ライフサイエンスコーパス: autofluorescence

1 photography, spectral-domain OCT, and fundus autofluorescence.

2 rates compared with GA arising from abnormal autofluorescence.

3 ence (increased autofluorescence), and mixed autofluorescence.

4 the target sRNA-FISH signal from background autofluorescence.

5 tata females label free by harnessing tissue autofluorescence.

6 bility on funduscopy, ultrasound, and fundus autofluorescence.

7 n vivo imaging because they eliminate tissue autofluorescence.

8 rowth of atrophic lesions measured by fundus autofluorescence.

9 of atrophic lesions as determined by fundus autofluorescence.

10 poor tissue penetration and high background autofluorescence.

11 /or the interference generated by biological autofluorescence.

12 ference associated with light scattering and autofluorescence.

13 er of a paracentral ring of increased fundus autofluorescence.

14 y useful property of e-liquids, namely their autofluorescence.

15 and other undesirable effects such as tissue autofluorescence.

16 oteins and lipids, which are also sources of autofluorescence.

17 ed tissue scattering, and diminishing tissue autofluorescence.

18 of poorly demarcated questionably decreased autofluorescence.

19 al separation, less phototoxicity, and lower autofluorescence.

20 e-gated detection is not needed to avoid the autofluorescence.

21 e in assessing areas of definitely decreased autofluorescence.

22 ated combined NADH and NADPH (i.e., NAD(P)H) autofluorescence.

23 ally overlapped immunofluorescence and Abeta autofluorescence.

24 for poorly demarcated questionably decreased autofluorescence.

25 onic generation (SHG) and two photon excited autofluorescence.

26 engths, primarily due to drastically reduced autofluorescence.

27 t scattering, absorption, phototoxicity, and autofluorescence.

28 transparent (92% transparency), and has low autofluorescence.

29 totoxicity, and interference from background autofluorescence.

30 y, indocyanine green angiography, and fundus autofluorescence.

31 ral imaging and dotdotdot to mask lipofuscin autofluorescence.

32 py number analytes in biospecimens with high autofluorescence.

33 Almost 30% of eyes showed minimal change autofluorescence 3 years before GA.

34 is(6) to separate the signal from background autofluorescence(7), which typically limits sensitivity.

35 Utilizing autofluorescence (AF) as a discriminating parameter, we

36 ss and safety were evaluated through OCT and autofluorescence (AF) as well as BCVA.

37 ry image was compared with the corresponding autofluorescence (AF) images at 488 nm (SW-AF) and at 78

38 and demarcated RPE atrophy on serial fundus autofluorescence (AF) images were included.

39 ce, the acute blue light diminished the mean autofluorescence (AF) intensity in both fundus short-wav

40 evaluating FACS-based side population (SP), autofluorescence (AF+) and Alde-red assays for CSCs, and

41 computed tomography detecting near-infrared autofluorescence allows in vivo monitoring of intraplaqu

42 .964 (0.929, 0.999) for definitely decreased autofluorescence and 0.268 (0.000, 0.730) for poorly dem

43 dge of the estimated endmembers to learn the autofluorescence and background fluorescence spectra on

44 pping emission spectra from contamination by autofluorescence and background fluorescence.

45 With a FOV now similar to autofluorescence and color fundus imaging, SS OCT imagin

46 miR-204-/- eyes evidenced areas of hyper-autofluorescence and defective photoreceptor digestion,

47 Positive samples were observed by autofluorescence and electron microscopy, analyzed by me

48 Fundus autofluorescence and electrophysiological testing (ERG a

49 Autofluorescence and fundus photography showed a lower p

50 better photon penetration, diminished tissue autofluorescence and high contrasts, its molecular mass

51 dye FRET pairs efficiently suppressed sample autofluorescence and improved the signal-to-background r

52 ISH data in complex tissues while overcoming autofluorescence and increasing multiplexing capacity.

53 was delineated on the basis of serial fundus-autofluorescence and infrared-reflectance images.

54 1 portfolio specifically designed to measure autofluorescence and luciferase inhibition.

55 gns and symptoms, visual acuity (VA), fundus autofluorescence and OCT findings, ERG phenotype, and mi

56 ical notes, retinal imaging including fundus autofluorescence and OCT, electroretinography (ERG), and

57 uorescent lesion types (definitely decreased autofluorescence and poorly demarcated questionably decr

58 tion, deep-tissue imaging, but is limited by autofluorescence and scattering.

