戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 onnected group of molecules for a high-titer autoantibody response.
2 region suggests a genetic restriction of the autoantibody response.
3 n of any of these phosphatases results in an autoantibody response.
4 ficient CD40-mediated signal 2 to support an autoantibody response.
5 not detectable within the strong Tg-specific autoantibody response.
6 nts; P=0.02), with marked attenuation of the autoantibody response.
7     B cell depletion reduced the PG-specific autoantibody response.
8 PAD-4 polymorphisms influence the anti-PAD-4 autoantibody response.
9 lung, the target tissue associated with this autoantibody response.
10  for examining the role of T cells in the RF autoantibody response.
11 ing a novel window into the initiation of an autoantibody response.
12 y mature, complex humoral autoimmune anti-Sm autoantibody response.
13 D4+ T cell repertoire required to induce the autoantibody response.
14 , as central to the maturation of the Ab and autoantibody responses.
15 ith both H(+)K(+)ATPase and intrinsic factor autoantibody responses.
16 regulate two different sets of nephritogenic autoantibody responses.
17 uated systemic inflammatory mediators and to autoantibody responses.
18 mmunologic, histologic, transcriptional, and autoantibody responses.
19 fferentiation processes that underpin Ab and autoantibody responses.
20 ss-switched and hypermutated (mature) Ab and autoantibody responses.
21 oped for autoantigen discovery and profiling autoantibody responses.
22  the development of IgG(4)- and IgE-specific autoantibody responses.
23 ) regulatory T cells (Tregs) that suppressed autoantibody responses.
24  cell stimuli can influence the induction of autoantibody responses.
25 form germinal centers, and produce secondary autoantibody responses.
26 e maturation, rather than the initiation, of autoantibody responses.
27 rce of snoRNP to initiate and maintain these autoantibody responses.
28 s occurs in the face of an anti-Neu5Gc "xeno-autoantibody" response.
29  NZM2328 mice developed 1) accelerated dsDNA autoantibody response, 2) early onset and severe prolife
30 develop autoimmune ovarian disease (AOD) and autoantibody response 5 weeks later.
31 ral tolerance permits a cross-reactive HIV-1 autoantibody response able to neutralize HIV-1.
32 e show here that ablation of miR-155 reduced autoantibody responses accompanied by a decrease in seru
33 by ELISA, we found a consistent and specific autoantibody response against Dsg1 and other keratinocyt
34   Patients with UC, and not CD, show mucosal autoantibody response against hTM isoforms, particularly
35                                     A common autoantibody response against K(ir)3.4 protein was detec
36 4 wk, AOD progressed to ovarian atrophy with autoantibody response against multiple oocyte Ag of earl
37 e current study, we further investigated the autoantibody response against SPAG16-a protein with unkn
38                          We have studied the autoantibody response against the enzyme transglutaminas
39                                              Autoantibody response against the small nuclear ribonucl
40 ody targets revealed marked heterogeneity in autoantibody responses against islet cell autoantigens i
41 hesis that NZW genes act to class-switch the autoantibody response, an effect that appears to contrib
42 ssion of Th1-mediated events exacerbated the autoantibody response and augmented IgG1, IgE, and IL-4
43 le manifestations, Th1 cells drive the early autoantibody response and IL-17-producing cells may be r
44 during apoptosis as potential targets of the autoantibody response and our results identify poly(ADP-
45                               In this study, autoantibody response and tumor antigen expression are a
46                      This study has examined autoantibody responses and autoimmune pathology in a mur
47 e would be able to assess the specificity of autoantibody responses and define whether exposure to E.
48  germinal center (GC), plasma cell (PC), and autoantibody responses and disease.
49 sociated with the development of spontaneous autoantibody responses and immunopathology in systemic l
50 s heterozygous mice also showed reduction of autoantibody responses and immunopathology.
51 sulted in increased anti-mouse thyroglobulin autoantibody responses and increased expression of IFN-g
52  Tfr cells repress arthritis, Tfh cells, and autoantibody responses and that SFB can mitigate this re
53 h different forms of IgE and tested anti-IgE autoantibody responses and their specificities.
54  the critical role of Tfh cells in promoting autoantibody responses and unveil, (to our knowledge), a
55 parations induced high-avidity anti-CCR5 IgG autoantibody responses, and all five immunized macaques
56 to study the specificity and pathogenesis of autoantibody responses, and to identify and define relev
57 tin, full-blown pathogenic maturation of the autoantibody response appears to require additional inpu
58 autoantigen expression in the cancer and the autoantibody response are associated.
59                                Although most autoantibody responses are dominated by IgG (and mainly
60 suggesting that at the clonal level, chronic autoantibody responses are dynamic and episodic, much li
61         To determine how pathogenic anti-GPS autoantibody responses are regulated, we generated an Ig
62 es encoded across the genome that trigger an autoantibody response associated with tumor development.
