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1 AGATCT arises from the C-terminal non-finger basic domain.
2 membrane targeting through an effect on the basic domain.
3 activity has been ascribed to the conserved basic domain.
4 locked by a mutant c-Jun protein lacking the basic domain.
5 binding and conformation of the C-terminal, basic domain.
6 like its human counterpart, also contains a basic domain.
7 supporting specific DNA binding by the CREB basic domain.
8 ore they can bind to DNA through an upstream basic domain.
9 e very distal part, including the C-terminal basic domain.
10 assays, that dCA directly binds to SIV Tat's basic domain.
11 at K-cyclin interacted with Cdk9 through its basic domain.
12 confer noncanonical E box binding to the E12 basic domain.
13 required to alleviate the inhibition by the basic domain.
14 minal activation domain 1 and the C-terminal basic domain.
15 recognized a clade B-specific epitope in the basic domain.
16 ombinations of fatty acids, isoprenoids, and basic domains.
17 and contains two extensively hydrophilic and basic domains.
18 hich one is bipartite, and two widely spaced basic domains.
19 action between the phosphorylated acidic and basic domains.
20 h the tail enables the close approach of the basic domains.
21 this hypothesis, mutations in the N-terminal basic domain (29/31KE) or deletion of the membrane-targe
25 Tat carrying alanine substitutions in the basic domain (AKKAAQAAA) remained localized in the cytos
27 A synthetic peptide encompassing the TRF2 basic domain also binds to DNA four-way junctions, where
28 now demonstrate that mutations in the highly basic domain also retarget virus particle formation to t
29 This catalogue of point mutants reveals that basic domain amino acids play distinct functions in bind
30 This association is specific, requiring the basic domain (amino acids 122 to 189) of K-bZIP and a sp
31 ty has been mapped to two amino acids in the basic domain, an alanine and threonine, referred to as t
32 the C terminus, and these are followed by a basic domain and a C-terminal domain that is highly simi
33 lmodulin, CaM53, with an extended C-terminal basic domain and a CTIL CaaX-box motif which are require
34 ininamide, a linear peptide mimic of the Tat basic domain and a cyclic peptide that potently inhibits
35 ide, a proline-rich domain, a histidine-rich basic domain and a cysteine-containing 'PAC' domain that
37 carboxyl-proximal amino acid position of the basic domain and a position within the amino-proximal se
38 nd with IkappaB-alpha through its 15-aa bZIP basic domain and adjacent sequences, respectively, and t
39 a HIF-1alpha construct with deletions of the basic domain and carboxy terminus blocked reporter gene
41 shortest isoform, VEGF120, lacks this highly basic domain and is freely diffusible upon secretion.
45 trating peptide constructs such as HIV-1 TAT basic domain and related peptides have been developed to
47 that RPGR(ORF15) variants, which disrupt its basic domain and the interaction with TTLL5, also impair
48 inding of the Sema III immunoglobulin- (Ig-) basic domain and the unrelated ligand, vascular endothel
52 ins numerous alanine-rich, proline-rich, and basic domains and has limited homology to an expressed s
53 association reaction suggested that the two basic domains and the linker region between the subunits
54 band shift assays has demonstrated that both basic domains and the three leucine zipper motifs are ne
55 ed glycosylation site, a central lysine-rich basic domain, and a C-terminal tail containing 10 cystei
56 ch (C/H) domain found just N-terminal to the basic domain, and a carboxy-terminal amphipathic alpha-h
57 ptad repeat of five leucines (L(1)--L(5)), a basic domain, and a conserved alanine characterize the d
58 inger motif, a serine/proline-rich domain, a basic domain, and a leucine-zipper-like domain and is ex
60 erminal acidic domain and a carboxy-terminal basic domain, and displaying partial homology to the HMG
61 both activation domain 2 and the C-terminal basic domain, and p53-(DeltaPRDDeltaBD), which lacks bot
62 -associated protein glycine-rich domain, the basic domain, and serine/proline-rich (SP-rich) domain.
63 hypothesis that the functions of this highly basic domain are mediated by neutralization of linker DN
64 evealed that the PHD, CXXC, coiled-coil, and basic domains are identical between mouse and human, whi
66 the proline-rich domain, and the C-terminal basic domain, are only required for inducing apoptosis.
67 ce the electrostatic fields produced by this basic domain at the membrane interface and may play a ro
73 rodimers with other bHLH proteins, and their basic domain binds to a DNA sequence element, the E-box,
75 Mutations in either the VP5 hydrophobic or basic domain blocked VP5-directed permeability of cells.
77 l APC sequences lacking or including the APC basic domain but encompassing the EB1 binding region in
78 s, and includes the two adjacent zinc-finger/basic domains characteristic of the GATA factor family.