59 Tissues were imaged using multiphoton autofluorescence and second harmonic generation microsco

60 best-corrected visual acuity (BCVA), fundus autofluorescence and spectral domain-optical coherence t

61 Fundus autofluorescence and spectral-domain optical coherence t

62 d morphologic examinations, including fundus autofluorescence and spectral-domain optical coherence t

63 ot well understood, in part because of their autofluorescence and the absence of well-defined surface

64 predominant hyperautofluorescence (increased autofluorescence), and mixed autofluorescence.

65 magnification microscopy, overcoming tissue autofluorescence, and allowing the detection of low-abun

66 rve head (ONH), infrared reflectance, fundus autofluorescence, and color fundus photographs (CFP).

67 omicroscopy, OCT and OCT angiography, fundus autofluorescence, and fluorescein and indocyanine green

68 n is challenging due to the plant cell wall, autofluorescence, and low effector abundance.

69 inical funduscopy as well as by pseudocolor, autofluorescence, and OCT imaging.

70 , and full-field electroretinography, fundus autofluorescence, and optical coherence tomography findi

71 incident GA size, area of surround abnormal autofluorescence, and presence of reticular pseudodrusen

72 Color fundus photographs, fundus autofluorescence, and spectral-domain OCT were obtained,

73 erval with near-infrared reflectance, fundus autofluorescence, and spectral-domain OCT.

74 ange from baseline in microperimetry, fundus autofluorescence, and spectral-domain optical coherence

75 fundus photography, fluorescein angiography, autofluorescence, and spectral-domain optical coherence

76 without a priori knowledge of background or autofluorescence; and (iii) 3D reconstruction of live wh

77 erve the microstructure of the near-infrared autofluorescence (AO-IRAF) from the RPE layer in healthy

78 leaching, broad emission spectra, and sample autofluorescence are disadvantages that cannot be easily

79 In the live animal, tissue autofluorescence arises from a number of biologically im

80 luorescence (DDAF and questionably decreased autofluorescence) at first visit was 2.6 (2.8) mm2.

81 Autofluorescence, attributed to nicotinamide adenine din

82 oherence tomography (OCT), conventional blue autofluorescence (B-AF), and NIR-AF imaging.

83 rared fundus autofluorescence (NIR-AF), blue autofluorescence (B-AF), optical coherence tomography (O

84 of increased autofluorescence on blue fundus autofluorescence (B-FAF).

85 However, because of the high autofluorescence background of many tissue samples, it i

86 visualized, is made less sensitive by tissue autofluorescence background, and is usually incompatible

87 me enables time-gated (TG) detection without autofluorescence background.

88 ), fluorescein angiography (FA), blue fundus autofluorescence (BFAF), en face OCT, and OCT angiograph

89 photography, near-infrared reflectance, blue autofluorescence, blue reflectance, and spectral-domain

90 after cell fixation can effectively suppress autofluorescence, but this approach is not conducive to

91 ia in COS-7 cells and showed that background autofluorescence can be identified through its distinct

92 ther achieved if the detection background of autofluorescence can be removed.

93 onclusion, our data suggest that e-cigarette autofluorescence can be used as a marker of e-cigarette

94 f-target binding of FISH probes and cellular autofluorescence-can become limiting in a number of impo

95 Because autofluorescence changes with metabolic state, it can be

96 methods perform poorly when confronted with autofluorescence-contaminated images.

97 The total mean (SD) area of decreased autofluorescence (DDAF and questionably decreased autofl

98 Areas of definitely decreased autofluorescence (DDAF) and questionably decreased autof

99 Areas of definitely decreased autofluorescence (DDAF) and questionably decreased autof

100 -wavelength light resulted in reduced fundus autofluorescence, decreased HPLC-quantified A2E, outer n

101 Fundus autofluorescence demonstrated interposed, reduced, and i

102 to fluorescent microscopy and found (i) that autofluorescence differs widely between e-liquids, (ii)

103 spite apparent differences in morphology and autofluorescence emission with traditional two-photon mi

104 both navigate to nodules and also to perform autofluorescence endomicroscopy.

105 ce tomography (OCT), OCT-Angiography, fundus autofluorescence (FAF) and fluorescein-angiography (FA).

106 east 4 years and had undergone annual fundus autofluorescence (FAF) and OCT imaging using Heidelberg

107 ange in GA over 12 months measured by fundus autofluorescence (FAF) at 3 timepoints: baseline, month

108 corroboration for visual fields, and fundus autofluorescence (FAF) can show damage topographically.

