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1 s relatively spared in experimental diabetic autonomic neuropathy.
2 ibodies and had idiopathic or paraneoplastic autonomic neuropathy.
3 logic markers of various forms of autoimmune autonomic neuropathy.
4 educed HRV identifies diabetic patients with autonomic neuropathy.
5 ood flow would be impaired in diabetics with autonomic neuropathy.
6 with long-standing diabetes and established autonomic neuropathy.
7 abetic distal symmetrical polyneuropathy and autonomic neuropathy.
8 evidence of definite and 10 (31%) had early autonomic neuropathy.
9 tential pathogenetic mechanisms for diabetic autonomic neuropathy.
10 31.3% Child B, and 60% Child C had definite autonomic neuropathy.
11 in patients with advanced liver disease and autonomic neuropathy.
12 t, and parasymN dysfunction is a hallmark of autonomic neuropathy.
13 pastic paraplegia and hereditary sensory and autonomic neuropathy.
14 core for cardiovascular disease, and cardiac autonomic neuropathy.
15 ia, cardiovascular disease risk, and cardiac autonomic neuropathy.
16 lternate phenotype of hereditary sensory and autonomic neuropathy.
17 en genetically linked with human sensory and autonomic neuropathy.
18 astating genetic disease featuring a bladder autonomic neuropathy.
19 arrhythmias associated with diabetic cardiac autonomic neuropathy.
20 eta as a new therapeutic target for diabetic autonomic neuropathy.
21 Half had autonomic neuropathy.
22 l dysautonomia (FD), a recessive sensory and autonomic neuropathy.
23 ulin treatment, metabolic abnormalities, and autonomic neuropathy.
24 nglionic antibody likely mediates autoimmune autonomic neuropathy.
25 tribute to the development of human diabetic autonomic neuropathy.
26 in the pathogenesis of experimental diabetic autonomic neuropathy.
27 Moreover, in hereditary sensory and autonomic neuropathies.
28 iabetic large and small fiber peripheral and autonomic neuropathies.
29 neuronal dysfunction and loss in the sensory/autonomic neuropathies.
30 nesis, pathophysiology and treatment of some autonomic neuropathies.
31 ), and arterial stiffness and cardiovascular autonomic neuropathy (24 [1.8%] vs 13 [1.0%]; p=0.015).
32 mmon signs were peripheral neuropathy (47%), autonomic neuropathy (31%), cerebellar ataxia (26%), sub
33 inating neuropathy (2.8; 1.6-5.1; P = .001), autonomic neuropathy (4.2; 1.4-12.3; P = .009), and mono
34 6 patients with idiopathic or paraneoplastic autonomic neuropathy (41 percent), in 6 of 67 patients w
35 stural tachycardia syndrome, 211; peripheral autonomic neuropathy, 463; myalgic encephalomyelitis/chr
37 ic gastrointestinal dysmotility, or diabetic autonomic neuropathy (9 percent), and in none of 44 pati
38 ients (13 female, 5 male) who had autoimmune autonomic neuropathy (AAN) and ganglionic acetylcholine
44 ave implications for both long-term diabetic autonomic neuropathies and insulin-induced hypoglycemia,
45 es patients with various forms of autoimmune autonomic neuropathy and distinguishes these disorders f
50 nerve abnormalities, frequent optic atrophy, autonomic neuropathy and upper and lower motor neurone s
52 omia (FD) is a severe hereditary sensory and autonomic neuropathy, and all patients with FD have a sp
53 treatment of Parkinson's disease-associated autonomic neuropathy, and antioxidant therapies have bee
54 processes, including hepatitis C infection, autonomic neuropathy, and drug side effects, that can re
55 y aid in early detection of both sensory and autonomic neuropathy, and perhaps in the case of patient
60 ion, axonal sensory-motor polyneuropathy and autonomic neuropathy are commonly seen in patients with
62 pathy, peripheral neuropathy, cardiovascular autonomic neuropathy, arterial stiffness, and hypertensi
64 s' CNFL correlated with the severity of both autonomic neuropathy assessed by the Compound Autonomic
66 abetic distal symmetrical polyneuropathy and autonomic neuropathy but that the clinical role of ARIs
68 rmine the association between cardiovascular autonomic neuropathy (CAN) and indices of left ventricle
69 y on the prevalence and incidence of cardiac autonomic neuropathy (CAN) in former DCCT intensive and
72 s studies have suggested that cardiovascular autonomic neuropathy (CAN) may predict rapid kidney func
76 myloidosis characterized by sensorimotor and autonomic neuropathy, cardiac conduction defects, and in
77 ly progressive gastrointestinal symptoms and autonomic neuropathy caused by autosomal dominant, hered
78 Familial dysautonomia (FD) is a sensory and autonomic neuropathy caused by mutations in elongator co
80 mmon member of a group of congenital sensory/autonomic neuropathies characterized by widespread senso
81 ary motor neuropathy, hereditary sensory and autonomic neuropathy, complicated hereditary spastic par
87 duals with long-standing type I diabetes and autonomic neuropathy has not been assessed, nor has it b
