ライフサイエンスコーパス: augmentation index

1 n independent predictor of higher hs-CRP and augmentation index.

2 ts were change in flow-mediated dilation and augmentation index.

3 ere seen in either flow-mediated dilation or augmentation index.

4 increased insulin sensitivity and decreased augmentation index.

5 o influence on augmentation index (change in augmentation index, -0.4%; 95% CI, -1.7 to 0.8; P=0.5) o

6 0.01 to 0.03), p = 0.36; arterial stiffness (augmentation index), -1.1% (-2.5 to 0.3), p = 0.097; sys

7 NO3(-) reduced aortic augmentation index (132.2+/-16.7% versus 141.4+/-21.9%;

8 1%; P = 0.003), systemic arterial stiffness (augmentation index: -2.24%; 95% CI: -3.97%, -0.61%; P =

9 ry flow reserve, (3) pulse wave velocity and augmentation index, (4) circulating NT-proBNP (N-termina

10 +/- 1.0 m/s vs. -0.1 +/- 0.9 m/s, p < 0.01), augmentation index (-5.2 +/- 6.1% vs. -1.4 +/- 5.9%, p <

11 orearm mitochondrial oxidative function, and augmentation index (a marker of arterial wave reflection

12 sterone, and endothelin (ET)-1 together with augmentation index, a measure of arterial stiffness, wer

13 Secondary outcomes included augmentation index, abdominal aortic calcification, BP,

14 The peak reductions in peripheral augmentation index after the WA, AO, and CO meals (-9.5%

15 01), AI d (r = -0.17, P = 0.06), the central augmentation index (AI c ) (r = 0.61, P < 0.001) or AI c

16 AI rd , a combination of AI r and diastolic augmentation index (AI d ) with a weight alpha, to achie

17 Radial augmentation index (AI r ) can be more conveniently meas

18 Augmentation index (AI(a)) and timing of the reflected p

19 tid-femoral pulse wave velocity (cf-PWV) and augmentation index (AI) at a steady state.

20 Central aortic BP and augmentation index (AI) were noninvasively assessed via

21 ity of existing vascular measures, including augmentation index (AI), pulse wave velocity (PWV), and

22 emoral pulse wave velocity (PWV) and carotid augmentation index (AI)].

23 , PWVcf, PWVba) and derivation of the aortic augmentation index (AIao); (b) bilateral cIMT assessment

24 t 3 h (-0.5 +/- 0.2 m/s, P = 0.003), whereas augmentation index (AIx) also improved after 14 d (-7.0

25 ness measures: carotid-femoral (PWV), aortic augmentation index (AIX) and ankle-brachial index (ABI).

26 t the hypothesis that age-related changes in augmentation index (AIx) are more prominent in younger i

27 Augmentation index (AIx) is a measure of systemic arteri

28 Augmentation index (AIx) is widely used as a measure of

29 pressure (BP), pulse wave velocity (PWV) and augmentation index (AIx) were assessed in 130 subjects (

30 Pulse wave velocity (PWV) and the augmentation index (AIx) were assessed noninvasively and

31 and dose on the change in blood pressure and augmentation index (AIx) were determined.

32 s, LV wall thickness and dimensions, central augmentation index (AIx), aortic pulse wave velocity (aP

33 Pulse wave analysis was used to determine augmentation index (AIx), which provides a measure of sy

34 using pulse wave velocity (PWV) and central augmentation index (AIx).

35 emoral pulse wave velocity (PWV), and aortic augmentation index (AIx).

36 alter fasting haemodynamics, it lowered the augmentation index (AIx75, P = 0.024) and increased the

37 % vs. placebo -0.69 +/- 2.8%; p = 0.017) and augmentation index (allopurinol -2.8 +/- 5.1% vs. placeb

38 hanges in air pollution were associated with augmentation index and augmentation pressure at several

39 t-term exposure to air pollution and central augmentation index and augmentation pressure, correlates

40 ate capabilities for measuring radial artery augmentation index and pulse pressure velocity.

41 proved after EVT, with significantly reduced augmentation index and pulse wave velocity and increased

42 elocity and radial tonometry-derived central augmentation index and subendocardial viability ratio we

43 arterial stiffness (pulse wave velocity and augmentation index) and blood pressure were also not sig

44 erial stiffness (pulse wave velocity, aortic augmentation index, and aortic distensibility) at either

45 ntral pulse pressure, augmentation pressure, augmentation index, and mean arterial pressure.

46 tic modulus, impedance, pulse wave velocity, augmentation index, and pulse pressure amplification) ar

47 6 mm Hg (95% confidence interval, 2.4-20.7), augmentation index, and pulse wave velocity without chan

48 unction was measured by arterial compliance, augmentation index, and pulse wave velocity.

49 Brachial stiffness, augmentation index, and systemic arterial compliance wer

50 -femoral pulse wave velocity (cfPWV), aortic augmentation index, and systemic arterial compliance.

