ライフサイエンスコーパス: base excess

1 resuscitation, shock index, coagulation, and base excess.

2 pressure, heart rate, Po(2), Pco(2), pH, and base excess.

3 ange in chloride and the degree of change in base excess.

4 ), as rapidly detected by a negative alactic base excess.

5 dysfunction, as rapidly detected by alactic base excess.

6 ge enough to explain the degree of change in base excess (0.8 +/- 2.3 to -2.7 +/- 2.9).

7 es included UA pH less than 7.2, UA Pao2, UA base excess, 1- and 5-minute Apgar scores, and neonatal

8 bonate (ACZ + HCO(3)(-) ; pH: -0.01 0.04 and base excess: -1.5 2.1 mEq.l(-1) , trial effects: P = 1.0

9 rchloremia by 8-12 hrs (mean sodium-chloride base excess, -10.0 mmol/L).

10 re acidosis [ie, umbilical artery pH <7.0 or base excess -12.0 mmol/L or less], 5-minute Apgar score

11 ficantly lower, whereas nonsurvivor standard base excess (-17.9 +/- 5.1 vs. -2.9 +/- 4.4 mEq/L, p < .

12 (more negative) sodium chloride-partitioned base excess, 2) maintained a greater urine output, and 3

13 0 mL/kg (P=0.019, odds ratio [OR]=13.79) and base excess 30-min after reperfusion less than -16 (P=0.

14 ) arterial pH and [H(+)]a, and a significant base excess (-4.5 +/- 2.7 mEq/L), consistent with compen

15 7 degrees C [95% CI, 39.4-39.9]), and higher base excess (-5.9 mEq/L [95% CI, -8.0 to -3.8] vs -11.2

16 The baseline pH was 7.27 +/- 0.11 and base excess -5.9 +/- 5.0 mmol/L.

17 well with a systemic indicator of recovery, base excess, 5.4 +/- 4.7 (MalPEG-Hb), 1.7 +/- 3.8 (SB),

18 at 250 mg every 8 h (ACZ; pH: -0.07 0.04 and base excess: -5.7 1.9 mEq.l(-1) , trial effects: P < 0.0

19 d anions at admission (mean unmeasured anion base excess, -9.2 mmol/L) to predominant hyperchloremia

20 ic acidosis was common at presentation (mean base excess, -9.7 mmol/L) and persisted for 48 hrs.

21 Population based excess absolute risks are estimated for a number o

22 dent predictors for mortality, together with base excess and Glasgow Coma Scale.

23 ry mechanism, weighted-Revised Trauma Score, base excess and hemoglobin, ACT-predicted red blood cell

24 1 mEq/L (95% CI 0.76-2.06; p=0.0002) in mean base excess and increase of 1.65 mmol/L (0.47-2.8; p=0.0

25 resuscitation with saline, arterial standard base excess and plasma apparent strong ion difference we

26 In about one-sixth of the patients, base excess and plasma bicarbonate were normal.

27 d outcomes in blood pressure, lactate level, base excess and plasma protein concentration compared to

28 r 30-day mortality in comparison to standard base excess and strong ion gap.

29 uration (SaO(2)), bicarbonate concentration, base excess, and alveolar-arterial oxygen difference wer

30 lobin, oxygen content, lactate, pH, standard base excess, and arginine vasopressin levels were determ

31 stric intramucosal pH, arterial pH, arterial base excess, and arterial lactate concentrations were me

32 ith Ringer's solution resulted in a standard base excess, and Cl(-) between that of saline and Hexten

33 resuscitation room: age, Glasgow Coma Scale, base excess, and prothrombin time.

34 ric pressure has a substantial effect; PCO2, base excess, and respiratory quotient have small effects

35 to limitations in bedside monitoring tools (base excess, anion gap).

36 We defined the "alactic base excess," as the sum of lactate and standard base ex

37 They also had lower base excess at admission.

38 We compared the base excess (BE) and anion gap (AG) methods with the les

39 nous fluid resuscitation (r = .44), with the base excess changing, on average, by -0.4 mmol/L for eac

40 ressure, arterial bicarbonate concentration, base excess, fibrinogen concentration, and platelet coun

41 ributions from age and sex, and of admission base excess for maximal creatinine concentration.

42 The mean improvement in base excess from 0 to 24 hours was significantly greater

43 The primary outcome was mean change in base excess from 0 to 24 hours.

44 with baseline, acidosis of pH 7.1 decreased base excess from 6.6 +/- 0.5 mM to -12.4 +/- 0.5 mM; red

45 Furthermore, bosentan decreased blood base excess in Nx animals (0.1 +/- 0.3 to -0.12 +/- 0.03

46 correlated with raised serum bicarbonate and base excess, indicating compensated respiratory acidosis

47 mergency department pH < or = 7.26, standard base excess < or = -7.3 mEq/L, lactate > or = 5 mmol/L,

48 excess," as the sum of lactate and standard base excess.Measurements and Main Results: Organ dysfunc

49 Base excess missed serious acid-base abnormalities in ab

50 d venous blood gases; electrolytes; lactate; base excess; oxygen delivery, consumption, and extractio

51 , injury severity score, Glascow Coma Scale, base excess, platelet count and hemoglobin, adrenaline,

52 Glascow Coma Scale, systolic blood pressure, base excess, platelet count, hemoglobin, prehospital pla

53 elated with Scvo2 (r=.80), lactate (r=-.78), base excess (r=.80), and shed blood volume (r=-.75).

54 administered was compared with the change in base excess (r2 = .93; p < .0001).

55 ions by expressing them in terms of standard base excess (SBE in mM), which quantifies the metabolic

56 ted tantalum catalyst that avoids the use of bases, excess substrate, or additives.

57 During acute base excess, the renal collecting duct beta -intercalate

58 .90-7.00), whereas 20.5% (31/151) selected a base excess threshold (mean, -15.62+/-0.78; median, -16;

59 M infusion corrected pH to 7.40 +/- 0.02 and base excess to 2.6 +/- 0.9 mM (p < .05).

60 oxygen saturation (Scv(O(2))), lactate, and base excess to better determine the origin of lactate.Me

61 4 mm Hg) but no significant difference in UA base excess, UA pH less than 7.2, Apgar scores, or neona

62 Trends toward lower base excess values and higher occurrences of deaths only

63 normal saline administered and the change in base excess was found (r2 = .86; p < .0001), although no

64 Base excess was partitioned using abridged Stewart equat

65 In contrast, positive values of alactic base excess were associated with a relative reduction of

66 concentration and "anion gap," the other on "base excess," with a third method based on physicochemic