88 ity to pain (CIP) and hereditary sensory and autonomic neuropathies (HSAN) are clinically and genetic
90 acts distinctly from hereditary sensory and autonomic neuropathy (HSAN)-causing SPTLC2 variants by c
94 of gene mutations of hereditary sensory and autonomic neuropathies I, III and IV could lead to more
95 he gene mutations for hereditary sensory and autonomic neuropathies I, III, and IV are now known and
96 hemical sympathectomy in mice resulted in BM autonomic neuropathy, impaired Lin(-)cKit(+)Sca1(+) (LKS
97 he neuropathological hallmark of sympathetic autonomic neuropathy in diabetic rats, increased 9- to 1
98 opathologic hallmark of diabetic sympathetic autonomic neuropathy in human and several insulinopenic
100 neuritic dystrophy, the hallmark of diabetic autonomic neuropathy in mouse prevertebral sympathetic g
112 ility (AGID) is a limited form of autoimmune autonomic neuropathy occurring idiopathically or in a pa
113 patients with liver disease had evidence of autonomic neuropathy; of these, 12 (36%) had evidence of
114 haracterised by a progressive peripheral and autonomic neuropathy often with associated cardiac failu
116 elation was observed between the severity of autonomic neuropathy or the duration of diabetes and the
118 progressive distal symmetric polyneuropathy, autonomic neuropathy, radiculo-plexopathies, and mononeu
120 a (FD), a devastating hereditary sensory and autonomic neuropathy, results from an intronic mutation
121 ng cholinergic systems, including autoimmune autonomic neuropathy, seizures, dementia, and movement d
122 king improvement in the severity of diabetic autonomic neuropathy shown with IGF-I treatment in these
123 inflammation, advanced glycation endpoints, autonomic neuropathy, sleep-disordered breathing, and ge
125 much greater risk of developing a painful or autonomic neuropathy than those individuals with no chan
126 It is indistinguishable from the subacute autonomic neuropathy that may accompany lung cancer or o
127 lial dysautonomia (FD) is a rare sensory and autonomic neuropathy that results from a mutation in the
128 ith idiopathic and paraneoplastic autoimmune autonomic neuropathy, the severity parallels serum level
129 f a larger disease process, such as diabetic autonomic neuropathy, traumatic brain injury, and spinal
131 ants cause peripheral hereditary sensory and autonomic neuropathy type 1 (HSAN1) due to the synthesis
133 concentrations cause hereditary sensory and autonomic neuropathy type 1 (HSAN1), an axonal neuropath
134 PT are known to cause hereditary sensory and autonomic neuropathy type 1 (HSAN1), we examined 10 addi
136 mic the mutations seen in hereditary sensory autonomic neuropathy type 1 as well as a third mutant N1
137 ations in human SPT cause hereditary sensory autonomic neuropathy type 1, a disease characterized by
138 l diseases, including hereditary sensory and autonomic neuropathy type 1A (HSAN1A), GM3 synthase defi
139 clinical spectrum for hereditary sensory and autonomic neuropathy type 1E (HSAN1E) and a potential pa
145 amilial dysautonomia (hereditary sensory and autonomic neuropathy type III), we found that the techni
147 ered in patients with Hereditary Sensory and Autonomic Neuropathy type V (HSAN V), caused by the 661C
148 h factor (NGF) causes Hereditary Sensory and Autonomic Neuropathy type V, a rare disease characterize
149 monogenic disorders: Hereditary Sensory and Autonomic Neuropathy type VI (caused by a loss of DST-a)
150 tions in SPTLC1 cause hereditary sensory and autonomic neuropathy (type I) (HSAN1), an adult onset, a
151 d with autosomal-dominant hereditary sensory autonomic neuropathy, type 1A, by disrupting an essentia
152 our hypothesis, we evaluated the presence of autonomic neuropathy using a battery of tests in 33 pati
153 an in vivo streptozotocin model of diabetic autonomic neuropathy using an unbiased counting method t
154 wth factor I (IGF-I) on diabetic sympathetic autonomic neuropathy was examined in an experimental str
155 ensorimotor) neuropathy was found in 71% and autonomic neuropathy was found in 48% of the patients.
157 To address the pathogenesis of diabetic autonomic neuropathy, we have examined the sympathetic n
158 nly dangerous as contributors to sensory and autonomic neuropathies when elevated by inherited SPT mu
159 subsequently were found to have sensorimotor autonomic neuropathy, whereas 2 others had subclinical a
163 tion are likely to have a high prevalence of autonomic neuropathy with an associated increase in mort
164 al dominant diseases: hereditary sensory and autonomic neuropathy with dementia and hearing loss type
165 ration in one form of hereditary sensory and autonomic neuropathy with dementia and hearing loss.
167 be a valuable model of diabetic sympathetic autonomic neuropathy with unambiguous, rapidly developin