ライフサイエンスコーパス: aureus

1 oli and methicillin-resistant Staphylococcus aureus).

2 lorhexidine solutions against E. coli and S. aureus.

3 d from the PLA in children is Staphylococcus aureus.

4 , Pseudomonas aeruginosa, and Staphylococcus aureus.

5 ile and methicillin-resistant Staphylococcus aureus.

6 nt for mild-to-moderate PUs infected with S. aureus.

7 such as methicillin-resistant Staphylococcus aureus.

8 robial diversity and reduced abundance of S. aureus.

9 enes, Salmonella enterica and Staphylococcus aureus.

10 d colonisation of implants by Staphylococcus aureus.

11 , but not glutamate promote the growth of S. aureus.

12 crobial activity against only Staphylococcus aureus.

13 ed with methicillin-resistant Staphylococcus aureus.

14 ith potent anti-virulence effects against S. aureus.

15 TNFR2pos Treg after culture in vitro with S. aureus.

16 in the same way as SstD from Staphylococcus aureus.

17 actors important for host defense against S. aureus.

18 atinocytes exhibited increased binding to S. aureus.

19 issed' PJI at the time of bacteremia with S. aureus (1.1%).

20 reptococcus pneumoniae (33%), followed by S. aureus (22%).

21 he most common pathogens were Staphylococcus aureus (34%) and Pseudomonas aeruginosa (17%), whereas b

22 gets with a PPA of <100% were Staphylococcus aureus (34/37 [91.9%]), Streptococcus pneumoniae (10/11

23 was most frequently caused by Staphylococcus aureus (43%), followed by streptococci (26%) and Gram ne

24 at pathogens encompassing 288 Staphylococcus aureus, 456 Pseudomonas aeruginosa, and 1588 Escherichia

25 s pneumoniae (9/44 [20%]) and Staphylococcus aureus (7/14 [50%]) were the predominant pathogen identi

26 A (97%), followed by P. aeruginosa (81%), S. aureus (79%) and Candida spp (72%), with lower reduction

27 cluding methicillin-resistant Staphylococcus aureus, a common cause of human infections.

28 ntimicrobial activity against Staphylococcus aureus, a common pathogen co-isolated with P. aeruginosa

29 Decreased S. aureus abundance during dupilumab treatment correlated w

30 High baseline S aureus abundance in turn predicts an increase in AD seve

31 or larger studies linking skin pH and skin S aureus abundance to understand driving factors of diseas

32 High S aureus abundance was associated with skin pH 5.7-6.2.

33 encing of 16S ribosomal RNA, and absolute S. aureus abundance was measured by quantitative PCR.

34 Each compound inhibits all S. aureus accessory gene regulator (agr) alleles (IC(50) 2-

35 the effects of type I and type II IFNs on S. aureus adherence and invasion.

36 Staphylococcus aureus adhesion to the host's skin and mucosae enables a

37 ected mice compared to mice infected with S. aureus alone.

38 ected mice compared to mice infected with S. aureus alone.

39 Introducing these mutations into S. aureus alters the ability of the bacterium to resist sup

40 We found that both wild-type S. aureus and a DeltahemB SCV prototype potently activate g

41 pathogenic bacterial species Staphylococcus aureus and antibiotic resistant Acinetobacter baumannii

42 the morphologically distinct Staphylococcus aureus and Bacillus subtilis species, using live cells a

43 some atopic patients can act similarly to S aureus and damage the skin by expression of a cysteine p

44 Staphylococcus aureus and Enterobacterales were the most common pathoge

45 s than half of patients, with Staphylococcus aureus and enterococcus bacteremia associated with worse

46 Staphylococcus aureus and enterococcus had the highest 1-year mortality

47 s treated with valve surgery, Staphylococcus aureus and Enterococcus spp. were associated with valve

48 ly validate sulfur acquisition systems in S. aureus and establish their importance during pathogenesi

49 gate mechanisms of acquired resistance in S. aureus and identify key residues in FabI that stabilize

50 host cell death that failed to eradicate S. aureus and instead promoted DeltahemB SCV pathogenicity.

51 ar cells were stimulated with Staphylococcus aureus and Mycobacterium tuberculosis before, as well as

52 nce factors to the surface of Staphylococcus aureus and other medically significant bacterial pathoge

53 quently, increased opsonic phagocytosis of S aureus and other pathogens.