109 Fundus autofluorescence (FAF) decays were detected in short (49

110 Eyes were evaluated on fundus autofluorescence (FAF) for GA.

111 Fundus autofluorescence (FAF) images from a subset of AREDS2 pa

112 ous fundus photographs and SD OCT and fundus autofluorescence (FAF) images of eyes affected with tube

113 The 200 degrees pseudocolor and fundus autofluorescence (FAF) images were captured on the Optos

114 and after 6 weeks, respectively, and fundus autofluorescence (FAF) images were obtained to visualize

115 olume scans centered at the fovea and fundus autofluorescence (FAF) images were obtained.

116 Fundus autofluorescence (FAF) imaging is crucial to the diagnos

117 ical coherence tomography (SDOCT) and fundus autofluorescence (FAF) imaging.

118 cal coherence tomography (SD OCT) and fundus autofluorescence (FAF) imaging.

119 marily by color fundus photography or fundus autofluorescence (FAF) imaging.

120 Most studies of fundus autofluorescence (FAF) in geographic atrophy (GA) have b

121 responding to focal dots of decreased fundus autofluorescence (FAF) surrounded by increased FAF.

122 ptical coherence tomography (SD OCT), fundus autofluorescence (FAF), and fluorescein angiography/indo

123 ptical coherence tomography (SD-OCT), fundus autofluorescence (FAF), and infrared reflectance (IR) to

124 nventional multimodal imaging (color, fundus autofluorescence (FAF), and infrared reflectance [IR] im

125 ptical-coherence-tomography (SD-OCT), fundus autofluorescence (FAF), and near-infrared-reflectance (I

126 rared (NIR) and short-wavelength (SW) fundus autofluorescence (FAF), and NIR reflectance (REF).

127 or fundus photography (CFP), confocal fundus autofluorescence (FAF), confocal near-infrared reflectan

128 phthalmological examination including fundus autofluorescence (FAF), dynamic simultaneous fluorescein

129 for at least 1 examination modality: fundus autofluorescence (FAF), spectral-domain (SD) optical coh

130 OCT, fluorescein angiogram (FA), and fundus autofluorescence (FAF).

131 ithelium (RPE) is the major source of fundus autofluorescence (FAF).

132 area in untreated eyes with CHM using fundus autofluorescence (FAF).

133 em of SC lesions based on SS-OCTA and fundus autofluorescence findings.

134 B-scan ultrasonography, fundus photography, autofluorescence, fluorescein angiography (FA), optical

135 ing included pseudocolor photography, fundus autofluorescence, fluorescein angiography, and indocyani

136 Remarkably, because of the autofluorescence-free background and high-throughput sig

137 mple preparation, higher-order multiplexing, autofluorescence-free detection, and increased dynamic r

138 er it expressed, even in the specimens where autofluorescence from environment severely interferes fl

139 ng intrinsically expressed GFP fluorescence, autofluorescence from Flavin proteins, and exogenous ant

140 agellate fresh water green alga, but intense autofluorescence from photosynthesis pigments has hinder

141 Clinical data, color, infrared and autofluorescence fundus imaging, optical coherence tomog

142 s photography, and widefield pseudocolor and autofluorescence fundus imaging.

143 and retinal imaging by OCT, pseudocolor, and autofluorescence fundus photography.

144 Fundus autofluorescence, fundus color photography, and spectral

145 ear-infrared reflectance (NIR), green fundus autofluorescence (G-FAF), confocal pseudocolor, and retr

146 giography, near-infrared reflectance, fundus autofluorescence, high-resolution OCT, and ultrawide-fie

147 image registration, we identified changes in autofluorescence images before the onset of GA.

148 eye at the most recent visit, and (2) fundus autofluorescence images for at least 2 visits with a min

149 avelength (SW-AF) and near-infrared (NIR-AF) autofluorescence images of ten patients with mutations i

150 arial-network model can transform wide-field autofluorescence images of unlabelled tissue sections in

151 A series of 3 fundus autofluorescence images using 3 different acquisition pa

152 ical coherence tomography (SDOCT) and fundus autofluorescence images were acquired every 6 months.

153 Quantitative autofluorescence images were also evaluated using a dedi

154 patients, 215 had at least 2 gradable fundus autofluorescence images with atrophic lesion(s) present

155 , including color fundus photographs, fundus autofluorescence images, and spectral-domain OCT images.

