ライフサイエンスコーパス: bacterial surface

1 involved in the acquisition of hTSP-1 to the bacterial surface.

2 regulator, complement factor H (CFH), to the bacterial surface.

3 phologically distinct pilus structure on the bacterial surface.

4 on results in lower deposition of C3b on the bacterial surface.

5 diacylglycerol and mycolate ester wax to the bacterial surface.

6 d for complement factor C3 deposition on the bacterial surface.

7 plasminogen into plasmin and binds it to the bacterial surface.

8 er membrane proteins (OMPs) expressed on the bacterial surface.

9 correlated with higher C3b deposition on the bacterial surface.

10 the presence of tubes extending out from the bacterial surface.

11 ule as prominent targets of these Abs on the bacterial surface.

12 e-binding adhesin, which is expressed on the bacterial surface.

13 ociated protein form extended fibrils on the bacterial surface.

14 he Fc-binding proteins were removed from the bacterial surface.

15 d the levels of complement deposition on the bacterial surface.

16 ave a discrete, punctate localization on the bacterial surface.

17 ccessible to EsaD-specific antibodies on the bacterial surface.

18 TonB-dependent transporters expressed at its bacterial surface.

19 s and their subsequent polymerization on the bacterial surface.

20 otherwise periplasmic tether mutants to the bacterial surface.

21 a particular lipopolysaccharide (LPS) on the bacterial surface.

22 ds most likely lie in close proximity on the bacterial surface.

23 ipid A increasing the negative charge of the bacterial surface.

24 hlighting its role in the maintenance of the bacterial surface.

25 LT binds to lipopolysaccharide (LPS) on the bacterial surface.

26 position and activation of complement on the bacterial surface.

27 link pilin subunits and assemble pili on the bacterial surface.

28 s the presence of needles extending from the bacterial surface.

29 to thin fibrils and denser aggregates on the bacterial surface.

30 n of the Hia trimeric autotransporter on the bacterial surface.

31 nt down-regulatory protein, factor H, to the bacterial surface.

32 ructure, comprising the YscF protein, on the bacterial surface.

33 filaments that protrude up to 1 mum from the bacterial surface.

34 BS NeuA reduced O-acetylation of Sias on the bacterial surface.

35 anism of evasion is to mobilize actin to the bacterial surface.

36 tion of some of the secreted proteins on the bacterial surface.

37 ld-type levels of the SpaA-type pilus on the bacterial surface.

38 electrostatic attraction between AAF and the bacterial surface.

39 on and a hollow 'needle' protruding from the bacterial surface.

40 amic properties of metal complexation on the bacterial surface.

41 itiated by lipooligosaccharides (LOS) on the bacterial surface.

42 or rearrangement of and modifications to the bacterial surface.

43 in blood and on complement deposition on the bacterial surface.

44 ates delivery of the passenger domain to the bacterial surface.

45 e mechanism by which HMW1 is anchored to the bacterial surface.

46 er show that MxiH and IpaD colocalize on the bacterial surface.

47 hysiological consequences of fH bound to the bacterial surface.

48 xtracellular assembly of Mfa fimbriae on the bacterial surface.

49 nergy, the virus interacts directly with the bacterial surface.

50 N-terminal host-colonization domain from the bacterial surface.

51 ificity for carbohydrate determinants on the bacterial surface.

52 ntly decreasing complement deposition on the bacterial surface.

53 te at and neutralize negative charges on the bacterial surface.

54 croscopy confirmed their localization on the bacterial surface.

55 membrane protein OmpD-specific IgG2a to the bacterial surface.

56 to the generation of an invasive proteolytic bacterial surface.

57 ide to transport across membrane and bind to bacterial surface.

58 ks can be achieved by engineering the native bacterial surface.

59 e presence of carbohydrate structures on the bacterial surface.

60 es are randomly oriented with respect to the bacterial surface.

61 arge serine-rich repeat protein, Hsa, to the bacterial surface.

62 ights into the interactions occurring at the bacterial surface.

63 located proteins, thus anchoring them to the bacterial surface.

64 g activation of the classical pathway on the bacterial surface.

