ライフサイエンスコーパス: back pain

1 scores indicating more disability related to back pain).

2 controls and patients with pain (chronic low back pain).

3 and the outcome, the incident occurrence of back pain.

4 adults with acute, subacute, or chronic low back pain.

5 and harms of SMT for acute (</=6 weeks) low back pain.

6 nd the role of MBSR in the management of low back pain.

7 ommendations on noninvasive treatment of low back pain.

8 al disc degeneration (IVDD) is linked to low back pain.

9 cacy and safety of MBSR in patients with low back pain.

10 ty as a primary outcome in patients with low back pain.

11 therapy, she reported 3 months of worsening back pain.

12 d nonpharmacologic treatment options for low back pain.

13 were abdominal pain, vomiting, diarrhea, and back pain.

14 anagement practice for patients with chronic back pain.

15 be a lifelong task for patients with chronic back pain.

16 aminations by rheumatologists due to chronic back pain.

17 oung and middle-aged adults with chronic low back pain.

18 eatment option for patients with chronic low back pain.

19 ial activation, in patients with chronic low back pain.

20 rheumatologist, with symptoms of the chronic back pain.

21 aches, vague upper abdominal pain, and lower back pain.

22 degeneration is the leading cause of chronic back pain.

23 a 3-month history of fatigue and unremitting back pain.

24 tervertebral disc (IVD) degeneration and low back pain.

25 d hot flushes, alopecia, abdominal pain, and back pain.

26 tern United States with a 3-month history of back pain.

27 respiratory tract infection, influenza, and back pain.

28 n of muscle fat content in patients with low back pain.

29 Here, we identify a subcortical signature of back pain.

30 and to ameliorate IVD-associated chronic low back pain.

31 generation are believed to contribute to low back pain.

32 SAID allergy on OUD in patients with chronic back pain.

33 controlled trial of acupuncture for chronic back pain.

34 the relationships of BMI and height with low back pain.

35 tability of acupuncture to patients with low back pain.

36 and they can be responsible for chronic low back pain.

37 ty can identify patients prone to persistent back pain.

38 a clinically effective treatment for chronic back pain.

39 as they considered prognosis studies of low back pain.

40 nal injections of methylprednisolone for low back pain.

41 y during movement-evoked pain in chronic low back pain.

42 heimer's disease, headache disorder, and low back pain.

43 ey presented with a 3-month history of lower back pain.

44 ctural integrity and elicit debilitating low back pain.

45 ey presented with a 3-month history of lower back pain.

46 ogic and nonpharmacologic treatments for low back pain.

47 nditions, including osteoarthritis and lower back pain.

48 ercise program for patients with chronic low back pain.

49 ute or chronic nonradicular or radicular low back pain.

50 ute or chronic nonradicular or radicular low back pain.

51 line addressed pharmacologic options for low back pain.

52 tes including osteoarthritis and chronic low back pain.

53 sociated with modest effects for chronic low back pain.

54 ditions owing to non-specific arthralgia and back pain.

55 rug treatments for patients with chronic low back pain?

56 ith indigestion (0.74, 0.58-0.95; p=0.0018), back pain (0.76, 0.58-0.99; p=0.044), diabetes (0.63, 0.

57 ith indigestion (0.71, 0.56-0.89; p=0.0033), back pain (0.77, 0.59-0.99; p=0.040), diabetes (0.71, 0.

58 nificantly after MR imaging for inflammatory back pain (14% vs 76%, before vs after; P < .001), mecha

59 s or older with a new primary care visit for back pain (2011-2013) in 3 US health care systems.

60 Of 47,114 patients with chronic back pain, 3,620 (7.7%) had a reported NSAID ADR.

61 38 [20%] of 189 in the bicalutamide group), back pain (35 [19%] vs 34 [18%]), and hot flush (27 [15%

62 patients were headache (61 [35%] patients), back pain (38 [22%]), and nausea (19 [11%]).