63  We hypothesized that TBI generated variable autoantibody responses between individuals that would co
64 multiple autoimmunity risk alleles, promotes autoantibody responses both by increasing B cells' coope
65 on suppressed lung mucosa-associated Tfh and autoantibody responses by increasing the gut-homing alph
66 e of type I IFN-mediated signaling, systemic autoantibody responses can be dissociated from glandular
67                                Nevertheless, autoantibody responses can have their own unique isotype
68              Surprisingly, they had a robust autoantibody response comparable with that of the wild-t
69 onged and significant increase of anti-dsDNA autoantibody responses compared with WT mice (week 4 to
70  always had reduced anti-mouse thyroglobulin autoantibody responses, compared with Tg(-) littermates,
71 bserved in vivo, suggesting that a metatypic autoantibody response could play a physiological role in
72  undertaken to determine whether this strong autoantibody response depends on T cell differentiation
73 rm large-scale multiplex characterization of autoantibody responses directed against structurally div
74                            Modulation of the autoantibody response disrupts pathogenesis by preventin
75                            However, although autoantibody responses driven by acute B cell-targeted d
76 se disparate clinical outcomes, anticollagen autoantibody responses during CIA did not differ among t
77                    KIR4.1 is a target of the autoantibody response in a subgroup of persons with mult
78                       The specificity of the autoantibody response in different autoimmune diseases m
79 ally incite autoimmunity, we have traced the autoantibody response in human SLE back in time, prior t
80 ell differentiation to dampen the pathogenic autoantibody response in lupus.
81 ur goal was to analyze the regulation of the autoantibody response in male and female W/B mice bearin
82 tal mercury elicits a genetically restricted autoantibody response in mice that targets the nucleolar
83 study, we showed that the class-switched IgG autoantibody response in MRL/Fas(lpr/lpr) and C57/Sle1Sl
84 trategy to confirm KIR4.1 as a target of the autoantibody response in multiple sclerosis and to show
85  of this type could plausibly perpetuate the autoantibody response in myasthenia gravis, and are a ra
86 n important role in driving the anti-TG2 IgA autoantibody response in patients with celiac disease.
87 ncentrations were negatively correlated with autoantibody response in patients with immunoreactivity
88                  These data suggest that the autoantibody response in scleroderma is the immune marke
89 pitope-specific Ab responses and alternative autoantibody responses in a model system in which an ant
90 dy identifies axopathic and/or demyelinating autoantibody responses in a subset of patients with mult
91                       Increased diversity of autoantibody responses in acute EAE predicted a more sev
92  of Sle1b on Spt-GC, follicular Th cell, and autoantibody responses in B6.Sle1b mice was B cell auton
93 oked strong germinal centre alloantibody and autoantibody responses in C57BL/6 recipients and develop
94         The aim of this study was to compare autoantibody responses in diabetic and healthy control d
95 ome' microarrays to profile the evolution of autoantibody responses in experimental autoimmune enceph
96 ulated systemic Th17 immunity and heightened autoantibody responses in joints.
97  mechanisms may be responsible for eliciting autoantibody responses in lpr/lpr mice.
98 depletes autoreactive Tfh cells and prevents autoantibody responses in lupus-prone mice.
99 used to study islet and other organ-specific autoantibody responses in parallel.
100                                              Autoantibody responses in patients and mice were dose-de
101 bodies, which correlated with anti-human RNA autoantibody responses in patients with systemic lupus e
102 urther evidence that TLRs play a key role in autoantibody responses in SLE, we found that autoimmune-
103 ction and could therefore promote pathogenic autoantibody responses in SLE.
104 -dose rIL-2 to deplete Tfh cells and prevent autoantibody responses in SLE.
105 hese Th17 mediators correlated directly with autoantibody responses in synovial fluid, providing a po
106 FNs might be critical for amplifying overall autoantibody responses in systemic lupus erythematosus.
107 particle (VLP)-based immunogens could induce autoantibody responses in well-characterized transgenic
108                                   In 6 of 10 autoantibody responses, in 5 of 8 diseases, the investig
109                                 Anti-nuclear autoantibody responses, including anti-dsDNA, were seen
110 ion of linear epitopes and the complexity of autoantibody responses, including the broad spectrum of
111             Functional evaluation of several autoantibody responses indicated that they neutralized t
112  further investigate the pathogenesis of the autoantibody response induced by peptide immunization, w
113                          The anti-Smith (Sm) autoantibody response is highly specific for systemic lu
114 nducible regulatory T cells that can control autoantibody responses is a potential avenue for the tre
115                   The role of T cell help in autoantibody responses is not well understood.
116 beyond IgG are scarcely investigated in many autoantibody responses, leaving substantial gaps in our
117 ection of a self-molecule as a target for an autoantibody response may be a consequence of the proinf
118       It has been suggested that these novel autoantibody responses may be immune system reactions to
119 A vaccines decouple SARS-CoV-2 immunity from autoantibody responses observed during acute COVID-19.
120  of IL-17 and IL-21 and the inflammatory and autoantibody responses observed in these autoimmune mice
121  Autoimmune ovarian disease (AOD) and oocyte autoantibody response of day 3-thymectomized (d3tx) mice
122 0-CD40L interactions are required to support autoantibody responses of B cells whose anergy has been
123  Little is known about the anti-alpha-fodrin autoantibody response on a molecular level.
124                      Proteomic monitoring of autoantibody responses provides a useful approach to mon
125 es, the relative contributions of T cell and autoantibody responses remain largely undefined.
