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1 rototypical antiferroptotic agents including baicalein.
2 cells caused by the compound) as compared to baicalein.
3 rils of alpha-synuclein are disaggregated by baicalein.
4  cytotoxicity is less than that observed for baicalein.
5 d in the interaction of alpha-synuclein with baicalein.
6 nificantly activated by EGF and inhibited by baicalein.
7 L), eicosatetraynoic acid (1 micromol/L), or baicalein (10 micromol/L), desensitization of C-fibers u
8 d mice were randomized to receive vehicle or baicalein (12/15-lipoxygenase inhibitor) at 10-15 minute
9 100 microM) and a 12-lipoxygenase inhibitor, baicalein (5 microM), suggesting that opioids acted via
10                                              Baicalein (5,6,7-trihydroxyflavone), a predominant bioac
11      Its effects were compared with those of baicalein (a flavonoid isolated from Scutellaria baicale
12                                              Baicalein, a 12-LO enzyme inhibitor, dose-dependently in
13 c acid, a pan inhibitor of lipoxygenases and baicalein, a selective inhibitor of 12-lipoxygenase, red
14    Among the active TCMs, we discovered that baicalein, a specific flavonoid from Scutellaria baicale
15 previously observed inhibition properties of baicalein against alpha-synuclein fibrillation.
16 d 23 is also a more selective inhibitor than baicalein against P-gp 170, because its cytotoxicity is
17          The present study demonstrated that baicalein alleviated the severity of TNBS-induced coliti
18 25 micro M for celecoxib, and 75 micro M for baicalein and indomethacin.
19 e most dominant being p-hydroxybenzoic acid, baicalein and kaempferol (T. aestivum), epicatechin and
20         The effect of two 12-LOX inhibitors, Baicalein and N-benzyl-N-hydroxy-5-phenylpentamide (BHPP
21                                              Baicalein and phenidone attenuated the increase in 12-HE
22                                         Both baicalein and phenidone attenuated the protective effect
23 genin and is responsible for biosynthesis of baicalein and scutellarein in roots and aerial parts of
24              These observations suggest that baicalein and similar compounds may have potential as th
25  components with potent antifungal activity, baicalein and wogonin, were identified in Ou-gon.
26 n the presence of the lipoxygenase inhibitor baicalein and/or exogenous 12(S)HETE.
27 also blocked by inhibitors of lipoxygenases (baicalein) and CYP4A (17-octadecynoic acid), but not of
28 w here that low micromolar concentrations of baicalein, and especially its oxidized forms, inhibit th
29 esence of LOX inhibitors (NDGA, AA-861, CDC, baicalein, and PD146176) vs. vehicle-treated and mock-tr
30                   Three compounds, apigenin, baicalein, and quercetin, decreased Gli1 mRNA concentrat
31     Acetylated compounds are more toxic than baicalein, and their potency against cell growth is comp
32 ore propose that resveratrol, genistein, and baicalein are attractive candidates for improved chemoth
33     In our study, we successfully identified baicalein as a potent structure-correcting agent.
34                 Postinjury administration of baicalein attenuated oxidation of arachidonic/adrenic ac
35 dy provided evidence for the first time that baicalein attenuated TNBS-induced colitis, at least in p
36                   4'-deoxyflavones including baicalein, baicalin, wogonin, wogonoside, chrysin and 4'
37                        Progressively adapted baicalein (BLN)-resistant parasites (pB(25)R) show overe
38                                              Baicalein blocked EGF-dependent translocation and activa
39 roxyeicosatetraenoic acid in the presence of Baicalein blocked loss of pRB, whereas 12(S)-HETE alone
40 oncert with these results, apigenin, and not baicalein, blocked the localization of MUC1-C to the nuc
41                       At the upstream level, baicalein bound to the hydrophobic region of the myeloid
42 eicosatetraynoic acid) and lipoxygenase (LO; baicalein) but not cyclooxygenase (indomethacin).