63 [5%] vs 1 [1%]), fatigue (3 [4%] vs 3 [4%]), back pain (4 [5%] vs 3 [4%]), arthralgia (4 [5%] vs 1 [1

64 76%, before vs after; P < .001), mechanical back pain (4% vs 49%, P < .001), spondylitis (7% vs 76%,

65 puncture syndrome (3/8 [38%] vs 8/24 [33%]), back pain (4/8 [50%] vs 4/24 [17%]), and nausea (0/8 [0%

66 of 1941 patients in the letrozole group) and back pain (44 [2%] vs 38 [2%]).

67 [1%]), bone giant cell tumour (6 [1%]), and back pain (5 [1%]).

68 PP group had a significant reduction of low back pain (66.2% vs 50.0%; P = 0.04) and analgesic consu

69 3, and October 24, 2014, and had chronic low back pain, a positive diagnostic block at the facet join

70 erative disc disease often causes severe low-back pain, a public health problem with huge economic an

71 s included a cough that varied over the day, back pain/aches/discomfort, early satiety, appetite loss

72 Pain complaints (arms, legs, joints, back pain) affected the majority of men and women, and s

73 Non-specific low back pain affects people of all ages and is a leading co

74 ty were divided according to symptoms of low back pain alone and symptoms of low back pain with objec

75 and disability in patients with chronic low back pain, although this difference became nonsignifican

76 link between antecedent depression and later back pain among female military nurses.

77 tistically-significant predictor of incident back pain among female subjects (odds ratio [OR]: 1.75,

78 le of depression as a potential predictor of back pain among nurses appears understudied.

79 potential therapeutics for treatment of low back pain and disc degeneration.

80 ewly diagnosed AIDS presented with months of back pain and fever.

81 ual care, resulted in greater improvement in back pain and functional limitations at 26 weeks, with n

82 ationship between self-management of chronic back pain and health-related quality of life (HRQoL); (2

83 y in patients presenting with acute chest or back pain and high blood pressure.

84 ebral disc degeneration (IDD) causes chronic back pain and is linked to production of proinflammatory

85 onditions, such as fibromyalgia, chronic low back pain and myofascial pain.

86 ety, musculoskeletal disorders including low back pain and neck pain, diabetes, and cirrhosis--increa

87 Back pain and OUD were identified using administrative d

88 positive affect, the affective component of back pain and pain tolerance.

89 20-year-old man presented with 1 week of low back pain and progressive lower extremity weakness.

90 mages, intracranial pressure processing, low back pain and real-time tumour tracking; (3) outcome pre

91 rapid progression to coma preceded by lower back pain and recurrent falls.

92 Adults with chronic back pain and reported NSAID ADRs are at a higher risk o

93 associated with worse self-reported health, back pain and stop-smoker status.

94 SOE, moderate) are effective for chronic low back pain and strengthens previous findings regarding th

95 most important factor leading to chronic low back pain and subsequent disability after discectomy.

96 e sacroiliac joints in patients with chronic back pain and suspected axial spondyloarthritis.

97 een migraine and vaginal dryness and between back pain and VMS.

98 on (10 kHz SCS) in subjects with chronic low back pain and/or leg pain and performed post hoc analysi

99 us pulposus' or 'lumbar disc herniation' or 'back pain' and their age range was between 18 and 64 yea

100 y local DRG inflammation (a rat model of low back pain) and by a peripheral paw inflammation model.

101 imaging for patients with uncomplicated low back pain) and using the results for public reporting an

102 ic resonance imaging (MRI) (for headache and back pain), and referrals to other physicians (for all 3

103 ed, duloxetine was effective for chronic low back pain, and benzodiazepines were ineffective for radi

104 uloskeletal pain, nasopharyngitis, headache, back pain, and diarrhea.

105 Leg pain, back pain, and disability were converted to common scale

106 t acetaminophen is ineffective for acute low back pain, and duloxetine is associated with modest effe

107 50 mg dose (one subject) and abdominal pain, back pain, and eczema after multiple doses of 800 mg avo

108 se events (six vaginal symptoms, one case of back pain, and one case of abdominal pain) and one unexp

109 occurred in nine (3%) patients, and anaemia, back pain, and pain in extremities, each of which occurr

110 1.10-1.42), and preoperative pain disorders (back pain: aOR, 1.57; 95% CI, 1.42-1.75; neck pain: aOR,

111 Several systemic medications for low back pain are associated with small to moderate, primari

112 acologic therapies for primarily chronic low back pain are associated with small to moderate, usually

113 n-related conditions, but its effects on low back pain are uncertain.