54 r more than 259 days and 147 days against S. aureus and P. aeruginosa, respectively, compared to 70 d

55 ntibacterial activity against Staphylococcus aureus and Pseudomonas aeruginosa and with their geograp

56 Staphylococcus aureus and Pseudomonas aeruginosa were isolated in 10.5

57 or fatty acid biosynthesis in Staphylococcus aureus and represents a promising target for the develop

58 We sought to characterize S aureus and S epidermidis colonization and biofilm propen

59 mon implicated pathogens were Staphylococcus aureus and S. epidermidis.

60 Staphylococcus aureus and Staphylococcus epidermidis are the most abund

61 ia coli, Salmonella enterica, Staphylococcus aureus and Streptococcus pneumoniae were also isolated.

62 the interactions between S epidermidis and S aureus and that the balance between these two species, a

63 lstein Friesian cows were inoculated with S. aureus and treated intramammarily with vehicle (NEG; day

64 enic microorganisms including Staphylococcus aureus and two Candida strains.

65 pacity of neutrophils to kill Staphylococcus aureus and worse clinical outcomes.

66 learance after infection with Staphylococcus aureus and, by licensing encephalitogenic Th17 cells, pl

67 he ability to inhibit both gram-positive (S. aureus) and gram-negative (E. coli) bacteria on solid an

68 cus species (eg, S epidermidis, S capitis, S aureus), and enrichment in metabolic pathways (eg, branc

69 cens, Salmonella typhimurium, Staphylococcus aureus); and fungal enzymes under acid-stress (Terfezia

70 lomerans, Pseudomonas putida, Staphylococcus aureus, and Bacillus subtilis was observed when the assa

71 our study to a second bacterial pathogen, S. aureus, and demonstrate that CP also inhibits iron uptak

72 bacteria media (blank broth, Staphylococcus aureus, and E. coli).

73 uding Pseudomonas aeruginosa, Staphylococcus aureus, and Escherichia coli We have previously demonstr

74 g Mycobacterium tuberculosis, Staphylococcus aureus, and Escherichia coli, and identify thousands of

75 EOS by group B Streptococcus, Staphylococcus aureus, and Escherichia coli.

76 t antimicrobial activity against E. coli, S. aureus, and S. typhi in in vitro antimicrobial tests, fo

77 Moreover, after epicutaneous Staphylococcus aureus application, impaired S1pr2(-/-) mouse epidermal

78 Pseudomonas aeruginosa and Staphylococcus aureus are opportunistic bacterial pathogens that cause

79 otoxins of the major pathogen Staphylococcus aureus as a prototype, we randomly fragmented and separa

80 against methicilin resistant Staphylococcus aureus ATCC 43300 and Candida albicans MTCC 227.

81 Staphylococcus aureus bacteraemia (SAB) is associated with high mortali

82 cohort study of patients with Staphylococcus aureus bacteremia (SAB) and gram-negative bacteremia (GN

83 Staphylococcus aureus bacteremia (SaB) causes significant disease in hu

84 Staphylococcus aureus bacteremia (SAB) is uniquely characterized by foc

85 Patients with septic shock and S. aureus bacteremia admitted directly from the emergency d

86 structure infections (SSSI), Staphylococcus aureus bacteremia, and right-sided endocarditis infectio

87 the changing epidemiology of Staphylococcus aureus bacteremia, as well as the variables associated w

88 inosa and Gram stain-positive Staphylococcus aureus bacteria, inducing 95 +/- 5% and 83 +/- 12% cell

89 iameter) and nearly spherical Staphylococcus aureus bacterium.

90 and SLE keratinocytes exhibited increased S. aureus-binding integrins.

91 udy provides maiden evidence that chronic S. aureus biofilm infection in wounds results in impaired g

92 ion between primary human neutrophils and S. aureus biofilms and provides insight into how S. aureus

93 ce mechanisms associated with Staphylococcus aureus biofilms are becoming better understood.

94 (PSM) comprise the structural scaffold of S. aureus biofilms through self-assembly into functional am

95 s that converge to promote the ability of S. aureus biofilms to evade killing by neutrophils.

96 -Asp and D-Glu was studied on Staphylococcus aureus biofilms.