65 understanding nanoparticle interaction with bacterial surfaces.

66 structural remodeling of lipid A anchored on bacterial surfaces.

67 bacter actinomycetemcomitans displays on the bacterial surface a nonfimbrial adhesin, EmaA, which is

68 ysiological and morphological changes at the bacterial surface, a phase shift with an altered array o

69 HMW1 is essential for HMW1 tethering to the bacterial surface, a prerequisite for HMW1-mediated adhe

70 at tie motor activity to mechanical cues and bacterial surface adaptation.

71 nvaded cultured HCT116 CRC cells through the bacterial surface adhesin Fap2.

72 The adherence function of the protein, named bacterial surface adhesin of GBS (BsaB), depended both o

73 heir regulatory effects on the expression of bacterial surface adhesins.

74 -7, indicating the involvement of additional bacterial surface adhesive components.

75 ing both on intracellular targets and on the bacterial surface, also being more efficient at interact

76 needle, made of MxiH and protruding from the bacterial surface, anchored in both bacterial membranes

77 for detection of each fusion molecule on the bacterial surface and as an indicator for complement-dep

78 age infection, VapA was observed at both the bacterial surface and at the membrane of the host-derive

79 ococcal recruitment of soluble hTSP-1 to the bacterial surface and binding of pneumococci to host cel

80 ositol (3,4)-bisphosphate [PI(3,4)P2] on the bacterial surface and by interactions between the C-term

81 amino terminus (alpha-domain) of IcsA on the bacterial surface and contributes to cell-to-cell spread

82 s the expression of mosGCTLs, which coat the bacterial surface and counteract AMP activity.

83 ecreased rat and rabbit C3 deposition on the bacterial surface and decreased group C bactericidal tit

84 ion is mediated by a preformed ligand on the bacterial surface and driven entirely by host cell proce

85 mechanisms, including reduced binding to the bacterial surface and efflux pump activity.

86 of protein effectors across membranes to the bacterial surface and environment.

87 o bind effectively to the negatively charged bacterial surface and exhibit high antimicrobial activit

88 noglobulin from mice was able to bind to the bacterial surface and exhibited complement-dependent bac

89 crucial for the binding of fibrinogen to the bacterial surface and for survival in equine and human b

90 ase resistance could be measured both on the bacterial surface and in solution, enabling the method t

91 t that protrudes several nanometers from the bacterial surface and is capped at its distal end by the

92 tructure, the needle, that projects from the bacterial surface and is linked to the base by the inner

93 a surface antigen which extends out from the bacterial surface and is tightly attached to the bacteri

94 ound that HMW1 forms hair-like fibres on the bacterial surface and is usually present as pairs that a

95 eading to rapid pilus disappearance from the bacterial surface and loss of pili-mediated functions.

96 Such proteins need to project away from the bacterial surface and resist significant mechanical forc

97 SICM results find heterogeneities across the bacterial surface and significant differences among the

98 nase (PGK) is both secreted and bound to the bacterial surface and simultaneously binds plasminogen a

99 genic protein that expresses epitopes on the bacterial surface and that induces potentially protectiv

100 of pili that are covalently attached to the bacterial surface and the elucidation of the residues li

101 the repertoire of MSCRAMMs expressed on the bacterial surface and the fluid mechanical shear rates p

102 The RhsT protein was exposed on the bacterial surface and translocated into phagocytic cells

103 nzyme is removed from its native site on the bacterial surface and truncated to improve solubility.

104 ussis inhibited binding of antibodies to the bacterial surface and was required for B. parapertussis

105 olysaccharides are dominant features of most bacterial surfaces and are displayed in different format

106 oteins C3 and C4, which covalently attach to bacterial surfaces and initiate opsonization and killing

107 s that Hoc could attach the phage capsids to bacterial surfaces and perhaps also to other organisms.

108 s its ability to attach to a variety of oral bacterial surfaces and to colonize subgingival dental pl

109 , an external needle that protrudes from the bacterial surface, and a tip complex that caps the needl

110 nd less factor I, generated less iC3b on the bacterial surface, and bound fewer C3 fragments.

