戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 lmuramyl-L-alanine amidase, the pneumococcal autolysin.
2 es suggested as being involved in triggering autolysin.
3 P60, an established SecA2-dependent secreted autolysin.
4 hTSP-1 and Vn, respectively, and a bacterial autolysin.
5 s accelerated by the addition of lysozyme or autolysin.
6 r/stationary-phase survival (Rpf/Sps) family autolysin.
7 ng AmiC's purported function as a gonococcal autolysin.
8 of S. mitis known to possess pneumolysin and autolysin.
9 ffects chain length through activation of an autolysin.
10 ease (MF-1), a putative nuclease (MF-3), and autolysin.
11 ypeptidases and the cell wall degradation by autolysins.
12 specialized functions of essential cell wall autolysins.
13 can O-acetylation and repression of cellular autolysins.
14 pholipid (PL) degradation, and activation of autolysins.
15 ions in peptidoglycan structure or levels of autolysins.
16 o explore the binding mechanism of the major autolysin Acm2 from the probiotic bacterium Lactobacillu
17 of pneumolysin from WU2 was not dependent on autolysin action.
18 rine protease inhibitor) showed an increased autolysin activity.
19  at a pH non-permissive for other gonococcal autolysins, an autolytic activity was detectable in the
20 R, which "repurposes" SinR as a repressor of autolysin and motility genes, are discussed.
21 biofilms, where both are mediated by the Atn autolysin and the GelE protease.
22 um and regulates the reciprocal synthesis of autolysins and autolysin inhibitors to co-ordinate growt
23 ttle is known about the factors that control autolysins and how penicillins subvert this regulation t
24 dy, we demonstrate that binding domains from autolysins and lysins can be fused to the Fc region of h
25                                              Autolysins and phage lysins are peptidoglycan hydrolases
26                                  Thus, these autolysins and phage-entry enzymes have a shared chemica
27 n TiO2 NP, limiting the activity of released autolysins and preventing further lytic activity.
28  in counteracting the deleterious effects of autolysins and reactive oxygen species in beta-lactam-tr
29 y controlling the transcription of genes for autolysins and their inhibitors.
30                                  Among known autolysins and their regulators, we found that arlRS rep
31 ll envelope stress decreases the activity of autolysins and thereby reduces the rate of antibiotic-de
32 ociated beta-lactamase, lipoprotein, lipase, autolysin, and an ABC transporter lipoprotein.
33     Although some factors, such as sortases, autolysin, and extracellular DNA (eDNA), have been assoc
34  the controlled secretion and proteolysis of autolysin, and this developmental program appears to be
35 r coagulase-negative staphylococci that some autolysins are able to bind polymer surfaces, these data
36 mary, we reveal the hydrolytic activities of autolysins are associated with the surface display of S.
37 omplestatin-treated cells, highlighting that autolysins are required to liberate Spa during cell divi
38  hydrolases involved in this process (called autolysins) are potentially lethal enzymes that can caus
39  a condition which is reported to inactivate autolysin, as well as most known pneumococcal phage lysi
40  to peptidoglycan and blocks the activity of autolysins, as a chemical probe of autolysin function.
41 ectrometry revealed the major staphylococcal autolysin Atl as a bacterial binding protein for hTSP-1.
42               Staphylococcus aureus secretes autolysin (Atl) to complete cell division by hydrolyzing
43 ns extracellular adherence protein (Eap) and autolysin (Atl), both surface-exposed proteins containin
44                We show here that Esp cleaves autolysin (Atl)-derived murein hydrolases and prevents s
45  may function independently from several key autolysins (Atl, LytM, and LytN) and regulators (ArlRS,
46 Staphylococcus aureus, the so-called "major" autolysin, Atl, has long been associated with host adhes
47 study, we identify the major cell separation autolysin AtlA as an SCC-binding protein.
48                       The bifunctional major autolysin AtlA of Staphylococcus aureus cleaves the bact
49        We have identified a gene encoding an autolysin (atlA) from Neisseria gonorrhoeae.
50 ing downstream interactions with the primary autolysin, AtlA.
51  partial collapse of a pathway consisting of autolysins, AtlA and Sle1, a transmembrane sugar permeas
52 n, primary attachment, and expression of the autolysin AtlE, but lacked delta-toxin production.