97 hes are critically needed for Staphylococcus aureus bloodstream infections (BSIs), particularly for m

98 y, we randomly assigned 121 patients with S. aureus BSI/endocarditis to receive a single dose of exeb

99 nly fail to efficiently kill phagocytosed S. aureus, but also induce tolerance to multiple antibiotic

100 oli and methicillin-resistant Staphylococcus aureus by recognizing corresponding antimicrobial resist

101 ociated methicillin-resistant Staphylococcus aureus (CA-MRSA) is threatening public health as it spre

102 ociated methicillin-resistant Staphylococcus aureus (CA-MRSA) SSTI, their household contacts, and pet

103 This study investigated Staphylococcus aureus carriage in patients with microbial keratitis (MK

104 vel, ecological therapies for controlling S. aureus carriage.

105 fy Acrs capable of inhibiting Staphylococcus aureus Cas9 (SauCas9), an alternative to the most common

106 In Staphylococcus aureus-caused endocarditis, the pathogen secretes staphy

107 udy demonstrates that characterization of S. aureus CC and virulence genes helps to predict the likel

108 benefit of diagnostics tools to identify S. aureus CC during bovine mastitis.

109 us Given that neutrophils are crucial for S. aureus clearance, understanding the mechanism(s) driving

110 -fold in a CodY-deficient strain in three S. aureus clinical isolates and in S. epidermidis.

111 nd is ubiquitous in representation within S. aureus clinical isolates.

112 s with experimentally induced Staphylococcus aureus clinical mastitis.

113 A-sequencing expression profiles from two S. aureus clinical strains (TCH1516 and LAC).

114 The primary end point was concordant S aureus colonization by 90 days, defined as neonatal acqu

115 barrier disruption that allows for higher S. aureus colonization in SLE skin.

116 This improved understanding of S. aureus colonization is an important first step toward th

117 Parents may expose neonates to S aureus colonization, a well-established predisposing fac

118 To define the impact of IFNs on S. aureus colonization, we examined the effects of type I a

119 Neonates (n = 236) with S aureus-colonized parent(s) were enrolled.

120 in the opportunistic pathogen Staphylococcus aureus comprises nine proteins, called iron-regulated su

121 defective in vitro killing of Staphylococcus aureus, consistent with a specific granule deficiency.

122 We generated an S. aureus ddh/ldh1/ldh2 triple Tn mutant that cannot produc

123 diversity increased and the abundance of S. aureus decreased.

124 Mice failed to eliminate S. aureus deficient in vWbp, but clearance of these same mi

125 Additionally, S. aureus did not develop resistance to auranofin after rep

126 Although bacterial isolates of S. aureus differ in their virulence potential it is largely

127 stablished predisposing factor to invasive S aureus disease.

128 Cleavage of this probe by the secreted S. aureus enzyme micrococcal nuclease results in emission o

129 us biofilms and provides insight into how S. aureus evades the neutrophil response to cause persisten

130 , our study describes mechanisms by which S. aureus EVs induce inflammasome activation and reveals an

131 Pore-forming toxins associated with S. aureus EVs were critical for NLRP3-dependent caspase-1 a

132 togramin-resistant strains of Staphylococcus aureus, exhibits decreased rates of acetylation in vitro

133 We find that YoeB-YefM complex from S.aureus exists as two distinct oligomeric assemblies: het

134 el method was developed, for isolation of S. aureus from complex (food) samples using molecular impri

135 P. aeruginosa, we show that heme protects S. aureus from CP-mediated inhibition of iron uptake and ir

136 quently, MIPs were used for extraction of S. aureus from milk and rice.

137 substrate protein, encoded in a subset of S. aureus genomes, has been functionally characterized.

138 te sex bias in the neutrophil response to S. aureus Given that neutrophils are crucial for S. aureus

139 mic and epidemiological studies show that S. aureus has jumped between host species many times over i

140 h Chlamydophila pneumoniae or Staphylococcus aureus, have received antibacterial drug therapy prior t

141 esult in overaccumulation of phosphate by S. aureus However, it does reduce the ability of S. aureus

142 elihood of the occurrence of CM following S. aureus IMI and highlights the potential benefit of diagn

143 00 (58.5%) samples, including Staphylococcus aureus in 22% of samples and Haemophilus influenzae in 1

144 se to physiological stress experienced by S. aureus in diverse environments.

145 describe the genetic variation of bovine S. aureus in Europa.

146 hat is significantly anti-correlated with S. aureus in infants and mothers.

147 The detection of Staphylococcus aureus in the concentration range from 50 to 10(7) CFU/m

148 sition strategy supports proliferation of S. aureus in these organs.

