on wasp strain.
132 e conditions, including Breast Cancer, Heart Attack, Diabetes, etc.
133 ape noxious stimuli, such as parasitoid wasp attacks, Drosophila melanogaster larvae generate a curli
134 t the MeApv-VmH synapses selectively control attack duration, whereas the MeApv-BNST synapses modulat
135 rols binding and reveal a site for oxidative attack during catalysis.
136 comes of acute stroke and transient ischemic attack during pregnancy or within 6 weeks of childbirth
137 roxylation and ring cleavage of DOM by HO(.) attack during the AOP and the influence on DBP formation
138 eavage, sigma-bond metathesis, electrophilic attack, etc.
139 e frequency distribution of P. vivax malaria attacks experienced by each individual over 12 months.
140  the ability to withstand targeted or global attacks; extinctions; or shocks is vital to the survival
141 er syndrome, is characterised by sudden pain attacks, followed by patchy muscle paresis in the upper
142 on, whereas the MeApv-BNST synapses modulate attack frequency, both with no effect on social behavior
143                         She had daily asthma attacks from the age of fourteen, and the addition of or
144 oreover, they cooperate, forming "swarms" to attack fungi that are larger than individual neutrophils
145 sequence, in part because RIP preferentially attacks GC-poor long duplicates that interact in three d
146 H analysis expands Woerpel's "inside/outside attack" glycosylation model by considering the stereoele
147 d sporadic attacks; and 1 reported recurrent attacks (&gt;4 attacks/year).
148 ately, proposed countermeasures against such attacks have failed to be effective, as they do not cons
149 ry heart disease, stroke, transient ischemic attack, heart failure hospitalization, cardiac revascula
150 rforms an I See You (ISY) signal that deters attacking hornets.
151  modeled as a chemical arms race of pathogen attack, host chemical counterattack, and pathogen defens
152 old temperatures, since they are confined to attacking hosts pupating and overwintering in relatively
153                             During the acute attack, hyperreflective cellular structures consistent w
154 nd are hypothesized to be the site of immune attack in AA.
155 were assayed for aggressiveness (tendency to attack in resident-intruder tests) and then experienced
156 ibition restores tumor sensitivity to T cell attack in vitro and in melanoma xenografts.
157 te relapses and for recovery from autoimmune attacks in the CNS.
158 cy and at least 2 investigator-confirmed HAE attacks in the first 56 days of a prospective run-in per
159  porphyria diagnosis and had experienced >=3 attacks in the prior 12 months or were receiving prophyl
160                              After two heart attacks in three years, an associate professor discusses
161 ischemic and hemorrhagic)/transient ischemic attack incidence and in-hospital mortality were extracte
162 gy, proximity to the substrate, and angle of attack (inclination).
163 n validated for the measurement of the heart attack indicator cardiac troponin I and is shown to succ
164 uding cardiovascular diseases (such as heart attacks), injuries, mental and behavioral disorders, and
165  and a positive control (emamectin benzoate 'Attack' insecticide).
166 indispensable element for the initial allene attack involving allenic C(sp(3))-H bond cleavage, but i
167 xygen species generated by respiratory burst attack iron-sulfur cluster-containing proteins, includin
168 -initiated photooxidation, wherein oxidative attack is powered by photogenerated and electrically-inj
169       We found that predators took longer to attack larvae that were resting in a twig-like posture t
170 s in the probe compounds for the monodentate attack leading to an ozone adduct.
171 rosophila melanogaster varies with strain of attacking Leptopilina heterotoma wasp.
172 from females and either AR is sufficient for attacking males.
173 lnerability, but hydraulic failure or biotic attack may dominate the process during the end stages of
174 d endogenous sources of immunity to parasite attack may represent an underappreciated consequence of
175 ytotoxic effectors, YT cells loaded with GrB attacked MDA-MB-231 target cells, and active GrB influen
176                         However, traditional attack methods to create adversarial examples do not ext
177  prominent cranial autonomic features during attacks (miosis OR 11.24, 95% CI 3.21 to 41.34; eyelid o
178 e Effects of ACT-246475 in Adults With Heart Attack; NCT03487445, 2018-000765-36 [EudraCT]).
179 cy for treating these malignancies, but also attack normal B cells leading to immune deficiency.
180   In 8 enrolled patients, the mean number of attacks normalized per month was lower during C1-INH tre
181  (11 steps, 46% global) are the nucleophilic attack of a hindered tertiary alkoxide, a ring-closing m
182 ternative outer-sphere process, nucleophilic attack of a metal-phosphido (M-PR(2)) group on an unsatu
183 n of methylenecyclopropanes and nucleophilic attack of an enamine to realize the construction of spir
184 yndrome (CIS) patients present with a single attack of inflammatory demyelination of the central nerv
185 ituents, due to reversibility of the initial attack of the amines at the electrophiles followed by ra
186 became faster, which allowed the rate of the attack of the amines at the electrophiles to be determin
187                    However, the nucleophilic attack of the anion on carbonates induces an oxidative b
188 4% positive effect of the symbiont following attack of the fly host by the Lh14 strain of wasp to 21%
189 lation reaction takes place via nucleophilic attack of the terminal alkyne to the C2 carbon of the ac
190 he gas phase and occurs through nucleophilic attack of water on the carbonyl carbon.
