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1 yslipidemia to 8.3 (95% CI, 4.6 to 14.9) for avascular necrosis.
2 ine patients had MR findings consistent with avascular necrosis.
3 ataracts; 1%, posttransplant diabetes; 0.2%, avascular necrosis; 0.2%, posttransplant lymphoprolifera
4 with a 1.2-fold increase in the risk of both avascular necrosis (95% CI 1.1, 1.4) and stroke (95% CI
6 gesics and physical therapy for treatment of avascular necrosis, and use of angiotensin-converting en
8 eology and the development of bone infarcts, avascular necrosis (AVN) and other bone diseases associa
9 xperience a high rate of fracturing and some avascular necrosis (AVN), but little is known about the
12 yocardial infarction, stroke, heart failure, avascular necrosis, cataract, or glaucoma) as the compos
13 cluded chronic osteomyelitis, growth arrest, avascular necrosis, chronic dislocation, and pathologic
14 recipients had significantly lower rates of avascular necrosis, cytomegalovirus, cataracts, new-onse
16 suffer from chronic pain syndromes including avascular necrosis, leg ulcers, and intractable pain.
23 recognized acute complication of treatment, avascular necrosis of weight-bearing bones, is still not
24 lipped capital femoral epiphysis (SCFE), and avascular necrosis/osteonecrosis (AVN) in 4598 children
28 up, and 0% of the methylprednisolone group), avascular necrosis (seen in 11% of the cyclophosphamide
29 d HIV act as common risk factors for stroke, avascular necrosis, severe splenic dysfunction, pulmonar
30 s suppression resulted in severe hypoxia and avascular necrosis that are incompatible with progressiv