156 escent AZOOR line in short-wavelength fundus autofluorescence images, delineating the peripapillary l

157 ry sparing is a consistent feature on fundus autofluorescence images, whereas the same region is less

158 OCT imaging and were hyperautofluorescent on autofluorescence images.

159 lary sparing as consistent feature on fundus autofluorescence images.

160 Fundus autofluorescence imaging and optical coherence tomograph

161 We suggest that near-infrared autofluorescence imaging is a novel technology that allo

162 re measured with reflection spectroscopy and autofluorescence imaging methods, respectively.

163 rescence lifetime imaging microscopy (FLIM), autofluorescence imaging microscopy and inflammatory res

164 Here, we demonstrate two-photon autofluorescence imaging of NAD(P)H and FAD to nondestru

165 Quantitative fundus autofluorescence imaging revealed characteristic qAF lev

166 reduced-illuminance and conventional fundus autofluorescence imaging showed good concordance in asse

167 POD levels were measured by a two-wavelength autofluorescence imaging technique.

168 These results establish label-free autofluorescence imaging to quantify dynamic metabolism,

169 nts underwent spectral-domain OCT and fundus autofluorescence imaging using the Spectralis HRA+OCT (H

170 In selected cases, fundus autofluorescence imaging was performed.

171 signs are optimally displayed en face using autofluorescence imaging whereas cross-sectional OCT rev

172 l responses were measured using flavoprotein autofluorescence imaging, and ageing-related changes in

173 ctroretinography, fundus photography, fundus autofluorescence imaging, and optical coherence tomograp

174 ally by wide-field color photography, fundus autofluorescence imaging, and spectral-domain optical co

175 graphy and SD OCT, and some were imaged with autofluorescence imaging, fluorescein angiography, indoc

176 py, fundus photography, spectral-domain OCT, autofluorescence imaging, fundus fluorescein angiography

177 Retinal imaging included OCT, blue-light autofluorescence imaging, fundus photography, and widefi

178 s on optical coherence tomography and fundus autofluorescence imaging, retinal function assessed by f

179 examination, wide-field photography, fundus autofluorescence imaging, sedated electroretinography, o

180 domain OCT, color fundus photography, fundus autofluorescence imaging, visual field testing, full-fie

181 hthalmoscopy infrared reflectance and fundus autofluorescence imaging.

182 The dots were hyperautofluorescent on fundus autofluorescence imaging.

183 tical coherence tomography (OCT), and fundus autofluorescence imaging.

184 th Stargardt disease as determined by fundus autofluorescence imaging.

185 ed a striking leopard-spot pattern on fundus autofluorescence imaging.

186 th marked loss of autofluorescence on fundus autofluorescence imaging.

187 and growth of GA were measured using fundus autofluorescence imaging.

188 come the common problem of the wide range of autofluorescence in embedded plant tissue using linear s

189 , however, due to substantial scattering and autofluorescence in tissue at visible (350-700 nm) and n

190 onstrated interposed, reduced, and increased autofluorescence in traumatic pigment epitheliopathy.

191 that monitored atrophy progression by fundus autofluorescence in untreated eyes with STGD1 for 6 mont

192 gical hydrogel, exhibiting new green and red autofluorescence in vitro and in vivo without the use of

193 wing key terms: "parathyroid, near infrared, autofluorescence" in various search engines such as PubM

194 na included color fundus photography, fundus autofluorescence, intravenous fluorescein angiography, s

195 Fundus autofluorescence is a valuable imaging tool in the diagn

196 ent GA that arises from predominantly normal autofluorescence is associated with faster enlargement r

197 Here we show that near-infrared autofluorescence is associated with the presence of intr

198 rkedly suppressed photon scattering and zero-autofluorescence is reported, which enables visualizatio

199 nd subcellular resolution by evaluating NADH autofluorescence kinetics during the mitochondrial redox

200 nd subcellular resolution by evaluating NADH autofluorescence kinetics during the mitochondrial redox

201 was larger than both the ONL-slab and fundus autofluorescence lesion areas.

202 e in vivo and this study explores the use of autofluorescence lifetime (AFL) measurements to provide

203 Autofluorescence lifetime imaging can be used to non-des

204 2) significantly decreased the excited-state autofluorescence lifetime of enzyme-bound reduced NADH a

205 Here, we show that imaging of the autofluorescence lifetime signals of quiescent and activ

206 cific activation-state-dependent patterns of autofluorescence lifetime.