126 nce and B cell tolerance must be defeated in autoantibody responses requiring T cell help.
127 es, we studied the role of T cells in murine autoantibody responses resulting from acute B cell-speci
128 inea pigs not only develop antimitochondrial autoantibody responses similar to human PBC, but also de
129 CD4(+) T cells, and a robust, variegated IgG autoantibody response targeting multiple components of c
130 in 0 (P0), which was the principal target of autoantibody responses targeting nerve proteins.
131  Overall, the highest correlations among the autoantibody responses tested were in the endemic contro
132  WT mice, CD28(-/-) mice have lower anti-MTg autoantibody responses than do WT mice.
133 lyclonal and sustainable anti-mTNF-alpha IgG autoantibody response that lasts for at least 40 weeks.
134  by which a toxin such as mercury elicits an autoantibody response that predominantly targets a singl
135 Ns drove T cell-mediated inflammation and an autoantibody response that targeted abundant, tissue-res
136 its a genetically restricted, anti-nucleolar autoantibody response that targets fibrillarin, a 34-kDa
137 ritical in the initiation and maintenance of autoantibody responses that are a hallmark of systemic l
138 g linked to foreign Th epitopes induces weak autoantibody responses that are predominantly of the IgM
139 e substantial variation between (and within) autoantibody responses, this unprecedented comparison sh
140               Estrogen enhances antibody and autoantibody responses through yet to be defined mechani
141  hTSHR A-subunit protein fails to divert the autoantibody response to a nonpathogenic form.
142                             We conclude that autoantibody response to diverse native GBM Ags was indu
143 ed approach to identify the initiation of an autoantibody response to identify the sites and cell dif
144 bably mediated through an O volvulus induced autoantibody response to multiple proteins.
145              In this study, we evaluated the autoantibody response to native insulin after administra
146 cobacterial T cell epitope elicited a strong autoantibody response to native ubiquitin.
147  selvagem [FS]) in which the pathogenic IgG4 autoantibody response to the self-antigen desmoglein 1 (
148 be a straightforward HLA-class II-restricted autoantibody response to the thyroid stimulating hormone
149                                              Autoantibody responses to apoA-I can be polyclonal and i
150 atosus clearly dissociate genetic control of autoantibody responses to classic lupus antigens and kid
151                                              Autoantibody responses to DNA topoisomerase I (Topo I) a
152                            We concluded that autoantibody responses to EGFR hold the potential of ful
153 uggest that measurement of T-cell as well as autoantibody responses to FcepsilonRIalpha could improve
154 e inverse relationship between IFN-gamma and autoantibody responses to FcepsilonRIalpha may signify d
155                                IFN-gamma and autoantibody responses to FcepsilonRIalpha were inversel
156                        HLA-A*24 genotype and autoantibody responses to insulin (IAA), glutamate decar
157 cally diagnosed with type 2 diabetes exhibit autoantibody responses to islet autoantigens.
158 esponses, using cellular immunoblotting, and autoantibody responses to islet proteins, before transpl
159 esponses, using cellular immunoblotting, and autoantibody responses to islet proteins, before transpl
160 observations that concomitant or coupling of autoantibody responses to proteins which are associated
161 molecular-intrastructural diversification of autoantibody responses to the components of Ul snRNPs in
162 l help may play a crucial role in regulating autoantibody responses to the HA in HA104 mice.
163                                              Autoantibody responses to these MAA have been shown to r
164                               The breadth of autoantibody responses, together with the absence of con
165 f specific autoantigens that might stimulate autoantibody responses under proinflammatory conditions.
166                                  The humoral autoantibody response was associated with C3d complement
167 active B cells depend on T cells to generate autoantibody responses, we studied the role of T cells i
168                                       Strong autoantibody responses were also induced by conjugated p
169                                      Similar autoantibody responses were detected in the seminal plas
170                                        Serum autoantibody responses were measured by radioligand bind
171                                              Autoantibody responses were measured in serum samples.
172                              Anti-galectin-3 autoantibody responses were predominantly of the IgG(4)
173 elopment of SAT and anti-mouse thyroglobulin autoantibody responses were reduced.
174  Myd88 is required for anti-DNA and anti-RNA autoantibody responses, whereas Fcer1g is not expressed
175 including T follicular helper (Tfh) cell and autoantibody responses, whereas mice carrying the Sle2c2
176 ete Freund's adjuvant enhanced their insulin autoantibody response with a higher level and longer per
177                The association of a specific autoantibody response with distinct disease phenotypes i
178 clear ribonucleoprotein (snRNP) and anti-DNA autoantibody responses with some changes in isotype swit

 
Page Top