111 ne, fewer FimH and fimbriae expressed on the bacterial surface, and decreased bacterial adhesion unde

112 nation, the stalk projects the head from the bacterial surface, and the anchor provides the export fu

113 showed enhanced complement C3 deposition on bacterial surfaces, and protection was dependent upon an

114 acteria by means of seven antibodies against bacterial surface antigens associated with Salmonella en

115 onducive for motility and the substratum and bacterial surface are similarly hydrophobic or hydrophil

116 ts suggest that the mechanical properties of bacterial surfaces are greatly affected by the presence

117 the helical propellers that extend from the bacterial surface, are a paradigm for how complex molecu

118 bacteriophages engage flagella to reach the bacterial surface as an effective means to increase the

119 due to significant reduction of FimQ on the bacterial surface, as the aggregation was not observed i

120 cused on the binding of plasminogen (Plg) to bacterial surfaces, as it has been shown that this inter

121 hPm localizes on the bacterial surface, assisting bacterial dissemination via

122 Bacterial surface-associated proteins play crucial roles

123 nellae have focused primarily on its role in bacterial surface attachment and chronic infection; howe

124 tility, which when blocked decreases initial bacterial surface attachment but subsequently leads to t

125 acquire heme from hemoglobin directly at the bacterial surface, B. anthracis secretes IsdX1 to captur

126 teria to their carbohydrate receptor through bacterial surface binding lectins that significantly enh

127 infection, the alpha C protein (ACP) on the bacterial surface binds to host cell surface heparan sul

128 The bacterial surface-bound plasmin was more resistant to th

129 nfection, the capsule exists attached to the bacterial surface but also free in the host tissues.

130 d iC4b into C4dg, which remains bound to the bacterial surface but no longer forms a convertase compl

131 at Borrelia lipoproteins are targeted to the bacterial surface by default, but can be retained in the

132 onally active and could be released from the bacterial surface by specific proteolytic cleavage into

133 reptococcus gordonii that is exported to the bacterial surface by the accessory Sec system.

134 des, which have a close association with the bacterial surface, CA forms a loosely associated sacchar

135 mutation of major antigenic proteins on the bacterial surface can be a signature of selection for fu

136 ed into the gut lumen, and direct binding to bacterial surfaces can be detected by immunogold analysi

137 in the number of nanoparticles that bind to bacterial surface carbohydrates, causing lower shifts in

138 via complementary affinity-purification and bacterial-surface centered enrichment strategies and qua

139 surface of bacteria; while a low reversal of bacterial surface charge suggested intercalation of CCOE

140 s preference showed a linear relationship to bacterial surface charge, suggesting that microvilli res

141 h showed that surface-adherent AgNPs inhibit bacterial surface colonization, a precursor to biofilm f

142 ition, reductions in antibody binding to the bacterial surface, complement deposition, and passive pr

143 teric viruses, we initially investigated how bacterial surface components might improve CoV infection

144 Accordingly, bacterial surface components that mediate attachment of

145 ts in other proteins have been shown to bind bacterial surface components, we hypothesized that BAI1

146 transcripts, including those associated with bacterial surface components.

147 duced the efficiency of initial adhesion and bacterial surface coverage by >85%.

148 al hyphal network) resulted in (i) increased bacterial surface coverage, (ii) effective degradation o

149 ns beyond the contamination zone even at low bacterial surface coverage.

150 Indeed, the peptide caused bacterial surface damage and killed community-associated

151 ion which affected N-WASP recruitment to the bacterial surface, decreased the number of bacteria disp

152 Bacterial surface density was affected by the presence a

153 likely because resistant mutations affected bacterial surface determinants important for infectivity

154 mplex interactions between nanoparticles and bacterial surface determine the colorimetric response of

155 teins through a 'needle' protruding from the bacterial surface directly into eukaryotic cells after a

156 Here we review strategies for bacterial surface display and describe how they have bee

157 host hemostatic factor prothrombin, and the bacterial surface display of agglutinins, proteins that

158 ionally designed BH3 peptide libraries using bacterial surface display to identify selective binders

159 ive point-mutagenesis of peptide substrates, bacterial surface-display, cell sorting, and deep sequen

160 Binding of polymerizing hGBP1 to the bacterial surface disrupts the O-antigen barrier, thereb

161 bproteome; 30 individual OMPs present on the bacterial surface during growth in human urine were iden

162 mably because F1 protein can detach from the bacterial surface easily.

