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1 ment duration for infections associated with bacteremia.
2 ntly causes severe invasive disease, such as bacteremia.
3 e of bacteremia, especially during S. aureus bacteremia.
4 mplicated PNA, UTI, or ABSSSI and associated bacteremia.
5 ation of NESp and increased virulence during bacteremia.
6 ents specifically with Staphylococcus aureus bacteremia.
7 ad short, 28% intermediate, and 9% prolonged bacteremia.
8 s for the prevention of postdental procedure bacteremia.
9 provide for an effective cure the persistent bacteremia.
10 ISPR)-Cas9, sensitized P9 pups to E. coli K1 bacteremia.
11 aureus bacteremia, 48.5% (SE, 0.4%) had MRSA bacteremia.
12 e prompt initiation of effective therapy for bacteremia.
13 phy in consecutive patients with E. faecalis bacteremia.
14 me was the incidence of postdental procedure bacteremia.
15 ospitals between 2000-2011 with pneumococcal bacteremia.
16 PC-Kp infections, especially those involving bacteremia.
17 and in a subgroup excluding catheter-related bacteremia.
18 cells in culture and lethality during mouse bacteremia.
19 illin-resistant Staphylococcus aureus (MRSA) bacteremia.
20 in methicillin-susceptible S. aureus (MSSA) bacteremia.
21 ity and mortality in patients with S. aureus bacteremia.
22 mes develop into systemic infections such as bacteremia.
23 have shown improved length of stay (LOS) in bacteremia.
24 considered in all patients with E. faecalis bacteremia.
25 sed alveolar tissue destruction and systemic bacteremia.
26 The negative-control outcome was gut-origin bacteremia.
27 curred in 3.2% of cases, with no FMT-related bacteremia.
28 revalence of IE in patients with E. faecalis bacteremia.
29 uided antibiotic management in patients with bacteremia.
30 ter treatment for subjects with pneumococcal bacteremia.
31 ere correlated with mortality and persistent bacteremia.
32 outcomes in daptomycin-treated enterococcal bacteremia.
33 or clinicians in the management of S. aureus bacteremia.
34 nt in the treatment of Staphylococcus aureus bacteremia.
35 revalence of IE in patients with E. faecalis bacteremia.
36 lungs and airways, with repeated episodes of bacteremia.
37 reduce mortality rates in patients with MSSA bacteremia.
38 g rates of colonized patients progressing to bacteremia.
39 use in humans of urinary tract infection and bacteremia.
40 tam as definitive treatment of P. aeruginosa bacteremia.
41 tion of treatment to impact outcomes in MSSA bacteremia.
42 respiratory and urinary tract infections and bacteremia.
43 e to recurrent nontyphoidal Salmonella (NTS) bacteremia.
44 motes acute inflammation, tissue damage, and bacteremia.
45 g clinical isolates of S. marcescens causing bacteremia.
46 2018 among 352 hospitalized adults with MRSA bacteremia.
47 concerning clinical pathogen, causing fatal bacteremia.
48 based on the daily probability of acquiring bacteremia.
49 The negative control outcome was gut-origin bacteremia.
50 italized patients with Staphylococcus aureus bacteremia.
51 w, moderate, and high pretest probability of bacteremia.
52 . aureus and methicillin-sensitive S. aureus bacteremia.
54 mong adults with uncomplicated gram-negative bacteremia, 30-day rates of clinical failure for CRP-gui
56 ients with coagulase negative Staphylococcus bacteremia (5.5d and 4.5d vs 7.2d; P=0.003) in AXDX, AXD
58 ly alarming that probiotic strains can cause bacteremia(8,9), yet direct evidence for an ancestral li
61 osa from the bloodstream to the feces during bacteremia, a process that promotes transmission in this
63 nd compared with patients with uncomplicated bacteremia (absence of any of the risk factors and no kn
64 In patients hospitalized with gram-negative bacteremia achieving clinical stability before day 7, an
66 Patients with septic shock and S. aureus bacteremia admitted directly from the emergency departme
68 the least incidence of postdental procedure bacteremia among all oral or topical forms of prophylact
69 rventions in preventing postdental procedure bacteremia among all the oral/topical forms of intervent
70 the least incidence of postdental procedure bacteremia among all the prophylactic interventions (odd
71 rt that probiotic strains can directly cause bacteremia and adaptively evolve within ICU patients.