114 e events in evolocumab-treated patients were back pain, arthralgia, headache, muscle spasms, and pain

115 c pain, encompassing conditions, such as low back pain, arthritis, persistent post-surgical pain, fib

116 emselves in different ways such as joint and back pain, as well as deficiencies in skeletal bone qual

117 when patients present with new or worsening back pain before motor, sensory, bowel, or bladder defic

118 (1.71 billion people [1.68-1.80]), with low back pain being the most prevalent condition in 134 of t

119 ire [RDQ]; range, 0-23) and in self-reported back pain bothersomeness (scale, 0-10) at 26 weeks.

120 and assessment of disease progression in low back pain, brain tumours and primary epilepsy; (2) enhan

121 tive for short-term pain relief in acute low back pain but caused sedation.

122 height are linked to the pathogenesis of low back pain, but evidence-based confirmation is lacking.

123 is a commonly used treatment for chronic low back pain, but high-quality evidence for its effectivene

124 brain representation for a constant percept, back pain, can undergo large-scale shifts in brain activ

125 y of questionnaires to patients with chronic back pain (CBP) and collected repeated sessions of resti

126 e of the acute stress response of 16 chronic back pain (CBP) patients and 18 healthy individuals expo

127 d the volume of human hippocampus in chronic back pain (CBP), complex regional pain syndrome (CRPS),

128 n-trait and Emote-trait-were associated with back pain characteristics and could be related to distin

129 +/- 13 years old; 13F, 12M) with chronic low back pain (cLBP) and 27 healthy control subjects (48.9 +

130 ated thalamocortical networks in chronic low back pain (cLBP) given its non-specific etiology and com

131 Patients with chronic low back pain (cLBP) or amyotrophic lateral sclerosis (ALS)

132 ral activity in individuals with chronic low back pain (cLBP) remains scarce and results are inconsis

133 s effective for mild to moderate chronic low back pain (cLBP), but its comparative effectiveness with

134 ommendation 2: For patients with chronic low back pain, clinicians and patients should initially sele

135 pain in 16 patients with chronic nonspecific back pain (CNBP) and in 16 age- and gender-matched healt

136 (D2/D3R) function in chronic non-neuropathic back pain (CNBP) by comparing CNBP patients and healthy

137 including several (such as headache and low back pain) commonly encountered by internal medicine pro

138 nically important improvement in chronic low back pain compared with a standardized exercise program

139 er prevalence of neck, hand/wrist, and lower back pain compared with family medicine physicians; repe

140 iotics (for URIs), radiography (for URIs and back pain), computed tomography (CT) or magnetic resonan

141 d, 44-year-old Puerto Rican man with chronic back pain, diabetes, hypertension, asthma, and a history

142 We randomized 24 patients with chronic back pain diagnosed with lumbar disk degeneration and un

143 In contrast, the history of back pain did not predict the frequency of VMS.

144 e events, mostly mild self-limited joint and back pain, did not differ between the yoga and PT groups

145 Secondary outcomes were self-reported low back pain, disability, global improvement, satisfaction,

146 Because non-specific low back pain does not have a known pathoanatomical cause, t

147 h exercise but not with rest; awakening from back pain during the second half of the night only; and

148 der adults with a new primary care visit for back pain, early imaging was not associated with better

149 adults aged 20 to 70 years with chronic low back pain enrolled between September 2012 and April 2014

150 study, individuals who developed an intense back pain episode were followed over a 1-year period, du

151 eturn to work, global improvement, number of back pain episodes or time between episodes, patient sat

152 arrassed, Mr. M. reported that during recent back-pain exacerbations he occasionally resorted to purc

153 Subjects with recent onset of back pain exhibited emergence of kD only when the pain b

154 , hydronephrosis (three [2%] vs seven [4%]), back pain (five [3%] vs three [2%]), pathological fractu

155 up, n = 94), to subjects who have lived with back pain for >10 years (chronic back pain group, n = 59

156 roximately 2 months with no prior history of back pain for 1 year (early, acute/subacute back pain gr

157 e 36 years or older was each associated with back pain for male and female nurses.

