ライフサイエンスコーパス: avian influenza virus

1 well as therapeutic treatments of HPAI H7N7 avian influenza virus.

2 during human infection with pathogenic H7N7 avian influenza virus.

3 vaccine is urgently needed against the H7N9 avian influenza virus.

4 ls to effectively control a modified form of avian influenza virus.

5 ns, the avian protozoan Eimeria tenella, and avian influenza virus.

6 erity of both seasonal and highly pathogenic avian influenza virus.

7 ed by furin, a hallmark of highly pathogenic avian influenza virus.

8 esence of a PB2 gene segment derived from an avian influenza virus.

9 DCs in the pathogenesis of highly pathogenic avian influenza virus.

10 and ferrets higher than that in an authentic avian influenza virus.

11 its coding sequences are very like those of avian influenza virus.

12 the high mortality of human infected by H5N1 avian influenza virus.

13 uction of neutralizing Abs specific for H5N1 avian influenza virus.

14 rus, whatever its origin, is very similar to avian influenza virus.

15 tects ferrets against H5N1 highly pathogenic avian influenza virus.

16 lerable vaccine with broad immunogenicity to avian influenza virus.

17 vaccines may help promote protection against avian influenza virus.

18 h the severity of infection with seasonal or avian influenza virus.

19 PAI viruses and cocirculating low-pathogenic avian influenza viruses.

20 with activity against highly pathogenic H5N1 avian influenza viruses.

21 hanisms of the differential pathogenicity of avian influenza viruses.

22 ent barrier against zoonotic transmission of avian influenza viruses.

23 ection with both low- and high-pathogenicity avian influenza viruses.

24 ing of the species barrier compared to other avian influenza viruses.

25 so that it is similar to that observed from avian influenza viruses.

26 the virus is a reassortant of H7N9 and H9N2 avian influenza viruses.

27 infections highlight the threat of emerging avian influenza viruses.

28 n of the neuraminidase (NA) of H2N2 and H9N2 avian influenza viruses.

29 tained replication in humans attenuates H5N1 avian influenza viruses.

30 minants that govern airborne transmission of avian influenza viruses.

31 velopment of pandemic influenza strains from avian influenza viruses.

32 time is posed by the highly pathogenic H5N1 avian influenza viruses.

33 ght confer heterosubtypic protection against avian influenza viruses.

34 ring therapeutic protection against human or avian influenza viruses.

35 at human disease caused by highly pathogenic avian influenza viruses.

36 redict the pandemic potential of circulating avian influenza viruses.

37 thought to potentiate antigenic diversity in avian influenza viruses.

38 clear distinction between human-adapted and avian influenza viruses.

39 ltry farms in northwest Iowa for exposure to avian influenza viruses.

40 s virus can also reassort with H5N1 and H9N2 avian influenza viruses.

41 the assessment of the pandemic potential of avian influenza viruses.

42 s containing various combinations of Cal and avian influenza virus A/chicken/Nanchang/3-120/01 (H3N2)

43 Here, we characterize a low-pathogenicity avian influenza virus, A/chicken/Israel/810/2001 (H9N2)

44 mportant role in the evolutionary biology of avian influenza viruses-a manifestation of the "storage

45 Our data provide a glimpse into avian influenza virus adaptation in mammals, with broad

46 rase genes are known to play a major role in avian influenza virus adaptation to mammalian hosts.

47 nd continuance today of a highly lethal H5N1 avian influenza virus (AIV) causing human disease has ra

48 ement of the human-origin PA gene segment in avian influenza virus (AIV) could overcome barriers to c

49 r rapid, sensitive and specific detection of avian influenza virus (AIV) H5N1.

50 Vs in fruit bats and serological evidence of avian influenza virus (AIV) H9 infection in frugivorous

51 H9N2 avian influenza virus (AIV) has an extended host range,

52 ainst H7 (52%), H5 (55%) and H9 (6%) subtype avian influenza virus (AIV) in egg yolk samples, and 45%

53 arly diagnosis of the highly pathogenic H5N1 avian influenza virus (AIV) is significant for preventin

54 Avian influenza virus (AIV) subtype H5N1 attracts partic

55 Avian influenza virus (AIV) subtype H5N1 was first disco

56 ve bird markets (LBMs) are major targets for avian influenza virus (AIV) surveillance programmes.