72 and who had a history of serious infection (bacteremia and associated sternal osteomyelitis, infecti
73 of blood proteins at initial presentation of bacteremia and disease severity outcomes using 2 cohorts
75 nvasive organ dissemination during S. aureus bacteremia and for studying bacterial dynamics during mi
76 ical strategies for persistent and relapsing bacteremia and found that a persister killer, but not a
78 identified patients at low and high-risk for bacteremia and fungemia using routinely collected electr
80 cantly suppressed in patients with S. aureus bacteremia and in S. aureus-challenged primary human mac
88 streptococcus, or GBS) is a common cause of bacteremia and sepsis in newborns, pregnant women, and i
93 nses to BPI can arise acutely in response to bacteremia and that this association is not limited to P
95 etermine prevalence and common etiologies of bacteremia and to inform a diagnostic approach to reliev
98 onizing bacteria across the small intestine, bacteremia, and invasion of the meninges, with animals f
101 ure infections (SSSI), Staphylococcus aureus bacteremia, and right-sided endocarditis infections asso
104 ab, the rate of a CPE carrier progressing to bacteremia; and deltac, the progression rate to nonbacte
105 above 65 years were more likely to have NTS bacteremia (AOR, 1.54 [95% CI, 1.46 to 1.67]; 2.57 [95%
106 Mortality rates from Staphylococcus aureus bacteremia are high and have only modestly improved in r
111 anging epidemiology of Staphylococcus aureus bacteremia, as well as the variables associated with poo
113 isolates recovered from separate episodes of bacteremia at a single academic institution in Toronto,
114 e, 4.5%; 95% CI, -3.7% to 12.7%); persistent bacteremia at day 5 was observed in 19 of 166 (11%) vs 3
115 a 90-day composite of mortality, persistent bacteremia at day 5, microbiological relapse, and microb
116 mortality at days 14, 42, and 90; persistent bacteremia at days 2 and 5; acute kidney injury (AKI); m
118 models resulting in resolving or persistent bacteremia, based on the total SA exceeding a detection
119 er >=38 degrees C for >=12 h and/or S. Typhi bacteremia) between participants challenged with wild-ty
120 sms are a major cause of gastroenteritis and bacteremia, but little is known about maternally acquire
122 he average LOS that would be observed if all bacteremia cases could be prevented was multiplied by th
123 Anaerobes were identified in 57% of the bacteremia cases from the NB-PC group by conventional me
124 total number of extra ICU days caused by 666 bacteremia cases was estimated at 2453 (95% confidence i
129 ured by the numbers of Staphylococcus aureus bacteremia, Clostridium difficile infection, and vancomy
131 Patients with risk factors for complicated bacteremia (community acquisition, persistently positive
132 uding adults hospitalized with gram-negative bacteremia conducted in 3 Swiss tertiary care hospitals
133 r endocarditis was protective and persistent bacteremia constituted the sole risk factor for relapse.
136 ntext of longitudinal surgical registers and bacteremia data, we describe a valuable approach to adju
137 , in a patient who developed a break-through bacteremia despite taking antibiotics to which the S. ep
138 mphiphilic pathogen biomarkers indicative of bacteremia, directly in aqueous blood, by mimicking inna
139 esent, most streptococci or Enterobacterales bacteremias do not require routine follow-up blood cultu
140 st-PCV eras, the prevalence of S. pneumoniae bacteremia dropped across all age groups (from 32.4% to
141 De-escalation in patients with monomicrobial bacteremia due to Enterobacteriaceae was not associated
147 ibiotic therapy did not significantly affect bacteremia duration; however, time to source-control pro
148 ended in syndromes with a high likelihood of bacteremia (eg, endovascular infections) and those with
150 sk of becoming infected during an episode of bacteremia, especially during S. aureus bacteremia.