163 d selected M proteins, displaced FH from the bacterial surface, enhanced alternative pathway activati

164 C8 targets capsular carbohydrate on the bacterial surface, enhancing opsonophagocytosis.

165 Recognition of bacterial surface epitopes by host receptors plays an im

166 which stimulates vesiculation that expedites bacterial surface exchange and adaptation to the host en

167 tein is the bgaC product of Streptococcus, a bacterial surface-exposed beta-galactosidase.

168 that the two monoclonal antibodies recognize bacterial surface-exposed epitopes of naturally folded P

169 dicate the presence of at least two distinct bacterial surface-exposed neutralization epitopes in P44

170 by c-di-GMP signaling, which stabilizes some bacterial surface-exposed proteins against proteases.

171 ule serotype independent and mediated by the bacterial surface-exposed proteins, as pretreatment of p

172 dent degradation of particular, but not all, bacterial surface-exposed proteins, including TRP120, wh

173 Our results show that, although these bacterial surface features influence the frequency of ad

174 ion of a select group of operons controlling bacterial surface features such as lipopolysaccharide (L

175 Bacterial surface fibers (pili) permit adherence to biot

176 hous fibrillar structures emanating from the bacterial surface forming physical bridges between bacte

177 The presence of chlorine influenced bacterial surface functional groups and further prevente

178 ites, demonstrate that Cd is mostly bound to bacterial surface functional groups by forming inner-sph

179 cellular spread, actin polymerization at the bacterial surface generates protrusions of the plasma me

180 een previously demonstrated that bacteria or bacterial surface glycans can enhance poliovirus virion

181 Several recently identified bacterial surface glycans, such as PS-I and PS-II, are p

182 similar processes are used to assemble other bacterial surface glycoconjugates.

183 production of many hexuronic acid-containing bacterial surface glycostructures.

184 in displayed increased levels of ActA on the bacterial surface in protrusions.

185 in displayed increased levels of ActA on the bacterial surface in protrusions.

186 C-synthesized M1-linked ubiquitin transforms bacterial surfaces into signalling platforms for antibac

187 step in the delivery of HMW1 and HMW2 to the bacterial surface involves targeting to the HMW1B and HM

188 hesis and presence of type 1 fimbriae at the bacterial surface is only partially responsible for the

189 , we show that local assembly of C5b6 at the bacterial surface is required for the efficient insertio

190 , the principal constituent of virtually all bacterial surfaces, is a specific molecular signature re

191 Bacterial surface layer (S-layer) proteins self-assemble

192 small sized (5-20 nm) Au nanoparticles using bacterial surface-layer proteins as a template.

193 ich is formed by the interaction between the bacterial surface lectin LecA and its cellular receptor,

194 r system that senses iron levels to regulate bacterial surface lifestyle.

195 During invasion, the InlB bacterial surface ligands closely interact with host cel

196 ptide region (a1a2) of a specific subtype of bacterial surface M protein, present in all GAS pattern

197 or layer for the specific recognition of the bacterial surface markers lipopolysaccharide (LPS) and l

198 Putative host receptors for bacterial surface molecules known to induce squid develo

199 Stomatal guard cells of Arabidopsis perceive bacterial surface molecules, which requires the FLS2 rec

200 twitching motility, a pilus-mediated form of bacterial surface movement, is required for Pseudomonas

201 components, a variable chain exposed at the bacterial surface, named polysaccharide pellicle (PSP),

202 tected more than 100 proteins exposed at the bacterial surface of M. avium subsp. hominissuis.

203 a promising vaccine candidate antigen on the bacterial surface of M. catarrhalis.