57 RNA oligonucleotide sequences related to the avian influenza virus (AIV) type H5N1.

58 tion of oligonucleotide sequences related to avian influenza virus (AIV) type H5N1.

59 y nodes, we infer that the internal genes of avian influenza virus (AIV) underwent a global selective

60 ratory birds in the ecology and evolution of avian influenza virus (AIV), there is a lack of informat

61 l determination of DNA sequence derived from Avian Influenza Virus (AIV), type H5N1.

62 which 84 were real-time RT-PCR positive for avian influenza virus (AIV).

63 fy an interaction between specific CHIRs and avian influenza virus (AIV).

64 sence of detectable antibodies in serum) for avian influenza viruses (AIV) among 4,485 birds, from 11

65 focus of surveillance activities monitoring avian influenza viruses (AIV) circulating in poultry.

66 Transmission of pathogenic avian influenza viruses (AIV) from wild birds to domesti

67 d, quantitative, and label-free detection of avian influenza viruses (AIV) H5N1.

68 glycoproteins and internal gene segments of avian influenza viruses (AIV) sampled from wild birds.

69 olution to understand virulence evolution in avian influenza viruses (AIV).

70 Novel reassortants of H7N9, H10N8, and H5N6 avian influenza viruses (AIVs) are currently circulating

71 Several subtypes of avian influenza viruses (AIVs) are emerging as novel hum

72 Our surveillance of avian influenza viruses (AIVs) at Delaware Bay, USA, rev

73 ces can result in variable susceptibility of avian influenza viruses (AIVs) carrying resistance-assoc

74 Outbreaks of highly pathogenic strains of avian influenza viruses (AIVs) cause considerable econom

75 H9N2 avian influenza viruses (AIVs) circulate in poultry thro

76 H7N9 avian influenza viruses (AIVs) continue to evolve and re

77 TANCE The frequency of human infections with avian influenza viruses (AIVs) has increased in recent y

78 Avian influenza viruses (AIVs) have been pivotal to the

79 ce a neutralizing antibody (NAb) response to avian influenza viruses (AIVs) in rhesus macaques.

80 viously found during OS and ZAN selection in avian influenza viruses (AIVs) of the N3 to N9 subtypes

81 Phylogenetic analysis of these two novel avian influenza viruses (AIVs) suggested that their geno

82 he best way to predict and identify emerging avian influenza viruses (AIVs) that pose a potential thr

83 of the hemaglutinin and neuramidase genes of avian influenza viruses (AIVs) to identify sequences tha

84 Due to the recent concern about pandemic avian influenza virus and because CD4 T cells specific f

85 ential for reassortment of H1N1 viruses with avian influenza virus and emphasize the need for continu

86 zed human sera against the tl/TX/079/07 H3N8 avian influenza virus and observed low but detectable an

87 to primary challenge with highly pathogenic avian influenza virus and onward transmission dynamics w

88 protection in mice against clade 0, 1, and 2 avian influenza viruses and also protected against seaso

89 ccines (pLAIV) representing four subtypes of avian influenza viruses and found that pLAIVs replicate

90 ion and poultry adaptation of H9N2 and other avian influenza viruses and helps us understand the stri

91 fections may represent a portal of entry for avian influenza viruses and highlights the need to bette

92 response to the continuing evolution of H5N1 avian influenza viruses and human infections, new candid

93 ever, the results clearly indicate that H9N2 avian influenza viruses and pH1N1 viruses, both of which

94 nn Arbor/6/60 (H2N2) could be transferred to avian influenza viruses and produce partially attenuated

95 early apoptosis of PAM limits the spread of avian influenza viruses and that PB1-F2 could play a con

96 the innate and adaptive immune responses to avian influenza viruses and their role in disease and re

97 pandemic H1N1 virus, highly pathogenic H5N1 avian influenza virus, and the recently emerged H7N9 str

98 Titers to avian influenza virus antigens increased with age and wi

99 thern Vietnam, we tested for antibodies to 5 avian influenza virus antigens, using a protein microarr

100 Human infections with avian influenza viruses are a serious public health conc

101 Highly pathogenic H5N1 avian influenza viruses are associated with severe disea

102 Highly pathogenic H5N1 avian influenza viruses are associated with severe disea

103 hosts for avian influenza viruses.IMPORTANCE Avian influenza viruses are capable of crossing the spec

104 A(H5N1) and A(H9N2) avian influenza viruses are enzootic in Egyptian poultry

105 H9N2 avian influenza viruses are enzootic in poultry across A

106 Although most avian influenza viruses are harmless for humans, some (s

107 ze to the mitochondria while PB2 proteins of avian influenza viruses are nonmitochondrial.

