ライフサイエンスコーパス: avian leukosis virus

1 egration site in B cell lymphomas induced by avian leukosis virus.

2 to the novel envelope gene of the subgroup J avian leukosis virus.

3 est of this hypothesis, the requirements for avian leukosis virus A (ALV-A) infection were examined.

4 We have analyzed pol- mutants of avian leukosis virus (ALV) and murine leukemia virus (Mu

5 n to bind CCAAT/enhancer elements within the avian leukosis virus (ALV) and Rous sarcoma virus (RSV)

6 The avian leukosis virus (ALV) entry mechanism is controvers

7 day-old chicken embryos with the recombinant avian leukosis virus (ALV) EU-8 induces a high incidence

8 Avian leukosis virus (ALV) has been used as a model syst

9 Avian leukosis virus (ALV) has endogenized prior to chic

10 we provide new evidence for the presence of avian leukosis virus (ALV) in both CEF supernatants and

11 Avian leukosis virus (ALV) induces bursal lymphoma in ch

12 Avian leukosis virus (ALV) induces bursal lymphoma in tu

13 Avian leukosis virus (ALV) induces tumors by integrating

14 st chicken strains are highly susceptible to avian leukosis virus (ALV) induction of bursal lymphoma,

15 Avian leukosis virus (ALV) infection induces bursal lymp

16 but the factors that mediate alpharetroviral avian leukosis virus (ALV) integration are unknown.

17 Avian leukosis virus (ALV) is detrimental to poultry hea

18 The avian leukosis virus (ALV) long terminal repeat (LTR) co

19 ct with different regions of the RSV and the avian leukosis virus (ALV) LTRs.

20 In this study, we identified avian leukosis virus (ALV) proviral integration sites in

21 al chromosome-transgenic mice expressing the avian leukosis virus (ALV) receptor TVB, fused to monome

22 monstrated that bridge proteins comprised of avian leukosis virus (ALV) receptors fused to epidermal

23 We also found that endogenous avian leukosis virus (ALV) retroviral insertions were no

24 A new subgroup of avian leukosis virus (ALV) that includes a unique env ge

25 An avian leukosis virus (ALV) was found in some chicken emb

26 A new subgroup of avian leukosis virus (ALV), designated subgroup J, was i

27 characteristics of a eukaryotic retrovirus, avian leukosis virus (ALV), offers a robust, eukaryotic

28 Avian leukosis virus (ALV), previously shown to be nonin

29 ion-deficient retroviral vector based on the avian leukosis virus (ALV), we inserted into the chicken

30 An avian leukosis virus (ALV)-based retroviral vector syste

31 interference experiments have indicated that avian leukosis virus (ALV)-E may utilize a cellular rece

32 s is a common retroviral integration site in avian leukosis virus (ALV)-induced B-cell lymphomas orig

33 In 2010, sporadic cases of avian leukosis virus (ALV)-like bursal lymphoma, also kn

34 ptor for the cytopathic subgroups B and D of avian leukosis virus (ALV-B and ALV-D), as a tumor necro

35 us avian retrovirus (EAV) and the endogenous avian leukosis virus (ALV-E), which originate from the c

36 Subgroup J avian leukosis virus (ALV-J) is a recently identified av

37 nd inter-host transmission of the subgroup J avian leukosis virus (ALV-J) may be the most important i

38 nvelope glycoprotein (Env) of the subgroup J avian leukosis virus (ALV-J) play an essential role in t

39 in, is a cellular receptor of the subgroup J avian leukosis virus (ALV-J).

40 cellular receptors for subgroup B, D, and E avian leukosis viruses (ALV) encoded by the s1 allele of

41 Host susceptibility to subgroup B, D, and E avian leukosis viruses (ALV) is determined by specific a

42 TVA, the cellular receptor for subgroup A avian leukosis viruses (ALV-A) can mediate viral entry w

43 ar domain of the TVA receptor for subgroup A avian leukosis viruses (ALV-A), fused to the MR1 single-

44 Subgroups B, D, and E avian leukosis viruses (ALV-B, -D, and -E) share the sam

45 containing RNA of both subgroup E endogenous avian leukosis viruses (ALV-E) and endogenous avian viru

46 cterized the interactions between subgroup A avian leukosis virus [ALV(A)] envelope glycoproteins and

47 The interactions between the subgroup A avian leukosis virus [ALV(A)] envelope glycoproteins and

48 studied genomic RNAs of wild-type and mutant avian leukosis viruses (ALVs) in an attempt to (i) bette

49 B and D and noncytopathic subgroup E of the avian leukosis viruses (ALVs).

50 ansgene encoding the receptor for subgroup A avian leukosis virus and controlled by the astrocyte-spe

51 By infecting both clones and subclones with avian leukosis virus and using a PCR-based assay to dete

52 The avian leukosis virus DeltaLR-9 causes a high frequency o

53 rendering mice susceptible to infection with avian leukosis virus-derived gene vectors.

54 set B-cell lymphomas induced by the nonacute avian leukosis virus EU-8.

55 ar domain of the TVB receptor for subgroup B avian leukosis virus fused to epidermal growth factor (E

56 d at a common retroviral integration site in avian leukosis virus-induced lymphomas and has been impl

57 Avian leukosis virus induces lymphoma in chickens after

58 vian retroviral [i.e., replication-competent avian leukosis virus long terminal repeat with splice ac

59 n using the retroviral replication-competent avian leukosis virus long terminal repeat, splice accept

60 binding the A1 CCAAT/enhancer motif from the avian leukosis virus long terminal repeat.

61 ng locations while other viruses such as the avian leukosis virus, MAYV and several unclassified viru

62 or, tv-a, to permit infection by recombinant avian leukosis virus produced by the replication-compete

63 We have adapted the avian leukosis virus RCAS (replication-competent avian s

64 ryocyte-lineage expression of the subgroup A avian leukosis virus receptor, TVA.

65 mino-terminal domain of the alpharetrovirus, avian leukosis virus, revealing a previously undetected

66 Replication-competent avian leukosis virus splice-acceptor (RCAS)/cellular rec

67 irus A (tva), which encodes the receptor for avian leukosis virus subgroup A (ALV/A), we provide dire

68 Cell killing by avian leukosis virus subgroup B (ALV-B) in cultures has

69 Avian leukosis virus subgroup J (ALV-J) is a simple retr

70 Avian leukosis virus subgroup J (ALV-J) is an important

71 ific region, termed the E element or XSR, of avian leukosis virus subgroup J (ALV-J), a member of avi

72 Avian leukosis virus subgroup J (ALV-J), an avian oncoge

73 otransferrin, a serine/threonine kinase, and avian leukosis virus subgroup J glycoprotein.

74 Among all subgroups, avian leukosis virus subgroup J is one of the most patho

75 differences in receptor usage among the many avian leukosis virus subgroups.

76 o cell lineages by infection with subgroup A avian leukosis virus vectors in lines of transgenic mice

77 ific transfer of genes carried by subgroup A avian leukosis virus vectors.