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1 llary thymic epithelial cells expressing the autoimmune regulator.
2 ng the T cell repertoire and the role of the autoimmune regulator Aire in thymic MPO expression, we c
4 heckpoint occurs through the activity of the autoimmune regulator AIRE, which promotes central T cell
6 such as insulin, we are seeing the result of autoimmune regulator (AIRE) activity and the workings of
7 eted of T cells, showed a notable absence of autoimmune regulator (AIRE) and Foxp3(+) regulatory T ce
11 Both humans and mice with a mutation in the autoimmune regulator (aire) gene develop multiorgan auto
15 dividuals with thymic abnormalities, such as autoimmune regulator (AIRE) gene mutations, frequently d
17 dullary thymic epithelial cells (mTECs), the Autoimmune regulator (Aire) gene plays an essential role
19 of mature lymphocytes from mice lacking the autoimmune regulator (aire) gene was performed to examin
20 e type I (APS-I), caused by mutations in the autoimmune regulator (AIRE) gene, and myasthenia gravis
21 syndrome, which is caused by mutation of the autoimmune regulator (AIRE) gene, is a highly variable d
22 yndrome type 1 is caused by mutations in the autoimmune regulator (AIRE) gene, located on chromosome
31 y thymic epithelial cells (mTECs) expressing autoimmune regulator (AIRE) have a critical role in self
40 pression of insulin under the control of the Autoimmune Regulator (Aire) is thought to be a key compo
42 sis-ectodermal dystrophy (APECED), caused by autoimmune regulator (AIRE) mutations, manifests with ch
50 -antigens, regulated at least in part by the autoimmune regulator (AIRE) protein, is crucial for comp
51 PECED patients are defined by defects in the autoimmune regulator (AIRE) that mediates central T cell
52 also interacts with the transcription factor autoimmune regulator (AIRE) to promote central T-cell to
53 significant reduction in self-Ag-presenting, autoimmune regulator (AIRE)(+) medullary thymic epitheli
54 ciated with a reduction in expression of the autoimmune regulator (Aire), a critical mediator of cent
56 rome caused by loss-of-function mutations of autoimmune regulator (AIRE), a key central tolerance med
57 specific antigens that are controlled by the autoimmune regulator (AIRE), a key factor in central tol
61 layered mechanism for condensate assembly by autoimmune regulator (Aire), an essential transcriptiona
62 ed antigens in the thymus, driven in part by autoimmune regulator (Aire), is critical for the protect
63 thymic epithelial cell transcription factor autoimmune regulator (Aire), its colocalization with the
65 Here we show that in the presence of the autoimmune regulator (AIRE), the class III VNTR haplotyp
66 ctopic expression of TSAs largely depends on autoimmune regulator (Aire), which is expressed in matur
68 m our laboratory and others have established autoimmune regulator (Aire)-deficient mice as a useful m
71 ing type I IFNs due to rare inborn errors of autoimmune regulator (AIRE)-driven T cell tolerance were
72 e selection of T cells, with the presence of autoimmune regulator (Aire)-expressing medullary thymic
79 ecessive disorder caused by mutations in the autoimmune regulator (AIRE, which regulates immune toler
81 mice revealed that the combined loss of the autoimmune regulator, AIRE, with PD-1 unleashed a sponta
82 the results indicate that OCA-B is a potent autoimmune regulator and a promising target for pharmaco
83 epithelial cells (mTEC(high)) expressing the autoimmune regulator are targets of donor T-cell alloimm
84 olleagues presents new insights into how the Autoimmune Regulator carries out its molecular activity
85 ssue antigens in a manner similar to that of autoimmune regulator-deficient (Aire-deficient) mice.
86 ctive form of the transcription factor AIRE (autoimmune regulator) develop multiorgan autoimmune dise
87 als, in spite of the inability to induce the autoimmune regulator, displayed a significant ameliorati
88 Finally, we described a thymic hematopoietic autoimmune regulator-expressing APC population resemblin
89 f group 3 innate lymphoid cells, extrathymic autoimmune regulator-expressing cells and, potentially,
90 is indispensable for the differentiation of autoimmune regulator-expressing medullary thymic epithel
91 e mouse thymus could be assigned to strongly autoimmune regulator-expressing, mature medullary thymic
92 tioned with anti-CD3epsilon mAb, we detected autoimmune regulator expression together with the absenc
97 variable number of tandem repeats) and AIRE (autoimmune regulator) have been associated with the modu
99 d expression of genes regulated by DEAF1, an autoimmune regulator-like transcription factor required
100 interactions between developing T cells and autoimmune regulator-positive medullary thymic epithelia
102 s) in the thymus is under the control of the autoimmune regulator protein (AIRE), and polymorphisms i
105 cent lessons learned from the study of AIRE (autoimmune regulator), the gene responsible for the rare
106 hymi from NOD mice lacking expression of the autoimmune regulator transcription factor, which develop
107 llary thymic epithelial cells expressing the autoimmune regulator were detected within 10 days of gen
108 affinity for the transcription factor AIRE (autoimmune regulator), which is highly expressed in thym
109 LNSC subset expressing the highest levels of autoimmune regulator, which responds potently to bystand