108 Emerging avian influenza viruses are of global concern because th

109 Avian influenza viruses are widespread in birds, contagi

110 humans, some (such as highly pathogenic H5N1 avian influenza viruses) are capable of infecting humans

111 explanation for the loss of CpG motifs from avian influenza viruses as they adapt to mammalian hosts

112 etween antigenic drift and viral fitness for avian influenza viruses as well as the challenges of pre

113 was greater than human and low-pathogenicity avian influenza viruses, as reported by others.

114 virus differ from typical low-pathogenicity avian influenza viruses at only a small number of amino

115 lication of and immune response to human and avian influenza viruses at relevant physiological temper

116 A novel highly pathogenic avian influenza virus belonging to the H5 clade 2.3.4.4

117 etain fusion and attachment properties of an avian influenza virus but displayed robust growth and co

118 are that a highly pathogenic strain of H7N1 avian influenza virus can be adapted to become capable o

119 d ecology of viruses in this host.IMPORTANCE Avian influenza viruses can jump from wild birds and pou

120 uctive and virulent infection of humans with avian influenza viruses can occur.

121 We also demonstrated that avian influenza viruses carrying the PB1 and PB2 mutatio

122 Infection of naive ferrets with H5N1 avian influenza virus causes a rapid and lethal systemic

123 H7N9 avian influenza virus causes severe infections and might

124 mologous vaccine against a highly pathogenic avian influenza virus challenge.

125 etically distinct, this virus shares several avian influenza virus characteristics suggesting a more

126 of full genome sequences from low pathogenic avian influenza viruses circulating in Egypt, underscori

127 Avian influenza viruses continue to evolve and acquire m

128 Pathogenic H7N9 avian influenza viruses continue to represent a public h

129 Highly pathogenic H5N1 avian influenza viruses continue to spread via waterfowl

130 dominated the 2009 flu season, and the H5N1 avian influenza virus continues to kill both people and

131 Most avian influenza viruses do not replicate efficiently in

132 suggest that the Eurasian H5N8 clade 2.3.4.4 avian influenza virus emerged in late 2013 in China, spr

133 Highly pathogenic H5N1 avian influenza viruses emerged in 1996 and have since e

134 However, information on avian influenza virus evolution and transmission during

135 In 2017, low-pathogenicity H7N9 avian influenza viruses evolved to a high-pathogenicity

136 We found that the H5Nx highly pathogenic avian influenza viruses exhibited high virulence in mice

137 these parameters among a panel of human and avian influenza viruses exhibiting diverse respiratory d

138 s utility for monitoring the evolution of H9 avian influenza viruses from China between 2005 and 2015

139 Our results indicate that the H7N8 avian influenza viruses from Indiana are able to replica

140 ough mutation to which functional components avian influenza viruses gain the ability to grow efficie

141 Avian influenza viruses generally replicate at higher te

142 c analyses revealed the global prevalence of avian influenza virus genes whose proteins differ only a

143 ) against several NAs of wild-type human and avian influenza viruses (H1N1, H3N2, H5N1, and H7N9), al

144 Prototypic avian influenza viruses (H3N2, H9N2, and H5N1) and swine

145 t HA (short and long) from highly pathogenic avian influenza virus H5N1 and the anti-H5 HA monoclonal

146 ng contrast, immunization of humans with the avian influenza virus H5N1 induced broadly cross-reactiv

147 Finally, highly pathogenic avian influenza virus H5N1 polymerase activity was teste

148 Avian influenza virus H5N1, a serious worldwide threat t

149 intranasal infection with highly pathogenic avian influenza virus (H5N1 [A/Viet Nam/1203/2004]) in f

150 Avian influenza virus H9N2 is prevalent in waterfowl and

151 hile M2 has previously been shown to protect avian influenza virus HA proteins of the H5 and H7 subty

152 protein has previously been shown to protect avian influenza virus HA proteins that contain a polybas

153 We showed that the H9N2 avian influenza viruses harboring 190V in the HA exhibit

154 of-function' experiments on high-consequence avian influenza viruses has highlighted the role of ferr