207 ing Spectralis-based FLIO was used to detect autofluorescence lifetimes in short (SSC; 498-560 nm) an

208 Q at a concentration of 46 mM exhibited long autofluorescence lifetimes of around 1100 ps in either s

209 Additionally, the autofluorescence lifetimes of HCQ were measured in a cuv

210 However, endogenous cellular autofluorescence masks a useful fluorescence signal, lim

211 Additionally, quantitative autofluorescence may have potential for use as an outcom

212 Stargardt disease with DDAF lesions, fundus autofluorescence may serve as a monitoring tool for inte

213 IL4 plus IL13 in 2D culture, confirming that autofluorescence measurements capture known metabolic ph

214 splasia using in vivo volumetric multiphoton autofluorescence microscopy and second harmonic generati

215 um in diameter by integrating nondestructive autofluorescence microscopy and spatially targeted liqui

216 Autofluorescence microscopy provides the visualization o

217 and followed wound closure up to 6 hours by autofluorescence multiphoton microscopy.

218 for poorly demarcated questionably decreased autofluorescence (n = 12).

219 2.04 +/- 1.87 mm(2) for definitely decreased autofluorescence (n = 15) and 1.86 +/- 2.14 mm(2) for po

220 Other methods, including fundus autofluorescence, near-infrared reflectance, and color i

221 Fundus autofluorescence, near-infrared reflectance, and spectra

222 h autofluorescence (SW-AF) and near-infrared autofluorescence (NIR-AF) modalities correlating with re

223 horoid together with decreased near-infrared autofluorescence (NIR-AF) provided evidence for retinal

224 inal imaging, including near-infrared fundus autofluorescence (NIR-AF), blue autofluorescence (B-AF),

225 al imaging (color fundus photography, fundus autofluorescence, OCT), electrophysiologic assessment, a

226 from background signals, such as the typical autofluorescence of biological samples.

227 20 The latter likely accounts for the strong autofluorescence of Ca Entotheonella filaments.

228 Furthermore, we used the autofluorescence of e-liquids as a marker for tracking e

229 hic signs of myopic CNV, and the presence on autofluorescence of multiple hyper-autofluorescent spots

230 eveals novel aspects of uEV analysis such as autofluorescence of podocyte origin.

231 In tissue engineering, autofluorescence of polymer scaffolds often lowers the i

232 More interestingly, the strong red autofluorescence of the as-prepared hydrogel allows for

233 blished, taking advantage of the distinctive autofluorescence of these cells.

234 Autofluorescence of these precipitates was observed poss

235 tomography (SDOCT) and an area of increased autofluorescence on blue fundus autofluorescence (B-FAF)

236 ient that was associated with marked loss of autofluorescence on fundus autofluorescence imaging.

237 (OCT), en face near-infrared imaging, fundus autofluorescence, optical coherence tomography angiograp

238 ondria by efficiently suppressing endogenous autofluorescence or overexpressed cytosolic unmodulatabl

239 udies using color fundus photography, fundus autofluorescence, or OCT (P = 0.35-0.99).

240 opy (P = 0.001), ultrasound (P = 0.013), and autofluorescence (P = 0.002).

241 pendently delineated boundaries of preserved autofluorescence (PAF) and preserved ellipsoid zone (EZ)

242 Interestingly, no changes in macular fundus autofluorescence pattern were evident after optical cohe

243 in both mice and non-human primates, termed autofluorescence-positive (AF(+)) and negative (AF(-)).

244 graphic atrophy evolving from minimal change autofluorescence precursor lesions was associated with f

245 predominant hypoautofluorescence (decreased autofluorescence), predominant hyperautofluorescence (in

246 lesions, were classified into minimal change autofluorescence, predominant hypoautofluorescence (decr

247 ns based on changes in the endogenous tissue autofluorescence profile.

248 ntrol subjects underwent quantitative fundus autofluorescence (qAF) imaging using a confocal scanning

249 ants were examined using quantitative fundus autofluorescence (qAF) imaging with a modified confocal

250 undus autofluorescence (SW-AF), quantitative autofluorescence (qAF), and full-field electroretinogram

251 intensities, measured as quantitative fundus autofluorescence (qAF), indicated chronic impairment in

252 fundus autofluorescence (quantitative fundus autofluorescence [qAF]) and spectral-domain optical cohe

253 uorescence (DDAF) and questionably decreased autofluorescence (QDAF) were outlined and quantified.

