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1 tion how a foreign Ag (gluten) can induce an autoimmune response.
2 networks consistent with an adrenal-targeted autoimmune response.
3 hat transform an alloimmune reaction into an autoimmune response.
4 nfection may culminate in an T-cell-mediated autoimmune response.
5 cleic acids as well as for prevention of the autoimmune response.
6 ansportation by MZ-P B cells to stimulate an autoimmune response.
7 n (ATG) might be effective for reducing this autoimmune response.
8 evelopment of a T cell-mediated inflammatory autoimmune response.
9 infection, followed by a bile duct-targeted autoimmune response.
10 epitope is not sufficient to halt an ongoing autoimmune response.
11 now characterized by a more intense humoral autoimmune response.
12 roxyvitamin D(3) may not play a role in this autoimmune response.
13 ich TREM receptors regulate inflammation and autoimmune response.
14 atin is abnormally exposed, facilitating the autoimmune response.
15 odorant binding protein 1a, targeted by the autoimmune response.
16 ial for the perpetuation of the CNS-targeted autoimmune response.
17 alpha5NC1 subunits, which in turn elicits an autoimmune response.
18 in particular, are major contributors to the autoimmune response.
19 generation of memory cells that control the autoimmune response.
20 prior to the generation of a robust adaptive autoimmune response.
21 ion involved in the development of the human autoimmune response.
22 rogressive survival of Teff cells during the autoimmune response.
23 and the functional roles of IFNgamma in the autoimmune response.
24 ization process and favor environment for an autoimmune response.
25 ells and their potential role in shaping the autoimmune response.
26 consequence of a dysregulated and persistent autoimmune response.
27 rt of a feedback circuit that limits further autoimmune responses.
28 ost may be crucial to limit inflammatory and autoimmune responses.
29 here is little data on possible IgA-mediated autoimmune responses.
30 at microbiota are also capable of regulating autoimmune responses.
31 ce peripheral immune tolerance that prevents autoimmune responses.
32 icularly in suppressing harmful allergic and autoimmune responses.
33 ) proteolytic activity in the suppression of autoimmune responses.
34 (OPN), a pleiotropic cytokine implicated in autoimmune responses.
35 rocess and present self-peptides to suppress autoimmune responses.
36 ed that the DC-HIL/SD-4 pathway may regulate autoimmune responses.
37 clear whether it serves a functional role in autoimmune responses.
38 from the circulation might serve to trigger autoimmune responses.
39 eakdown in self-tolerance and propagation of autoimmune responses.
40 sion, but they often also are accompanied by autoimmune responses.
41 while also contributing to inflammatory and autoimmune responses.
42 ller cell-mediated killing modulates humoral autoimmune responses.
43 that directly contribute to a modulation of autoimmune responses.
44 otential for activation and recruitment into autoimmune responses.
45 antigenic responses and chronic inflammatory autoimmune responses.
46 B cells are important for the regulation of autoimmune responses.
47 ions in the host limit pathogenic Th1-driven autoimmune responses.
48 D exacerbated only genetically predetermined autoimmune responses.
49 major sources of IL-17A in antimicrobial and autoimmune responses.
50 play a significant role in inflammatory and autoimmune responses.
51 dysfunction but also may trigger detrimental autoimmune responses.
52 but also to the development of IL-17-driven autoimmune responses.
53 erons (IFNs) are important for antiviral and autoimmune responses.
54 (T(H)1) and T(H)17 cells are associated with autoimmune responses.
55 tribute to viral clearance and regulation of autoimmune responses.
56 t sufficient to protect against Th1-mediated autoimmune responses.
57 here are many parallels between allergic and autoimmune responses.
58 ens, yet prevents deleterious NK cell-driven autoimmune responses.
59 s frequently associated with microbe-related autoimmune responses.
60 T cells that are crucial for the control of autoimmune responses.
61 DCs limit T cell activation and thus prevent autoimmune responses.
62 s frequently the target of antibody-mediated autoimmune responses.
63 ed suppression that leads to amelioration of autoimmune responses.
64 relationship in MHC-II-dependent normal and autoimmune responses.
65 t for controlling excessive inflammation and autoimmune responses.
66 nd might lead to the development of aberrant autoimmune responses.