155 enetic clades, while reassortment with other avian influenza viruses has led to the emergence of new

156 acid substitutions in highly pathogenic H5N1 avian influenza viruses have been shown to contribute to

157 Highly pathogenic H5N1 avian influenza viruses have caused outbreaks among poul

158 Notably, certain avian influenza viruses have evolved to escape this rest

159 se in southern China, highly pathogenic H5N1 avian influenza viruses have posed a continuous threat t

160 nuated influenza vaccines (LAIVs) expressing avian influenza virus hemagglutinins (HAs) prime for str

161 by a human isolate of the highly pathogenic avian influenza virus (HPAI) and 1918 pandemic influenza

162 way to prevent large-scale highly pathogenic avian influenza virus (HPAI) H5N1 outbreaks in the human

163 H5N1 highly pathogenic avian influenza virus (HPAIV) causes periodic outbreaks

164 experienced several recent highly pathogenic avian influenza virus (HPAIV) epizootics.

165 e sustained circulation of highly pathogenic avian influenza virus (HPAIV) H5N1 A/goose/Guangdong/199

166 The highly pathogenic avian influenza virus (HPAIV) H5N1 A/goose/Guangdong/199

167 Highly pathogenic avian influenza virus (HPAIV) H5N1 can infect mammals vi

168 hown to be associated with highly pathogenic avian influenza virus (HPAIV) H5N1 outbreaks in South-Ea

169 major policies to control highly pathogenic avian influenza virus (HPAIV) infections in chickens.

170 Highly pathogenic avian influenza virus (HPAIV) subtype H5N1 causes severe

171 on of the H5 HA of an H5N1 highly pathogenic avian influenza virus (HPAIV), A/Vietnam/1203/04 (VN1203

172 Highly pathogenic avian influenza viruses (HPAIV) induce severe inflammati

173 umans infected by the highly pathogenic H5N1 avian influenza viruses (HPAIV) present unusually high c

174 High-pathogenic avian influenza viruses (HPAIVs) evolve from low-pathoge

175 2.2 Eurasian-lineage H5N1 highly pathogenic avian influenza viruses (HPAIVs) were first detected in

176 t commonly considered intermediate hosts for avian influenza viruses.IMPORTANCE Avian influenza virus

177 t, efforts to control highly pathogenic H5N1 avian influenza virus in poultry and in humans have fail

178 Maintenance of avian influenza virus in waterfowl populations requires

179 Active surveillance of avian influenza viruses in Bangladeshi live poultry mark

180 nfections in humans, as well as detection of avian influenza viruses in birds in the United States.

181 s of numerous outbreaks of highly pathogenic avian influenza viruses in commercial poultry farms.

182 to the low activity of the RNA polymerase of avian influenza viruses in mammalian cells.

183 The restrictions to transmissibility of avian influenza viruses in mammals are multigenic, and o

184 e essential for the efficient replication of avian influenza viruses in mammals.

185 ow cocirculating with highly pathogenic H5N1 avian influenza viruses in many parts of the world, rais

186 e and genetic diversity of H7N9, we surveyed avian influenza viruses in poultry in Jiangsu province w

187 gated serological profiles against human and avian influenza viruses in the general population using

188 estrict the emergence and perpetuation of HP avian influenza viruses in these natural reservoirs.

189 lls support transcription and replication of avian influenza viruses, in contrast to human cells, in

190 an AS03-adjuvanted versus nonadjuvanted H5N1 avian influenza virus inactivated vaccine.

191 Avian influenza viruses, including H5N1 and H7N9, have b

192 We found that a subset of avian influenza viruses, including potentially pandemic

193 ry T cell numbers were decreased in modified avian influenza virus-infected mice.

194 In avian influenza virus-infected patients, the host immune

195 Highly pathogenic avian influenza virus infection is characterized by a ma

196 negative regulatory signals during modified avian influenza virus infection.

197 he host cell surface in the initial stage of avian influenza virus infection.

198 tance of H9N2 viruses as the source of novel avian influenza virus infections in humans requires cont

199 se of the pathogenicity and low incidence of avian influenza virus infections in humans, the immune c

200 severe and prolonged disease associated with avian influenza virus infections in humans.

201 ary endothelial cells in the pathogenesis of avian influenza virus infections is largely unknown.