149 ent elective surgical discharges, 180-day S. aureus incidence was 1.19% (0.25% BSI, 0.75% SSI no BSI,

150 hown to affect many cellular processes in S. aureus, including autolysis, biofilm formation, capsule

151 ned for quorum quenching activity against S. aureus, including direct protein output assessment (delt

152 d endocarditis infections associated with S. aureus, including methicillin resistant S. aureus (MRSA)

153 Multi-drug resistant Staphylococcus aureus, including methicillin-resistant S. aureus (MRSA)

154 Interestingly, S aureus-induced ER stress response was found to be depend

155 e provide evidence that co-culturing with S. aureus induces a decrease in the activity of ClpXP in P.

156 schemic methicillin-resistant Staphylococcus aureus infected delayed healing wounds in rats with DM2.

157 one of the most common sites of invasive S. aureus infection and a unique environment characterized

158 e (1.0%) in the control group developed an S aureus infection before colonization.

159 and 100% specificity for the diagnosis of S. aureus infection by ELISA.

160 ted in host defense following Staphylococcus aureus infection, but precise mechanisms of host protect

161 hospitalizations, 50.3% had a Staphylococcus aureus infection, compared with 19.4% of IE hospitalizat

162 flammation and mortality upon Staphylococcus aureus infection, recapitulating the human disease.

163 ritical for the innate immune response to S. aureus infection.

164 in swarm formation following Staphylococcus aureus infection.

165 egulatory element of SrrB function during S. aureus infection.

166 The high burden of S. aureus infections after both inpatient and outpatient el

167 Staphylococcus aureus infections can lead to diseases that range from l

168 to a surge in drug-resistant Staphylococcus aureus infections, both in the hospital and community se

169 CD163(-/-) mice are highly susceptible to S aureus infections, demonstrating the relevance of CD163

170 tuberculosis, leprosy, P. aeruginosa and S. aureus infections, where it develops via missense mutati

171 bial discovery to fight Gram-positive and S. aureus infections.

172 serious methicillin-resistant Staphylococcus aureus infections.

173 sition of any S aureus strain and neonatal S aureus infections.

174 tcomes with non-inoculated and historical S. aureus-inoculated controls.

175 le for survival during osteomyelitis, and S. aureus instead has a critical need for anaplerosis.

176 mast cells, resulting in release of viable S aureus into the extracellular space.

177 Staphylococcus aureus is a leading cause of bacterial pneumonia, and we

178 Staphylococcus aureus is a leading cause of health care-associated infe

179 Staphylococcus aureus is a leading cause of pneumonia.

180 Staphylococcus aureus is a significant human pathogen due to its capaci

181 Staphylococcus aureus is also a "high priority" pathogen which is a maj

182 Staphylococcus aureus is among the leading causes of bacterial infectio

183 Staphylococcus aureus is an important bacterial pathogen that can cause

184 Staphylococcus aureus is an opportunistic pathogen that can cause soft

185 ow that relative abundance of Staphylococcus aureus is associated with disease severity in the poster

186 Staphylococcus aureus is generally thought to divide in three alternati

187 S aureus is known to exacerbate AD, whereas S epidermidis

188 cted to clinical settings, drug resistant S. aureus is now one of the key causative agents of communi

189 At the same time, S. aureus is the most frequent cause of skin and soft tissu

190 Staphylococcus aureus (S. aureus) is a common colonizer of healthy skin and mucous

191 alone, including 22/92 (23.9%) additional S. aureus isolates and 25/92 (27.2%) H. influenzae isolates

192 Among the S. aureus isolates that exhibited antibiotic susceptibiliti

193 Staphylococcus aureus, known to induce IFN production, could play a rol

194 Staphylococcus aureus lacking this protease is attenuated in vivo, bein

195 Proliferating intracellular S aureus led to the expansion and eventual rupture of mast

196 found that IL-4 and IL-13 and Staphylococcus aureus lipoteichoic acid work in combination through p63

197 obiome dysbiosis, due to high Staphylococcus aureus loads, especially during flares.

198 sera from naive rabbits and rabbits with S. aureus-mediated osteomyelitis, and then we validated a p

199 icate that T7SS contribute to maintaining S. aureus membrane integrity and homeostasis when bacteria

200 against methicillin-resistant Staphylococcus aureus (MRSA) and bolsters the innate immune response to

201 such as methicillin-resistant Staphylococcus aureus (MRSA) and other healthcare-associated infections

202 ts with methicillin-resistant Staphylococcus aureus (MRSA) bacteremia.

