ライフサイエンスコーパス: baroreflex sensitivity

1 transdermal nor oral ERT had any effects on baroreflex sensitivity.

2 ither baseline cardiovascular parameters nor baroreflex sensitivity.

3 ence between control and occlusion groups in baroreflex sensitivity.

4 ied Oxford technique to assess cardiac vagal baroreflex sensitivity.

5 ivity and negatively correlated with cardiac baroreflex sensitivity.

6 in an increase in HRV and an improvement in baroreflex sensitivity.

7 an increase in parasympathetic activity and baroreflex sensitivity.

8 heart, leading to hypertension and depressed baroreflex sensitivity.

9 vs. sham-operated SHR) and an improvement in baroreflex sensitivity.

10 trual cycle had no influences on cardiovagal baroreflex sensitivity.

11 ssed according to heart rate variability and baroreflex sensitivity.

12 renal sympathetic nerve activity and to test baroreflex sensitivity.

13 ors with a possible reduction in sympathetic baroreflex sensitivity.

14 amic Starling mechanism and arterial-cardiac baroreflex sensitivity.

15 ood pressure and a smaller change in cardiac baroreflex sensitivity.

16 ivity to higher pressures without changes in baroreflex sensitivity.

17 , employing heart rate variability (HRV) and baroreflex sensitivity.

18 ympathetic nerve activity without changes in baroreflex sensitivity.

19 r the Fontan operation, with reduced HRV and baroreflex sensitivity.

20 d more impaired autonomic balance, ie, lower baroreflex sensitivity (1.4 +/- 1.3 versus 5.0 +/- 1.5 m

21 hostatic intolerance had lower cardiac vagal baroreflex sensitivity (12+/-1 versus 25+/-4 ms/mm Hg; P

22 antecedent hypoglycemia leads to 1) reduced baroreflex sensitivity (16.7 +/- 1.8 vs. 13.8 +/- 1.4 ms

23 resulted in greater improvements in resting baroreflex sensitivity (2.3 ms/mm Hg [95% CI, 1.3 to 3.3

24 ntal Young's elastic modulus (-17%; P=0.03), baroreflex sensitivity (+28%; P=0.049), heart rate varia

25 , OR, 10; 95% CI, -1 to 21), lower mean (SD) baroreflex sensitivity (3.77 [0.79] vs 9.76 [2.92] s-3mm

26 One marker of these adaptations is decreased baroreflex sensitivity, a strong predictor of post-MI mo

27 Baroreflex sensitivity (adjusted odds ratio, 0.9; p = 0.

28 served baroreceptor gain could maintain high baroreflex sensitivity after ischaemic injury.

29 c hypertrophy, pulmonary congestion, loss of baroreflex sensitivity (alpha index: SHAM = 3.64 +/- 1.0

30 eart rate [HR] recovery, HR variability, and baroreflex sensitivity) among 46-year-old adults from th

31 We have previously shown that depressed baroreflex sensitivity, an established marker of reduced

32 cal responsiveness to ACTH, but had enhanced baroreflex sensitivity and augmented plasma catecholamin

33 Fortunately, emerging data suggest that baroreflex sensitivity and autonomic function may be res

34 rt rate and blood pressure were recorded and baroreflex sensitivity and heart rate variability were d

35 Hemodynamics and ARIs, baroreflex sensitivity and intrinsic cardiac neural acti

36 cardiac vagal function, as measured by both baroreflex sensitivity and intrinsic cardiac neural acti

37 ostatic intolerance have lower cardiac vagal baroreflex sensitivity and marginally lower blood volume

38 Ala-(1-5) increases baroreflex sensitivity and produces a long-lasting (~6 h

39 Baroreflex sensitivity and the sympathetic response to h

40 goal was to test sympathetic and cardiovagal baroreflex sensitivity and the transduction of sympathet

41 ngiotensin II levels, inflammation, impaired baroreflex sensitivity, and autonomic dysfunction, as we

42 oexcitation, autonomic dysfunction, impaired baroreflex sensitivity, and enhanced blood pressure, whe

43 biomarkers included heart rate variability, baroreflex sensitivity, and flow-mediated dilation, alon

44 stress, impaired sympathetic and cardiovagal baroreflex sensitivity, and increased inflammation.

