コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 e a mortality rate comparable to spontaneous bacterial peritonitis.
2 testinal bleeding, and 65.5% for spontaneous bacterial peritonitis.
3 s due to Entamoeba histolytica as well as to bacterial peritonitis.
4 enteric infection, and possibly spontaneous bacterial peritonitis.
5 lammation and sepsis that are symptomatic of bacterial peritonitis.
6 f peritoneal macrophages, and (iii) clearing bacterial peritonitis.
7 , esophageal variceal bleed, and spontaneous bacterial peritonitis.
8 t worsen vasodilatation, such as spontaneous bacterial peritonitis.
9 hallenge with other TLR agonists, as well as bacterial peritonitis.
10 ophilus influenzae rarely causes spontaneous bacterial peritonitis.
11 contribute to optimal survival during acute bacterial peritonitis.
12 from hospitalized patients with spontaneous bacterial peritonitis.
13 al in murine models of acute endotoxemia and bacterial peritonitis.
14 trocytic ascites is a variant of spontaneous bacterial peritonitis.
15 the host's systemic inflammatory response to bacterial peritonitis.
16 vity significantly increase the lethality of bacterial peritonitis.
17 crease of mesothelial EGFR expression during bacterial peritonitis.
18 who have hepatorenal syndrome or spontaneous bacterial peritonitis.
19 testinal bleeding, and 48.8% for spontaneous bacterial peritonitis.
20 ects the outcome of sterile inflammation and bacterial peritonitis.
21 significantly increased in a mouse model of bacterial peritonitis after Cx43 inhibition and in Cx43
23 utrophils are recruited normally to sites of bacterial peritonitis and chemical dermatitis, indicatin
24 scites and hepatorenal syndrome, spontaneous bacterial peritonitis and hepatic encephalopathy, as wel
26 duces mortality in patients with spontaneous bacterial peritonitis and improves outcome following lar
27 neal dialysis patients presenting with acute bacterial peritonitis and monitoring individuals before
28 vo response to a well-characterized model of bacterial peritonitis and sepsis induced by cecal ligati
29 ovement in c-statistic: <.01 for spontaneous bacterial peritonitis and variceal hemorrhage to ascites
30 mRNA levels were increased in mice following bacterial peritonitis, and blocking an endogenous tumor
31 of cirrhosis, including ascites, spontaneous bacterial peritonitis, and esophageal variceal hemorrhag
33 al bleeding, serious infections, spontaneous bacterial peritonitis, and hepatorenal syndrome (RR = 0.
34 ly community-acquired pneumonia, spontaneous bacterial peritonitis, and IgE-mediated food allergy.
35 with interrupted uteroplacental blood flow, bacterial peritonitis, and older age but not in fetuses
36 xis, acute bacterial meningitis, spontaneous bacterial peritonitis, and outpatient gonococcal infecti
37 ith an escalated risk of sepsis, spontaneous bacterial peritonitis, and the use of last-line antibiot
38 lbumin, blood urea, ascites, and spontaneous bacterial peritonitis as factors significantly predictiv
39 l of normal C57BL/6 mice in a model of acute bacterial peritonitis, cecal ligation and puncture (CLP)
40 dict clinical outcomes (ascites, spontaneous bacterial peritonitis, Child-Turcotte-Pugh score >or=7 o
41 enteric infection, and possibly spontaneous bacterial peritonitis, community-acquired pneumonia, and
42 factor were analyzed in patients with overt bacterial peritonitis during the first 5 days of infecti
44 variceal bleeding, paracentesis, spontaneous bacterial peritonitis, hepatic encephalopathy, hepatoren
45 ld-Turcotte-Pugh score, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, variceal
46 iagnoses as symptomatic ascites, spontaneous bacterial peritonitis, hepatorenal syndrome, or fulminan
47 (HR, 0.47 [95% CI, 0.40-0.56]), spontaneous bacterial peritonitis (HR, 0.55 [95% CI, 0.46-0.65]), pa
48 in such as infections other than spontaneous bacterial peritonitis, hyponatremia, HE, prevention of p
49 arices, hepatorenal syndrome, or spontaneous bacterial peritonitis identified by ICD-9/CPT codes.