203 Vs from methicillin-resistant Staphylococcus aureus (MRSA) in an environment with or without stressor

204 read of methicillin-resistant Staphylococcus aureus (MRSA) in urban areas.

205 trol of methicillin-resistant Staphylococcus aureus (MRSA) infections remain challenging.

206 Methicillin-resistant Staphylococcus aureus (MRSA) is an important cause of ventilator-associ

207 f novel methicillin resistant Staphylococcus aureus (MRSA) strains.

208 Live methicillin-resistant Staphylococcus aureus (MRSA) was inoculated into the tail vein of rats.

209 such as methicillin-resistant Staphylococcus aureus (MRSA)(1-3).

210 s aureus, including methicillin-resistant S. aureus (MRSA), has become a worldwide, major health care

211 used by methicillin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant enterococcus (VRE),

212 (MSSA), methicillin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant Enterococcus (VRE),

213 yed for methicillin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant Enterococcus faecali

214 cularly methicillin-resistant Staphylococcus aureus (MRSA).

215 . aureus, including methicillin resistant S. aureus (MRSA).

216 ), particularly for methicillin-resistant S. aureus (MRSA).

217 tients with SaB (47 methicillin-resistant S. aureus [MRSA], 12 methicillin-sensitive S. aureus [MSSA]

218 n for methicillin-susceptible Staphylococcus aureus (MSSA) (19/24 [79%]) and avoidance of antibiotics

219 stent methicillin-susceptible Staphylococcus aureus (MSSA) bacteremia, including immediate clearance

220 lth importance of methicillin-susceptible S. aureus (MSSA) in selected communities.

221 pecies [methicillin-sensitive Staphylococcus aureus (MSSA), methicillin-resistant Staphylococcus aure

222 . aureus [MRSA], 12 methicillin-sensitive S. aureus [MSSA]) from 2015-2017.

223 of osteomyelitis, we examined survival of S. aureus mutants deficient in central metabolic pathways,

224 (LA) elicited an increased inhibition of S. aureus mutants lacking T7SS effectors EsxC, EsxA and Esx

225 acteria trapped in NETs is facilitated by S. aureus nuclease (Nuc)-mediated degradation of NET DNA.

226 racterization of the C-terminal domain of S. aureus OatA.

227 Clinically important Staphylococcus aureus obtains iron by extracting heme from hemoglobin (

228 e for detection of Staphylococcus aureus (S. aureus) or Streptococcus pneumoniae (S. pneumoniae), res

229 In conclusion, S. aureus organisms that escape the infected bone may recov

230 dant when detected at low concentrations (S. aureus, P < 0.001; H. influenzae, P < 0.0001) and in spu

231 P < 0.0001) and in sputum-type specimens (S. aureus, P < 0.05).

232 In Staphylococcus aureus, pentaglycine cross-bridges are synthesized by th

233 protein complex that spatially regulates S. aureus peptidoglycan synthesis.

234 Infections caused by Staphylococcus aureus pose a serious and sometimes fatal health issue.

235 Incubation of S. aureus-positive blood culture samples with the P2&3TT pr

236 S. aureus produce membrane-bound, spherical, nano-sized, MV

237 S. aureus produces an array of bicomponent pore-forming tox

238 r the sensitive and specific diagnosis of S. aureus prosthetic joint infection.

239 ults: Haemophilus influenzae, Staphylococcus aureus, Pseudomonas aeruginosa, and Aspergillus infectio

240 d inhibitory activity against Staphylococcus aureus, Pseudomonas aeruginosa, and Enterococcus faecali

241 omplement, as well as their receptors, in S. aureus recognition and clearance, we investigated their

242 Staphylococcus aureus relies on quorum sensing to exert virulence to es

243 nd Pseudomonas aeruginosa and Staphylococcus aureus, representing Gram-positive and Gram-negative bac

244 Both P. aeruginosa and S. aureus require iron to infect the mammalian host.

245 and of skeletal cells, we discovered that S. aureus requires glycolysis for survival in bone.

246 In contrast, S. aureus resistance to mupirocin emerged rapidly.

247 describe the binding mode of Staphylococcus aureus RsfS to the large ribosomal subunit and present a

248 Staphylococcus aureus (S. aureus) is a common colonizer of healthy skin

249 or lytA gene for detection of Staphylococcus aureus (S. aureus) or Streptococcus pneumoniae (S. pneum

250 scherichia coli (E. coli) and Staphylococcus aureus (S. aureus), which decreased first and then incre

251 ptive immune response against Staphylococcus aureus (SA) skin infection substantially improved system

252 Here we show that S. aureus secretes RNA and DNA molecules that are mostly pr

253 rly activation of Treg during Staphylococcus aureus sepsis induces CD4+ T-cell impairment and increas

254 mice, which were previously infected with S. aureus, showed faster monocyte recruitment, increased ba

255 tical for the pathogenesis of Staphylococcus aureus skin and soft tissue infection.