45 ormalized indexes of sympathetic outflow and baroreflex sensitivity, and reduced the incidence of apn

46 is an active posture test used in assessing baroreflex sensitivity, and the array of patients a phys

47 c nervous system--heart rate variability and baroreflex sensitivity--are reviewed, and the clinical a

48 Cardiovagal baroreflex sensitivity assessed during decreasing BP (i.

49 Sympathetic baroreflex sensitivity assessed with burst incidence was

50 The spontaneous baroreflex sensitivity at baseline was significantly low

51 aimed to evaluate left ventricular function, baroreflex sensitivity, autonomic modulation, and inflam

52 m, mean and maximum (+/-s.d.) supine control baroreflex sensitivities averaged 5 +/- 3, 18 +/- 6, and

53 fferences in the cardiovagal and sympathetic baroreflex sensitivities between phases under any condit

54 Similar sympathetic baroreflex sensitivity between sexes and phases was also

55 alysis of heart rate variability [HRV]), and baroreflex sensitivity (bolus phenylephrine method and a

56 systolic blood pressure, cardiac parameters, baroreflex sensitivity (BRS) and hyperinsulinemia in the

57 Although heart rate variability (HRV) and baroreflex sensitivity (BRS) are recognized as independe

58 This study sought to evaluate cardiac baroreflex sensitivity (BRS) as a predictor of response

59 Background- Cardiovagal baroreflex sensitivity (BRS) declines with age but is fa

60 lex function as indicated by the blunting of baroreflex sensitivity (BRS) following the antagonizatio

61 y on autonomic cardiovascular control and on baroreflex sensitivity (BRS) have not been studied.

62 Hemispheric influences on baroreflex sensitivity (BRS) have not yet been evaluated

63 muscle sympathetic nerve activity (MSNA) and baroreflex sensitivity (BRS) in treated patients with hy

64 Sympathetic baroreflex sensitivity (BRS) is greater during decreasin

65 ter standard tests of autonomic function and baroreflex sensitivity (BRS) measurement, diabetic parti

66 Impaired baroreflex sensitivity (BRS) predicts cardiovascular mor

67 Sympathetic baroreflex sensitivity (BRS) was assessed.

68 Baroreflex sensitivity (BRS) was quantified from the R-R

69 Changes in heart rate variability (HRV) and baroreflex sensitivity (BRS) were also examined.

70 Also, we analysed sympathetic baroreflex sensitivity (BRS) with burst occurrence and a

71 bute to sympathetic overactivity, attenuated baroreflex sensitivity (BRS), and excessive ventilation

72 ventilatory recruitment threshold (VRT-CO2), baroreflex sensitivity (BRS), blood pressure, and blood

73 Arterial baroreflex sensitivity (BRS), muscle sympathetic nerve a

74 Cardiovagal baroreflex sensitivity (BRS), the arterial baroreflex-me

75 ailure (CHF) results in blunting of arterial baroreflex sensitivity (BRS), which arises from alterati

76 ise-induced muscle chemoreflex activation on baroreflex sensitivity (BRS).

77 es in sympathetic activity and reductions in baroreflex sensitivity (BRS).

78 mental stress, as well as impaired arterial baroreflex sensitivity (BRS).

79 od pressure or heart rate, or to cardiovagal baroreflex sensitivity, but correlated with muscle sympa

80 /DeltaMAP, which does not involve changes in baroreflex sensitivity, but may involve changes in chemo

81 t-negative SNARE protein (dnSNARE) increased baroreflex sensitivity by 70% (p < 0.001).

82 nism for the decrease in spontaneous cardiac baroreflex sensitivity (cBRS) during exercise in humans.

83 Sympathetic and cardiovagal baroreflex sensitivities change during the 28-day course

84 brainstem and peripheral ganglionic loci to baroreflex sensitivity changes remain underexplored.

