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1 us, Strongyloides stercoralis, or Wuchereria bancrofti.
2 or cause of lymphatic filariasis, Wuchereria bancrofti.
3 opheles koliensis are the only vectors of W. bancrofti.
4 n appears to be a new ligand of TLR4 from W. bancrofti.
5 etection of microfilaremia due to Wuchereria bancrofti (2 of 46 samples positive by both blood filtra
6 sed with 327 filarial infections (Wuchereria bancrofti = 6, Onchocerca volvulus = 33, Loa loa = 150,
7 ocerciasis or river blindness, or Wuchereria bancrofti and Brugia malayi and related parasitic nemato
11 ing the cycles of transmission of Wuchereria bancrofti and Brugia spp. through the application of ann
12 l strategies aimed at eliminating Wuchereria bancrofti and in guiding research that will facilitate a
13 reatment with ALB is macrofilaricidal for W. bancrofti and leads to sustained reductions in Mf counts
15 of the related filarial parasite Wuchereria bancrofti and predict 14,907 L. loa genes on the basis o
16 ion increases childhood susceptibility to W. bancrofti and skews filaria-specific immunity toward a T
17 susceptibility to infection with Wuchereria bancrofti and to microfilaremia in a village of the Repu
18 whole blood from individuals with Wuchereria bancrofti and/or Mansonella perstans infections (Fil(+);
20 s of L. loa, Onchocerca volvulus, Wuchereria bancrofti, and Brugia malayi available, new drug targets
22 quinquefasciatus genes responsive to WNV, W. bancrofti, and non-native bacteria facilitated an unprec
23 n 1 year, who tested negative for Wuchereria bancrofti antigen (determined by Filariasis Test Strip)
24 uding 143 individuals with a diagnosis of W. bancrofti circulating filarial antigens (CFAs) and 44 wh
25 , where transmission intensity of Wuchereria bancrofti differed by 63-fold (37 vs 2355 L3 per person
26 of filarial infection and the presence of W. bancrofti DNA in anopheline mosquitoes before and after
27 Antenatal immune experience with Wuchereria bancrofti due to maternal filariasis may influence susce
28 e-treated bed nets is a valuable tool for W. bancrofti elimination in areas in which anopheline mosqu
29 Twenty-five individuals from a Wuchereria bancrofti-endemic area of Brazil were classified as bein
30 lariasis, 19 adult residents of a Wuchereria bancrofti-endemic island who had been diagnosed 17 years
32 um), lymphatic filariasis (Brugia,Wuchereria bancrofti), giardiasis (Giardia), toxoplasmosis (Toxopla
34 the same period, the rate of detection of W. bancrofti in anopheline mosquitoes decreased from 1.8% t
35 k of infection by and immunity to Wuchereria bancrofti in children, we performed a cross-sectional st
38 ation coefficients were assessed for both W. bancrofti infection and microfilaremia by controlling fo
40 ased assay will be useful in diagnosis of W. bancrofti infection in a variety of clinical settings.
42 in Leogane, Haiti, an area where Wuchereria bancrofti infection is endemic, and from 67 North Americ
43 Treatment of patients with patent Wuchereria bancrofti infection results in an acute clinical reactio
44 CR)-based method for diagnosis of Wuchereria bancrofti infection, blood, plasma, and paraffin-embedde
45 ilariasis, frequently caused from Wuchereria bancrofti infection, is endemic in several parts of the
47 e context of a systemic helminth (Wuchereria bancrofti) infection such that patent infection altered
48 Brazilians from an area in which Wuchereria bancrofti is endemic were classified as asymptomatic mic
54 eatly reduce the concentration of Wuchereria bancrofti microfilariae in the blood for months to years
55 ein (MfP) was isolated from the sheath of W. bancrofti microfilariae through ultrafiltration, followe
57 1998, reduced the transmission of Wuchereria bancrofti, one of the nematodes that cause lymphatic fil
59 esponsible for LF (Brugia malayi, Wuchereria bancrofti) or onchocerciasis (Onchocerca volvulus) is do
64 the glutathione-S transferase of Wuchereria bancrofti (WbGST), a major lymphatic filarial pathogen o