50 revalence and natural history of spontaneous bacterial peritonitis in asymptomatic patients with asci
51 sment of diagnostic criteria for spontaneous bacterial peritonitis in outpatients may be required.
53 NLRP3 inflammasome is activated during acute bacterial peritonitis in patients on PD, and this activa
56 We describe a typical case of spontaneous bacterial peritonitis in which the causative organism wa
58 me paracentesis, at diagnosis of spontaneous bacterial peritonitis, in acute kidney injury, and in he
59 ations (ascites, encephalopathy, spontaneous bacterial peritonitis) increased with severity of AKI.
60 OFA score, bleeding, female sex, spontaneous bacterial peritonitis, intermediate increases in serum c
61 ascites, the annual incidence of spontaneous bacterial peritonitis is 11% and of hepatorenal syndrome
65 om individuals with a history of spontaneous bacterial peritonitis, known to have defective cellular
67 the cells that produced IFN-gamma in a mouse bacterial peritonitis model and determine the effects of
69 event (SAE) occurred; cecal perforation and bacterial peritonitis occurred following colonoscopic FM
70 ysis, antibiotic prophylaxis for spontaneous bacterial peritonitis (odds ratio [OR] 8.3, P=0.002), po
71 f other complications, including spontaneous bacterial peritonitis or other bacterial infections, is
72 ermined by diagnoses of ascites, spontaneous bacterial peritonitis, or esophageal variceal hemorrhage
73 utpatient diagnoses for ascites, spontaneous bacterial peritonitis, or esophageal variceal hemorrhage
74 tion, renal replacement therapy, spontaneous bacterial peritonitis, positive blood culture, and infec
75 e liver disease 25, preoperative spontaneous bacterial peritonitis prophylaxis, and antimicrobial exp
77 act infection (UTI) (28.5%), and spontaneous bacterial peritonitis (SBP) (22.5%) were the most preval
78 e major infectious diseases were spontaneous bacterial peritonitis (SBP) (645, 49.8%), urinary tract
79 D) score over 20 points and with spontaneous bacterial peritonitis (SBP) as a precipitating event was
81 ntibiotic prophylaxis to prevent spontaneous bacterial peritonitis (SBP) in patients colonized with m
82 own to decrease the incidence of spontaneous bacterial peritonitis (SBP) in patients with cirrhosis a
89 , urinary tract infection (UTI), spontaneous bacterial peritonitis (SBP), and sepsis without specific
96 Urinary tract infections (52%), spontaneous bacterial peritonitis (SBP, 23%) and spontaneous bactere
99 dition, malignancy-related ascites (13%) and bacterial peritonitis specimens (5.8%) occasionally yiel
100 , ascites, hepatorenal syndrome, spontaneous bacterial peritonitis, the cardiopulmonary complications
102 30% for secondary prophylaxis of spontaneous bacterial peritonitis, to 90% for assays for cell number
103 k of infection: prior episode of spontaneous bacterial peritonitis, upper gastrointestinal bleeding,
104 ly; P < 0.0001), particularly in spontaneous bacterial peritonitis, urinary tract infection, and pneu
105 lications of cirrhosis (ascites, spontaneous bacterial peritonitis, varices, variceal hemorrhage, enc
106 techniques for the diagnosis of spontaneous bacterial peritonitis, we prospectively compared three b
107 arcinoma, variceal bleeding, and spontaneous bacterial peritonitis were associated with a higher mort
108 blood vessels, restricted dietary intake, or bacterial peritonitis were compared with tissues from TM
109 involved in the pathogenesis of spontaneous bacterial peritonitis, which is a common complication of
110 ients with refractory ascites or spontaneous bacterial peritonitis while other data suggests a surviv