85 at 0.6 of gestation; however, fetal cardiac baroreflex sensitivity decreased with advancing gestatio

86 ared with baseline euglycemic conditions, 1) baroreflex sensitivity decreases significantly (19.2 +/-

87 Sympathetic baroreflex sensitivity did not differ between sexes (P =

88 d pressure and baroreflex threshold, reduced baroreflex sensitivity, diminished plasma catecholamine

89 d HR increase, systolic function impairment, baroreflex sensitivity drop, as well as pulse interval v

90 MSNA, haemodynamic responses and baroreflex sensitivity during early tilting were not dif

91 athetic neural responses but not sympathetic baroreflex sensitivity during orthostasis, though uprigh

92 x sensitivity with OC differ from changes in baroreflex sensitivity during the normal menstrual cycle

93 We tested sympathetic and cardiovagal baroreflex sensitivity during the placebo or "low-hormon

94 sibly 'steady-state' conditions, human vagal baroreflex sensitivity fluctuates in a major way, at ver

95 t during brief periods of observation, human baroreflex sensitivity fluctuates widely and rhythmicall

96 Fast Fourier transforms indicated that baroreflex sensitivity fluctuations (expressed as percen

97 The periodicity of very low frequency baroreflex sensitivity fluctuations was not influenced s

98 rterial pressure, sympathovagal balance, and baroreflex sensitivity for control of heart rate.

99 ween arrhythmic events and predictive tests (baroreflex sensitivity, heart rate turbulence, heart rat

100 Mean values of baroreflex sensitivity, heart rate variability, intracra

101 mpathetic nerve activity and reduced cardiac baroreflex sensitivity heighten cardiovascular risk, alt

102 In addition, baroreflex sensitivity, hemodynamic responses to bolus i

103 response remained unchanged; and indices of baroreflex sensitivity improved.

104 er, intravenous CV-11974 failed to alter the baroreflex sensitivities in area postrema-lesioned SHRs.

105 We measured HRV and baroreflex sensitivity in 22 consecutive patients (8 mal

106 es were associated with baseline measures of baroreflex sensitivity in both CLBP and NP participants.

107 nin activity, aldosterone, urine sodium, and baroreflex sensitivity in both groups.

108 y 5959 affects the control of heart rate and baroreflex sensitivity in conscious dogs with pacing-ind

109 Thus, the calcium promoter restores arterial baroreflex sensitivity in HF.

110 hough studies have examined resting arterial baroreflex sensitivity in older subjects, little attenti

111 mpathetic nerve activity and reduced cardiac baroreflex sensitivity in patients with RA compared to m

112 of ANA-12 into the dmNTS greatly diminished baroreflex sensitivity in sham rats, whereas it had less

113 e, and normalized heart rate variability and baroreflex sensitivity in TGA-PE rats compared to contro

114 In addition, EX increased arterial baroreflex sensitivity in the CHF group (heart rate slop

115 s measurements of heart rate variability and baroreflex sensitivity in the neuromonitoring setting of

116 AT(1) receptor blockade enhanced baroreflex sensitivity in the non-EX CHF rabbits but had

117 Sympathetic baroreflex sensitivity increased from supine to upright

118 upled with impairments in renal function and baroreflex sensitivity, increased neuroinflammatory mark

119 To test the hypothesis that arterial baroreflex sensitivity increases during exercise-induced

120 on against sympathetic overdrive and loss of baroreflex sensitivity, independent markers of mortality

121 with laryngopharyngeal symptoms had reduced baroreflex sensitivity, indicating diminished vagal cont

122 eart rate variability, endothelial function, baroreflex sensitivity, inflammation, and platelet funct

123 Baroreflex sensitivity is a strong indicator of post-myo

124 Baroreflex sensitivity is associated with conditioned pa

125 Reduced baroreflex sensitivity is associated with nicotinamide a

126 Not only sympathetic but also cardiovagal baroreflex sensitivity is similar between sexes and mens

127 t, as measured by heart rate variability and baroreflex sensitivity, is significantly associated with

128 ns of physiological abnormalities: depressed baroreflex sensitivity low LF/HF low LF/(HF + LF) low al

129 Enhanced arterial baroreflex sensitivity may contribute to this reduction.

130 Thus, individual differences in baroreflex sensitivity may explain the hemodynamic varia

131 function, including decreased cardiac vagal baroreflex sensitivity, may contribute directly to morta

132 ing age, left ventricular ejection fraction, baroreflex sensitivity, mean RR interval, standard devia

133 thetic nerve activity (MSNA) and sympathetic baroreflex sensitivity (MSNA-diastolic pressure relation

134 ivity (n=38), heart rate variability (n=34), baroreflex sensitivity (n=20), and ergoreflex activity (

135 Measuring baroreflex sensitivity, now possible in routine clinical

136 roreflex dysfunction (defined by spontaneous baroreflex sensitivity of <6 ms mm Hg).