191 ological autoimmune disease characterized by attacks of CNS inflammation that are often severe and pr
192  Our findings have parallels to the suicidal attacks of social insects [6-9], which are also performe
193                                    Terrorist attacks often fuel online hate and increase the expressi
194                         Patients experienced attacks often requiring treatment in a health care facil
195 ioxide to give a bicyclic product by P-CH(2) attack on CO(2) combined with internal P=O to boron coor
196  demonstrate the feasibility of an inference attack on differentially private query results by utiliz
197 the incidence or severity of microbiological attack on exposed timber and have significant impact on
198 ate that infection with MHV induces a severe attack on host cell NAD(+) and NADP(+) Finally, we show
199  unimolecular ones involving stereoselective attack on naked glycosyl oxocarbenium ions.
200 catalytic activity for tyrosine nucleophilic attack on phosphorus.
201  is rate-determining, and reaction occurs by attack on the central carbon of the keteniminium cation
202 antibiotic resistance genes and initiate its attack on the human host.
203 cial interactions among predators can reduce attacks on aposematic prey and therefore influence selec
204  We tested a simple Markov model of migraine attacks on headache diary data and estimated transition
205  found larger nesting clusters and decreased attacks on outgroup competitors in the Bonobo condition
206 earch has focused on the impact of terrorist attacks on prejudiced attitudes toward groups linked to
207  man had been suffering from recurrent fever attack once in 1-2 months during childhood.
208 g episodic migraine attacks, and identifying attack onset, a sound theoretical model of a migraine at
209 lanocyte stem cells is independent of immune attack or adrenal stress hormones.
210 ompounds to protect themselves from pathogen attack or herbivore predation.
211 ad experienced at least one documented NMOSD attack or relapse in the past 12 months and had a score
212  ischemia-reperfusion injury (IRI), in heart attack or stroke settings, but also as an unavoidable co
213 er the Plesioteuthis died from the pterosaur attack or survived for some time with the broken tooth l
214  = 0.35, 95% CI = 0.13-0.90), fewer wheezing attacks (OR = 0.40, 95% CI = 0.17-0.97; >3 vs <=3 times/
215  event (either stroke or transient ischaemic attack) or a further ICH following study entry.
216 arotid revascularization, transient ischemic attack, or stroke.
217 emonstrated improvement in EDSS following an attack (P = 0.006).
218 set, a sound theoretical model of a migraine attack, paired with a uniform standard for counting them
219 epend on the interplay of symbiont, host and attacking parasite genotypes.
220 ultiprotein complexes, called supramolecular attack particles (SMAPs), from CTLs to target cells.
221 specialized structures termed supramolecular attack particles (SMAPs).
222 r month; P < .001) relative to placebo (2.35 attacks per month).
223 ttacks per month; P = .024) and 150 mg (1.31 attacks per month; P < .001) relative to placebo (2.35 a
224 eduction in attack rate at both 110 mg (1.65 attacks per month; P = .024) and 150 mg (1.31 attacks pe
225 sociated with the coleoid when the pterosaur attacked Plesioteuthis at or near the water surface.
226 ent work has pinpointed the enzymes that ROS attack, plus an array of clever protective strategies th
227 cluding the discovery of guidance rules that attacking predators use to intercept mobile prey, and co
228 ities when live prey interact with ballistic attacking predators, and find this diversion effect sign
229 (in particular, B cell-depleting agents) for attack prevention has greatly improved the long-term out
230                                 Although the attack-prevention treatment regimens were considerably d
231                         Although dragonflies attack prey dorsally, we show that demoiselles attack pr
232 tack prey dorsally, we show that demoiselles attack prey frontally.
233 in administration (median [range] annualized attack rate 2.0 [0.0-37.0]).
234 transmission (P = .009) and higher secondary attack rate among household contacts (P = .03), after ad
235 stat demonstrated a significant reduction in attack rate at both 110 mg (1.65 attacks per month; P =
236               Least-squares mean monthly HAE attack rate for lanadelumab was compared with placebo.
237 e and for antibody waning, we estimate a 66% attack rate in June, rising to 76% in October.
238 ing the index case), and 1 in Spain (overall attack rate in the chalet: 75%).
239 ted during the follow-up period, yielding an attack rate on board of 85.2% (104/122 individuals).
240                                          The attack rate there is an estimate of the final size of th
241         Lanadelumab efficacy was durable-HAE attack rate was consistently lower vs placebo, from the
242               During days 0-69, mean monthly attack rate was significantly lower with lanadelumab (0.
243                          The safety profile, attack rate, and duration of illness make GI.1 Lot 001-0
244 2 on a fishing vessel associated with a high attack rate.