158 iofrequency denervation to treat chronic low back pain from these sources.

159 A 57 years-old patient presented with back pain, general discomfort, polydipsia, polyuria, fat

160 y for back pain in the early, acute/subacute back pain group is limited to regions involved in acute

161 me, whereas in the persistent acute/subacute back pain group, activity diminished in acute pain regio

162 volved in acute pain, whereas in the chronic back pain group, activity is confined to emotion-related

163 In the recovered acute/subacute back pain group, brain activity diminished in time, wher

164 those who recover (recovered acute/sub-acute back pain group, n = 19) and those in which the back pai

165 ck pain persists (persistent acute/sub-acute back pain group, n = 20; based on a 20% decrease in inte

166 lived with back pain for >10 years (chronic back pain group, n = 59).

167 back pain for 1 year (early, acute/subacute back pain group, n = 94), to subjects who have lived wit

168 were aged 18 and over, suffered from chronic back pain, had opted in to the clinic and had sufficient

169 We found that patients with back pain have alterations in brain dopamine function th

170 wever, significant associations remained for back pain (hazard ratio, 1.13 [99% CI, 1.03-1.24]), migr

171 Patients were classified with inflammatory back pain if they had >/=2 positive responses to 4 valid

172 hat most patients with acute or subacute low back pain improve over time regardless of treatment, cli

173 ted included reduction or elimination of low back pain, improvement in back-specific and overall func

174 20; based on a 20% decrease in intensity of back pain in 1 year).

175 al obscurations occurred in 68% of patients, back pain in 53%, and pulse synchronous tinnitus in 52%.

176 r a history of depression predicted incident back pain in a population of military registered nurses

177 We examined the prevalence of low back pain in adolescents and its association with BMI an

178 ht was associated with increased risk of low back pain in both genders.

179 Low back pain in children and adolescents is a common proble

180 k during 2011-2014 without evidence of prior back pain in clinical records.

181 ed mechanisms of pain regulation, in chronic back pain in human subjects.

182 er BMI was significantly associated with low back pain in males (for overweight, odds ratio = 1.097,

183 We observed that brain activity for back pain in the early, acute/subacute back pain group i

184 Odds ratios for low back pain in the tallest group compared with the shortes

185 to be a further, modifiable risk factor for back pain in this population.

186 ad', 'tumor', 'spine', 'classification' and 'back pain' in the title and abstract of the manuscripts

187 , -4.8 points [CI, -8.9 to -0.7 points]) and back pain intensity (relative difference, -1.0 points [C

188 outcomes included the extent of disability, back-pain intensity, and quality-of-life measures at pre

189 Low back pain is associated with degeneration of the interve

190 Acute low back pain is common and spinal manipulative therapy (SMT

191 Back pain is common in the general population, but only

192 ceptability of acupuncture treatment for low back pain is complex and multifaceted.

193 The most common cause of low back pain is degenerative disease of the intervertebral

194 Back pain is linked to intervertebral disc (IVD) degener

195 The clinical course of low back pain is often favourable, thus many patients requir

196 Low back pain is often the direct result of degeneration of

197 Observations: Low back pain is rarely seen in youth before they reach scho

198 afternoon in 47 subjects without current low back pain (IVDs = 230; age range, 20-71 years) after obt

199 us, and chronic pain conditions (chronic low back pain, knee osteoarthritis, and fibromyalgia).

200 ed to screen for risk factors for future low back pain (LBP) -related disability and work loss respec

201 l disc (IVD) degeneration and consequent low back pain (LBP) are common and costly pathological proce

202 st-effective primary care treatments for low back pain (LBP) are required to reduce the burden of the

203 differs between people with and without low back pain (LBP) during a low-load lifting task.

204 Low back pain (LBP) in children and adolescents is a common

205 Low back pain (LBP) is a common debilitating condition which

206 Low back pain (LBP) is a widespread debilitating disorder of

207 Low back pain (LBP) is common in primary care.

208 Low back pain (LBP) is responsible for more than 2.5 million

209 ted with a specific phenotype of chronic low back pain (LBP).

