ライフサイエンスコーパス: axoplasmic

1 on, CV was highly sensitive to variations in axoplasmic and membrane resistivities, the formation of

2 jury index, a quantitative representation of axoplasmic and myelinic pathologies, was significantly l

3 to estimate neurotubule density (rho(RNFL)), axoplasmic area (A(x)) mode, axon area (A(a)) mode, slop

4 ope (u) of the number of neurotubules versus axoplasmic area (neurotubule packing density), fractiona

5 ns of action potentials transiently elevates axoplasmic [C(2+)] around nodes of Ranvier.

6 wild-type axons induced a dramatic spike in axoplasmic Ca(2+) and termination of mitochondrial movem

7 Our results point to axoplasmic Ca(2+) concentrations as a determinant for th

8 High focal axoplasmic Ca(2+) levels correlate with focal aggregatio

9 The results reveal that dramatically higher axoplasmic Ca(2+) levels occur at the sites of axonal sp

10 els but significantly attenuates the rise of axoplasmic Ca(2+) that occurs during degeneration.

11 Cardiac spillover, reuptake, vesicular-axoplasmic exchange, and tissue stores of NE were assess

12 tion of neuronal anterograde (or retrograde) axoplasmic flow, leading to axonal degeneration, especia

13 leton while kinetic energy is carried by the axoplasmic fluid.

14 cular (PVN) nuclei neurons in colchicine (an axoplasmic inhibitor)-untreated animals.

15 ld(S) for the first time in vivo, and higher axoplasmic levels in transgenic mice with Wld(S) redistr

16 d with mitochondria versus synaptic vesicles/axoplasmic matrix reveals significant differences in the

17 of 98 nm that could nevertheless accommodate axoplasmic mitochondria.

18 e competes with protein-conjugated beads for axoplasmic motor(s).

19 xons that lack Na+/K+ ATPase cannot exchange axoplasmic Na+ for K+ and are incapable of nerve transmi

20 ated axons causes sequential deregulation of axoplasmic Na, K and Ca; i.e., an initial influx of Na a

21 to myosin V, this myosin is likely to be an axoplasmic organelle motor.

22 Equilibrium density fractionation of motile axoplasmic organelle preparations has revealed that p196

23 Because axoplasmic organelles move on actin filaments, they must

24 Axoplasmic organelles obtained from the squid giant axon

25 orates the cytoplasmic surface of 21% of the axoplasmic organelles, as demonstrated by immunogold ele

26 myosin antibody in Western blots of isolated axoplasmic organelles, has been previously proposed to b

27 in the axon, and is associated with isolated axoplasmic organelles.

28 ament in neurons), with segregation of other axoplasmic organelles.

29 t and maintenance of neurofilament-dependent axoplasmic organization that is critical for preserving

30 In addition, TTX significantly attenuated axoplasmic pathology at both 4 and 24 hr after injury.

31 The modest axoplasmic peri-nodal [Ca(2+)] elevations measured in in

32 rayfish medial giant axon (MGA), we analyzed axoplasmic proteins separately from proteins of the glia

33 PA receptors, release of toxic Ca2+ from the axoplasmic reticulum, overactivation of ionotropic and m

34 Densely opaque axoplasmic sentinels are arranged along normoxic/hypoxic

35 or availability of the NE transporter to the axoplasmic side of the membrane, causing massive carrier

36 nse to axonal injury also involves intrinsic axoplasmic signals.

37 d axonal transport, associated with upstream axoplasmic swelling and eventual axonal detachment.

38 Enrichment is achieved via enhanced axoplasmic translation of CaMKII mRNA, through a mechani

39 e known to inhibit neuronal fast anterograde axoplasmic transport (FAAT) in a reversible and dose-dep

40 Impaired axoplasmic transport (IAT) and neurofilament compaction

41 neurofilament compaction (NFC) and impaired axoplasmic transport (IAT) in distinct populations of ax

42 ong-term effects of infraorbital nerve (ION) axoplasmic transport attenuation with vinblastine on the

43 bre nociceptors following the application of axoplasmic transport blockers (colchicine and vinblastin

44 rats, suggesting a diminution of anterograde axoplasmic transport by adrenal transplants.

45 port, we hypothesized that the disruption of axoplasmic transport by nerve inflammation could cause t

46 y neurodegenerative disorders yet changes to axoplasmic transport in disease models have not been qua

47 e inflammation that causes the disruption of axoplasmic transport in patients with painful conditions

48 e body (MGB) was discovered in the cat using axoplasmic transport methods combined with immunocytoche

49 halamus, midbrain, and cerebral cortex using axoplasmic transport methods.

50 To monitor axoplasmic transport of HSV, we used the giant axon of t

51 newly synthesized proteins destined for fast axoplasmic transport pass through the Golgi apparatus.

52 separate series of experiments, the block of axoplasmic transport proximal to a localized neuritis si

53 the latter is dependent exclusively on slow axoplasmic transport to maintain protein mass in a stead

54 icate that although transient attenuation of axoplasmic transport with vinblastine has limited effect

55 Carried by fast anterograde axoplasmic transport, APP will pool at regions of impair

56 Because nerve inflammation also disrupts axoplasmic transport, we hypothesized that the disruptio

57 biosynthesis in the motoneuron cell body and axoplasmic transport.

58 pealei, a well known system for the study of axoplasmic transport.

59 processing of polypeptides destined for fast axoplasmic transports, the fragmentation of the organell

60 Ranvier characterized by an accumulation of axoplasmic vesicles under the nodal membrane.

61 ent, suggesting that both were released from axoplasmic vesicular stores.

62 Fast and slow axoplasmic waves have been known for decades, but altern

63 axoplasm isolated from myelinated fibers as axoplasmic whole mounts and delipidated spinal nerve roo

64 of anaesthetized transgenic mice expressing axoplasmic yellow fluorescent protein were stimulated el