137 Hypoxia had no significant effect on baroreflex sensitivity or 'set point' for the control of

138 No significant differences in cardiovagal baroreflex sensitivity or vascular transduction were obs

139 ent of heart rate variability (P<0.0001) and baroreflex sensitivity (P=0.03), and overactive ergorece

140 , it still decreased heart rate and restored baroreflex sensitivity (PI/SAP slope, 12.7+/-2.8 ms/mm H

141 central chemoreceptor sensitivity, arterial baroreflex sensitivity, plasma norepinephrine, epinephri

142 pathetic activity and increasing sympathetic baroreflex sensitivity plays a key role in promoting the

143 long the vagal efferent pathway can maintain baroreflex sensitivity post-cardiac ischaemia.

144 (integrated relaxation pressure) and reduced baroreflex sensitivity (r = -0.33; 95% CI, -0.58 to -0.0

145 0.15 Hz) (r=-.52, P=.006) and inversely with baroreflex sensitivity (r=-.60, P=.005).

146 ver, their specific contribution to arterial baroreflex sensitivity remains unclear.

147 Spontaneous baroreflex sensitivity (SBR) was evaluated as the slope

148 emetry), autonomic function, and spontaneous baroreflex sensitivity (SBRS) were not significantly dif

149 Baroreflex sensitivity showed no correlation with intrac

150 fects heart rate, blood pressure regulation, baroreflex sensitivity, tissue oxygenation, and vascular

151 cardiovascular disease biomarkers including baroreflex sensitivity to quantify the influence of the

152 endent astroglial release of ATP to modulate baroreflex sensitivity via P2Y(1) receptors.

153 at process afferent information and modulate baroreflex sensitivity via the release of ATP.

154 Sympathetic baroreflex sensitivity was assessed by intravenous doses

155 Sympathetic baroreflex sensitivity was assessed by lowering and rais

156 Baroreflex sensitivity was assessed every 10 seconds usi

157 Baroreflex sensitivity was assessed in control and in HF

158 Baroreflex sensitivity was assessed in the time domain w

159 Cardiovagal baroreflex sensitivity was defined as the slope relating

160 However, sympathetic baroreflex sensitivity was greater and mean arterial pre

161 Furthermore, sympathetic baroreflex sensitivity was greater during the ML than th

162 Similarly, cardiovagal baroreflex sensitivity was greater in the LH than in the

163 Baroreflex sensitivity was lower in patients (12+/-1 ver

164 Baroreflex sensitivity was lower in susceptible dogs (10

165 Baroreflex sensitivity was measured by the alpha-index m

166 Sympathetic baroreflex sensitivity was quantified using the slope of

167 Sympathetic baroreflex sensitivity was quantified using the slope of

168 Baroreflex sensitivity was similar in COI and control su

169 s measurements of heart rate variability and baroreflex sensitivity we aimed to test whether autonomi

170 s of RR interval variability and spontaneous baroreflex sensitivity were also computed.

171 y-four-hour ambulatory BP, SND, and arterial baroreflex sensitivity were measured before and after 8

172 t subjects, moderate ongoing fluctuations of baroreflex sensitivity were punctuated by brief major pe

173 Sympathovagal balance and spontaneous baroreflex sensitivity were restored during vitamin C in

174 Similar changes in baroreflex sensitivity were seen.

175 Analyses of heart rate variability and baroreflex sensitivity were used to assess autonomic bal

176 art rate variability, heart rate turbulence, baroreflex sensitivity) were significant predictors of a

177 stiffness; (2) it is associated with reduced baroreflex sensitivity, which increases blood pressure v

178 Furthermore, changes in baroreflex sensitivity with OC differ from changes in ba

179 We estimated vagal baroreflex sensitivity with systolic pressure and R-R in

180 amic Starling mechanism and arterial-cardiac baroreflex sensitivity, without changing dynamic arteria