210 of the paraspinal muscles in people with low back pain (LBP); but so far, HDEMG has not been used to

211 ain patients, we followed brain activity for back pain longitudinally over a 1-year period, and compa

212 categorizing musculoskeletal pain into lower back pain, lower extremity (hips, knees, and feet/ankles

213 to evaluate the relationship between lumbar back pain, lumbar disc herniation, and erector spinae an

214 ng brain activity for rating fluctuations of back pain magnitude.

215 the largest number of YLDs in 2010 were low back pain, major depressive disorder, other musculoskele

216 Patients with low back pain may have fatty degeneration in erector spina a

217 nfidence for clinical features (inflammatory back pain, mechanical back pain, muscular back pain, rad

218 tain noncancer pain diagnoses, in particular back pain, migraine, and psychogenic pain.

219 noses of pain-related conditions (arthritis, back pain, migraine, neuropathy, headache or tension hea

220 health problems not listed, or complained of back pain, migraines, or digestive problems at baseline.

221 treatments for participants with chronic low back pain (Mint study) were conducted in 16 multidiscipl

222 duced mechanical pain behaviors induced by a back pain model and a model of peripheral inflammatory p

223 features (inflammatory back pain, mechanical back pain, muscular back pain, radicular back pain, spon

224 83 [50%]), skin disorders (n = 81 [48.8%]), back pain (n = 54 [32.5%]), and alopecia (n = 53 [31.9%]

225 at could be confused for musculoskeletal low back pain (nepholithiasis, urinary tract infection, oste

226 erse events were nasopharyngitis (12 [11%]), back pain (nine [8%]), and diarrhoea (eight [7%]) in the

227 acetaminophen was ineffective for acute low back pain, nonsteroidal anti-inflammatory drugs had smal

228 the patient does not develop the symptoms of back pain or leg pain during the injection.

229 dental but may be found in patients with low back pain or neuromuscular deficits.

230 were half as likely to have work-related low-back pain (OR=0.50, 95% CI 0.26-0.96) and nurses reporti

231 ical trials of participants with chronic low back pain originating in the facet joints, sacroiliac jo

232 rted acute on chronic onset of thoracolumbar back pain over a period of 24 hours.

233 In our experiment, chronic back pain patients and healthy controls completed an app

234 observed in a separate cohort of chronic low-back pain patients and was associated with dynamic chang

235 Low back pain patients are sometimes offered fusion surgery

236 e general population, but only a subgroup of back pain patients develops a disabling chronic pain sta

237 nome-wide-association studies in chronic low back pain patients identified sepiapterin reductase as a

238 sites and accurately classified chronic low-back pain patients in two additional independent dataset

239 We tracked brain properties in subacute back pain patients longitudinally for 3 years as they ei

240 producible across two cohorts of chronic low-back pain patients obtained from different sites and acc

241 g-state activity in the subacute and chronic back pain patients revealed loss of power in the slow-5

242 At baseline, subacute back pain patients showed altered local nucleus accumben

243 er number of healthy individuals and chronic back pain patients were also studied concomitantly, as p

244 Specifically, subacute back pain patients who are at risk for developing chroni

245 In a subset of subacute back pain patients, we followed brain activity for back

246 and affective dimensions of pain in chronic back pain patients.

247 egions predicts placebo analgesia in chronic back pain patients.

248 k pain group, n = 19) and those in which the back pain persists (persistent acute/sub-acute back pain

249 sitive responses to 4 validated inflammatory back pain questions: presence of morning stiffness >30 m

250 ry back pain, mechanical back pain, muscular back pain, radicular back pain, spondylitis, sacroiliiti

251 Occupational back pain rates are substantial among registered nurses,

252 First we compared back pain-related brain activity between subjects who ha

253 eration (IVDD) as major cause of chronic low back pain represent the most common degenerative joint p

254 CI, 1.6 to 6.3); and prolonged arm, leg, or back pain (RR, 4.0; 95% CI, 2.2 to 7.1).

255 ently associated with BASDAI question 2 (ie, back pain score) (beta = 0.45, P = .01), mean stiffness

256 to a person's health status) indicating low back pain severity were divided according to symptoms of

257 In addition, she reported progressive back pain since she was 5 years old.

258 For back pain, smaller differences favoring steroids compare

259 on, massage, and acupuncture for chronic low back pain (SOE, low to moderate).