67 gut integrity and the control of anti-islet autoimmune responses.
68 intenance of immune homeostasis and blocking autoimmune responses.
69 lian cell surface glycoconjugates triggering autoimmune responses.
70 therapeutic target to treat TH17 cell-driven autoimmune responses.
71 pients against both alloimmune and recurring autoimmune responses.
72 e differentiation and function of T cells in autoimmune responses.
73 des, and other peptides that are relevant in autoimmune responses.
74 iator of T cell functions in both immune and autoimmune responses.
75 ses capable of exacerbating self-destructive autoimmune responses.
76 ocal inflamed tissues/organs to suppress the autoimmune response after adoptive transfer, thereby avo
77 clerosis and suggest an initially protective autoimmune response against apoB with a progressive dera
80 ith APECED, loss of AIRE appears to cause an autoimmune response against enteric defensins and loss o
81 ic anemia (AA) has 2 key characteristics: an autoimmune response against hematopoietic stem/progenito
82 als that in psoriasis HLA-C*06:02 directs an autoimmune response against melanocytes through autoanti
84 e death is sufficient to trigger an adaptive autoimmune response against myelin, suggesting that a si
85 mon neurological disorder, the origin of the autoimmune response against myelin, which is the charact
87 lmark of systemic lupus erythematosus is the autoimmune response against self nuclear Ags, including
88 hepatobiliary viral infection followed by an autoimmune response against the bile duct epithelia.
89 ught to test the hypothesis that inducing an autoimmune response against the cardiac sodium channel (
90 nd subsequent effects on the induction of an autoimmune response against the eye, we examined CD4 T c
92 ed immune cells elaborate a specific humoral autoimmune response against the von Willebrand factor A
95 It has become increasingly appreciated that autoimmune responses against neuronal components play an
97 disorder classic Ehlers-Danlos syndrome, and autoimmune responses against the alpha1(V) chain are lin
99 at IL-11 might serve as a biomarker of early autoimmune response and a selective therapeutic target f
102 OCK2 inhibitor downregulates the Th17-driven autoimmune response and improved clinical symptoms in ps
103 at underlie the continued progression of the autoimmune response and islet destruction is critical.
105 lost Foxp3 expression during an inflammatory autoimmune response and might be involved in inadequate
106 Multiple sclerosis involves an aberrant autoimmune response and progressive failure of remyelina
108 y Toso as a unique regulator of inflammatory autoimmune responses and an attractive target for therap
109 (MS) and for exploring the interface between autoimmune responses and CNS tissue that ultimately lead
111 (IFNs) in disease, from antivirus defense to autoimmune responses and immuno-metabolic syndromes.
112 suggesting a role for DNA-PKcs in regulating autoimmune responses and maintaining AIRE-dependent tole
113 ransferred MDSCs to downregulate Ag-specific autoimmune responses and prevent diabetes onset, suggest
114 et al. have shown that PAHSAs both attenuate autoimmune responses and promote beta cell survival in N
115 ransplants revealed a complete abrogation of autoimmune responses and severe downregulation of alloim
116 ng in contrast, only modestly contributed to autoimmune responses and the disease process in these mi
117 cells play a central role in the control of autoimmune responses and their generation and function a
118 toreactive T cells primed during the primary autoimmune response, and demonstrate that local antigen
119 negative selection, the ability to prime an autoimmune response, and the elimination of the relevant
120 itory molecule involved in immune tolerance, autoimmune responses, and antiviral immune evasion.
121 participate in the priming of the allo- and autoimmune responses, and their depletion can thus be ad
122 oids as a carrier for DNA and potentiator of autoimmune responses, and we propose a novel link betwee
126 s in RA as both promoters and targets of the autoimmune response, as well as discussing the mechanist
127 these peripheral Tregs (pTregs) in averting autoimmune responses, as well as immunological mechanism
128 es may be disadvantageous by contributing to autoimmune responses associated with antibiotic-refracto
130 a preceding infection is the trigger of the autoimmune response, but the mechanism connecting the in
131 t significantly affect anti-islet Th1 or Th2 autoimmune responses, but markedly increased inflammator
132 t commensal flora can dramatically influence autoimmune responses, but the mechanisms behind this are
135 te experimental autoimmune encephalomyelitis autoimmune response by inhibiting immune cell infiltrati
137 cells can independently support Ag-specific autoimmune responses by CD4 T cells in EAE is lacking.