53 o assess the importance of the proteinacious autolysin (AtlE) and the polysaccharide intercellular ad
54 o analysis approach, we identified the major autolysin AtlS as a natural substrate of SdbA and showed
55 ne important roles for the major E. faecalis autolysin (Atn), eDNA, and sortase A (SrtA) during the d
56  the activity of cell wall hydrolases called autolysins, but how penicillins misactivate these deadly
57  predicted to represent the docking site for autolysins by modeling studies.
58 g the virulence determinants pneumolysin and autolysin classically associated with S. pneumoniae.
59 lation of the CwlO and LytE systems and that autolysins control different aspects of cell morphogenes
60 ent is not required for cell death since the autolysin-defective lytC, lytD, lytE, lytF strain fails
61 he culture medium, and the lack of effect of autolysin-deficient mutants.
62 by the coupling of massive PL degradation to autolysin-dependent killing and bacterial lysis or both.
63      The secreted Listeria monocytogenes p60 autolysin digests peptidoglycan and promotes bacterial i
64 ted by means of broth-enrichment culture and autolysin-encoding gene (lytA) quantitative polymerase c
65 e, undergoing degradation and remodeling by 'autolysins', enzymes that break down PG.
66      The growth of this PG polymer relies on autolysins, enzymes that create space within the structu
67 plestatin and corbomycin block the action of autolysins-essential peptidoglycan hydrolases that are r
68 rtate phosphatase (RapD), and a regulator of autolysin expression (LytR).
69     Further characterization revealed normal autolysin expression, localization, and triggering by de
70 al-PCR protocol (targeted at pneumolysin and autolysin) for EDTA blood samples.
71    A gene, designated atlS, encoding a major autolysin from Streptococcus gordonii, was identified an
72                          Here we report that autolysins from Gram-positive pathogenic bacteria, enzym
73 ontaining walls are similar, indicating that autolysin function is similar and suggesting that modula
74                          As such, disrupting autolysin function reduced the affinity of S. aureus for
75              We demonstrate that by blocking autolysin function, type V glycopeptide antibiotics are
76 tivity of autolysins, as a chemical probe of autolysin function.
77  We tested blood by PCR for the pneumococcal autolysin gene in children aged 1-59 months in the Pneum
78 rulence factor of pneumococci, and the major autolysin gene lytA, also considered an important virule
79      Real-time PCR targeting lytA (the major autolysin gene) and piaB (permease gene of the pia ABC t
80 , but none of them showed translation of the autolysin gene, which is located downstream of recA.
81 tch and exists in a SlrR(LOW) state in which autolysin genes are on, and a SlrR(HIGH) state in which
82 enes are on, and a SlrR(HIGH) state in which autolysin genes are repressed by SinR-SlrR.
83 d, rat appears to be a negative regulator of autolysin genes including lytM and lytN.
84 d expression in the stp1 mutant and included autolysin genes.
85  monoclonal antibodies against the S. aureus autolysin glucosaminidase (Gmd) domain, and subsequent t
86 ion conditions, mutations in pneumolysin and autolysin had different effects on virulence.
87 A, a protective antigen, and LytA, the major autolysin) have been well characterized.
88 l studies of lysostaphin, a PG lytic enzyme (autolysin), have suggested that residues in the active s
89   To elucidate the function of this putative autolysin, here named IsdP, we investigated its contribu
90 which [the p60 and N-acetylmuramidase (NamA) autolysins] hydrolyze bacterial peptidoglycan (PGN) and
91           The role played by pneumolysin and autolysin in pneumococcal meningitis is poorly understoo
92                  Downregulation of the major autolysin in Streptococcus pneumoniae leads to penicilli
93                           The lack of active autolysin in the mutant cells became apparent by impaire
94 d to bacteriophage endolysins and acts as an autolysin in the stationary phase.
95 ve bacteria that regulates the all-important autolysins in cell wall homeostasis.
96  LytM domain of DipM interacts with multiple autolysins, including the soluble lytic transglycosylase
97  and by wild-type cells after treatment with autolysin, indicating that they are localized to the per
98 s the reciprocal synthesis of autolysins and autolysin inhibitors to co-ordinate growth and division
99 that the amidase LytA, the main pneumococcal autolysin, inhibits complement-mediated immunity indepen
100  by a multimodular LysM domain from AtlA, an autolysin involved in cell division in the opportunistic
101                              The activity of autolysins is not restricted to the producer cells but c
102               Accordingly, the expression of autolysins is tightly regulated by several endogenous re
103 dunensis contains a gene encoding a putative autolysin located adjacent to the Isd operon.
104 of dividing cells from cleavage by the major autolysin LytA and occurs at the septal site.