152 port a markedly higher risk of Lactobacillus bacteremia for intensive care unit (ICU) patients treate
154 ly been associated with several outbreaks of bacteremia from contaminated pharmaceutical products.
155 infection (pyelonephritis) or urinary-source bacteremia, from non-invasive UPEC, defined as isolates
156 us aureus bacteremia (SAB) and gram-negative bacteremia (GNB) to compare the characteristics, outcome
159 r than that of a matched cohort whose KPC-Kp bacteremia had been treated with drugs other than CAZ-AV
160 o difference between MRSA and MSSA, but MRSA bacteremia had more readmission for bacteremia recurrenc
161 The gradient boosting machine model for bacteremia had significantly higher area under the recei
162 ivariate analysis of the 208 cases of KPC-Kp bacteremia identified septic shock, neutropenia, Charlso
163 tissue infection was present in 50%, sepsis/bacteremia in 52%, osteomyelitis in 10%, and endocarditi
165 munogenic and effective in reducing S. Typhi bacteremia in children 9 months to 16 years of age.
167 in-flora-related, or central-line-associated bacteremia in patients with hematological malignancies w
168 in flora-related, or central line-associated bacteremia in patients with hematological malignancies w
169 urgent need for a rapid method for detecting bacteremia in pediatric patients with co-morbidities to
171 up with over 30 times the occurrence rate of bacteremia in the low-risk group (27.4% vs 0.9%; p < 0.0
175 lar bacterial cell numbers (ie, the level of bacteremia), in patients at the time of clinical present
176 tion of meningitis, bacteremic pneumonia, or bacteremia (including hearing loss, developmental delay,
177 lin-susceptible Staphylococcus aureus (MSSA) bacteremia, including immediate clearance (<=24 hours) i
179 e further assessed, if PCT can reflect early bacteremia induced by non-surgical periodontal treatment
180 level cytomegalovirus viremia, gram-negative bacteremia, invasive mold infection, acute and chronic g
184 illin-resistant Staphylococcus aureus (MRSA) bacteremia is associated with mortality of more than 20%
189 ity testing (AST) in Gram-negative rod (GNR) bacteremia is compelling; however, evidence supporting i
191 for patients at risk of adverse events if a bacteremia is missed (eg, patient with pacemaker and sev
193 hallenge, although only 7% of mice presented bacteremia, LF and EF were detected in the blood of 100%
194 h a non-inferiority margin of 10%: recurrent bacteremia, local suppurative complication, distant comp
198 expression, and attenuated virulence in the bacteremia model as compared to their respective parenta
204 oup (n = 11/23, 48%), when compared with the bacteremia (n = 1/11, 9%; p = 0.03) and nonbacterial thr
206 atients with invasive Salmonella Typhimurium bacteremia (n = 7) and those with Staphylococcal bactere
207 eremia (n = 7) and those with Staphylococcal bacteremia (n = 7) with 100% correlation with confirmato
208 compared with three other conditions, namely bacteremia, nonbacterial thrombotic endocarditis, and he
210 -lactamase (ESBL)-producing Escherichia coli bacteremia occurred after they had undergone FMT in two
211 nt upon the growth kinetics or the levels of bacteremia of B. pseudomallei represent the next-generat
212 TGR in endothelial cells results in very low bacteremia, optimal sensitivity of qPCR for these ricket
215 We identified all patients diagnosed with bacteremia or sepsis, endocarditis, osteomyelitis or sep
216 impacts human gut microbiota as a prelude to bacteremia or whether antimalarials affect gut microbiot
217 CI: 1.26 to 4.40; p = 0.007), monomicrobial bacteremia (OR: 2.73; 95% CI: 1.23 to 6.05; p = 0.013),
218 ls were elevated in patients with persistent bacteremia (P < .0001), endovascular (P = .026) and meta
219 vated in mortality (P = .008) and persistent bacteremia (P = .034), while no difference occurred in I
220 on with an agr group III organism (P = .04), bacteremia (P = .04), delayed source control (P < .001),