138 is a pleiotropic cytokine that can regulate autoimmune responses by enhancing regulatory CD4(+)FoxP3
139 hematosus (SLE) and is postulated to enhance autoimmune responses by increasing access to intracellul
140 odel that could recapitulate T cell-specific autoimmune responses by initiating and sustaining inflam
141 icate that the DC-HIL/SD-4 pathway regulates autoimmune responses by mediating the T cell suppressor
143 ed that IDO2 expression by B cells modulates autoimmune responses by supporting the cross talk betwee
144 histocompatibility complexes (pMHC) blunted autoimmune responses by triggering the differentiation a
145 These injections also lead to an anti-AChR autoimmune response characterized by a significant produ
147 ticular, imaging of CD4+ T cells involved in autoimmune responses could be helpful in diagnosing myoc
148 However, the etiology and progression of autoimmune responses directed against these antigens are
149 spensible to sustain tolerance that prevents autoimmune responses directed at self-Ags during experim
152 indicate that pDCs are important in quieting autoimmune responses during EAE, and that trafficking in
153 isease remain unanswered, the development of autoimmune responses during infection clearly occurs in
154 40/Ox40L pathway drives cellular and humoral autoimmune responses during lupus nephritis in NZB/W F1
156 verting the effect of AR agonist on the Th17 autoimmune response from anti-inflammatory to proinflamm
157 olism (MYH, TRHDE, ALDH1A3), and nervous and autoimmune response (GRIA1, IL2, IL7, IL21, IL1R1) were
158 , SFB can promote IL-17-dependent immune and autoimmune responses, gut-associated as well as systemic
159 ay a critical role in the development of the autoimmune response, has not been extensively studied in
160 esides determining the target specificity of autoimmune responses, HLA molecules may influence diseas
161 t muscle degeneration due to an uncontrolled autoimmune response; however, the mechanisms leading to
163 tigens associated with the development of an autoimmune response in breast cancer has relevance to de
164 ated the statins' mechanisms that target the autoimmune response in humans, and evaluated their thera
170 to regulate alloimmunity and to abrogate the autoimmune response in NOD mice in different settings co
173 1 as a new cytokine that plays a role in the autoimmune response in the early phase of the disease.
178 f crosstalk between alloimmune responses and autoimmune responses in AILD is an important area that n
182 dy, we compared their effect on Th17 and Th1 autoimmune responses in experimental autoimmune uveitis,
187 SIgA-DCs are highly potent in inhibiting autoimmune responses in mouse models of type 1 diabetes
189 -secreting T cells play an important role in autoimmune responses in multiple sclerosis and the model
190 d examined their activities to stimulate the autoimmune responses in NOD mice, a model for human type
191 t insulinoma-released EXOs can stimulate the autoimmune responses in nonobese diabetic (NOD) mice, a
192 standing of the role of leptin in modulating autoimmune responses in SLE can open possibilities of le
193 ht the critical role of Stat4 Th1 signals in autoimmune responses in suppressing Foxp3(+) Treg respon
197 tem also has a key involvement in regulating autoimmune responses in the central nervous system.
198 effects of such a blockade on development of autoimmune responses in the CNS should be considered.