204 ant purified OppA recognized epitopes on the bacterial surface of the wild type but not the OppA knoc

205 nstrated that SBP2 expresses epitopes on the bacterial surface of the wild type but not the sbp2 muta

206 et of these proteins undergoes processing by bacterial surface omptins to be released into the supern

207 contrast, SipB and SipC were detected on the bacterial surface only subsequent to bacterial contact w

208 show that pre-existing PS-specific Igs, the bacterial surface or particulation, selective recruitmen

209 ould potentially include viral, cellular and bacterial surfaces or artificial surfaces displaying mul

210 ctionally active proteins to be displayed on bacterial surfaces or released into the culture supernat

211 can repress the elaboration of an oncogenic bacterial surface organelle.

212 Peptidoglycans (PGNs) are immunogenic bacterial surface patterns that trigger immune activatio

213 The bacterial surface polysaccharide poly-beta-(1-6)-N-acety

214 Bacterial surface polysaccharides are synthesized from l

215 Previous work indicated that bacteria and bacterial surface polysaccharides can stabilize viral pa

216 Understanding interactions of bacterial surface polysaccharides with receptor protein

217 These data suggest that poliovirus binds bacterial surface polysaccharides, enhancing cell attach

218 ng mechanisms, we find that poliovirus binds bacterial surface polysaccharides, which enhances virion

219 ers of bacteriophages can recognize specific bacterial surface polysaccharides.

220 icroscopy showed that SipD is present on the bacterial surface prior to bacterial contact with host c

221 es induced the fixation of complement on the bacterial surface, promoted phagocytosis by macrophages

222 ved between the two isolates indicating that bacterial surface properties play a role in how biochar

223 at mutations in these ORFs confer changes in bacterial surface properties.

224 Bacterial surface protein A (BspA) is a surface-expresse

225 the genetically linked high molecular weight bacterial surface protein Air.

226 into some human cells through binding of the bacterial surface protein InlB to the host receptor tyro

227 some mammalian cells through binding of the bacterial surface protein InlB to the host receptor tyro

228 f mammalian cells through interaction of the bacterial surface protein InlB with the cellular recepto

229 sed by Mycobacterium tuberculosis (Mtb) is a bacterial surface protein that is commonly used in nucle

230 l adherence and virulence factor B (PavB), a bacterial surface protein with orthologues in other stre

231 -specific polymorphisms present in the major bacterial surface protein, outer-membrane protein A (Omp

232 This process is usually mediated by specific bacterial surface proteins and host factors coating the

233 distinct hosts, ticks and mammals, in which bacterial surface proteins are expected to have a critic

234 Bacterial surface proteins are of primary importance in

235 macrophages is dependent upon the display of bacterial surface proteins attached to the cell wall by

236 Many bacterial surface proteins containing an LPXTG motif are

237 Bacterial surface proteins covalently attach to host cel

238 ing adhesive matrix molecules (MSCRAMMs) are bacterial surface proteins mediating adherence of the mi

239 s is mediated via a range of strain-specific bacterial surface proteins that bind to a variety of pla

240 st cells by facilitating degradation of some bacterial surface proteins via endogenous serine proteas

241 H. pylori urease from a pool of bacterial surface proteins was found to coprecipitate wi

242 The biotinylated bacterial surface proteins were isolated by streptavidin

243 The biotinylated bacterial surface proteins were isolated by streptavidin

244 and prototype for a family of Gram-positive bacterial surface proteins, ACP facilitates GBS entry in

245 After stochastic binding to bacterial surface proteins, HD6 undergoes ordered self-a

246 tein B (OmpB) to block ubiquitylation of the bacterial surface proteins, including OmpA, and subseque

247 e revealed several families of Gram-positive bacterial surface proteins, including serine-rich repeat

248 riers, is mediated by the interaction of two bacterial surface proteins, InlA and InlB, with their re

249 g phenotype characterized by upregulation of bacterial surface proteins.

250 activation by microbes, complement opsonizes bacterial surfaces, recruits professional phagocytes, an

251 protein forms antenna-like structures on the bacterial surface required for collagen adhesion.

252 ivery with both eukaryotic microvesicles and bacterial surface secretion systems.