221 tcomes of mortality (P=0.008) and persistent bacteremia (P=0.034), while no difference occurred in IL
222 ococcus aureus (MSSA and MRSA, respectively) bacteremia, particularly readmission, is scarce and requ
225 ts with uncomplicated Pseudomonas aeruginosa bacteremia, patients receiving short-course (median, 9 d
226 h persistent methicillin-resistant S. aureus bacteremia (PB) and resolving methicillin-resistant S. a
227 ed infections such as Pseudomonas aeruginosa bacteremia pose a major clinical risk for hospitalized p
228 ibutes to increased capacity to resolve MRSA bacteremia, potentially through a mechanism involving in
230 nd resolving methicillin-resistant S. aureus bacteremia (RB) matched by sex, age, race, hemodialysis
232 but MRSA bacteremia had more readmission for bacteremia recurrence (hazard ratio, 1.17 [95% confidenc
234 acteremia is associated with readmission for bacteremia recurrence, increased mortality, and longer h
236 antistaphylococcal beta-lactam on mortality, bacteremia, relapse, or treatment failure in patients wi
238 h the highest risk for treatment failure and bacteremia-related complications, providing a valuable t
240 wth of the same organism causing the initial bacteremia), restarting gram-negative-directed antibioti
241 study of patients with Staphylococcus aureus bacteremia (SAB) and gram-negative bacteremia (GNB) to c
242 -10 responses early in Staphylococcus aureus bacteremia (SaB) are associated with bacteremia duration
243 early in the course of Staphylococcus aureus bacteremia (SaB) are associated with bacteremia duration
248 disease management of Staphylococcus aureus bacteremia (SAB) was surveyed through the Emerging Infec
249 atients with high-risk Staphylococcus aureus bacteremia (SAB), because of the risk for metastatic inf
250 ured by the numbers of Staphylococcus aureus bacteremia (SAB), Clostridium difficile infection (CDI)
260 26% definite IE in patients with E. faecalis bacteremia, suggesting that echocardiography should be c
262 filtration at the infectious foci, increased bacteremia, systemic inflammatory response, and multiorg
264 ic treatment for uncomplicated gram-negative bacteremia to 7 days is an important antibiotic stewards
265 767 hospitalized patients with P. aeruginosa bacteremia treated with beta-lactam monotherapy during 2
267 udy focusing on intensive care unit-acquired bacteremia using data from 2 general intensive care unit
269 ssion were perinatal infections, candidemia, bacteremia, very low birth weight, prematurity, respirat
278 s that discriminate resolving and persistent bacteremia, we applied a machine learning approach and f
280 clinical outcomes in patients with S. aureus bacteremia, we evaluated the association between a panel
282 reus (MRSA), streptococcal, and pneumococcal bacteremia were found to significantly increase the risk
283 A total of 344 patients with E. faecalis bacteremia were included, all examined using echocardiog
285 otic treatment for PNA, UTI, and ABSSSI with bacteremia were not associated with increased overall ri
289 the lux signal in a mouse model of S. aureus bacteremia with a sensitivity of approximately 3 x 10(4)
291 ptococcus pneumoniae infection can result in bacteremia with devastating consequences including heart
292 od from positive blood culture broth for GNR bacteremia with electronic isolate-specific de-escalatio
293 ls of sepsis (cecal ligation and puncture or bacteremia with Escherichia coli or Streptococcus pneumo
294 n cohorts, demonstrating associations of Mtb bacteremia with progressive phenotypes of latent infecti
296 rdiac surgery, embolic events, or relapse of bacteremia with the primary pathogen, from the time of r
298 es of methicillin-resistant S. aureus (MRSA) bacteremia, with a rising proportion due to MSSA (55% gr
299 terleukin-10 (IL-10) than patients with MRSA bacteremia without DNMT3A mutation (A/C: 9.7038 pg/mL vs
300 Staphylococcus aureus is a leading cause of bacteremia, yet there remains a significant knowledge ga