199 nce for this mechanism and the role of these autoimmune responses in various liver diseases, includin
200 nstrated that Foxp3+ Tregs potently suppress autoimmune responses in vivo through inhibition of the a
201 e to blunt polyclonal, multiantigen-specific autoimmune responses in vivo without impairing systemic
202 onses can still be decoupled from pathologic autoimmune responses in vivo, which may provide novel in
207 isease focus mainly on downstream targets of autoimmune responses, including effector cells and cytok
208 -17-producing helper T (T(H)17) cells during autoimmune responses, including experimental autoimmune
209 generally thought to be driven by a systemic autoimmune response, increasing evidence suggests that i
213 n 2 unrelated TTP patients suggests that the autoimmune response is antigen driven, because the proba
217 y demonstrated that TSHR, the target of this autoimmune response, is also a key susceptibility gene f
218 er, is subverted during infection, injury or autoimmune response leading to increased population of M
219 hat absence of pDCs during the priming of an autoimmune response leads to increased mobilization of M
224 C3aR on APC and T cells, a heightened local autoimmune response occurs in which myelin destruction i
225 or oxidative/nitrosative stress to elicit an autoimmune response or to contribute to disease pathogen
226 human protein, which may also contribute to autoimmune responses or enhanced pathology in some scrub
228 indicate potentially related islet and blood autoimmune response phenotypes that coincide with and pr
229 sues leads to an attenuation of pathological autoimmune responses, possibly as a means to mitigate in
230 e model of SLE-like serology, that during an autoimmune response, RAG was reinduced in antigen-activa
231 stratification, together with enrichment of autoimmune response-related (nucleotide binding site-leu
236 C9orf72 regulates immune homeostasis and an autoimmune response reminiscent of systemic lupus erythe
237 d the induction of T-helper 17 cell-mediated autoimmune responses resembling those observed in patien
241 by the immune system in MS to perpetuate the autoimmune response, suggesting that inhibiting immune a
242 ic human gut bacteria that regulate adaptive autoimmune responses, suggesting therapeutic targeting o
243 case for the beta-cell as the trigger of an autoimmune response, supported by analogies in cancer an
245 y to microbes and xenobiotics, and regulates autoimmune responses that can affect the central nervous
247 changes occurred in the absence of adaptive autoimmune responses, the findings show that brain infec
248 cumulation of NLRs can result in unwarranted autoimmune responses, their cellular concentrations must
249 to HIV-1 restriction and suppression of the autoimmune response through direct cleavage of viral and
251 une privileges by suppressing alloimmune and autoimmune responses through its receptor, CD200R, expre
252 he gut commensal flora in sustaining ongoing autoimmune responses through the local fine tuning of T-
254 The question considered is, "What causes the autoimmune response to begin and what causes it to worse
257 ce of this strategy is the development of an autoimmune response to HLA-presented epitopes encoded do
259 mmune system by restraining inflammation and autoimmune response to intracellular Ags released from d
260 n environment or lifestyle alter the humoral autoimmune response to islet antigens should help explai
262 provide the first tangible evidence that an autoimmune response to retina is causally involved in pa
264 h progressive neurodegeneration caused by an autoimmune response to self-antigens in a genetically su
266 a critical role for HLA-DR3 in generating an autoimmune response to SmD and lupus nephritis in the NZ
270 n made in clarifying mechanisms of allo- and autoimmune responses to FVIII and in suppression of thes
272 enia gravis (EAMG) are caused by Ab-mediated autoimmune responses to muscle nicotinic acetylcholine r
273 antly delays and attenuates inflammatory and autoimmune responses to myelin Ags in the mouse experime
275 noma paradigm, little is known about whether autoimmune responses to normal tissue can induce rejecti
277 ivo lymphotoxin blockade link these systemic autoimmune responses to the formation of gut-associated
278 uggest that posttransplantation induction of autoimmune responses to tissue-specific antigens contrib
279 ients with cancer and frequently result from autoimmune responses triggered by the ectopic expression
281 ell lineage is important in inflammatory and autoimmune responses, via its ability to produce interle
282 on in heart tissue, and TnI-directed humoral autoimmune responses, was also present in 2 cases of ICI
284 ether oligodendrocyte death could cause this autoimmune response, we examined the oligodendrocyte abl
286 B cells play a crucial role in induction of autoimmune responses, we defined the role of B cells and
291 in T1D patients may account for the chronic autoimmune response when damage-associated molecular pat
292 mast cells has no discernible effect on the autoimmune response, which involves both innate and adap
295 patibility protein (pMHC) interactions limit autoimmune responses while enhancing T cell response to
296 lace to prevent such potentially deleterious autoimmune responses while preserving immunity integral
297 ular metabolism and detection, triggering an autoimmune response with an increase in cerebral IFN-alp
298 that preferentially target disease-relevant autoimmune responses within the CD8(+) T-cell compartmen
299 to wild-type mice induce a transient primary autoimmune response without apparent anti-nuclear Ab rea