254 sions by quantifying short-wavelength fundus autofluorescence (quantitative fundus autofluorescence [

255 s not associated with corresponding abnormal autofluorescence resolved without clinical scarring afte

256 OCT results, OCT angiography results, fundus autofluorescence results, ultra-widefield fluorescein an

257 Skin AGEs measured as skin autofluorescence (SAF) are a noninvasive reflection of l

258 s complications and can be estimated by skin autofluorescence (sAF).

259 ssessed noninvasively by measurement of skin autofluorescence (SAF).

260 eed, they have intrinsic limitations such as autofluorescence, scattering, and phototoxicity.

261 cancer imaging, significantly overcoming the autofluorescence/scattering issues for deep tissue molec

262 normalities documented with short-wavelength autofluorescence, SD-OCT, fluorescein and indocyanine gr

263 RNFL thinning and autofluorescence showed relative sparing of the temporal

264 Fundus autofluorescence showed zonal areas of hypoautofluoresce

265 The results show that the autofluorescence signals emitted from polycaprolactone (

266 s in living cells exhibiting high and uneven autofluorescence signals.

267 fication of cancerous tissue by its distinct autofluorescence signature that is associated with the a

268 Next, the autofluorescence, solvent compatibility, and biocompatib

269 g real-world spectral images contaminated by autofluorescence, SSASU greatly improved proportion inde

270 The autofluorescence suppressive effect does not substantial

271 F) intensity in both fundus short-wavelength autofluorescence (SW-AF) and near-infrared autofluoresce

272 In short-wavelength fundus autofluorescence (SW-AF) images, speckled hyperautofluor

273 try that presents on short-wavelength fundus autofluorescence (SW-AF) imaging with hyperautofluoresce

274 oherence tomography, short-wavelength fundus autofluorescence (SW-AF), quantitative autofluorescence

275 ography (SD-OCT) and short wavelength fundus autofluorescence (SW-AF).

276 clinically and imaged with short-wave fundus autofluorescence (SW-FAF), spectral-domain optical coher

277 perautofluorescent ring on short-wave fundus autofluorescence (SW-FAF).

278 herosclerotic plaques generate near-infrared autofluorescence that can be detected via emission compu

279 erformed without influence from the inherent autofluorescence that commonly affects fluorescence-base

280 ce lifetimes from the lifetime of the skin's autofluorescence, these two APIs are viable targets for

281 by HPLC analysis and quantitation of fundus autofluorescence; this effect is consistent with photoox

282 ssays, percent actives ranged from 0.5% (red autofluorescence) to 9.9% (luciferase inhibition).

283 generation) and elastin (two-photon excited autofluorescence), to obtain biochemical and structural

284 Raman scattering (CARS), two-photon excited autofluorescence (TPEF), and second-harmonic generation

285 This scheme simply avoids both autofluorescence under infrared excitation and many tedi

286 the disease extent on ultra-widefield fundus autofluorescence (UWF-FAF) in patients with ABCA4 Starga

287 nce in the form of luciferase inhibition and autofluorescence via multiple wavelengths (red, blue, an

288 2-0.61 mm2/y), and of total decreased fundus autofluorescence was 0.35 mm2/y (95% CI, 0.28-0.43 mm2/y

289 Relatively normal-appearing peripapillary autofluorescence was identified in all patients, indepen

290 Decreased autofluorescence was the most common change, although mi

291 and poorly demarcated questionably decreased autofluorescence) was measured.

292 Macular and peripheral patterns of autofluorescence were classified as (1) minimal change,

293 uorescence (DDAF) and questionably decreased autofluorescence were quantified by a reading center.

294 Fluorescein angiography and fundus autofluorescence were useful in determining lesion age a

295 , near-infrared reflectance, and blue fundus autofluorescence, were investigated.

296 roduce pre-culture SBB treatment to suppress autofluorescence, where SBB is applied to polymeric scaf

297 e stage or presence of widespread changes on autofluorescence widefield images.

298 ission of the bound UCNPs was imaged free of autofluorescence with anti-Stokes photoluminescence micr

299 trate that autoluminescence is solely due to autofluorescence with lifetimes of about 5 ns in the vis

300 all eyes, a cross-shaped increase in macular autofluorescence with variable intensity occurred after

301 off-target probe binding and in the cellular autofluorescence without detectable loss in RNA.