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1 M986 (surface binding negative, resistant to bacteriolysis).
2 recruits professional phagocytes, and causes bacteriolysis.
3 e complement cascade ultimately resulting in bacteriolysis.
4 ility of the organism to complement-mediated bacteriolysis.
5 susceptible to anti-NspA complement-mediated bacteriolysis.
6 iotics disrupt cell wall synthesis to induce bacteriolysis.
7 y to antibody-dependent, complement-mediated bacteriolysis.
8 f the Abs could activate complement-mediated bacteriolysis.
9 ortance of source of complement in eliciting bacteriolysis.
10 autolysin responsible for penicillin-induced bacteriolysis.
11 athogens by promoting opsonophagocytosis and bacteriolysis.
12 meningococcus to resist complement-mediated bacteriolysis.
13 increased sensitivity to complement-mediated bacteriolysis, a result that is consistent with recently
14 s that activate classical complement pathway bacteriolysis and also inhibit binding of the complement
15 gonococcal resistance to complement-mediated bacteriolysis and cationic antimicrobial peptides (CAMPs
16 tro studies to determine complement-mediated bacteriolysis and complement-mediated opsonization of an
17 Z232 (surface binding variable, resistant to bacteriolysis), and did not protect against strain M986
19 fant rats challenged by strains resistant to bacteriolysis, and the protective activity paralleled th
21 ligomeric FP, which is also dysfunctional in bacteriolysis but binds the AP proconvertase, C3 convert
22 hat were previously shown to be resistant to bacteriolysis by anti-NspA antibodies produced by immuni
23 _kpnM_05 and vB_kpnP_08 provided significant bacteriolysis for longer duration (8 h) than its monopha
25 s synergized for peptidoglycan digestion and bacteriolysis in the native enzyme or when expressed ind
26 s insufficient to elicit complement-mediated bacteriolysis in vitro but sufficient to confer protecti
27 ainst outer surface protein B (OspB), causes bacteriolysis of Borrelia burgdorferi in the absence of
28 t analysis of resultant human antibodies for bacteriolysis, opsonophagocytosis, and protective effica
29 antibody to achieve 50% complement-mediated bacteriolysis than conjugate-induced antibody (P < 0.001
30 ndent, classical complement pathway-directed bacteriolysis than were organisms containing 2 copies.
32 e binding positive, susceptible to anti-NspA bacteriolysis), was poorly protective against strain BZ2
33 aboons, implying reduced complement-mediated bacteriolysis, whereas treated animals showed slightly i
34 piration-deficient strain exhibited elevated bacteriolysis within the host cytosol and NOX expression