260 puncture is modestly effective for acute low back pain (SOE, low).

261 Among patients with acute low back pain, spinal manipulative therapy was associated wi

262 cal back pain, muscular back pain, radicular back pain, spondylitis, sacroiliitis, and other) and ove

263 y had >/=1 of the following: chest/abdominal/back pain, syncope, perfusion deficit, and if AAS was in

264 After chronic low back pain, Temporomandibular Joint (TMJ) disorders are t

265 y drugs had smaller benefits for chronic low back pain than previously observed, duloxetine was effec

266 rse triage to identify the rare cases of low back pain that are caused by medically serious pathology

267 truction site manager experienced 6 weeks of back pain that was not responsive to over-the-counter no

268 As the most common cause of low back pain, the cascade of intervertebral disc (IVD) dege

269 spect to reductions in disability related to back pain, the changes in the Oswestry Disability Index

270 os ranging from 1.33 [99% CI, 1.22-1.45] for back pain to 2.61 [1.82-3.74] for psychogenic pain).

271 ies varied from a low of 31% (n = 8) for low back pain to a high of 68% (n = 23) for fibromyalgia.

272 terviews following acupuncture treatment for back pain to identify, understand and describe the eleme

273 ect patients classified to have inflammatory back pain to refer for early rheumatologic assessment.

274 Ophthalmologists may use these questions on back pain to select patients classified to have inflamma

275 many guidelines allow for older adults with back pain to undergo imaging without waiting 4 to 6 week

276 eviews and RCTs, for RCTs of adults with low back pain treated in ambulatory settings with SMT compar

277 Among adults with chronic low back pain, treatment with MBSR or CBT, compared with usu

278 rials showing modest effects for chronic low back pain; trials were not designed to assess serious ha

279 to a free clinic with acute exacerbation of back pain triggered by carrying heavy loads of trash at

280 re pulmonary embolism (three [4%] patients), back pain (two [2%]), and diarrhoea (two [2%]).

281 deviation]; age range, 20-79 years) with low back pain underwent standard 1.5-T MR imaging, which was

282 Prevalence of low back pain was 0.2% for both males and females with objec

283 population, and 35% (4-88) in workers; lower back pain was 18% (14-24), 31% (22-41), and 44% (33-55),

284 The incidence rate of back pain was 18.6 per 100 person-years and the period p

285 rs, 224 (65.7%) were women, mean duration of back pain was 7.3 years (range, 3 months-50 years), 123

286 -evoked pain in individuals with chronic low back pain was associated with longer reaction times, del

287 Lower back pain was prevalent (63%), followed by ankle/foot (5

288 Chest or back pain was the most commonly reported presenting symp

289 Seven RCTs involving 864 patients with low back pain were eligible for review.

290 xamined symptoms (neck lump, chest pain, and back pain) were consistently associated with increased o

291 habitual learning and motivation in chronic back pain, which helps explaining why chronic pain can b

292 oes not address noninvasive treatment of low back pain, which is covered by a separate ACP guideline

293 patients using ice-pack therapy for chronic back pain who developed erythematous, purpuric plaques a

294 tandard deviation, 43.2 years +/- 13.5) with back pain who fulfilled the Assessment of SpondyloArthri

295 commendation 3: In patients with chronic low back pain who have had an inadequate response to nonphar

296 ness >30 minutes in duration; improvement in back pain with exercise but not with rest; awakening fro

297 s of low back pain alone and symptoms of low back pain with objective corroborating findings.

298 Low back pain with or without objective findings was associa

299 ting (OP-8) reduced MR imaging rates for low back pain without conservative therapy in either Medicar

300 of MR imaging examinations performed for low back pain without history of conservative therapy.

301 early diagnostic imaging in older adults for back pain without radiculopathy is uncertain.