105                             The pneumococcal autolysin LytA is a key virulence factor involved in sev
106                             The pneumococcal autolysin LytA is a virulence factor involved in autolys
107  a process dependent on the suicidal amidase autolysin LytA.
108 pneumolysin but depended on the pneumococcal autolysin LytA.
109 ids (LTAs) that bind and sequester the major autolysin LytA.
110 hat do not express pneumolysin (DeltaPly) or autolysin (LytA(-)) caused very mild or no disease.
111    Immunodetection studies revealed that the autolysin (LytA), normally located on the cell wall, was
112  pneumolysin (Pln), pyruvate oxidase (SpxB), autolysin (LytA), pneumococcal surface protein A, or neu
113 rial whole-cell screen based on pneumococcal autolysin-mediated lysis induction was developed to iden
114                                              Autolysin-mediated peptidoglycan hydrolysis, in particul
115  the genetic determinant of the pneumococcal autolysin (N-acetylmuramic acid-L-alanine amidase).
116                             The lytA-encoded autolysin (N-acetylmuramoyl-L-alanine amidase) of Strept
117 were resistant to hydrolysis by pneumococcal autolysin (N-acetylmuramyl-L-alanine amidase) but were c
118 umolysin-negative derivative (P-1), and into autolysin-negative derivative (A-1).
119                              Acm2, the major autolysin of Lactobacillus plantarum, is a tripartite pr
120 e strain by adding purified recombinant AtlA autolysin of S. mutans but was only partially restored b
121                   The lytA gene encoding the autolysin of Streptococcus pneumoniae may be a virulence
122 ; and (ii) indirectly, through adsorption of autolysins on TiO2 NP, limiting the activity of released
123 s type I, also shows reduced secretion of an autolysin, p60.
124   On the basis of LCA models, sensitivity of autolysin PCR and pneumolysin PCR was 82% and 89%, respe
125 strong in vivo evidence that pneumolysin and autolysin play crucial roles in the pathogenesis of pneu
126                 Peptidoglycan hydrolases, or autolysins, play a critical role in cell wall remodeling
127  transcript levels of lytA, which encodes an autolysin previously implicated in biofilms, and also th
128 competence-stimulating peptide (CSP) induced autolysin production and cell lysis of its own non-compe
129 ption of genes that function in motility and autolysin production.
130  that appeared to be mainly due to decreased autolysin production.
131 he Streptococcus mutans atlA gene encodes an autolysin required for biofilm maturation and biogenesis
132  of lytic transglycosylases (LTs), a type of autolysin responsible for breaking down PG glycan chains
133 ptococcus pneumoniae (Sp), LytA is the major autolysin responsible for penicillin-induced bacteriolys
134 ome highly susceptible to degradation by the autolysins, resulting in thinning of the cell wall.
135 s for ligand restriction by the pneumococcal autolysin, revealing for the first time an importance of
136              It was previously proposed that autolysin's primary role in the virulence of pneumococci
137                             A mutant lacking autolysin showed the same pattern of pneumolysin release
138 e mother cell, a process that depends on the autolysin SpoIID and two proteins of unknown function, S
139 h aids in bacterial survival, as it prevents autolysins such as lysozyme from cleaving the PG.
140           These studies establish IsdP as an autolysin that functions in heme acquisition and describ
141 e we present evidence that SpoIIP is also an autolysin, that it acts in tandem with SpoIID, and that
142 wall protein extract containing concentrated autolysin to exponentially growing TA-containing and TUA
143 reB homolog, MreBH, which localizes the LytE autolysin to the RodA-containing elongasome complex.
144 misactivation of cell wall hydrolases called autolysins to induce cell lysis.
145 riety of endogenous cell wall hydrolases, or autolysins, to remodel their cell walls during processes
146                                 We show that autolysins trim the outermost peptidoglycan fragments an
147 emonstrate that the peptidoglycan-remodeling autolysins under sigma(D) control, LytC, LytD, and LytF,
148 herefore the probability of being cleaved by autolysins varies with orientation of the chain on the c
149 were derived from PGN remodeled by bacterial autolysins, was recognized.
150                          ClpB, lysozyme, and autolysin were detected in the culture supernatant of th
151 Unchecked, ComX upregulates bacteriocins and autolysins, while simultaneously suppressing immunity pe
152               Cell separation is mediated by autolysins, whose genes are under the negative control o
153           As such, FlgJ represents the first autolysin with this activity to be characterized from a
154 n hydrolase activity and biofilm maturation, autolysin zymography was performed, which revealed an al

 
Page Top