ライフサイエンスコーパス: avian leukosis

1 The transmembrane subunit (TM) of the avian leukosis and sarcoma virus (ALSV) envelope glycopr

2 completely defective in mediating subgroup A avian leukosis and sarcoma virus (ALSV-A) entry.

3 Tva is the cellular receptor for subgroup A avian leukosis and sarcoma virus (ALSV-A).

4 The avian leukosis and sarcoma virus long terminal repeat (L

5 endent; pathologically, it appeared to mimic avian leukosis but is free of exogenous ALV infection; i

6 h myc in chicken B-cell lymphomas induced by avian leukosis proviral integrations.

7 y define cis-acting determinants involved in avian leukosis sarcoma virus packaging RNA binding to Ga

8 s to mice that express TVA, the receptor for avian leukosis sarcoma virus subgroup A (ALSV-A), under

9 We are using avian leukosis-sarcoma virus (ALSV) vectors to generate

10 The avian leukosis-sarcoma virus (ALV) group of retroviruses

11 rity at the amino acid level) to that of the avian leukosis-sarcoma virus family, it retains several

12 be important for the efficient packaging of avian leukosis-sarcoma virus RNA.

13 In avian leukosis-sarcoma virus, however, we have shown tha

14 sociated with infection by subgroups B and D avian leukosis-sarcoma viruses (ALVs).

15 ptide substrates that are derivatives of the avian leukosis/sarcoma virus nucleocapsid-protease cleav

16 OMMA-1D cells were engineered to express the avian leukosis subtype A receptor, tv-a, to permit infec

17 We have analyzed pol- mutants of avian leukosis virus (ALV) and murine leukemia virus (Mu

18 n to bind CCAAT/enhancer elements within the avian leukosis virus (ALV) and Rous sarcoma virus (RSV)

19 The avian leukosis virus (ALV) entry mechanism is controvers

20 day-old chicken embryos with the recombinant avian leukosis virus (ALV) EU-8 induces a high incidence

21 Avian leukosis virus (ALV) has been used as a model syst

22 Avian leukosis virus (ALV) has endogenized prior to chic

23 we provide new evidence for the presence of avian leukosis virus (ALV) in both CEF supernatants and

24 Avian leukosis virus (ALV) induces bursal lymphoma in ch

25 Avian leukosis virus (ALV) induces bursal lymphoma in tu

26 Avian leukosis virus (ALV) induces tumors by integrating

27 st chicken strains are highly susceptible to avian leukosis virus (ALV) induction of bursal lymphoma,

28 Avian leukosis virus (ALV) infection induces bursal lymp

29 but the factors that mediate alpharetroviral avian leukosis virus (ALV) integration are unknown.

30 Avian leukosis virus (ALV) is detrimental to poultry hea

31 The avian leukosis virus (ALV) long terminal repeat (LTR) co

32 ct with different regions of the RSV and the avian leukosis virus (ALV) LTRs.

33 In this study, we identified avian leukosis virus (ALV) proviral integration sites in

34 al chromosome-transgenic mice expressing the avian leukosis virus (ALV) receptor TVB, fused to monome

35 monstrated that bridge proteins comprised of avian leukosis virus (ALV) receptors fused to epidermal

36 We also found that endogenous avian leukosis virus (ALV) retroviral insertions were no

37 A new subgroup of avian leukosis virus (ALV) that includes a unique env ge

38 An avian leukosis virus (ALV) was found in some chicken emb

39 A new subgroup of avian leukosis virus (ALV), designated subgroup J, was i

40 characteristics of a eukaryotic retrovirus, avian leukosis virus (ALV), offers a robust, eukaryotic

41 Avian leukosis virus (ALV), previously shown to be nonin

42 ion-deficient retroviral vector based on the avian leukosis virus (ALV), we inserted into the chicken

43 An avian leukosis virus (ALV)-based retroviral vector syste

44 interference experiments have indicated that avian leukosis virus (ALV)-E may utilize a cellular rece

45 s is a common retroviral integration site in avian leukosis virus (ALV)-induced B-cell lymphomas orig

46 In 2010, sporadic cases of avian leukosis virus (ALV)-like bursal lymphoma, also kn

47 ptor for the cytopathic subgroups B and D of avian leukosis virus (ALV-B and ALV-D), as a tumor necro

48 us avian retrovirus (EAV) and the endogenous avian leukosis virus (ALV-E), which originate from the c

49 Subgroup J avian leukosis virus (ALV-J) is a recently identified av

50 nd inter-host transmission of the subgroup J avian leukosis virus (ALV-J) may be the most important i

51 nvelope glycoprotein (Env) of the subgroup J avian leukosis virus (ALV-J) play an essential role in t

52 in, is a cellular receptor of the subgroup J avian leukosis virus (ALV-J).