202 A novel avian influenza virus, influenza A(H7N9), emerged in Chi

203 H9N2 avian influenza virus is a major cause of poultry produc

204 Full length viral PB1-F2 present only in avian influenza viruses is a virulence factor that targe

205 The number of humans infected with avian influenza viruses is increasing, raising concerns

206 cate that the number of humans infected with avian influenza viruses is much larger than the number o

207 sence of an aspartic acid in over 95% of all avian influenza viruses is not, resulting in a clear dis

208 Previous avian influenza virus isolates have carried glutamic aci

209 These results imply that when an avian influenza virus jumps the species barrier from bir

210 Nonstructural protein 1 (NS1) proteins from avian influenza viruses like the 1918 pandemic NS1 are c

211 ctural analysis.IMPORTANCE Low-pathogenicity avian influenza virus (LPAIV) subtypes can reassort with

212 t HA head glycosylation of low-pathogenicity avian influenza virus (LPAIV) subtypes.

213 sence of a copathogen such as low-pathogenic avian influenza virus (LPAIV).

214 Low-pathogenic avian influenza viruses (LPAIVs) are genetically highly

215 H5 and H7 subtypes of low pathogenic avian influenza viruses (LPAIVs) can mutate to highly pa

216 r role in the epidemiology of low-pathogenic avian influenza viruses (LPAIVs), which are occasionally

217 Vaccines against H7 avian influenza viruses may be more effective than HI an

218 Highly pathogenic H5N1 avian influenza viruses must acquire mutations to overco

219 Avian influenza viruses need several adaptive mutations

220 Avian influenza viruses occasionally infect and adapt to

221 ncern in Bangladesh, where highly pathogenic avian influenza viruses of the A(H5N1) subtype are endem

222 nd Sw/Korea/C13/08 viruses were derived from avian influenza viruses of the Eurasian lineage.

223 Highly pathogenic avian influenza viruses of the H5N1 subtype continue to

224 Highly pathogenic avian influenza viruses of the H5N1 subtype continue to

225 Since 1997, highly pathogenic avian influenza viruses of the H5N1 subtype have been tr

226 Avian influenza viruses of the H5N1 subtype pose a serio

227 Avian influenza viruses of the H7 hemagglutinin (HA) sub

228 r non-faradic impedimetric detection of AIV (avian influenza virus) oligonucleotides.

229 on of human influenza virus strains, whereas avian influenza viruses overcome these restriction facto

230 that subvirion inactivated vaccines against avian influenza viruses, particularly H5N1, are poorly i

231 titution of human-origin PA subunits into an avian influenza virus polymerase alleviates restriction

232 in human cells, and mutations that adapt the avian influenza virus polymerase for human cells also in

233 mpletely mapping amino-acid mutations to the avian influenza virus polymerase protein PB2 that enhanc

234 e reassortment to restore the activity of an avian influenza virus polymerase that is normally impair

235 in avian cells enhances the activity of the avian influenza virus polymerase.

236 s, in contrast to human cells, in which most avian influenza virus polymerases display limited activi

237 Avian influenza virus polymerases function poorly in mam

238 we suggest, instead, that the restriction of avian influenza virus polymerases in human cells is the

239 The two typical avian influenza virus polymerases used in this study wer

240 Highly pathogenic avian influenza viruses pose a continuing global threat.

241 Infection of chickens with avian influenza virus poses a global threat to both poul

242 The emergence of human-transmissible H5N1 avian influenza viruses poses a major pandemic threat.

243 y droplet transmission between ferrets of an avian influenza virus possessing an avian polymerase sub

244 It is thought that avian influenza viruses preferentially bind to N-acetyln

245 The hemagglutinins (HAs) of avian influenza viruses preferentially bind to sialic ac

246 ction for prepandemic vaccines.IMPORTANCE H7 avian influenza viruses present a serious risk to human

247 Avian influenza viruses rarely infect humans, but the re

248 Avian influenza virus reassortants resembling the 1918 h

249 Human and avian influenza viruses recognize different sialic acid-

250 avian-origin influenza virus polymerases and avian influenza virus replication.

251 the possible role of PAM in the mediation of avian influenza virus resistance, we compared the host e

252 easing, raising concerns of the emergence of avian influenza viruses resistant to neuraminidase (NA)

253 lly bind the hemagglutinin (HA) of human and avian influenza viruses, respectively, were detected on

254 -linked sialic acids recognized by human and avian influenza viruses, respectively.