245 t rule of thumb based on household secondary attack rate.
246 accompanied by a reduction in the angioedema attack rate.
247  prey activity, and rapid locomotion reduces attack rates and increases chances of surviving predator
248 and 150-mg doses of berotralstat reduced HAE attack rates compared with placebo and were safe and gen
249  researchers have shown that crypsis reduces attack rates on resting prey, predation risk increases w
250 r time, and crowded cities have larger total attack rates than less populated cities.
251 pathogens with variable seasonal phasing and attack rates, most existing process-based forecasting sy
252 ehaviour in response to a simulated predator attack relative to grandoffspring of control, unexposed
253 mab (0.41-0.76) vs placebo (2.04), including attacks requiring acute treatment (0.33-0.61 vs 1.66) an
254 oximate the progression of observed migraine attacks satisfactorily, and imputing on migraine-locked
255    Patients with AHP, regardless of clinical attacks, should be screened for HCC, beginning at age 50
256  Phages tend to be predators finely tuned to attack specific hosts, even down to the strain level, wh
257 ation ranging from autoimmune disease, which attacks specific tissues, to autoinflammatory diseases,
258 by other bacterial prey cells; this targeted attack strategy has been termed the T6SS tit-for-tat res
259  composite outcome of all-cause death, heart attack, stroke, or hospitalization for heart failure.
260 fe-threatening diseases (e.g., cancer, heart attack, stroke, wound healing).
261 duced host-weakening was important to beetle attack success we used an iso-demographic approach in En
262                           Awareness of heart attack symptoms also declined.
263 ovascular events (stroke, transient ischemic attack, systemic embolism, valve thrombosis, or hospital
264 mes were recurrent stroke/transient ischemic attack/systemic embolism, symptomatic intracerebral hemo
265          beta-Cell stress provokes an immune attack that has considerable negative effects on the sou
266 trong oxidizing power of sMMO-Q necessary to attack the highly inert C-H bonds of methane.
267 er adult female ran to the scene, physically attacked the snake (with bites and hits), and pulled the
268 ogenase as a key target of IAA, specifically attacking the catalytic Cys 152.
269 a critical need for effective treatment that attacks the aberrant kinome activation associated with r
270                                          HIV attacks the body's immune cells, frequently compromises
271                                           CO attacks the carbonyl carbon of acetaldehyde in the coupl
272 tter attacks, and 1984 Rajneeshee Salmonella attacks, the threat of future epidemics/pandemics and/or
273 in understanding how Orobanchaceae parasites attack their hosts and how the hosts mount a defense aga
274                                         Aip1 attacks these hypertwisted regions along their sides, di
275 ents were prior stroke or transient ischemic attack (TIA) (HR 1.42, 95% CI 0.96 to 2.10) and ipsilate
276  stroke risk in high-risk transient ischemic attack (TIA) patients assessed by ABCD2 score.
277 s with ischemic stroke or transient ischemic attack (TIA).
278 ogenic ischemic stroke or transient ischemic attack (TIA).
279 me was ischemic stroke or transient ischemic attack (TIA).
280 ew data in patients with transient ischaemic attack (TIA)/stroke.
281 rboxylated phenyl group makes a nucleophilic attack to form the pyrrole ring of the indole, followed
282 c data obtained from the attribute inference attack to infer the membership of a target in another ge
283 USV(+) mounting, and converted male-directed attack to mounting with USVs.
284 ant transition states indicates that alcohol attack to the more crowded side of the acyl-catalyst int
285 ntrol experiments revealed that nucleophilic attack to the pai-allylpalladium intermediate is the ena
286                  We compared the symptomatic attacks to AQP4-IgG-NMOSD (n=30) and MS (n=30).
287 sis, pulmonary embolism, transitory ischemic attacks to arteritis.
288 point that affects the ability of T-cells to attack tumors.
289 ic for a noncancer antigen are retargeted to attack tumors.
290 nd ionising radiation (IR) monotherapies may attack tumours in dissimilar, and complementary, ways.
291 vascular Events in Patients With Prior Heart Attack Using Ticagrelor Compared to Placebo on a Backgro
292 litus, previous stroke or transient ischemic attack, vascular disease, 65 to 74 years of age, female)
293 ality are frequently susceptible to pathogen attack, whereas enhancement of disease resistance genera
294 e that cryptic species are less likely to be attacked while at rest.
295                  Plants respond to arthropod attack with the rearrangement of their transcriptome whi
296 ort lasting unilateral neuralgiform headache attacks with conjunctival injection and tearing (SUNCT)
297 ort lasting unilateral neuralgiform headache attacks with cranial autonomic symptoms (SUNA).
298 rm prophylaxis and treatment of breakthrough attacks with favourable safety profile in Japanese patie
299  reported a median (range) of 6 (0-52) acute attacks, with 52 (46%) patients receiving hemin prophyla
300 ttacks; and 1 reported recurrent attacks (>4 attacks/year).

 
Page Top