79 tributions of the amino acid residues in the basic domain combine to determine DNA-binding specificit
81 s S111-I116, A124-R132, and K144-T146 in the basic domain contain short alpha-helical or beta-sheet s
85 that the in vivo inhibitory activity of the basic domain depends upon activation domain 1, such that
87 it was shown that vertebrate APC through its Basic domain directly collaborates with the formin mDia1
89 gy between the vertebrate and Drosophila APC Basic domains, Drosophila APC1 collaborates with Dia to
90 llectively, our data indicate that the small basic domain encompassing residues R(537) and R(538) con
91 Furthermore, we showed that deletion of the basic domain enhances, whereas a mutation in activation
93 mutations within either the cysteine-rich or basic domains exerted minimal effects on the endothelial
98 Membrane permeation peptides, such as Tat basic domain, have emerged as useful membrane transducti
100 nslational products of this gene contain the basic domain helix-loop-helix motif characteristic of a
101 he scl (also called tal-1 or TCL5) gene is a basic domain, helix-loop-helix (bHLH) transcription fact
102 nreported transcript, designated BHLHB1 (for basic domain, helix-loop-helix protein, class B, 1) that
104 argeted mutagenesis of basic residues within basic domain I caused loss of hr-dependent transactivati
107 sisting of mostly positive-charged residues (basic domain I) abolished hr-dependent transactivation.
109 sidues 180-207, and that a 40 residue highly basic domain, immediately preceding the zipper, is respo
110 show that despite the crucial role for Zta's basic domain in eliciting cell growth arrest, its amino
111 ic residues that are organized into a single basic domain in the folded MIP-1 beta monomer, three (R1
112 ocalize to nuclear bodies, share a conserved basic domain in their N termini that binds to the ankyri
113 s implicate a functional role for the agouti basic domain in vivo, possibly influencing the biogenesi
114 xamine vectorial transport properties of Tat basic domain in well-differentiated epithelial cells pos
116 interactions being required to bring the two basic domains in close register with the mTERF target DN
119 nding protein 3 (IGFBP3) by p53 and that the basic domain inhibits induction of this gene by p53.
122 fically between telomeric substrates; TRF2's basic domain is particularly important for this stimulat
126 nding domain in the middle of the protein, a basic domain just upstream of this domain, and several s
130 an eif3h mutant, including the mRNA for the basic domain leucine zipper (bZip) transcription factor
131 ntagonizing the enhancer action of NF-IL6, a basic domain leucine zipper transcription factor belongi
135 lear factor-IL6 (NF-IL6) expression, a human basic domain-leucine zipper-containing transcription fac
136 We have identified three membrane-associated basic domain/leucine zipper (bZIP) factors in Arabidopsi
139 f genes dependent on the membrane-associated basic domain/leucine zipper (bZIP) transcription factor,
140 lly Interesting New Gene finger proteins and basic domain/leucine zipper and basic helix-loop-helix t
142 nce supporting VIP2 interaction with VIP1, a basic domain/leucine zipper motif-containing protein req
143 re mediated in large part by the ABA-induced basic domain/leucine zipper transcription factor ABA INS
144 ivator, Tax, interacts specifically with the basic-domain/leucine-zipper (bZip) protein, cAMP respons
147 henotypes correlating with signal peptide or basic domain mutations, and more devastating phenotypes
151 first time that the Ala/Thr dipeptide of the basic domain of an invertebrate MRF behaves as a myogeni
155 icrotubule-binding assays, we found that the basic domain of dynactin moves progressively along micro
157 through the second zinc finger and adjacent basic domain of GATA-4 and the N-terminal domain of Smad
158 that the C-terminal zinc finger and adjacent basic domain of GATA-4 is bifunctional, modulating both
159 NA and that the protein residues outside the basic domain of Hexim1 are involved in specific RNA inte
161 cally interacted with GATA proteins, and the basic domain of HRT was critical for physical associatio
162 Here we show that purified carboxyl-terminal basic domain of human APC protein (APC-basic) bound dire
163 p on Kar9p maps to a short region within the basic domain of Kar9p that contains a conserved phosphor
164 a functional relationship between the highly basic domain of MA (amino acids 17 to 31) and residues 8
166 AML2 protein chimera in which the N-terminal basic domain of MAML2 is replaced by the N-terminal doma
168 the first time we provide evidence that the basic domain of p53 is inhibitory in vivo as has been de
170 diated E-selectin up-regulation required the basic domain of Tat and was inhibited by a Tat antibody.