256 ently poorly understood whether localized S. aureus skin infections persistently alter the resident M

257 Staphylococcus aureus small colony variants (SCVs) are frequently assoc

258 use of different variants of Staphylococcus aureus sortase A for a range of ligation reactions and d

259 Staphylococcus aureus ST45 is a major global MRSA lineage with huge str

260 IgG) interaction with virulence factor of S. aureus, staphylococcal protein A (SpA) in the presence o

261 (45.5%) in the control group acquired any S aureus strain (HR, 0.57 [95% CI, 0.31 to 0.88]), and 1 n

262 comes included neonatal acquisition of any S aureus strain and neonatal S aureus infections.

263 were infected with methicillin-resistant S. aureus strain AW7 via the endotracheal tube, extubated,

264 the phage-host interaction of Staphylococcus aureus strain FS159 with a virulent phage JK2 (=812K1/42

265 ays, defined as neonatal acquisition of an S aureus strain that was the same strain as a parental str

266 ed against the multiresistant Staphylococcus aureus strain USA300 for which they displayed moderate t

267 em (T7SS) is conserved across Staphylococcus aureus strains and plays important roles in virulence an

268 T7SS gene cluster and is found across all S. aureus strains as well as in Listeria and Enterococci.

269 Staphylococcus aureus strains carrying enterotoxin A gene (sea) causes

270 were differentially stimulated by various S. aureus strains independently of their isolation sites or

271 he cellular redox environment to regulate S. aureus survival and pathogenesis.

272 diversity and higher overall abundance of S. aureus than nonlesional skin.

273 or pleural fluid, with S. pneumoniae and S. aureus the leading pathogens identified.

274 of Cd-SrtB and also SrtB from Staphylococcus aureus The serine residue indispensable for SrtB activit

275 tive tools that fight against Staphylococcus aureus, the results have not been successful.

276 In Staphylococcus aureus, the transcription factor CodY modulates the expr

277 illus subtilis, and inhibited biofilms by S. aureus to a lesser extent.

278 increases the sensitivity of Staphylococcus aureus to calprotectin-mediated manganese sequestration.

279 significant impairment in the ability of S. aureus to cause infection in both a subcutaneous and sep

280 us However, it does reduce the ability of S. aureus to grow in phosphate-replete defined medium.

281 known about ClpXP in the pathogenesis of S. aureus to include the respiratory tract.

282 onella infection model, where exposure of S. aureus to LL-37 abolished the antimicrobial effect of va

283 on of PhoPR would diminish the ability of S. aureus to resist nutritional immunity and cause infectio

284 ein results in the loss of the ability of S. aureus to secrete cytolytic toxins, protect itself from

285 ay serve as a novel secretory pathway for S. aureus to transport protected cargo in a concentrated fo

286 pneumoniae serotype 12F, and Staphylococcus aureus types 5 and 8 capsular polysaccharides.

287 ent system (TCS) is a global regulator of S. aureus virulence and critical for survival under environ

288 s isolate to be a vancomycin-intermediate S. aureus (VISA) strain, and reduced Ifnb was observed with

289 S aureus was captured by extracellular traps and entered m

290 Staphylococcus aureus was the most common pathogen (53%).

291 S. aureus was the most common pathogen identified in previo

292 helix transporter, NorC, from Staphylococcus aureus We identified this antibody in a yeast display sc

293 s in participants' homes, swabs to detect S. aureus were collected from participants, environmental s

294 Most S. aureus were resistant to penicillin.

295 coli (E. coli) and Staphylococcus aureus (S. aureus), which decreased first and then increased in agr

296 Isogenic mutant strains of S. aureus with varying degree (DeltarexB > USA300 > Deltasa

297 Thus, isogenic mutant stains of S. aureus with varying degree of biofilm formation ability

298 , Pseudomonas aeruginosa, and Staphylococcus aureus, with up to 3.7 logs of biomass reduction.

299 nduced myositis and a clinically relevant S. aureus wound infection murine model.

300 the coordinated defense against invading S. aureus, yet they have a limited life span with replaceme