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1 sion signature consistent with an underlying autoimmune mechanism.
2 eta-cell death and dysfunction induced by an autoimmune mechanism.
3 the faulty BBB and neuronal death through an autoimmune mechanism.
4 al fullness, and believed to be caused by an autoimmune mechanism.
5 lity to detect BB: in the joint implicate an autoimmune mechanism.
6 athogenic mechanisms but not a single global autoimmune mechanism.
7 onimmune bronchial injury and alloimmune and autoimmune mechanisms.
8 The most favored hypothesis is based on autoimmune mechanisms.
9 men is unknown but may involve microbes and autoimmune mechanisms.
10 ) in the course of HSE, suggesting secondary autoimmune mechanisms.
11 , suggesting a possible role for traditional autoimmune mechanisms.
12 lls in pancreatic islets that is mediated by autoimmune mechanisms.
13 onal damage, excitotoxicity and inflammatory/autoimmune mechanisms.
14 vide a unique opportunity to study potential autoimmune mechanisms.
15 ory synovitis whose pathogenesis may involve autoimmune mechanisms.
16 be a genetic susceptibility and a link with autoimmune mechanisms.
17 represent a useful tool in dissecting basic autoimmune mechanisms.
18 on these cytokines play in antimicrobial and autoimmune mechanisms.
19 lls in pancreatic islets that is mediated by autoimmune mechanisms.
20 lating and tissue antigens in HS suggests an autoimmune mechanism and uncovers putative therapeutic t
22 -kappaB pathways, type I interferon-enhanced autoimmune mechanisms and/or dysregulated adaptive immun
23 s such as oxidative injury, inflammatory and autoimmune mechanisms, and genetic predisposition also i
24 tive destruction of pancreatic beta-cells by autoimmune mechanisms, and intracellular pathways driven
27 biliary cirrhosis (PBC), but the postulated autoimmune mechanisms behind these conditions are poorly
33 e pathogenesis of this disease to support an autoimmune mechanism in its underlying pathogenesis.
35 n with factor H's active site may support an autoimmune mechanism in some cases previously considered
36 ted with autoimmune disease, but the role of autoimmune mechanisms in disease development is not well
38 r, has afforded an opportunity to understand autoimmune mechanisms in myocarditis, with autoreactive
40 of CVD decades later, suggesting a role for autoimmune mechanisms in the development of CVD in T1DM,
41 of genetic predisposition, inflammation, and autoimmune mechanisms in the development of pulmonary ar
42 etection of allelic inclusion (an implicated autoimmune mechanism) in healthy individuals and (iii) t
44 In some patients, there is evidence that an autoimmune mechanism may be responsible for eliciting da
45 These findings support the hypothesis that autoimmune mechanisms may contribute to the pathogenesis
46 cell-mediated or humoral (antibody-mediated) autoimmune mechanisms may predominate in effecting a giv
47 at CASPR2 and LGI1 modulation via genetic or autoimmune mechanisms may share common intermediate mole
48 the spinal cord or the brain and discuss the autoimmune mechanisms of complement-mediated myopathies,
50 nical model for understanding alloimmune and autoimmune mechanisms, optimizing immunosuppressive stra
51 pathogen was identified, we examined whether autoimmune mechanisms played a role in initiating or mai
53 d autoantibodies will aid in elucidating the autoimmune mechanism(s) leading to the development of HG
54 e Syndrome (GBS) is believed to be caused by autoimmune mechanisms that are predominantly T-cell medi
55 gress has been made in dissecting the 2 main autoimmune mechanisms that drive the pathogenesis of CSU
56 s model will be a useful tool for addressing autoimmune mechanisms underlying gender-related differen
57 ens, autoantibodies, and immune cells as the autoimmune mechanisms underlying various forms of myocar
58 roid response may suggest an inflammatory or autoimmune mechanism, underscoring the importance of con
59 Programmed cell death is an important anti-autoimmune mechanism used to delete autoreactive lymphoc
61 l and clinical results, we concluded that an autoimmune mechanism was responsible for neutropenia.
62 reatment of non-neoplastic diseases in which autoimmune mechanisms were considered important to patho
64 ered by secondary effects of acute COVID-19, autoimmune mechanisms with chronic inflammation, viral p