255 number of viruses that infect lungs, such as avian influenza virus, SARS-associated coronavirus, and

256 nction (GOF) research with highly pathogenic avian influenza virus, severe acute respiratory syndrome

257 Recurring reports of avian influenza viruses severely affecting humans have s

258 nly 1918 PB2 impacts the pathogenicity of an avian influenza virus sharing high homology to the 1918

259 uenza virus strain, a highly pathogenic H5N1 avian influenza virus strain, and a recently emerging H7

260 Influenza A(H5N1) virus and other avian influenza virus strains represent major pandemic t

261 experimental evidence that highly pathogenic avian influenza virus subtype H5 can acquire the ability

262 to sustain circulation of highly pathogenic avian influenza virus subtype H5N1 (HPAIV H5N1).

263 Highly pathogenic avian influenza virus subtype H5N1 is endemic in Asia, w

264 H5N1 and H9N2 avian influenza virus subtypes top the World Health Orga

265 pendent cellular cytotoxicity (ADCC) against avian influenza virus subtypes, including H7N9 and H5N1,

266 One likely scenario for an avian influenza virus, such as A/H5N1, to evolve to one

267 Avian influenza viruses, such as A(H5N1) and A(H7N9), ar

268 r zoonotic and pandemic emergence.IMPORTANCE Avian influenza viruses, such as H9N2, cause disease in

269 hical range seen in these viruses.IMPORTANCE Avian influenza viruses, such as H9N2, cause huge econom

270 Our results can inform better avian influenza virus surveillance efforts as well as co

271 Because pigs are more readily infected with avian influenza viruses than humans, it would seem that

272 the acquisition of the NS segment of an H5N1 avian influenza virus that had previously been overlooke

273 The zoonotic outbreak of H7N9 subtype avian influenza virus that occurred in eastern China in

274 Avian influenza viruses that cause infection and are tra

275 ows H7N9 and H5N1 as the latest in a line of avian influenza viruses that cause serious disease in hu

276 influenza viruses and for highly pathogenic avian influenza viruses that circulate in poultry, but m

277 Two studies of H5N1 avian influenza viruses that had been genetically engine

278 Antigenically novel avian influenza viruses that infect and cause disease in

279 d as a reservoir of internal genes for other avian influenza viruses that infect humans, and several

280 virus was similar to Eurasian avian lineage avian influenza viruses, the virus had been circulating

281 ution PB2-A588V may be a new strategy for an avian influenza virus to adapt mammalian hosts.

282 use in the event of transmission of an H3N8 avian influenza virus to humans.

283 We analyzed the adaptation of avian influenza viruses to a mammalian host by passaging

284 t insight into the potential for emerging H7 avian influenza viruses to acquire the ability to cause

285 The ability of avian influenza viruses to adapt to new host species is

286 been shown to play a role in the ability of avian influenza viruses to cross the species barrier, an

287 The inability of avian influenza viruses to effectively bind human-like s

288 s use of the codon usage biases of human and avian influenza viruses to generate a human-derived infl

289 NAI resistance among specific NA subtypes of avian influenza viruses to help guide clinical managemen

290 Furthermore, regular transmission of avian influenza viruses to humans increases the risk of

291 between antigenic drift and the potential of avian influenza viruses to infect humans.

292 for HA conformational change, may facilitate avian influenza virus transmission via respiratory dropl

293 ild birds were sampled and highly pathogenic avian influenza virus was detected in 1.3% (n = 63).

294 14, a Eurasian strain H5N8 highly pathogenic avian influenza virus was detected in poultry in Canada.

295 The pH of HA activation of highly pathogenic avian influenza viruses was greater than human and low-p

296 omparing human seasonal influenza strains to avian influenza viruses, we provide greater insight into

297 In contrast, selected avian influenza viruses were able to escape IFITM3 restr

298 ESEV is the consensus PBM sequence of avian influenza viruses, while RSKV is the consensus seq

299 lobal concern persists that these or similar avian influenza viruses will evolve into viruses that ca

300 in ferrets, demonstrating that contemporary avian influenza viruses with 1918 virus-like proteins ma

301 These findings demonstrate that avian influenza viruses within H7 and H5 subtypes are ca