171 of the protein, and peptides containing this basic domain of Tat protein can bind TAR RNA with high a
172 to bind specifically to the highly conserved basic domain of Tat, which also mediates binding to the
173 d domain located immediately adjacent to the basic domain of the bHLHZip region is required for SREBP
174 he LR domain of HIV-1 Vpr, when fused to the basic domain of the cellular transcription factor CREB,
178 phosphorylation/dephosphorylation within the basic domain of the linker region is not directly involv
179 The reduction of positive charge in the NC basic domain of the M1-2/BR virus adversely affects both
184 e U937 cells stably transfected with deleted basic domain of TRF2 is partially sensitive to Pu-27 but
187 iple Glu-Gly tandem repeats and a C-terminal basic domain of unknown function, localizes to the conne
189 th HIV-1 and HIV-2 Tat demonstrated that the basic domains of both the HIV-1 and HIV-2 Tat proteins w
190 embly, but low sequence homology between the Basic domains of Drosophila and vertebrate APC has left
191 novel cellular protein interacting with the basic domains of EB1 and c-Jun, and competing of their b
192 ng to mitotic chromosomes, we identified the basic domains of EBNA-1 within amino acids 1-89 and 323-
195 tive method to prevent errors of omission in basic domains of intensive care unit management that mig
196 terminal cysteine-rich and carboxyl-terminal basic domains of ORF31 mediate the ORF31-ORF34 interacti
197 ulin dimers; both the N-terminal CAP-Gly and basic domains of p150(Glued) are required in tandem for
198 st, tat mutants in the activation, core, and basic domains of Tat did not stimulate HIV-1 gene expres
199 onal antibodies directed against the RGD and basic domains of Tat, but not against the proline-rich d
200 Making use of various combinations of three basic domains of the receptors (i.e., exofacial, transme
204 of Gag proteins with mutations in either the basic domain or between residues 84 and 88 was rescued b
206 activation domain 2 and lacks the C-terminal basic domain, p53-(DeltaAD2DeltaBD), which lacks both ac
209 Furthermore, we identified arginines in the basic domain (RKKRRQRRR) of Tat as essential for (1) tar
210 ma domain, immunoglobulin domain (Ig), short basic domain, secreted, (semaphorin) 3D (SEMA3D) was sig
211 ainly with clade B and with only one clade B basic domain sequence, which included the rare amino aci
212 RGD sequence but contains cysteine-rich and basic domains similar to HIV-1 Tat, induced aggregation
214 n modeling of a missense mutation within the basic domain suggests DLF1 protein functions through DNA
216 A Tat protein containing a deletion of the basic domain (Tat(delta)49-57) localized exclusively to
217 ve identified the RASSF1A region harboring a basic domain that appears to mediate the interactions be
218 eted to the plasma membrane by an N-terminal basic domain that binds phosphatidylinositol-4,5-bisphos
219 the presence of domain II plus an additional basic domain that can be represented by an NLS or a basi
221 ers from Suv39h1 by containing an N-terminal basic domain that facilitates retention at mitotic chrom
222 alanine-threonine (Ala-Thr) dipeptide of the basic domain that is known in vertebrates as the myogeni
223 * required for pore formation: an N-terminal basic domain that permits VP5* to peripherally associate
224 arginine residues within the conserved pUS9 basic domain that were essential for binding the molecul
225 synaptic targeting requires three N-terminal basic domains that bind F-actin and acidic phospholipids
228 ne the functional significance of the agouti basic domain, the entire 29-aa region was deleted from t
229 s domain involved in lipin activation is its basic domain, the interaction domain is mapped to the N-
232 d alpha-helix in CREB that spans most of its basic domain to include amino acid residues localized to
233 ibutions of NC's zinc fingers and N-terminal basic domain to the two major components of chaperone ac
236 ined deletion of activation domain 1 and the basic domain was required to alleviate the inhibition by
237 arginine residues by alanines in the M-Twist basic domain was sufficient to abolish both the binding
239 Interestingly, we showed that the C-terminal basic domain, which is required for wild-type p53 activi
240 tions were identified, three residing in the basic domain, which is responsible for DNA binding, and
242 hat only the C-terminal zinc finger (Cf) and basic domain, which together constitute the GATA-binding
243 Here, we show that substitution of the MyoD basic domain with that of E12 does not prevent interacti