253 ar to the activating MBL.MASP complex on the bacterial surface so that, following recruitment of C2,

254 okaryotic post-translational modification of bacterial surface structures and the unidentified role t

255 have determined that these highly conserved bacterial surface structures are expressed by all M. cat

256 Type 1 pili are representative of a class of bacterial surface structures assembled by the conserved

257 ly biofilms the density and rupture force of bacterial surface structures can trigger cell sorting ba

258 urfactant production or by the appearance of bacterial surface structures that might power sliding mo

259 ed by bacteria or bind to negatively charged bacterial surface structures, where they can impair bind

260 may play a larger role in the decoration of bacterial surface structures.

261 on of C-terminus amidated D-amino acids onto bacterial surfaces substantially reduced the cell wall s

262 ed release of a C-terminal fragment from the bacterial surface, suggesting a novel mechanism of actio

263 organelles contribute positive charge to the bacterial surface, suggesting that dispersin's role in f

264 ubpopulations of lipooligosaccharides on the bacterial surface that are determinants of biofilm forma

265 LUBAC is recruited via its subunit HOIP to bacterial surfaces that are no longer shielded by host m

266 oach to AST based on signal amplification of bacterial surfaces that enables phenotypic AST within 5

267 , the specific association of ESAT6 with the bacterial surface, the binding of ESAT6 to laminin and t

268 During its transport to the bacterial surface, the phosphate groups of the lipid A a

269 bp MAb binds to sparsely exposed fHbp on the bacterial surface, there appears to be insufficient comp

270 nts to reduce the net negative charge of the bacterial surface, thereby promoting cationic antimicrob

271 By binding bacterial surfaces, this novel method allows more accura

272 and distribution of the IcsA protein on the bacterial surface through proteolytic cleavage of IcsA.

273 fection and how they are translocated to the bacterial surface through the five distinct type VII sec

274 is the needle, which is a hollow tube on the bacterial surface through which effectors are secreted,

275 al organs by potentiating C3 opsonization on bacterial surfaces, through the increase of hepatic C3 e

276 ydrate-mediated interactions directly on the bacterial surface, thus preserving the native environmen

277 mechanisms whereby SpA is released from the bacterial surface to access the host's immune system are

278 to deposit sufficient complement C3b on the bacterial surface to elicit bactericidal activity requir

279 complement regulator factor H (FH) onto the bacterial surface to evade complement-mediated cell lysi

280 /3-mediated actin filament nucleation at the bacterial surface to generate a branched network that wi

281 bular structures that protrude away from the bacterial surface to interact with the host cell and/or

282 sence of calcium but could be flipped to the bacterial surface upon calcium chelation.

283 In the throat, IgG was mostly bound to the bacterial surface via Fc, whereas in the blood IgG was m

284 , thus implying that RcsF does not reach the bacterial surface via OmpA.

285 eins that allow host plasmin assembly on the bacterial surface, viz. a high affinity plasminogen (Pg)

286 nction to modify the antigenic nature of the bacterial surface, warranting additional investigation o

287 alization of T4SS pilus protein VirB2 on the bacterial surface was demonstrated for the first time in

288 ral groups of proteins whose presence on the bacterial surface was either constitutive or appeared to

289 rthermore, C4b deposited from serum onto AP1 bacterial surfaces was processed into C4c/C4d fragments,

290 Through in situ stabilization on the bacterial surface, we demonstrate rapid isolation of sta

291 Most autotransporters are localised to the bacterial surface where they promote colonisation of hos

292 ng with anionic LUVs mimicking Gram-negative bacterial surface, where this peptide adopts alpha-helic

293 is often caused by molecular changes at the bacterial surface, which alter the nature of specific dr

294 We found that HIV-1 was trapped on the bacterial surface, which led to internalization of HIV-1

295 d a reduced amount of IcsA detectable on the bacterial surface with antibody.

296 d forms large clusters of 100 A pores in the bacterial surface with Perforin-2 cleavage products pres

297 tingly, FBN9 formed dimers that bound to the bacterial surfaces with different affinities.

298 of novel methods that allow modification of bacterial surfaces with small non-biological compounds.

299 mic material, and they are released from the bacterial surface without loss of membrane integrity.

300 suggest that OspA emerges tail-first on the bacterial surface, yet independent of a specific C-termi