43 elivery of inhibitors targeting 12-LOX (CDC, Baicalein), but not 5-LOX (Zileuton) dose-dependently at
44                Our findings demonstrate that baicalein can reconfigure existing TDP-43 aggregates int
45                            In disaggregation baicalein causes fragmentation throughout the length of
46                                              Baicalein, curcumin, and rifampicin showed concentration
47 atment and prevention abilities of apigenin, baicalein, curcumin, epigallocatechin 3-gallate (EGCG),
48                                              Baicalein decreased ferroptotic phosphatidylethanolamine
49 notoxic effects, resveratrol, genistein, and baicalein did not cause mutagenesis, which is a major si
50 he results indicate that alkylation of R5 of baicalein does not have a major impact on the interactio
51                                    In vitro, baicalein down-regulated the TLR4/MyD88 signaling cascad
52                           In the presence of baicalein, EGF triggered an asymmetric phosphorylation o
53 s fisetin, luteolin, quercetin, eriodictyol, baicalein, galangin and EGCG, and the synthetic flavonoi
54                                              Baicalein, however, restores the functionality of TDP-43
55 of 12(S)-HETE protected both cell lines from Baicalein-induced apoptosis, whereas other LOX metabolit
56                       It has been known that baicalein induces the formation of alpha-synuclein oligo
57                                The flavonoid baicalein inhibits fibrillation of alpha-synuclein, whic
58                                              Baicalein is a flavonoid with antioxidant properties; up
59 of SbCYP82D1.1 is knocked down, baicalin and baicalein levels are reduced significantly while chrysin
60                            The impact of the baicalein modifications on activity against the growth o
61 However, little is known about the effect of baicalein on 2,4,6-trinitrobenzene sulfonic acid (TNBS)-
62                                              Baicalein, one of the major flavones, was found to be re
63 , an effect reversed by the 12-LOX inhibitor baicalein or after chemical desensitization of local sen
64                               Treatment with Baicalein or BHPP resulted in a dose-dependent decrease
65                        Treatment with either Baicalein or BHPP resulted in significant apoptosis in b
66  was markedly abrogated in mice treated with baicalein or mice lacking 12-LOX.
67                                 Furthermore, Baicalein or N-benzyl-N-hydroxy-5-phenylpentamide, speci
68           They were then treated with either baicalein or phenidone, 2 selective 12-LO inhibitors.
69  with and without the LOX pathway inhibitor, baicalein, or lacking 12-LOX.
70                 The 12-lipoxygenase blocker, baicalein, prevents epidermal growth factor (EGF)-induce
71 alization signal) peptide-p53 gene, onto the baicalein pure crystals, followed by coating with glucur
72 n molecules have been covalently modified by baicalein quinone to form a Schiff base with a lysine si
73                                 Furthermore, baicalein reduced NOD-like receptor 3 (NLRP3) inflammaso
74                                              Baicalein showed potent antifungal activity against the
75                                              Baicalein-stabilized oligomers are beta-sheet-enriched a
76 mbrane permeability tests suggested that the baicalein-stabilized oligomers had a mild effect on the
77                                        These baicalein-stabilized oligomers, added to the solution of
78 der to evaluate the structural properties of baicalein-stabilized oligomers, we purified oligomer spe
79 cts of Scutellaria baicalensis compared with baicalein suggest the synergistic effects among componen
80  well as celecoxib and indomethacin, but not baicalein, suppressed proliferation cell nuclear antigen
81 dants, including resveratrol, genistein, and baicalein, that are currently used or investigated for t
82 otic executor gene p53 and anti-inflammatory baicalein to PASMCs to alleviate PAH.
83 an autoinductive loop, and apigenin, but not baicalein, treatment was associated with down-regulation
84                               The binding of baicalein was abolished by conversion of the Tyr residue
85 y contrast, the structurally related flavone baicalein was ineffective in blocking the formation of M
86  of micro-opioid inhibition by both 4-AP and baicalein was reduced.
87         The hydroxyl groups of the A ring of baicalein were systematically alkylated in order to asse
88 ne or both, could enhance the interaction of baicalein with P-gp 170 as well as the amount of intrace
89 selective platelet-type 12-LO inhibitor than baicalein, with no measurable nontargeted effects on the
90                                              Baicalein, wogonin, and their glycosides are major bioac