51 Augmentation index (as %) and augmentation pressure (in

52 erences in pulse wave velocity at 12 months, augmentation index at 12 months, BP, B-type natriuretic

53 n of either meal significantly decreased the augmentation index at 2 and 4 h (P < 0.002) and signific

54 a and augments pressure in late systole [ie, augmentation index = (augmented pressure/pulse pressure)

55 mpliance estimates but may underestimate the augmentation index because the latter requires greater f

56 ulse wave velocity (beta = -0.09, p = 0.04), augmentation index (beta = -0.11, p = 0.03), and subendo

57 differences in blood lipids, blood pressure, augmentation index, blood glucose, endothelin, adhesion

58 , blood pressure, central (aortic) pressure, augmentation index, blood glucose, endothelin, proprotei

59 artery (CCA-IMT), pulse wave velocity (PWV), augmentation index, blood pressure (BP), and vascular bi

60 arkedly change wave reflection amplitude and augmentation index by altering stiffness of the muscular

61 including pulse wave velocity (PWV), central augmentation index (CAI), and central aortic pressure (C

62 tic impedance [Zc]) and late-systolic loads (augmentation index [cAI]; late pressure-time integral [P

63 ral aortic systolic pulse (CASP) and central augmentation index (cAIx) estimates with precision and a

64 y arterial pulse-wave velocity (Doppler) and augmentation index (carotid tonometry) declined with ver

65 Calculations included the carotid augmentation index, carotid artery compliance and the di

66 pulse wave velocity [PWV]), wave reflection (augmentation index, carotid-brachial pressure amplificat

67 tion, carotid artery intima-media thickness, augmentation index, central blood pressure, subendocardi

68 In contrast, augmentation index, central pulse pressure, and pulse pr

69 -1.9 to 1.0], P=0.6) and had no influence on augmentation index (change in augmentation index, -0.4%;

70 HLS increased the augmentation index compared with the other test meals (P

71 ial stiffness (pulse wave velocity [PWV] and augmentation index corrected for heart rate [AI@75]) wer

72 The secondary outcomes were CFPWV, FWA, and augmentation index during examination cycle 8.

73 ex, height, weight, end-diastolic LV volume, augmentation index, end-systolic pressure, and cardiovas

74 oCor Mx system was used to derive the aortic augmentation index from radial artery pulse pressure wav

75 I >= 1.67 = normal endothelial function) and augmentation index (higher AIx = worse arterial elastici

76 Central augmentation index improved significantly with allopurin

77 sodilatation was calculated as the change in augmentation index in response to an endothelium-depende

78 essure augmentation was determined using the augmentation index in the ascending aorta (AIaa) and dis

79 arterial stiffness (pulse wave velocity and augmentation index) in 20 adult patients with hypertensi

80 Augmentation index increased after transplant and was gr

81 a strong inverse relationship between HR and augmentation index, indicative of increased wave reflect

82 dpoints [including flow-mediated dilatation, augmentation index, lipoprotein status (by nuclear magne

83 levels and 3 measures of vascular function (augmentation index, mean arterial pressure, and pulse pr

84 , 1.6 [95% CI, 1.3-2.0] per 1 SD; P < .001), augmentation index (OR, 1.7 [95% CI, 1.4-2.0] per 1 SD;

85 ention did not significantly change CCA-IMT, augmentation index, or BP, but pulse pressure variabilit

86 eactive protein (P(interaction) < 0.001) and augmentation index (P = 0.06) values at or above the 75t

87 had higher hs-CRP (P=0.014), higher central augmentation index (P=0.015), and lower glutathione leve

88 function (P=0.009), and improved the central augmentation index (P=0.015).

89 lood pressure, mean arterial pressure (MAP), augmentation index, pulse wave velocity (PWV), and intim

90 Augmentation index, pulse wave velocity, and arterial co

91 = [Reflected/Forward wave amplitude] x 100), augmentation index ([Second/First systolic peak] x 100)

92 rterial compliance to 6 +/- 7% accuracy, and augmentation index to within -7% points (30 +/- 45% accu

93 a decrease in pulse wave velocity (PWV) and augmentation index up to 26 h after the walk.

94 nts/cm(3) IQR increase) were associated with augmentation index values that were 0.8% (95% confidence

95 The augmentation index was 6% higher in the EP group and ass

96 In healthy volunteers, PWV and augmentation index were associated both with black carbo

97 is cohort, higher aortic stiffness, FWA, and augmentation index were associated with higher risk of i

98 Pulse wave velocity and augmentation index were improved only after anti-IL-12/2

99 Pulse wave velocity and aortic augmentation index were measured by SphygmoCor Pulse Wav

100 Aortic pulse wave velocity (PWV) and carotid augmentation index were reduced only with SR (p < 0.05).

101 Six months after RD aortic augmentation and augmentation index were significantly reduced by -11 mm

102 y [CFPWV], forward wave amplitude [FWA], and augmentation index) were examined over a 7-year period i

103 ticles and PM2.5, and an increase in PWV and augmentation index with NO2 and ultrafine particles.