53 cterized the interactions between subgroup A avian leukosis virus [ALV(A)] envelope glycoproteins and

54 The interactions between the subgroup A avian leukosis virus [ALV(A)] envelope glycoproteins and

55 est of this hypothesis, the requirements for avian leukosis virus A (ALV-A) infection were examined.

56 ansgene encoding the receptor for subgroup A avian leukosis virus and controlled by the astrocyte-spe

57 By infecting both clones and subclones with avian leukosis virus and using a PCR-based assay to dete

58 The avian leukosis virus DeltaLR-9 causes a high frequency o

59 set B-cell lymphomas induced by the nonacute avian leukosis virus EU-8.

60 ar domain of the TVB receptor for subgroup B avian leukosis virus fused to epidermal growth factor (E

61 Avian leukosis virus induces lymphoma in chickens after

62 vian retroviral [i.e., replication-competent avian leukosis virus long terminal repeat with splice ac

63 n using the retroviral replication-competent avian leukosis virus long terminal repeat, splice accept

64 binding the A1 CCAAT/enhancer motif from the avian leukosis virus long terminal repeat.

65 or, tv-a, to permit infection by recombinant avian leukosis virus produced by the replication-compete

66 We have adapted the avian leukosis virus RCAS (replication-competent avian s

67 ryocyte-lineage expression of the subgroup A avian leukosis virus receptor, TVA.

68 Replication-competent avian leukosis virus splice-acceptor (RCAS)/cellular rec

69 irus A (tva), which encodes the receptor for avian leukosis virus subgroup A (ALV/A), we provide dire

70 Cell killing by avian leukosis virus subgroup B (ALV-B) in cultures has

71 Avian leukosis virus subgroup J (ALV-J) is a simple retr

72 Avian leukosis virus subgroup J (ALV-J) is an important

73 ific region, termed the E element or XSR, of avian leukosis virus subgroup J (ALV-J), a member of avi

74 Avian leukosis virus subgroup J (ALV-J), an avian oncoge

75 otransferrin, a serine/threonine kinase, and avian leukosis virus subgroup J glycoprotein.

76 Among all subgroups, avian leukosis virus subgroup J is one of the most patho

77 differences in receptor usage among the many avian leukosis virus subgroups.

78 o cell lineages by infection with subgroup A avian leukosis virus vectors in lines of transgenic mice

79 ific transfer of genes carried by subgroup A avian leukosis virus vectors.

80 ng locations while other viruses such as the avian leukosis virus, MAYV and several unclassified viru

81 mino-terminal domain of the alpharetrovirus, avian leukosis virus, revealing a previously undetected

82 rendering mice susceptible to infection with avian leukosis virus-derived gene vectors.

83 d at a common retroviral integration site in avian leukosis virus-induced lymphomas and has been impl

84 egration site in B cell lymphomas induced by avian leukosis virus.

85 to the novel envelope gene of the subgroup J avian leukosis virus.

86 cellular receptors for subgroup B, D, and E avian leukosis viruses (ALV) encoded by the s1 allele of

87 Host susceptibility to subgroup B, D, and E avian leukosis viruses (ALV) is determined by specific a

88 TVA, the cellular receptor for subgroup A avian leukosis viruses (ALV-A) can mediate viral entry w

89 ar domain of the TVA receptor for subgroup A avian leukosis viruses (ALV-A), fused to the MR1 single-

90 Subgroups B, D, and E avian leukosis viruses (ALV-B, -D, and -E) share the sam

91 containing RNA of both subgroup E endogenous avian leukosis viruses (ALV-E) and endogenous avian viru

92 studied genomic RNAs of wild-type and mutant avian leukosis viruses (ALVs) in an attempt to (i) bette

93 B and D and noncytopathic subgroup E of the avian leukosis viruses (ALVs).